ライフサイエンスコーパス: wheeze

1 iagnosed (76 with asthma, 122 with transient wheeze).

2 mples of children who went on to have atopic wheeze.

3 e childhood-onset persisting, and continuous wheeze.

4 ow known to be present in young children who wheeze.

5 ferent RV species during an acute episode of wheeze.

6 for FEF25-75) compared with never/infrequent wheeze.

7 ment of allergic sensitization and recurrent wheeze.

8 nce than simply the observation of recurrent wheeze.

9 72 hours, including 13 with parent-reported wheeze.

10 with childhood asthma, independent of infant wheeze.

11 M-sensitized children did not have asthma or wheeze.

12 tudy were used to define asthma, eczema, and wheeze.

13 RSV-induced illness and subsequent recurrent wheeze.

14 28]), and we associated them with asthma and wheeze.

15 nses at birth were associated with recurrent wheeze.

16 associated with later development of atopic wheeze.

17 t associations for allergic and non-allergic wheeze.

18 of asthma if started in the young child with wheeze.

19 sitization, and only five reported on asthma/wheeze.

20 abolite of di-isodecyl phthalate (DIDP), and wheezing.

21 of breath, chest tightness, coughing, and/or wheezing.

22 shortness of breath, chest pain, cough, and wheezing.

23 diagnostic tests for recurrent or persistent wheezing.

24 n and jaundice, itching, flushing, cough and wheezing.

25 he prevention of postbronchiolitis recurrent wheezing.

26 he occurrence of postbronchiolitis recurrent wheezing.

27 cts the airways and presents as coughing and wheezing.

28 to promote airway obstruction and recurrent wheezing.

29 l eczema and rhinoconjunctivitis, but not to wheezing.

30 r/infrequent wheeze (74.1%), early transient wheeze (12.7%), and persistent wheeze (13.1%).

31 rly transient wheeze (12.7%), and persistent wheeze (13.1%).

32 ficantly associated (p < 0.05) with allergic wheeze (18 positive, 1 negative) and 21 pesticides with

33 egative) and 21 pesticides with non-allergic wheeze (19 positive, 2 negative); 11 pesticides were ass

34 cara spp. seropositivity was associated with wheeze (2.97[1.45- 7.76]), hayfever (4.03[1.63-9.95]), e

35 r fever (20% vs 16%) and less likely to have wheeze (30% vs 38%; all P < .05).

36 ness of breath (1.86, 0.97-3.57; p<0.1), and wheeze (4.00, 1.52-10.50; p<0.05) after walking down Oxf

37 rs; 77% phlegm; 70% shortness of breath; 47% wheezing; 46% chest pain; 42% abnormal peak flow), 334 (

38 itudinal wheeze phenotypes: never/infrequent wheeze (74.1%), early transient wheeze (12.7%), and pers

39 hteen children developed asthma or recurrent wheeze: 98 of 405 (24.3%; 95% CI, 18.7%-28.5%) in the 44

40 DEHP) was positively associated with current wheeze (adjusted OR 1.56, 95% CI 1.03-2.37), whereas the

41 d odds ratio [OR], 1.26; 95% CI, 0.98-1.61), wheeze (adjusted OR, 1.02; 95% CI, 0.82-1.27), and SPT r

42 ificant associations of e-cigarette use with wheeze after adjustment for cigarette use.

43 Disease phenotypes included asthma, atopy, wheezing, altered lung function, and bronchial reactivit

44 ed pesticides with allergic and non-allergic wheeze among male farmers.

45 which we identified 27 children with atopic wheeze and 70 healthy control subjects at 5 years of age

46 its effectiveness in children with preschool wheeze and a corticosteroid responder phenotype has not

47 ver, studies investigating associations with wheeze and asthma in later childhood are scarce and did

48 in pregnant women on the risk of persistent wheeze and asthma in their offspring.

49 Although sexual dimorphism in wheeze and asthma prevalence are well documented, sex-sp

50 nd allow for a more personalized approach to wheeze and asthma treatment.

51 ng pregnancy increases the risk of childhood wheeze and asthma.

52 airway obstruction common to both transient wheeze and asthma.

53 ren (<4 years of age) at an acute episode of wheeze and at a follow-up visit (median 11 weeks later).

54 t 9 months of life regarding the presence of wheeze and cough.

55 al levels in infancy to repeated measures of wheeze and development of asthma and rhinitis by age 13,

56 We defined allergic wheeze as reporting both wheeze and doctor-diagnosed hay fever (n = 1,310, 6%) an

57 Data on wheeze and eczema were collected by means of questionnai

58 Pediatric ED visits for asthma or wheeze and for upper respiratory infections were associa

59 Contrasting changes were noted with falling wheeze and HDM sensitisation but rising rhinitis and gra

60 er, the mechanisms underlying development of wheeze and how and why only some children progress to as

61 We identified sex-specific predictors of wheeze and longitudinal wheeze patterns, which might hav

62 ll documented, sex-specific risk factors for wheeze and longitudinal wheeze phenotypes have not been

63 ren at risk of persistent asthma (defined as wheeze and presence of airflow limitation or airway hype

64 00 days of life on the development of atopic wheeze and provide additional support for considering mo

65 ssociation between allergic and non-allergic wheeze and self-reported use of 78 specific pesticides,

66 We estimated hazard ratios and, for wheeze and sensitization, odds ratios for an interquarti

67 ciations between prevalence of self-reported wheeze and shortness of breath and annual mean particula

68 lution was associated with the prevalence of wheeze and shortness of breath in this large study, with

69 ir pollution exposures and the prevalence of wheeze and shortness of breath using harmonized baseline

70 o NO2 also showed positive associations with wheeze and shortness of breath.

71 2012, prevalence of lifetime wheeze, current wheeze and those ever treated for asthma decreased by 15

72 oth relations persisted after adjustment for wheeze and total IgE at age 1.

73 Pulmonary examination revealed wheezes and decreased basilar air entry.

74 and early life are associated with recurrent wheezing and aeroallergen sensitivity and altered cytoki

75 t of physician-diagnosed asthma or recurrent wheezing and allergic sensitization to food or environme

76 asurements, and assessed physician-diagnosed wheezing and asthma by questionnaires.

77 es predictors of remission or persistence of wheezing and asthma from early childhood through adultho

78 n 3 expression and correlated inversely with wheezing and asthma in nonatopic subjects.

79 al exhaled nitric oxide (Feno), and risks of wheezing and asthma in school-aged children.

80 thma risk factors and the natural history of wheezing and asthma through childhood and beyond.

81 infancy is a major risk factor for recurrent wheezing and asthma.

82 man was admitted to our hospital because of wheezing and dyspnea.

83 respectively); rs3918396 was associated with wheezing and eczema comorbidity (p = 3.41*10(-4)).

84 Due to its association with wheezing and eczema comorbidity, ADAM33 may also be invo

85 stion, he experienced coughing, dyspnea, and wheezing and had to be treated by anti-histamine and ste

86 n of maternal stress/depression to recurrent wheezing and peripheral blood mononuclear cell cytokine

87 n, including the increased risk of childhood wheezing and subsequent asthma.

88 lergic sensitization, lung function, current wheeze, and asthma (n = 1405) were investigated using re

89 l geohelminths on the development of eczema, wheeze, and atopy during the first 3 years of life.

90 three of physician-diagnosed asthma, current wheeze, and current use of asthma treatment, reported by

91 occurrence of chest infections, bronchitis, wheeze, and eczema in children was assessed at ages 6 an

92 were more likely to have chest retractions, wheezing, and a history of underlying asthma/reactive ai

93 also observed between 2,5-dichlorophenol and wheezing, and between monocarboxynonyl phthalate, a meta

94 respiratory symptoms (difficulty breathing, wheezing, and cough) lasting >/= 2 days or requiring pre

95 yfish product (100g), he experienced nausea, wheezing, and erythema and had visited our hospital.

96 o farm animal sheds was inversely related to wheeze (aOR, 0.44; 95% CI, 0.33-0.60).

97 range increase in NO2 [2.2 ppb]) and current wheeze (aOR, 1.14; 1.02-1.28).

98 rom a major road was associated with current wheeze (aOR, 1.38; 95% CI, 1.06-1.80) and atopy (aOR, 1.

99 We defined allergic wheeze as reporting both wheeze and doctor-diagnosed hay

100 d hay fever (n = 1,310, 6%) and non-allergic wheeze as reporting wheeze but not hay fever (n = 3,939,

101 ment of allergic sensitization and recurrent wheeze assessed at age 3 years.

102 short illness, temperature, age, recession, wheeze, asthma, and vomiting (mnemonic STARWAVe; AUROC 0

103 Wheezing, asthma and AD were assessed from questionnaire

104 -term lung health, with potential effects on wheezing, asthma, and chronic lung disease.

105 itization is related to an increased risk of wheeze/asthma (pooled OR 2.9; 95% CI 2.0-4.0), eczema (p

106 Eczema, wheeze/asthma, and allergic rhinitis were diagnosed at t

107 ncreased risk of infantile eczema, childhood wheeze/asthma, eczema and allergic rhinitis and young ad

108 tization in the first 2 years and subsequent wheeze/asthma, eczema and/or allergic rhinitis.

109 cally significant change in the incidence of wheezing/asthma after introduction of smoke-free legisla

110 le reductions in the incidence of paediatric wheezing/asthma or RTIs following introduction of smoke-

111 A total of 1623 children had information on wheeze at 1 or more time points.

112 onary function and decrease the incidence of wheeze at 1 year of age.

113 Overall, 166 (36%) children had recurrent wheeze at age 3 years.

114 floor dust were associated with reduced: i) wheeze at any age; ii) fungal sensitization; and iii) as

115 hagia at 1 month, nausea at 3 and 12 months, wheezing at 6 months; and inability to belch at 12 month

116 asthmatic symptoms, as well as virus-induced wheezing, at any time before biomarker assessment at age

117 rain) and five main atopic outcomes (asthma, wheeze, atopic dermatitis, allergic rhinitis and IgE) we

118 Asthma and wheezing begin early in life, and prenatal vitamin D def

119 bjects (of whom 57 developed adulthood-onset wheeze between ages 16 and 46 yr).

120 10, 6%) and non-allergic wheeze as reporting wheeze but not hay fever (n = 3,939, 18%); men without w

121 were significantly associated with recurrent wheezing but not increased atopy or reduced antiviral re

122 ome children with rhinovirus (RV) infections wheeze, but it is unknown whether this is due to more vi

123 s not associated with the risk of persistent wheeze, but the number of episodes of troublesome lung s

124 ed with the lowest risk for asthma/recurrent wheeze by age 3 years compared with having an initial le

125 s (P = .03), as well as male sex (P = .025), wheezing causing shortness of breath (P = .002), and ACS

126 rhinovirus (RV) is most commonly detected in wheezing children thereafter.

127 se bronchodilators to manage childhood acute wheezing conditions in the emergency department (ED), an

128 included participants reporting symptoms of wheeze, cough, or breathlessness in the previous 12 mont

129 ariable airflow obstruction and intermittent wheezing, cough, and dyspnea.

130 od from 2001 to 2012, prevalence of lifetime wheeze, current wheeze and those ever treated for asthma

131 association of parental migrant status with wheezing disorders among children born in Hong Kong, a d

132 contribute to the development of early life wheezing disorders and asthma, and discuss the external

133 higher risk of hospitalization for childhood wheezing disorders compared to the native population, pa

134 risk of hospitalization for asthma and other wheezing disorders, compared to both parents being Hong

135 uting factor to the increasing prevalence of wheezing disorders.

136 the aetiology of asthma and other childhood wheezing disorders.

137 s (g/dL) in pregnancy with hayfever, eczema, wheezing, doctor-diagnosed asthma, allergic sensitisatio

138 0.40-0.97) was associated with lower odds of wheeze during childhood.

139 risk alleles at the 17q21 genetic locus who wheeze during rhinovirus illnesses have a greatly increa

140 Although wheezes, dyspnea or loss of consciousness are known to o

141 erized by episodic or persistent symptoms of wheezing, dyspnea, and cough.

142 Main Outcomes and Measures: Wheeze, eczema, allergic rhinitis, food allergy, allergi

143 s positively associated with risk for asthma/wheeze, eczema, and sensitization at 10 years; adjustmen

144 nal period and (ii) documentation of asthma, wheezing, eczema, or other atopic disease in the offspri

145 steroid-naive children with the first severe wheezing episode (90% hospitalized/10% emergency departm

146 piratory syncytial virus/rhinovirus-negative wheezing episode (adjusted OR, 8.0; P = .001), first whe

147 ment resulted in prolonged time to the third wheezing episode (P = .048) and in fewer days with respi

148 episode (adjusted OR, 8.0; P = .001), first wheezing episode at age less than 12 months (adjusted OR

149 to identify risk factors at the first severe wheezing episode for current asthma 7 years later and se

150 IL-8 levels, prolonged the time to the third wheezing episode, and reduced overall respiratory morbid

151 ical markers at the time of the first severe wheezing episode.

152 The occurrence of wheezing episodes was assessed monthly over the ensuing

153 Maternal stress, depression, and childhood wheezing episodes were assessed by quarterly questionnai

154 older than 5 years reported higher rates of wheeze ever (OR, 1.4; 95% CI, 1.05-1.8) and recent wheez

155 born children less than 5 years old reported wheeze ever compared with 39% of full term-born children

156 t-acting bronchodilators to treat asthma and wheeze exacerbations in children 0-18 years presenting t

157 t asthma, bronchodilator responsiveness, and wheeze followed similar patterns.

158 ed to identify sex-specific risk factors for wheeze from birth through midchildhood and identify dist

159 groups of preschoolers with severe recurrent wheeze: group 1, less than or equal to 36 months (n = 20

160 Early life aeroallergen sensitization and RV wheezing had additive effects on asthma risk at adolesce

161 The interviews also inquired about wheeze, hay fever, eczema, and food allergy.

162 n were further validated using virus-induced wheezing illness and asthma phenotypes in an independent

163 Early life rhinovirus (RV) wheezing illnesses and aeroallergen sensitization increa

164 ses that affect the development of recurrent wheezing illnesses and allergic sensitization.

165 o new strategies for the prevention of viral wheezing illnesses and perhaps reduce the subsequent ris

166 (eg, airway microbiome) promote more severe wheezing illnesses and the risk for progression to asthm

167 Early life aeroallergen sensitization and wheezing illnesses associated with virus and bacterial i

168 RSV-induced wheezing illnesses during infancy influence respiratory

169 The etiology and timing of viral wheezing illnesses during the first 3 years of life were

170 isease in childhood and is often preceded by wheezing illnesses during the preschool years.

171 Viral respiratory infections can cause acute wheezing illnesses in children and exacerbations of asth

172 Viral infections are closely linked to wheezing illnesses in children of all ages.

173 mage and yet is a significant contributor to wheezing illnesses in young children and in the context

174 was most strongly associated with outpatient wheezing illnesses with RV and aeroallergen sensitizatio

175 nd early life stress may influence childhood wheezing illnesses, potentially through effects on immun

176 The associations between viral wheezing illnesses, presence and pattern of aeroallergen

177 re associated with the development of atopic wheeze in a nonindustrialized setting.

178 effects of gut microbial dysbiosis on atopic wheeze in a population living in a distinct developing w

179 tte use with chronic bronchitis symptoms and wheeze in an adolescent population.

180 c dermatitis were associated with persistent wheeze in both sexes, but paternal asthma was associated

181 ternal asthma was associated with persistent wheeze in boys only (OR, 4.27; 95% CI, 2.33-7.83; P for

182 gnancy against the development of eczema and wheeze in early childhood, although there was evidence i

183 Virus-triggered wheeze in infancy predicts asthma in individuals with a

184 Outcomes were reported wheeze in last year, atopy assessed both by skin prick t

185 tamin D supplementation and asthma/recurrent wheeze in offspring at 3 years.

186 n pregnancy and the risk of asthma/recurrent wheeze in offspring.

187 GMR 2.37 (1.39, 4.06)], A. lumbricoides with wheeze in participants >/=5 years [aOR 6.36 (1.10, 36.63

188 and assessed its role in the development of wheeze in the child.

189 Age at onset of persistent wheeze in the first 3 years of life.

190 arly (< 4 months) and late-onset eczema; and wheeze in the first year of life.

191 astfeeding was protective against late-onset wheeze in the IoW cohort.

192 ater than 30 ng/mL, reduced asthma/recurrent wheeze in the offspring through age 3 years, suggesting

193 cally significant reduced risk of persistent wheeze in the offspring through age 3 years.

194 chronic cough, phlegm, or bronchitis) and of wheeze in the previous 12 months were examined in 2,086

195 rnal asthma was a stronger predictor of ever wheezing in boys (odds ratio [OR], 2.15; 95% CI, 1.74-2.

196 Wheezing in infancy has been associated with subsequent

197 causes respiratory diseases, including acute wheezing in infants, of which life-threatening cases hav

198 lementation in pregnancy on asthma/recurrent wheezing in offspring (P for interaction = .77).

199 ory tract infection and subsequent long-term wheezing in term infants.

200 ciated with an increased risk for persistent wheezing in the child until the age of 5.

201 tions and has been associated with decreased wheezing in the first years of life.

202 intake during pregnancy may protect against wheezing in the offspring, but the preventive effect of

203 The incidence of asthma and recurrent wheezing in their children at age 3 years was lower by 6

204 risk factors facilitating severe asthma and wheezing, including airborne viruses, smoke, indoor damp

205 ontroversies in the diagnostic evaluation of wheezing infants.

206 ild cohort LINA for whom maternal stress and wheezing information was available (n = 443).

207 Although early childhood wheeze is common, persistent asthma is less common.

208 ucture in preschoolers with severe recurrent wheeze is poorly described.

209 Infantile wheezing is a common problem, but there are no guideline

210 Wheezing is common in childhood.

211 Although infantile wheezing is common, there is a paucity of evidence to gu

212 = 1.2; 95% CI: 1.0, 1.5, respectively), and wheezing lasting >/= 2 days, resulting in a doctor visit

213 tically deregulated and could be linked with wheezing later in children's life.

214 In preschoolers with severe recurrent wheeze, markers of remodeling and inflammation are unrel

215 Compared with never/infrequent wheeze, maternal asthma, infant bronchiolitis, and atopi

216 Variable patterns of childhood wheezing might indicate differences in the cause and pro

217 tion (particularly by 2 years) in the asthma/wheeze models reduced 25(OH)D associations with these la

218 ne 2010 for 2- to 18-year-olds for asthma or wheeze (n = 189,816), and for 0- to 18-year-olds for bro

219 noea (n=513 [84%]), cough (n=500 [81%]), and wheezing (n=427 [70%]); 294 (48%) patients had fever.

220 f boiled jellyfish, he experienced erythema, wheezing, nausea, and abdominal pain.

221 CI], 0.06 [0.01-0.12]) and increased risk of wheezing (odds ratio [95% CI], 1.07 [1.00-1.14], per Z s

222 gative ARI, were more likely to present with wheezing (odds ratio [OR], 1.7; 95% CI, 1.23-2.35; P < .

223 sociations seen for PM2.5 with prevalence of wheezing {odds ratio (OR)=1.16 per 5mug/m(3) [95% confid

224 subcostal recession, and clinician-reported wheeze on auscultation.

225 ancy reduced the absolute risk of persistent wheeze or asthma and infections of the lower respiratory

226 The risk of persistent wheeze or asthma in the treatment group was 16.9%, versu

227 The primary end point was persistent wheeze or asthma, and the secondary end points included

228 aben concentrations with asthma or recurrent wheeze or food or environmental sensitization at age 3 y

229 of the prevalence and severity of recurrent wheezing or asthma in children aged up to 6 years.

230 associated with offspring hayfever, eczema, wheezing or asthma.

231 chronic bronchitis or COPD, and a history of wheezing or use of respiratory inhalers in the last 12 m

232 PFOA), was inversely associated with current wheeze (OR 0.64, 0.41-0.99).

233 was associated with ED visits for asthma or wheeze (OR = 1.013; 95% CI: 1.003, 1.023) and upper resp

234 ure were significantly more likely to report wheezing (OR = 1.92; 95% CI: 1.32, 2.79); headaches (OR

235 zziness (OR = 4.21; 95% CI: 2.69, 6.58), and wheezing (OR = 4.20; 95% CI: 2.86, 6.17).

236 1.1-1.9), and 26% versus 17% reported recent wheezing (OR, 1.7; 95% CI, 1.3-2.4).

237 ncentrations and childhood asthma, recurrent wheeze, or allergic sensitization in the overall study p

238 osed patients, aged 18-80 years, with cough, wheeze, or dyspnoea and less than 20% bronchodilator rev

239 a (OR, 1.06 [95% CI, 0.99-1.14]; n = 14438), wheezing, or allergy.

240 with asthma symptoms; and days of coughing, wheezing, or chest tightness) across 6, 9, and 12 months

241 ever (OR, 1.4; 95% CI, 1.05-1.8) and recent wheezing over the last 12 months than full-term control

242 3 were positively associated with recurrent wheeze (P < 0.05).

243 was significantly associated with recurrent wheezing (P </= 0.01).

244 aire symptom scores (p=0.037), and increased wheezing (p=0.018), but no evidence of an association wi

245 ecific predictors of wheeze and longitudinal wheeze patterns, which might have important prognostic v

246 Childhood wheezing phenotype was related to 1989, 1995, 2001, and

247 childhood and identify distinct longitudinal wheeze phenotypes and the sex-specific risk factors asso

248 fic risk factors for wheeze and longitudinal wheeze phenotypes have not been well elucidated.

249 eastmilk fatty acid composition and specific wheeze phenotypes or doctor-diagnosed asthma.

250 dentified sex-specific predictors of wheeze, wheeze phenotypes, and sex-specific predictors of these

251 We identified 3 longitudinal wheeze phenotypes: never/infrequent wheeze (74.1%), earl

252 g was assessed at each age, and longitudinal wheezing phenotypes (early-transient, late-onset, persis

253 l milk fatty acid composition with childhood wheezing phenotypes and asthma up to age 13 years using

254 Adjusted risk ratios with parent-reported wheezing phenotypes and doctor-diagnosed asthma were com

255 new insights into the physiology underlying wheezing phenotypes based on age of onset and duration o

256 We characterized childhood wheezing phenotypes from infancy to adolescence and thei

257 COPSAC study or with asthma or virus-induced wheezing phenotypes in the COAST study.

258 Six wheezing phenotypes were identified: never/infrequent, p

259 In conclusion, after a decrease in wheeze prevalence among Dutch schoolchildren between 198

260 t to many countries, a decrease in childhood wheeze prevalence was previously reported for the Nether

261 pregnancy is necessary for asthma/recurrent wheeze prevention in early life.

262 ed to derive phenotypes based on patterns of wheezing recorded at up to 14 time points from birth to

263 he development of allergic sensitization and wheezing respiratory tract illnesses caused by viruses a

264 Symptoms of wheeze, rhinitis, fever, and environmental exposures wer

265 The relative risks (RRs) of wheeze (RR, 1.20; 95% CI, 1.03-1.40; P = .02), chest inf

266 hma (RR 0.99 [95% CI: 0.77-1.27], P = 0.95), wheezing (RR 1.02 [95% CI: 0.89-1.17], P = 0.76) or rhin

267 Recurrent wheeze (RW) is frequent in childhood.

268 Mothers reported child wheeze symptoms over the past year approximately yearly

269 ripheral eosinophil counts (P = .03) but not wheezing symptoms, baseline spirometric indices, or resp

270 ere not associated with an asthma diagnosis, wheezing symptoms, lung function measures, or prior sick

271 tion of infants with recurrent or persistent wheezing that is not relieved or prevented by standard t

272 Early childhood-onset wheezing that persists into adolescence represents the c

273 nfant biomarkers to the history of recurrent wheezing, the Asthma Predictive Index and its subsequent

274 t of physician-diagnosed asthma or recurrent wheezing through 3 years of age and (2) third trimester

275 Asthma and recurrent wheezing until age 3 years were recorded.

276 ants were followed up for medically attended wheezing until they reached age 3 years.

277 imilarly observed in children with transient wheeze up to age 3 years without subsequent development

278 Prevalence of reported wheeze was 2% in under-fives and 5% in participants >/=5

279 In contrast, adulthood-onset wheeze was associated with accelerated FEV1 decline.

280 Midchildhood-onset (4(1/2) years) remitting wheeze was associated with BDR (OR, 1.77; 95% CI, 1.11-2

281 Of the 581 children, persistent wheeze was diagnosed during the first 3 years of life in

282 The presence of wheeze was independently associated with less frequent t

283 ), whereas the association between PM2.5 and wheeze was limited to lower-income participants [OR=1.30

284 Preschool-onset (18 months) remitting wheeze was only associated with FEV1/FVC ratio decrement

285 Early wheeze was positively associated with presence of older

286 Parental-reported wheezing was assessed at each age, and longitudinal whee

287 A physician's diagnosis of asthma/recurrent wheezing was noted in 67%, and 51% were receiving regula

288 Eczema and wheeze were reported for 17.7% and 25.9%, respectively,

289 not hay fever (n = 3,939, 18%); men without wheeze were the referent.

290 d serum samples from 121 children with acute wheezing were analyzed by means of serology.

291 We identified sex-specific predictors of wheeze, wheeze phenotypes, and sex-specific predictors o

292 it inflammation have efficacy for RV-induced wheezing, whereas the anti-RSV mAb palivizumab decreases

293 ren have high rates of allergic diseases and wheezing, which are diseases associated with type 2-bias

294 infection is a common trigger for childhood wheezing, which is a risk factor for subsequent asthma d

295 , aged 1 to 16 years, admitted for asthma or wheezing who identified as African American (n = 441) or

296 the risk is greatest for young children who wheeze with RV infections.

297 1), eczema (adjusted OR, 4.8; P = .014), and wheezing with rhinovirus (adjusted OR, 5.0; P = .035).

298 When adjusted for all viral etiologies, wheezing with RV (odds ratio = 3.3; 95% CI, 1.5-7.1), bu

299 as significantly associated with measures of wheeze, with no observed protective effect regardless of

300 ht be useful in disease modification for the wheezing young child at risk of persistent asthma.