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1                                    CoQ domains within cells were highly enriched adjacent to ER-mitochondria contact site
2                                               Peptides that were highly enriched against IgY from at least 4 out of 10 in
3                                           PD-L1 alterations were highly enriched among non-germinal center DLBCLs and exh
4                                                     Targets were highly enriched for APC-related functions, including mic
5 vated enhancers, marked by increased deposition of H3K27Ac, were highly enriched for cistromic elements associated with P
6                                  These ATP-responsive genes were highly enriched for defense-related Gene Ontology terms.
7                                             The annotations were highly enriched for disease heritability, but produced o
8                                                  SCAF cells were highly enriched for gene expression signatures associate
9 ith differential sensitivities between 2D and 3D conditions were highly enriched for genes that are mutated in lung cance
10 is in vivo Genes encoding some upstream activators of NOTCH were highly enriched for H3K27me3, which forms repressive chr
11                            We found that neutrophil genomes were highly enriched for heterochromatic genomic interactions
12                                            The target genes were highly enriched for insulin-related functions including
13          Shortly after challenge, IFN-gamma-producing cells were highly enriched for Ly6C(+)T-bet(+) cells in the viscera
14                                             Candidate genes were highly enriched for miR-200c seed pairing in their 3'UTR
15 tion of the Nfib locus, and differentially accessible sites were highly enriched for Nfib transcription factor binding si
16          Methylated genes in WT1mut primary patient samples were highly enriched for polycomb repressor complex 2 (PRC2)
17                                                 These genes were highly enriched for processes involved in pulmonary stru
18                                  These purified populations were highly enriched for replication-competent and intact pro
19   Candidate genes required for reovirus to cause cell death were highly enriched for sialic acid synthesis and transport.
20  times across several large recombination cold spots, which were highly enriched for signatures of selection-driven post-
21  immune-mediated disorders, including rheumatoid arthritis, were highly enriched for T-cell SEs versus typical enhancers
22                                                Lung signals were highly enriched for those seen in newborn (24 overlappin
23                                                 These genes were highly enriched for transcription factors and overlapped
24                 Down-regulated genes in BAB compared to BAN were highly enriched for vasculature-related genes, suggestin
25                           In contrast, LMX1A-eGFP(+) grafts were highly enriched for vmDA neurons, and importantly exclud
26 With Mito-AP, intact mitochondria from 0.5 g plant material were highly enriched in 30-60 min, considerably faster than w
27 wed that over 600 gRNAs including the ones targeting LGALS2 were highly enriched in cells survived after sublethal H(2)O(
28 peptide in proliferation assays and the proliferating cells were highly enriched in certain T-cell receptor sequences.
29                Rho family GTPases and Wnt signaling pathway were highly enriched in ChIP-seq and RNA-seq datasets and mem
30                         Progastrin expression and secretion were highly enriched in colon CSC isolated from human colorec
31 ify a number of significantly mutated genes in NSCLC, which were highly enriched in DNA damage repair, NF-kappaB pathway,
32                                       Their human orthologs were highly enriched in ISGs, particularly for highly inducib
33                                          Cas9-bound regions were highly enriched in NGG sites, a sequence required for ta
34 helial growth factor receptor/FLT1, E2F2 and PCM1 oncogenes were highly enriched in ovarian cancer patients compared with
35 nes bound PGR within 3 kb of the gene and PGR binding sites were highly enriched in proximal promoter regions in close pr
36 pid polymer assembly, cell wall organization, and loosening were highly enriched in reticulated skin tissue.
37                                                Eight miRNAs were highly enriched in RISC of both control and infected cel
38                                                Such rhythms were highly enriched in secreted proteins accumulating tightl
39 ines and that actin-related proteins and cysteine proteases were highly enriched in the group.
40 omposed of unusual hydroxylated C17 sphingoid bases (t17:0) were highly enriched in the infected cells, and their synthes
41                                                         LDN were highly enriched in the low-density (LD) fraction of adva
42           In addition, we identified molecular markers that were highly enriched in UCP1-positive human adipocytes, a set
43                                               These regions were highly enriched near protein coding genes and had signif
44                                                   Oncogenes were highly enriched on amplified ecDNA, and the most common
45                                  Strongly upregulated genes were highly enriched on chromosome X, consistent with sexuall
46 ion, we identified small regulatory microRNAs (miRNAs) that were highly enriched or exclusive in MNs.
47 ted loss-of-function (pLOF) and nonsynonymous variants that were highly enriched or private in the SWAI population.
48                                                        Both were highly enriched over concentrations in underlying soils,
49 evealed that potato and Arabidopsis BiP1 and BiP2 promoters were highly enriched with cis-regulatory elements (CREs) link
50 tation assays demonstrated that long-term repopulating HSCs were highly enriched within the gata2a(+) runx1(+) fraction.