1 We used (
2)H/(13)C metabolic flux analysis to quantify interm
2 We used 72 low-cost silicone wristbands as noninvasive passiv
3 For 2015 live births,
we used a compartmental model to estimate (1) exposure to mat
4 We used a mathematical model to predict the direct and indire
5 ficulty of rationally predicting beneficial mutations, here
we used a more comprehensive mutagenesis approach with the go
6 We used a soluble IN fusion protein to facilitate structural
7 We used all live singleton births in California spanning 2005
8 We used an HDM-driven asthma mouse model to compare the capac
9 define serum inflammatory and cardiovascular risk proteins,
we used an OLINK high-throughput proteomic assay to analyze m
10 he evolutionary adaptation of polar capsules to parasitism,
we used as a model organism Ceratonova shasta, which causes l
11 In these experiments,
we used backward conditioning and contingency reversal to est
12 Here,
we used chimeric proteins to test whether DAT and SERT N and
13 We used chronoamperometry to measure protease-digested GA sam
14 We used Cox proportional hazards models to investigate whethe
15 We used Cox proportional hazards regression models to assess
16 We used cross-species expression experiments to show that Alx
17 We used diffusion MR imaging data and the Tract-Based Spatial
18 We used Drosophila melanogaster and Saccharomyces cerevisiae
19 We used genomic sequencing to identify potentially pathogenic
20 Here,
we used human intracranial recordings and visual word-by-word
21 Specifically,
we used IL-23 in vivo gene transfer to induce arthritis in mi
22 In this study,
we used insurance claims for over one-third of the entire US
23 le mass via their activation of the Smad2/3 signaling axis,
we used local injection of adeno-associated viral vectors (AA
24 We used log-linear and nonlinear concentration-response coeff
25 We used low-angle x-ray diffraction at the European Synchrotr
26 Here,
we used micro-CT scans of extant mammals (47 species) and bir
27 We used micromodulated fluorimetry to examine the effect of c
28 We used multivariate analyses to evaluate clinicopathological
29 We used multivariate decoding of electroencephalography (EEG)
30 We used multivariate logistic regression with PCR-confirmed i
31 Materials and Methods
We used National Health Interview Study data from 2000, 2005,
32 To answer this question,
we used psychoacoustic tests and comparative analyses to inve
33 We used published data to create logistic regression models c
34 e whether behaviour change was related to group attendance,
we used random effects models to assess associations between
35 We used Scientific Registry of Transplant Recipients data to
36 We used sialic acid linkage-specific derivatization methods t
37 To examine this issue,
we used single glomerular stimulation in mouse olfactory bulb
38 To understand how these receptors distinguish ACh and Cho,
we used single-channel electrophysiology to measure resting a
39 Studying 213 archival biopsy samples from 17 patients,
we used somatic variants in hypermutable DNA regions to recon
40 We used steady-state oxic and anoxic chemostat cultures to de
41 To address this question,
we used the Atonal family of proneural transcription factors
42 We used the Cochran-Armitage test for trend to examine trends
43 We used the difference in delta(15) N between 20 ant conspeci
44 We used the fish rhabdovirus infectious hematopoietic necrosi
45 We used the Scoring Algorithm for Molecular Subphenotypes (SA
46 Here,
we used theoretical considerations and experimental models of
47 We used theory and experiments to address this question in th
48 entify potential fine differences between rHBeAg and HBeAg,
we used these Fabs in microscale immunoaffinity chromatograph
49 Here,
we used two-photon Ca(2+) imaging to study visual processing
50 We used whole-genome sequencing and phylogenetic analysis to