ライフサイエンスコーパス: we think

1 lfactory results, however, may not mean what we think.

2 may not always be as benign or beneficial as we think.

3 ave even more far-reaching consequences than we thought.

4 etastable liquid may be more ubiquitous than we thought.

5 more common in plants and also animals than we thought.

6 In naming population groups, we think a chief aim is to use terms that the group memb

7 le forms suggests that we need to adjust how we think about animal mating systems and the evolution o

8 Falkow's many contributions remade the way we think about bacterial pathogens, antibiotic resistanc

9 ft interfaces could profoundly influence how we think about biomaterials.

10 of medicine and has forever changed the way we think about disease.

11 gy have begun to dramatically change the way we think about evolution, development, health and diseas

12 died in real time, dramatically changing how we think about gene regulation on chromatin templates.

13 ealth law has transcending importance in how we think about government, politics, and policy.

14 gating, and pharmacology has changed the way we think about iGluR function.

15 ction call for some modifications in the way we think about innate immunity strategies.

16 Thus, our studies, revise how we think about iron import and export from the retina.

17 nd metamaterials have revolutionized the way we think about optical space (varepsilon,mu), enabling u

18 s, if any, the brain spontaneously uses when we think about others.

19 ural reuse has profound implications for how we think about our continuity with other species, for ho

20 eeping imposed on us probably influences how we think about patients.

21 tudies, there has been a major change in how we think about perioperative management of anticoagulati

22 to highlight some of the advances in the way we think about rhabdomyosarcoma (RMS).

23 he potential to revise significantly the way we think about seed plant evolution, especially with reg

24 d techniques that challenges notions for how we think about T-cell receptor signaling.

25 ve represents a significant shift in the way we think about the cellular bases of perception.

26 nd type 2 diabetes is fundamental to the way we think about the disease.

27 al, it could mark a turning point in the way we think about the evolution of the genome.

28 ics of eukaryotic microbial life changes how we think about the flow of genetic information and the e

29 r intended arm movements has changed the way we think about the order of operations in the sensory to

30 to a wealth of information that changes how we think about the pathogenesis of immune-mediated disea

31 behaving animals are revolutionizing the way we think about the role of Purkinje cells in sensori-mot

32 there has been a dramatic change in the way we think about the role of vascular smooth muscle cells

33 g studies have fundamentally changed the way we think about the vegetative and minimally conscious st

34 dea that prokaryotes may not form species as we think about them for plants and animals.

35 how this new information is changing the way we think about this fatal disorder.

36 cellular and system level and challenges how we think about vaccine protection against these infectio

37 uencing technologies have revolutionized how we think about viruses.

38 s are fundamental to the way that, nowadays, we think about vision, somato-sensory function, the spin

39 What we think actually happens is that there is an effective

40 However, we think additional mechanisms are important when people

41 ian hypothesis, that language influences how we think; and that the "language of thought" maps to spo

42 We also pose questions whose answers we think are pivotal to understanding this path, and we

43 nts with systemic lupus erythematosus (SLE), we thought F4+ IgG antibodies might constitute a useful

44 of a min slmA double deletion mutant, which we think is due to the elimination of polar Z rings (or

45 By reviewing what we have learned and what we think is going on during development, we hope to lure

46 swimming are similar processes, a comparison we think is helpful in understanding how cells migrate.

47 echanisms of INH resistance and propose what we think is the means by which INH kills M. tuberculosis

48 lower limbs associated with weight loss, and we think it is under-recognized; (ii) recovery from the

49 Thus, we think it likely that arrest in both mre11-1 and the c

50 Instead, we think it likely that both pathways contribute to esta

51 As unlikely as we think it might be as the result of natural selection,

52 ations in outer membrane protein biogenesis, we think it possible that Spy may be involved in this pr

53 of AVATAR therapy and, if proven effective, we think it should become an option in the psychological

54 ce on Broca's area as a major speech centre, we thought it important to re-inspect these brains to de

55 to show significant levels of neurogenesis, we thought it likely that BLBP expression would also be

56 s functionally independent from what and how we think, know, desire, act, and so forth".

57 We put forth a series of questions that we think need to be investigated if the conservation res

58 nvironment works, as distinguished from what we think of as (e) imitation (the copying of the demonst

59 When we think of extremophiles, organisms adapted to extreme

60 Typically, we think of policymakers looking to scientists for advic

61 We think of transcription factors as being confined to t

62 We think our findings could potentially aid therapeutic

63 We end with an example of how we think plasticity may play out in stickleback life his

64 results obtained from studying humans, whom we think provide an underutilized, yet critical, animal

65 O2 uptake and photosynthetic plant activity, we think that a minimum CO2 mixing ratio might be needed

66 We think that a subset of patients with asymptomatic bra

67 Making conservative assumptions, we think that about 100000 excess deaths, or more have h

68 We think that all patients with advanced-stage carcinoid

69 We think that an abrupt change in the adapted state of t

70 We think that DWMR have important role due to quantitati

71 We think that exposures to hazardous substances migratin

72 fforts to lower cholesterol should continue, we think that important biology may be reflected in the

73 We think that improvements in technology and workflow st

74 ncertainty that we have about these effects, we think that it is helpful for the NIH to engage in ope

75 ind a unified evolutionary theory promising, we think that long-term and large-scale, scientifically

76 Thus, we think that mechanism-based novel strategies should be

77 We think that our data provide insight into expression r

78 We think that our data provide thrombosis-relevant infor

79 We think that PTVI is a good alternative to repeated sur

80 We think that RACK1 is an important Src substrate that s

81 We think that recipient BM-HSC-derived hepatocyte repopu

82 tional approach to the analysis of teaching, we think that she ignores important features of the soci

83 Thus, we think that TCRs are structurally able to recognize a

84 We think that the actin cytoskeleton, in particular the

85 We think that the early intermediates with peaks at 155

86 and that osteocytes produce paracrine DKK1, we think that the G171V mutation may cause an increase i

87 However, we think that the growing number of oral anticoagulant c

88 Thus, we think that the hydrophobic effect and hydrogen bondin

89 We think that the negative cooperativity occurs when sat

90 We think that the probability index provides an immediat

91 Therefore, we think that the process of working through religious l

92 We think that the recognition and killing of target cell

93 We think that the unbiased reader will follow our argume

94 We think that these results could serve as valuable indi

95 We think that these results propose a new mechanism by w

96 We think that this difference underscores the potential

97 We think that this integrated and tiered approach will l

98 In view of the data obtained, we think that this is a significant and important step t

99 We think that this kind of quantitative analysis can be

100 We think that this should be linked to the concept of so

101 We think that this study presents a novel approach for (

102 We think that tyrosine phosphorylation of these proteins

103 We think that US and clinical findings may sometimes mis

104 At this time, we think that, for most conditions, surgical procedures,

105 t for efficient charging of methionine tRNA, we thought that 1.72 distortion is probably effected by

106 As we thought that endogenous somatostatin might control th

107 refore, require multiple angiogenic factors, we thought that injection of BM, which contains cells th

108 We thought that polymeric matrices, which release growth

109 We thought that terminal homology would promote bidirect

110 y selective for microtubule-targeted agents, we thought that the active compounds might inhibit cell

111 eletion events and multiple point mutations, we thought that these multiple-mutated loci might repres

112 ces contain determinants for aminoacylation, we thought that they might also play a role in editing t

113 Just as we thought that we know everything about superantigens,

114 nd web systems are invaluable resources, and we think the scientific community will benefit for this

115 We think the term monoclonal gammopathy of renal signifi

116 We thought the attack caused from rhinitis by Staphyloco

117 We think there is now compelling evidence that a full un

118 We think these muscle-synergy patterns can be used as ph

119 We think these novel revascularization strategies are li

120 ype structures with considerable Si content, we thought these metastables would be only of the diamon

121 We think they should be included within all evidence-bas

122 entations and musculoskeletal manifestations.We think this article will heighten awareness of these d

123 We think this benefit is largely due to these drugs' abi

124 ic progress and provide a perspective on why we think this is only the beginning of a new era in scie

125 We think this is possible if funding agencies around the

126 We think this mechanism has potentially significant impl

127 We think this pattern should change.

128 We thought to determine the underlying molecular causes

129 We thought to tackle this problem by taking a systems bi

130 Evidence that what we thought was a single species based on morphology is,

131 phosphate chemistry, as well as much of what we think we know about metabolism, will need rewriting.

132 While a resident, we thought we had found a treatment for multiple scleros

133 hrenia and related psychotic disorders that, we think, will be of interest to psychiatric clinicians

134 We then selected variables we thought would be associated with increased rates of 1