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1 e profiles of patients who recovered from moderate COVID-19 were enriched in tissue reparative growth factor signature A,
2 esting that these cells are primed to grow in the bone, and were enriched in skeletal sites of metastasis over soft tissu
3 ions and mRNA abundance associations with target genes, and were enriched in motif-rewiring mutations and structural vari
4 start sites and accessible chromatin of the host genome and were enriched in repressive chromatin marks.
7 II and in chronically infected individuals, beta7high cells were enriched in integrated and total HIV-1 DNA compared to b
8 e Gene Ontology proteome analysis revealed that SUM52 cells were enriched in proteins associated with cell metabolism and
14 Compared with controls, uniquely upregulated genes in HFpEF were enriched in mitochondrial adenosine triphosphate synthes
17 nile acinar and islet cells in donors without diabetes, LDs were enriched in islet alpha- and beta-cells from donors with
18 Only CD8alphaalpha IELs established in early life were enriched in cells bearing type B IELp TCR usage.
19 ets and mRNAs expressed at low levels with short half-lives were enriched in the polysomes of upf1 mutants, indicating th
20 s 53 tissues, genes in kidney function-associated GWAS loci were enriched in kidney (P=9.1E-8 for eGFR; P=1.2E-5 for urat
22 The resulting transcriptome datasets from LCM microvessels were enriched in known brain endothelial and pericyte markers
27 h anemia compared with control individuals, other mutations were enriched in the anemia cohort, including TP53 and SF3B1.
28 revealed that gene sets of layer 5b and 6 pyramidal neurons were enriched in DEGs of the mPFC downregulated in both NAc D
29 nts of the Fibroblast Growth Factor (FGF) signaling pathway were enriched in nascent myotubes in Drosophila embryos.
31 gment biosynthesis, and other secondary metabolite pathways were enriched in control and drought stressed PRLT2/89-33 at
32 osing solution, C6 and C7 perfluoroalkyl sulfonates (PFSAs) were enriched in dosed mouse serum, suggesting in vivo transf
34 uding TRAIL (TNFSF10), TL1A (TNFSF15), and their receptors, were enriched in E2F1(high) While TRAIL was equally expressed
36 und the genes most informative for predicting drug response were enriched in well-known cancer signaling pathways and hig
37 whereas N. aquilinus (a wood-feeder) gut microbiome samples were enriched in genes involved in carbohydrate metabolism an
38 es of 1,024 metastatic samples revealed that 13 focal SCNAs were enriched in metastatic samples, including gains in chrom
39 or tyrosine kinase (Erbb), and Toll-like receptor signaling were enriched in lysates of podocytes treated with the TrkC l
40 ia targeted expression profiling(17) that B cell signatures were enriched in the tumours of patients who respond to treat
44 In the presence of inflammation, portal tracts were enriched in CD3+, CD20+ but displayed fewer CD68+.
45 In humans and mice, sTDP43 transcripts were enriched in vulnerable motor neurons, and we observed a
49 Interestingly, rare, predicted-damaging LRP2 variants were enriched in a HLHS cohort; however, understanding their