ライフサイエンスコーパス: wear debris

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1 ue of the physiological effects of graphitic wear debris.

2 Fibroblasts phagocytosed particulate wear debris and responded to cytokine/chemokine stimulat

3 ated to differences in the type or amount of wear debris and was mitigated by anti-TNF therapy.

4 asts respond to stimulation with particulate wear debris and/or conditioned media obtained from patho

5 etal cations are the contact surface and the wear debris, and the latter contains important 'historic

6 ve therapeutic candidate to treat or prevent wear debris-associated osteolysis and aseptic loosening.

7 ited a distinct cellular response to implant wear debris compared with patients without RA.

8 es (mTi) of sufficient size to accumulate as wear debris could stimulate innate or adaptive immunity

9 positional characteristics of tribofilms and wear debris from an IL-lubricated steel-steel contact.

10 ace with oxygen to form an oxide interlayer, wear debris generation and breakdown, tribofilm growth v

11 Wear debris in joint replacements has been suggested as

12 pe immune responses and further suggest that wear debris in joint replacements may have Th2-type infl

13 The amount and type of wear debris in periprosthetic tissues were similar in pa

14 effects of OPG transgene against orthopedic wear debris-induced bone loss in a murine model of osteo

15 tial therapeutic agent for the prevention of wear debris-induced inflammation and osteolysis.

16 The effects of AdvIL-10 administration on wear debris-induced osteolysis in vivo were analyzed usi

17 in vitro, and the effects of these cells on wear debris-induced proinflammatory cytokine production

18 ly involved in periprosthetic osteolysis: 1) wear debris-induced proinflammatory cytokine production,

19 ng in the release of metallic ions and solid wear debris (mainly titanium dioxide) leading to peri-im

20 tanding of how contacting asperities lead to wear debris particle has remained elusive.

21 fy wear at the most fundamental level, i.e., wear debris particles.

22 fibroblasts respond directly to particulate wear debris, possibly via phagocytosis, expressing proin

23 d phagocytosed large amounts of polyethylene wear debris, suggesting a role for phagocytic stimuli in

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