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1 les of chemoenzymatically generated 3-ketopentanoyl-ACP (9) were incubated with SAM and BonMT2 from module 2 of the bongk
2 cetaminophen (N-acetyl-p-aminophenol, APAP) and (13)C6-APAP were incubated with rat liver microsomes, which are known to
3 luorometric method; passive HR-IgE-stripped donor basophils were incubated with participants' serum and histamine release
4                                                    Biopsies were incubated with gluten digested with chymotrypsin (modifi
5                                              Cardiomyocytes were incubated with alkaline phosphatase, subsequently reasse
6                                     Cultured cardiomyocytes were incubated with clinically relevant histone concentration
7                                                       Cells were incubated with 1:1 decafluorobutane (DFB)/octafluoroprop
8                                                       Cells were incubated with EtOH (50 mmol/L), CSE (20-40 mug/mL), or
9                                                       Cells were incubated with forskolin and H89 to activate or inhibit
10                                                 HT-29 cells were incubated with cell stressors such as DTT, IFN, and adhe
11                                          In addition, cells were incubated with NRG1-beta, a mediator of HER2-HER3 signal
12                                         Breast cancer cells were incubated with DOX azide and (68)Ga, after which cell vi
13                                          Fibrosarcoma cells were incubated with (18)F-FDG and exposed to Cy7 azide with s
14                                         Primary glial cells were incubated with leptomycin b and MG132 to block nuclear e
15                           In addition, human hepatoma cells were incubated with different propionate concentrations.
16                To determine the effect of IL-12, hPDL cells were incubated with recombinant human IL-12 (p70) in a dose-
17  (LPS) to induce expression of NLRP3, IL1B, and IL18; cells were incubated with LPS and adenosine triphosphate to activat
18                                 C2C12 or human muscle cells were incubated with palmitate or directly with ceramide for s
19                                                  Some cells were incubated with methyl-beta-cyclodextrin (to deplete chol
20                                                  Some cells were incubated with MKC-3946, an inhibitor of endoplasmic ret
21                                              CD4(+) T cells were incubated with antigen, or agonist to CD3 and dendritic
22 ith copper, and nuclear fluorescence was reduced when cells were incubated with the CuDox complex as compared with free d
23                                                  When cells were incubated with TNF before infection, the subsequent anti
24        Primary human HSCs and immortalized HSCs (LX2 cells) were incubated with conditioned medium derived from HCV-expos
25 n ex vivo human model of BP, normal human skin cryosections were incubated with purified human peripheral blood eosinophi
26                                           Human eosinophils were incubated with T cells that were stimulated with allogen
27 s, HBV-infected HepaRG cells, and primary human hepatocytes were incubated with interferon-gamma (IFNgamma) or tumor necr
28        Human hepatoma (Huh-7 and Huh-7.5) and primary HFLCs were incubated with TNF and/or recombinant IFNA2A, IFNB, IFNL
29                                                       HUVEC were incubated with human whole blood.
30                            Human endothelial cells (HUVECs) were incubated with (18)F-FDG or (18)F-FLT and cell character
31                             In contrast, when keratinocytes were incubated with 4MU, cell proliferation was decreased.
32                                                 Macrophages were incubated with pan-phosphatidylinositol 3-kinase inhibit
33 primary macrophages from mice and human primary macrophages were incubated with lipopolysaccharide (LPS) to induce expres
34                                        When these mediators were incubated with diseased cells, we only found a modest do
35               Primary enteric glial cells from C57BL/6 mice were incubated with gastrin and separated into nuclear and cy
36      Enteroids derived from control and ATP7B-knockout mice were incubated with excess Cu or with Cu-chelating reagents;
37 tic cancer cells derived from Pdx1-Cre;LSL-Kras(G12D) mice) were incubated with inhibitors of different kinases and analy
38                                      Epitrochlearis muscles were incubated with [(3)H]-2-deoxyglucose (2DG) +/- 100 micro
39                                           The nanoparticles were incubated with the initial growth medium, isolated secre
40 NA on Toll-like receptors-9 expression, healthy neutrophils were incubated with acetaminophen-induced acute liver failure
41                                        Isolated neutrophils were incubated with immune complexes, and activation and rele
42 acrophage cell line RAW 264.7, or mouse and human organoids were incubated with second mitochondrial activator of caspase
43 astrectomy or sleeve resection or gastric antral organoids) were incubated with interferon gamma (IFNG) or interferon bet
44                                    Toxin-containing samples were incubated with magnetic beads coated with multiple abrin
45               Eighteen aflatoxin-contaminated maize samples were incubated with potassium hydroxide, trifluoromethanesulf
46                                                  Human sera were incubated with RBC isolated from various genetically eng
47 ar R-SNARE or one of the three integrally anchored Q-SNAREs were incubated with the tethering/SM protein complex HOPS and
48    In contrast, there was no infection of tissues when they were incubated with lymphocyte-attached HIV-1.
49                    In addition, cultured human trophoblasts were incubated with (13)C-cholecalciferol to examine the intr
50 n response to phenylephrine that was abolished when vessels were incubated with a CSE inhibitor, propargylglycine.

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