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1 ly used variables (i.e. mixed venous oxygen, wedge pressure).
2 rdiac filling pressures (pulmonary capillary wedge pressure).
3 nary artery pressure and pulmonary capillary wedge pressure.
4 pressure, heart rate, or pulmonary capillary wedge pressure.
5 orse symptoms and higher pulmonary capillary wedge pressure.
6 and severity of emphysema, and directly with wedge pressure.
7 ar resistance as well as pulmonary capillary wedge pressure.
8 ar systolic pressure and pulmonary capillary wedge pressure.
9 elocities for estimating pulmonary capillary wedge pressure.
10 ic filling, despite high pulmonary capillary wedge pressure.
11 lmonary artery pressure and pulmonary artery wedge pressure.
12 nary artery pressure and pulmonary capillary wedge pressure.
13 ctional flow reserve and increasing coronary wedge pressure.
14 ltaneous measurements of pulmonary capillary wedge pressure.
15 patient age, and varying pulmonary capillary wedge pressure.
16 ction in mean pulmonary artery and pulmonary wedge pressures.
17 nge systemic arterial or pulmonary capillary wedge pressures.
19 in, p < 0.01), increased pulmonary capillary wedge pressure (17 +/- 7 vs. 14 +/- 6 mm Hg, p < 0.002)
20 g; P=0.0002), and higher pulmonary capillary wedge pressure (17+/-5 versus 9+/-3 mm Hg; P=0.0001).
21 m Hg to 98 +/- 13 mm Hg; pulmonary capillary wedge pressure: 17 +/- 6 mm Hg to 21 +/- 7 mm Hg; cardia
22 s. 108 +/- 14 mm Hg) and pulmonary capillary wedge pressure (18 +/- 2 vs. 12 +/- 4 mm Hg) versus prei
23 -4 mm Hg; P=0.0001), and pulmonary capillary wedge pressure (18+/-5 versus 10+/-4 mm Hg; P<0.0001) co
24 mg significantly reduced pulmonary capillary wedge pressure (-2.6+/-0.7, -5.4+/-0.7, and -4.6+/-0.7 m
25 tric LVH and estimated mean pulmonary artery wedge pressure (20 mm Hg vs. 16 mm Hg) and shorter isovo
26 p < 0.01 by ANOVA) and mean pulmonary artery wedge pressure (21+/-3 to 16+/-2 mm Hg, p < 0.05 by ANOV
27 arly reductions were sustained for pulmonary wedge pressure (24+/-9 to 15+/-5 mm Hg early; 12+/-6 mm
29 produced significant reductions in pulmonary wedge pressure (27% to 39% decrease by 6 h), mean right
30 s. 16 +/- 8 mm Hg, p = 0.007, mean pulmonary wedge pressure: 27 +/- 8 mm Hg vs. 20 +/- 7 mm Hg, p = 0
31 s. 13 mm Hg; p = 0.001), pulmonary capillary wedge pressure (29 vs. 24 mm Hg; p = 0.001), but similar
32 pressure (18+/-2 to 7+/-1 mm Hg), pulmonary wedge pressure (32+/-3 to 15+/-2 mm Hg), and systemic va
33 vascular resistance 25%, pulmonary capillary wedge pressure 33%, and central venous pressure 27% whil
34 nitrite reduced resting pulmonary capillary wedge pressure (-4+/-3 versus -1+/-2 mm Hg; P=0.002), im
35 uctions in 0-hour trough pulmonary capillary wedge pressure (-4.3 mm Hg; P=0.16), pulmonary artery di
36 sulted in an increase in pulmonary capillary wedge pressure (54 +/- 78% vs. 32 +/- 26%, p = NS), card
37 es significantly reduced pulmonary capillary wedge pressure (-6.4 +/- 4.1 mm Hg, -5.7 +/- 4.6 mm Hg,
38 icacy end point included pulmonary capillary wedge pressure (72 to 96 hours) and days alive out of ho
39 of ADMA correlated with pulmonary capillary wedge pressure and both systolic and diastolic pulmonary
41 e actual R(micro) and IMR after the coronary wedge pressure and collateral flow were incorporated int
42 -confirmed PH with elevated pulmonary artery wedge pressure and low pulmonary vascular resistance, we
43 ight atrial pressure and pulmonary capillary wedge pressure and lower cardiac index (CI) but not Paco
45 ssure-volume curves from pulmonary capillary wedge pressure and LV end-diastolic volume and Starling
47 d normative responses of pulmonary capillary wedge pressure and mean pulmonary arterial pressure to v
48 the largest increases in pulmonary capillary wedge pressure and mean pulmonary arterial pressure.
49 the presence of a normal pulmonary capillary wedge pressure and portal hypertension, is a known compl
50 dose-dependently reduced pulmonary capillary wedge pressure and pulmonary and systemic vascular resis
51 ulmonary exercise tests, pulmonary capillary wedge pressure and serum sodium were strong predictors o
52 and Starling curves from pulmonary capillary wedge pressure and SV during lower body negative pressur
54 ary artery catheter use, pulmonary capillary wedge pressure and the incidence of diabetes mellitus an
58 re, pulmonary vascular resistance, capillary wedge pressure, and cardiac index were also obtained at
60 cardiac output, decrease pulmonary capillary wedge pressure, and reduce pulmonary and systemic vascul
61 cardiac index, elevated pulmonary capillary wedge pressure, and renal impairment or substantial diur
62 Primary end point was pulmonary capillary wedge pressure, and secondary end points comprised cardi
63 diac output, heart rate, pulmonary capillary wedge pressure, and systemic vascular resistance were me
65 s that pulmonary artery and pulmonary artery wedge pressures are higher in SIPE-susceptible individua
66 tly greater reduction in pulmonary capillary wedge pressure at 3, 4, and 8 hours and during the presp
67 was no effect of age on pulmonary capillary wedge pressure at any point throughout the cardiac cycle
68 r than 40%, and a raised pulmonary capillary wedge pressure at rest (>15 mm Hg) or during exercise (>
69 e were no differences in pulmonary capillary wedge pressure at rest (13+/-4 versus 13+/-3 mm Hg, P=0.
70 d with directly measured pulmonary capillary wedge pressure at rest (r=0.63, P<0.0001) and during exe
71 ients had a reduction in pulmonary capillary wedge pressure at rest, 34 (58%) of 59 had a lower pulmo
72 y be changed by elevated pulmonary capillary wedge pressure, augmenting right ventricular pulsatile l
73 ose-related decreases in pulmonary capillary wedge pressure (average change -5.9+/-0.9 mm Hg and -5.3
74 ulmonary artery pressure or pulmonary artery wedge pressure between SIPE-susceptible subjects and con
75 roving cardiac index and pulmonary capillary wedge pressure, but statistical significance for the pri
76 ram per minute decreased pulmonary-capillary wedge pressure by 6.0 and 9.6 mm Hg, respectively (as co
77 systemic blood pressure, pulmonary capillary wedge pressure, cardiac index, and estimated glomerular
78 mean arterial pressure, pulmonary capillary wedge pressure, cardiac index, or systemic vascular resi
79 gnificant differences in pulmonary capillary wedge pressure, cardiac output, pulmonary vascular resis
80 artery, right atrial and pulmonary capillary wedge pressures, cardiac index, systemic and pulmonary v
81 al, pulmonary artery, or pulmonary capillary wedge pressures; cardiac index; respiratory rate; or hea
82 11; LVAD, 18+/-4 mm Hg) and pulmonary artery wedge pressure (CHF, 16+/-10; LVAD 5+/-3 mm Hg) were red
83 , 30+/-5 mm Hg) and mean pulmonary capillary wedge pressure (CHF, 31+/-11; LVAD, 14+/-6 mm Hg) were l
84 ated LV end-diastolic or pulmonary capillary wedge pressure, consistent with diastolic dysfunction, w
85 as the inverse was true for pulmonary artery wedge pressure (corresponding muPAWP were 21.5, 16.5, an
86 (-2)]; P<0.001) and mean pulmonary capillary wedge pressure decreased (from 15 mm Hg [IQR, 12-20 mm H
87 48 hours after implant, pulmonary capillary wedge pressure decreased 44%, systemic vascular resistan
88 decreased 19% (P=0.03), pulmonary capillary wedge pressure decreased 46% (P=0.002), and mean arteria
89 iac index increased 43%, pulmonary capillary wedge pressure decreased 52%, systemic vascular resistan
91 25.9 +/- 1.7 mm Hg; mean pulmonary capillary wedge pressure decreased from 25.1 +/- 1.1 to 13.2 +/- 1
95 sion (n=1009) and normal pulmonary capillary wedge pressure displayed a consistent R(PA)-C(PA) hyperb
97 (58%) of 59 had a lower pulmonary capillary wedge pressure during exertion, and 23 (39%) of 59 fulfi
99 Exercise elicits greater pulmonary capillary wedge pressure elevation compared with saline in HFpEF b
101 p < 0.01) and increased pulmonary capillary wedge pressure (from 25 +/- 2 to 29 +/- 3 mm Hg, p < 0.0
102 is predictive of a mean pulmonary capillary wedge pressure greater than 15 mm Hg with a 92% sensitiv
103 ction 21+/-1%) who had a pulmonary capillary wedge pressure >/=15 mm Hg and a cardiac index </=2.5 L
104 Patients with a baseline pulmonary capillary wedge pressure >/=15 mm Hg and a cardiac index </=2.5 L.
105 ventricular [RV] DD) or pulmonary capillary wedge pressure >/=18 mm Hg (left ventricular [LV] DD) wi
106 pressure <100 mm Hg and pulmonary capillary wedge pressure >/=24 mm Hg and dependent on >/=2 inotrop
107 ients in MI+DD (29%) had pulmonary capillary wedge pressure >15 (14+/-4 mm Hg), whereas none of the M
108 identified patients with pulmonary capillary wedge pressure >15 mm Hg (area under the curve: 0.73 to
109 ubgroups: (1) patients with pulmonary artery wedge pressure >15 mm Hg; (2) pulmonary vascular resista
110 ic balloon pump support, pulmonary capillary wedge pressure >20 mm Hg and serum creatinine >1.5 mg/dl
111 on class II-IV, elevated pulmonary capillary wedge pressure (>/=15 mm Hg at rest or >/=25 mm Hg durin
112 ise, an abnormal rise in pulmonary capillary wedge pressure (>25 mm Hg) was observed in 94% of MI+DD
113 80% of predicted and peak pulmonary arterial wedge pressure>/=20 mm Hg) with 31 age- and sex-matched
115 0.022), diastolic PAP - pulmonary capillary wedge pressure (HR, 2.19; 95% CI, 1.23-3.89 per 10 mm Hg
116 eart failure to decrease pulmonary capillary wedge pressure, improve cardiac output, stimulate natriu
118 In healthy subjects, pulmonary capillary wedge pressure increased from 10+/-2 to 16+/-3 mm Hg aft
120 y arterial pressure, and pulmonary capillary wedge pressure increased similarly with saline and exerc
123 volume overload, a treatment-induced drop in wedge pressure is often accompanied by a rapid drop in N
124 ch as ejection fraction, cardiac output, and wedge pressure, is available, but also in clinical trial
125 d-diastolic pressures and pulmonary arterial wedge pressures, is associated with remarkably increased
127 ndex, ejection fraction, pulmonary capillary wedge pressure, left ventricular dimensions, watts achie
129 pressure >/=25 mm Hg and pulmonary capillary wedge pressure </=15 mm Hg at right heart catheterizatio
132 furosemide, mannitol (if pulmonary capillary wedge pressure <20 mm Hg), and low-dose dopamine (n = 43
134 tage have revealed a normal pulmonary artery wedge pressure, marked elevation of pulmonary artery pre
135 the true IMR (IMR(true)), which incorporates wedge pressure measurement to account for collateral flo
136 th Revision procedure codes describing PA or wedge-pressure monitoring, measurement of mixed venous b
137 owever, elevation of the pulmonary capillary wedge pressure (n=8142) had a larger impact, significant
138 versus 32 +/- 12%) and mean pulmonary artery wedge pressure of < or = 18 versus > 18 mm Hg (84 +/- 6%
139 wo post-CBC variables: mean pulmonary artery wedge pressure of < or = 18 versus > 18 mm Hg (90 +/- 6%
140 dex (2.4 liters/min.m2), pulmonary capillary wedge pressure of 16 +/- 9 mm Hg (mean +/- SD) and maxim
141 ter, 127 patients with a pulmonary-capillary wedge pressure of 18 mm Hg or higher and a cardiac index
142 re of 40.5+/-11.4 mm Hg, pulmonary capillary wedge pressure of 22.6+/-8.9 mm Hg, and pulmonary vascul
143 x of 1.7 L/min per m(2), pulmonary capillary wedge pressure of 25.6 mm Hg, and left ventricular eject
144 c filling volume was the pulmonary capillary wedge pressure of the seniors lower than that of the you
146 0.02, r = 0.83), higher pulmonary capillary wedge pressure (p = 0.01, r = 0.58) and lower cardiac in
150 y reduced pulmonary arterial (p < 0.001) and wedge pressures (p < 0.01) and pulmonary vascular resist
151 dynamic profiles, including higher pulmonary wedge pressures (P=0.002) and lower cardiac indexes (P<0
152 dilatation, increase of pulmonary capillary wedge pressure, PAP and RAP were more pronounced in AF t
154 ly correlated with supine pulmonary arterial wedge pressure (PAWP; r=0.36; P<0.001) and demonstrated
157 intraarterial catheter, pulmonary capillary wedge pressure (Pcw), continuous cardiac output (Q), and
158 =2.5 l/min per m(2) and pulmonary capillary wedge pressure (PCWP) > or =15 mm Hg) who were admitted
160 nine (2.6 to 1.5 mg/dL), pulmonary capillary wedge pressure (PCWP) (32 to 14 mm Hg), and right atrial
161 by baseline measures of pulmonary capillary wedge pressure (PCWP) and cardiac index (CI), and by cha
162 ficant increases in both pulmonary capillary wedge pressure (PCWP) and central venous pressure (CVP).
163 sitive relationship with pulmonary capillary wedge pressure (PCWP) and left ventricular end-diastolic
164 ns were observed between pulmonary capillary wedge pressure (PCWP) and sole parameters of mitral flow
165 subjects for measures of pulmonary capillary wedge pressure (PCWP) and SV (thermodilution derived car
166 trial pressure (RAP) and pulmonary capillary wedge pressure (PCWP) are correlated in heart failure, i
167 n the prospective group, pulmonary capillary wedge pressure (PCWP) derived as: PCWP(Doppler) = LV(end
170 has been correlated with pulmonary capillary wedge pressure (PCWP) in a wide variety of cardiac condi
171 s noninvasive markers of pulmonary capillary wedge pressure (PCWP) in the setting of critical illness
172 invasive measurement of pulmonary capillary wedge pressure (PCWP) simultaneous with Doppler echocard
173 d ePVH groups had higher pulmonary capillary wedge pressure (PCWP) than the ePH group (P < 0.05).
176 ion fraction, 22 +/- 9%; pulmonary capillary wedge pressure (PCWP), 16 +/- 10 mm Hg; cardiac index (C
177 c nerve activity (RSNA), pulmonary capillary wedge pressure (PCWP), and mean arterial pressure (MAP)
178 in eight males from whom pulmonary capillary wedge pressure (PCWP), central venous pressure and SV (v
179 ressure, as expressed by pulmonary capillary wedge pressure (PCWP), during lower-body negative pressu
180 as associated with lower pulmonary capillary wedge pressure (PCWP), fewer symptoms, and greater quali
182 es of istaroxime lowered pulmonary capillary wedge pressure (PCWP), the primary end point (mean +/- S
184 etween the reductions in pulmonary capillary wedge pressure (PCWP; 25.4, 24.6, 24.0, 23.5, 23.4, 21.5
189 aphic severity correlated significantly with wedge pressure (r = 0.93, P < .001) and pulmonary arteri
190 rong inverse relation with PVR (r=-0.64) and wedge pressure (r=-0.73), and provides stronger predicti
192 VR) ratio had the best correlation with mean wedge pressure (r=0.79, P<0.001), as well as in 24 prosp
194 ed a steeper increase in pulmonary capillary wedge pressure relative to infused volume (25+/-12 mm Hg
195 ed a steeper increase in pulmonary capillary wedge pressure relative to volume infused (16+/-4 mm Hg.
196 n mean pulmonary artery and pulmonary artery wedge pressures, respectively, but no change in transpul
197 and it had no effect on pulmonary-capillary wedge pressure, right atrial pressure, heart rate, or ca
198 LV transmural pressure (pulmonary capillary wedge pressure-right atrial pressure), which reflects LV
199 trans-septal gradient (=pulmonary capillary wedge pressure-right atrial pressure; r=0.67; P=0.003),
201 in pulmonary artery and pulmonary capillary wedge pressures, suggesting abnormal compliance, with ma
202 c output, stroke volume, pulmonary capillary wedge pressure, systemic and pulmonary vascular resistan
203 g pulmonary arterial and pulmonary capillary wedge pressures than the remaining heart-failure patient
204 d pulmonary arterial and pulmonary capillary wedge pressures to a greater level than OMA alone or ACE
207 004), and the corresponding pulmonary artery wedge pressure was 11.0 mm Hg versus 18.8 mm Hg (P=0.028
209 L . min(-)(1) . m(-)(2), pulmonary capillary wedge pressure was 31.5 +/- 5.7 mm Hg, and heart failure
210 . kg(-)(1) . min(-)(1), pulmonary capillary wedge pressure was 5.9 +/- 4.6 mm Hg, and cardiac index
213 vascular resistance and pulmonary capillary wedge pressure was evident at 3 min after infusion in 70
216 r for the two groups but pulmonary capillary wedge pressure was slightly lower for Group C (22 vs. 24
217 administration, exercise pulmonary capillary wedge pressure was substantially improved by nitrite as
219 tion with measurement of pulmonary capillary wedge pressure waveform during 5 different loading condi
220 detailed analysis of the pulmonary capillary wedge pressure waveform obtained by right-heart catheter
221 ar resistance and normal pulmonary capillary wedge pressure, we make a weak recommendation for either
222 vascular resistance and pulmonary capillary wedge pressure were decreased, as compared with untreate
223 The cardiac index and pulmonary-capillary wedge pressure were elevated in the six patients in whom
224 (r=0.947), while PVP and pulmonary capillary wedge pressure were found to be moderately correlated (r
226 rtery pressure (PAP) and pulmonary capillary wedge pressure were obtained by standard techniques.
228 displayed an increase in pulmonary capillary wedge pressure with exercise from 20+/-6 to 34+/-7 mm Hg
229 ubjects displayed higher pulmonary capillary wedge pressure with exercise, but this was solely becaus
230 diac output, and reduces pulmonary capillary wedge pressure without causing deleterious increases in
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