ライフサイエンスコーパス: wheezing

1 diagnostic tests for recurrent or persistent wheezing.

2 n and jaundice, itching, flushing, cough and wheezing.

3 he prevention of postbronchiolitis recurrent wheezing.

4 he occurrence of postbronchiolitis recurrent wheezing.

5 ng is an important risk factor for recurrent wheezing.

6 valence or incidence of atopy, AD, asthma or wheezing.

7 group is generally referred to as preschool wheezing.

8 quelae of persistent airway inflammation and wheezing.

9 stress during pregnancy with early childhood wheezing.

10 sults and were not associated with childhood wheezing.

11 e heterogeneity of both paediatric and adult wheezing.

12 cts the airways and presents as coughing and wheezing.

13 over time in infants/toddlers with recurrent wheezing.

14 sociations between cortisol trajectories and wheezing.

15 stical modeling based on parentally reported wheezing.

16 to promote airway obstruction and recurrent wheezing.

17 l eczema and rhinoconjunctivitis, but not to wheezing.

18 of breath, chest tightness, coughing, and/or wheezing.

19 abolite of di-isodecyl phthalate (DIDP), and wheezing.

20 shortness of breath, chest pain, cough, and wheezing.

21 characteristics with the risks of preschool wheezing (1-4 years) and school-age asthma (5-10 years).

22 urance (85% vs. 43%) and a history of asthma/wheezing (28% vs. 18%) but not more severe illness.

23 ore likely to experience frequent attacks of wheezing, 3.44-fold (1.19-9.94) more likely to experienc

24 hildren (4-18 yr old) were enrolled; 28 with wheezing, 32 with acute rhinitis, and 14 without respira

25 rs; 77% phlegm; 70% shortness of breath; 47% wheezing; 46% chest pain; 42% abnormal peak flow), 334 (

26 We assigned classes as follows: no wheezing (53.3%), transient early wheeze (13.7%), late-o

27 ociated with a 54.8% increase in the odds of wheezing (adjusted odds ratio, 1.55; 95% CI, 0.91-2.63).

28 ion observed between BMI>/=35 and late-onset wheezing (adjusted OR, 1.87; 95% CI, 1.28-2.73).

29 ificantly higher risk of physician-confirmed wheezing after antibiotic prescription (hazard ratio [HR

30 etween cord serum 25(OH)D levels and asthma, wheezing, allergic rhinitis, and atopic dermatitis in th

31 Disease phenotypes included asthma, atopy, wheezing, altered lung function, and bronchial reactivit

32 , NVAS was associated with increased risk of wheezing among females (RR 1.80 [1.03-3.17], but not amo

33 and early life are associated with recurrent wheezing and aeroallergen sensitivity and altered cytoki

34 al and suggest new preventive strategies for wheezing and allergic diseases.

35 t of physician-diagnosed asthma or recurrent wheezing and allergic sensitization to food or environme

36 asurements, and assessed physician-diagnosed wheezing and asthma by questionnaires.

37 es predictors of remission or persistence of wheezing and asthma from early childhood through adultho

38 ay play a role in the inception of recurrent wheezing and asthma in childhood.

39 inked to subsequent development of recurrent wheezing and asthma in childhood.

40 n 3 expression and correlated inversely with wheezing and asthma in nonatopic subjects.

41 al exhaled nitric oxide (Feno), and risks of wheezing and asthma in school-aged children.

42 Wheezing and asthma information up to 8/10 years was col

43 thma risk factors and the natural history of wheezing and asthma through childhood and beyond.

44 infancy is a major risk factor for recurrent wheezing and asthma.

45 cidence of postviral sequelae like childhood wheezing and asthma.

46 in order to avoid chronic sequelae-recurrent wheezing and asthma.

47 te and homocysteine levels were observed for wheezing and asthma.

48 associated with the risk of transient early wheezing and atopic dermatitis by the age of 5 years, bu

49 with emergency department visits for asthma/wheezing and congestive heart failure than PM2.5.

50 episodic shortness of breath with expiratory wheezing and cough, is a serious health concern affectin

51 individuals not only suffer from consistent wheezing and coughing but are also believed to be more p

52 man was admitted to our hospital because of wheezing and dyspnea.

53 m 25(OH)D levels and risk of transient early wheezing and early- and late-onset atopic dermatitis, as

54 respectively); rs3918396 was associated with wheezing and eczema comorbidity (p = 3.41*10(-4)).

55 Due to its association with wheezing and eczema comorbidity, ADAM33 may also be invo

56 dictive values of SB-FENO for persistence of wheezing and exacerbations were superior to tidal-FENO ,

57 stion, he experienced coughing, dyspnea, and wheezing and had to be treated by anti-histamine and ste

58 with atopy, atopic dermatitis (AD), asthma, wheezing and impaired lung function in a prospective stu

59 sely associated with consecutive symptoms of wheezing and number of mild respiratory tract infections

60 n of maternal stress/depression to recurrent wheezing and peripheral blood mononuclear cell cytokine

61 Wheezing and physician-diagnosed eczema were annually ob

62 ly associated with higher risks of preschool wheezing and school-age asthma (P < .05).

63 n, including the increased risk of childhood wheezing and subsequent asthma.

64 qPCRs, was inversely associated with risk of wheezing and was significantly (inverted-U shape) associ

65 were more likely to have chest retractions, wheezing, and a history of underlying asthma/reactive ai

66 infants developing bronchiolitis, recurrent wheezing, and asthma following infection.

67 also observed between 2,5-dichlorophenol and wheezing, and between monocarboxynonyl phthalate, a meta

68 respiratory symptoms (difficulty breathing, wheezing, and cough) lasting >/= 2 days or requiring pre

69 , with self-reported lifetime asthma, recent wheezing, and current asthma using data from participant

70 anterior rhinorrhea, loss of sense of smell, wheezing, and dyspnea) and on quality-of-life scores, ir

71 yfish product (100g), he experienced nausea, wheezing, and erythema and had visited our hospital.

72 rs), symptoms of cough, nasal congestion and wheezing, and longer interval from illness onset to clin

73 uld at home were at increased risk for early wheezing aOR: 1.34 (1.03-1.75), whereas the major allele

74 use, individuals with SCD and with recurrent wheezing are at increased risk for subsequent morbidity

75 Wheezing, asthma and AD were assessed from questionnaire

76 -term lung health, with potential effects on wheezing, asthma, and chronic lung disease.

77 cally significant change in the incidence of wheezing/asthma after introduction of smoke-free legisla

78 extracted monthly counts of new diagnoses of wheezing/asthma and RTIs among children aged 0-12 years

79 366,642 new wheezing/asthma diagnoses and 4,324,789 RTIs were observ

80 le reductions in the incidence of paediatric wheezing/asthma or RTIs following introduction of smoke-

81 hagia at 1 month, nausea at 3 and 12 months, wheezing at 6 months; and inability to belch at 12 month

82 ation >/= 30 p.p.b. predicted persistence of wheezing at age 3 years with a sensitivity of 77%, a spe

83 s, aeroallergen sensitization, and recurrent wheezing at age 3 years.

84 asthmatic symptoms, as well as virus-induced wheezing, at any time before biomarker assessment at age

85 the effect of BMI and GWG on risk of asthma, wheezing, atopic eczema (AE), and hay fever in children

86 Asthma and wheezing begin early in life, and prenatal vitamin D def

87 siveness that leads to recurrent episodes of wheezing, breathlessness, chest tightness, and coughing.

88 were significantly associated with recurrent wheezing but not increased atopy or reduced antiviral re

89 ssociated with increased risk for persistent wheezing, but only long-term exposure to high levels of

90 ficantly less likely to experience recurrent wheezing by 3 years compared with those children without

91 s (P = .03), as well as male sex (P = .025), wheezing causing shortness of breath (P = .002), and ACS

92 Fifty-seven percent of wheezing children and 56% with acute rhinitis had nasal

93 rhinovirus (RV) is most commonly detected in wheezing children thereafter.

94 ive polymerase chain reaction in washes from wheezing children was 2.8-fold lower, but did not differ

95 With regard to recurrent wheezing, children with detectable TSLP at one year of a

96 se bronchodilators to manage childhood acute wheezing conditions in the emergency department (ED), an

97 Asthma, wheezing, cough, and atopic dermatitis were assessed usi

98 ariable airflow obstruction and intermittent wheezing, cough, and dyspnea.

99 association of parental migrant status with wheezing disorders among children born in Hong Kong, a d

100 contribute to the development of early life wheezing disorders and asthma, and discuss the external

101 Asthma and wheezing disorders are common chronic health problems in

102 higher risk of hospitalization for childhood wheezing disorders compared to the native population, pa

103 ntal reports helps to more accurately define wheezing disorders during childhood and whether incorpor

104 tematic review investigating risks of asthma/wheezing disorders in children born preterm, including t

105 rth was associated with an increased risk of wheezing disorders in unadjusted (13.7% versus 8.3%; odd

106 delivery by Caesarean section with childhood wheezing disorders may vary with setting and may not be

107 d with hospitalizations for asthma and other wheezing disorders to 12 years [hazard ratio (HR) 1.11,

108 ibutable risk of preterm birth for childhood wheezing disorders was >/=3.1%.

109 risk of hospitalization for asthma and other wheezing disorders, compared to both parents being Hong

110 the aetiology of asthma and other childhood wheezing disorders.

111 non-communicable diseases, including asthma/wheezing disorders.

112 association between preterm birth and asthma/wheezing disorders.

113 uting factor to the increasing prevalence of wheezing disorders.

114 s (g/dL) in pregnancy with hayfever, eczema, wheezing, doctor-diagnosed asthma, allergic sensitisatio

115 eczema, allergen sensitization, or recurrent wheezing during the first three years of life.

116 erized by episodic or persistent symptoms of wheezing, dyspnea, and cough.

117 ore than one symptom of COPD (cough, sputum, wheezing, dyspnoea, or chest tightness), with at least o

118 nal period and (ii) documentation of asthma, wheezing, eczema, or other atopic disease in the offspri

119 steroid-naive children with the first severe wheezing episode (90% hospitalized/10% emergency departm

120 piratory syncytial virus/rhinovirus-negative wheezing episode (adjusted OR, 8.0; P = .001), first whe

121 eline) within the first 48 hours of an acute wheezing episode (day 0) and 10 and 30 days later.

122 ment resulted in prolonged time to the third wheezing episode (P = .048) and in fewer days with respi

123 irate by using PCR, 79 children with a first wheezing episode at age 3 to 23 months were randomized t

124 episode (adjusted OR, 8.0; P = .001), first wheezing episode at age less than 12 months (adjusted OR

125 to identify risk factors at the first severe wheezing episode for current asthma 7 years later and se

126 cute, moderate-to-severe, rhinovirus-induced wheezing episode in young children.

127 omes were long term: new physician-confirmed wheezing episode within 2 months, number of physician-co

128 IL-8 levels, prolonged the time to the third wheezing episode, and reduced overall respiratory morbid

129 cute, moderate-to-severe, rhinovirus-induced wheezing episode.

130 of age, presenting to hospital with an acute wheezing episode.

131 Forty-three children experienced a wheezing episode.

132 ical markers at the time of the first severe wheezing episode.

133 rence in unscheduled medical attendances for wheezing episodes between children in the montelukast an

134 -treated participants experienced at least 3 wheezing episodes compared with 50% of participants in t

135 The occurrence of wheezing episodes was assessed monthly over the ensuing

136 Maternal stress, depression, and childhood wheezing episodes were assessed by quarterly questionnai

137 placebo, unscheduled medical attendances for wheezing episodes were reduced in children given montelu

138 thin 2 months, number of physician-confirmed wheezing episodes within 12 months, and initiation of re

139 lar positive associations with the number of wheezing episodes, wheezing patterns, and physician-diag

140 umber of unscheduled medical attendances for wheezing episodes.

141 ma Predictive Index (API) for persistence of wheezing, exacerbations and lung function change through

142 aled nitric oxide (FENO ) for persistence of wheezing, exacerbations, or lung function change over ti

143 he patient presented with swollen eyelid and wheezing following combined intake of orange and aspirin

144 A quarter of infants had recurrent wheezing (>/=3 episodes) and more frequent in the presen

145 Early life aeroallergen sensitization and RV wheezing had additive effects on asthma risk at adolesce

146 ffspring measured at 7 years of age (asthma, wheezing, hay fever, eczema, atopy, and total IgE).

147 models including age, asthma diagnosis, and wheezing histories.

148 s into asthma ever, recent asthma, or recent wheezing illness (recent asthma or recent wheeze).

149 n were further validated using virus-induced wheezing illness and asthma phenotypes in an independent

150 Children with an HRV-related wheezing illness had an increased risk of readmission wi

151 Children with histories of wheezing illness with rhinovirus infection before the th

152 0.76, 0.87) for 94 studies analyzing recent wheezing illness.

153 Early life rhinovirus (RV) wheezing illnesses and aeroallergen sensitization increa

154 ses that affect the development of recurrent wheezing illnesses and allergic sensitization.

155 us C (RV-C) species are more likely to cause wheezing illnesses and asthma exacerbations compared wit

156 n of CDHR3 protein, and an increased risk of wheezing illnesses and hospitalizations for childhood as

157 o new strategies for the prevention of viral wheezing illnesses and perhaps reduce the subsequent ris

158 (HRV) and respiratory syncytial virus (RSV) wheezing illnesses and tested for interactions between 1

159 Sex influences the risk of wheezing illnesses and the prevalence of asthma througho

160 (eg, airway microbiome) promote more severe wheezing illnesses and the risk for progression to asthm

161 ated with asthma in children who had had HRV wheezing illnesses and with expression of two genes at t

162 he 17q21 locus and virus-induced respiratory wheezing illnesses are associated with the development o

163 Early life aeroallergen sensitization and wheezing illnesses associated with virus and bacterial i

164 Wheezing illnesses cause major morbidity in infants and

165 RSV-induced wheezing illnesses during infancy influence respiratory

166 The etiology and timing of viral wheezing illnesses during the first 3 years of life were

167 isease in childhood and is often preceded by wheezing illnesses during the preschool years.

168 Viral respiratory infections can cause acute wheezing illnesses in children and exacerbations of asth

169 Viral infections are closely linked to wheezing illnesses in children of all ages.

170 nsistent with HRV-C causing recurrent severe wheezing illnesses in children who are more susceptible

171 mage and yet is a significant contributor to wheezing illnesses in young children and in the context

172 HRV-C-related wheezing illnesses were associated with an increased ris

173 ions between 17q21 genotypes and HRV and RSV wheezing illnesses with respect to the risk of asthma.

174 was most strongly associated with outpatient wheezing illnesses with RV and aeroallergen sensitizatio

175 nd early life stress may influence childhood wheezing illnesses, potentially through effects on immun

176 The associations between viral wheezing illnesses, presence and pattern of aeroallergen

177 were restricted to children who had had HRV wheezing illnesses, resulting in a significant interacti

178 330 mutation could be a risk factor for RV-C wheezing illnesses.

179 ention in preschool children with outpatient wheezing illnesses.

180 MSP increases risk of asthma and wheezing in adolescence; mechanisms go beyond reducing l

181 rnal asthma was a stronger predictor of ever wheezing in boys (odds ratio [OR], 2.15; 95% CI, 1.74-2.

182 was no effect on rates of medically attended wheezing in children aged 1-3 years (190 [14.9%] of part

183 isrupting chemical, has been associated with wheezing in children, but few studies have examined its

184 al or bacterial pathogens is associated with wheezing in children; however, the influence of both bac

185 breastfeeding on respiratory infections and wheezing in early infancy.

186 Wheezing in infancy has been associated with subsequent

187 causes respiratory diseases, including acute wheezing in infants, of which life-threatening cases hav

188 ironmental factors associated with recurrent wheezing in inner-city environments.

189 lementation in pregnancy on asthma/recurrent wheezing in offspring (P for interaction = .77).

190 associated with increased risk of asthma and wheezing in offspring but not with AE and hay fever, sug

191 Maternal BMI was also associated with wheezing in offspring, with the strongest association ob

192 Wheezing in SCD should be treated aggressively both in t

193 ory tract infection and subsequent long-term wheezing in term infants.

194 ciated with an increased risk for persistent wheezing in the child until the age of 5.

195 lected birth cohort), presence of eczema and wheezing in the child's first year and physician-diagnos

196 Wheezing in the children was annually examined by using

197 tions and has been associated with decreased wheezing in the first years of life.

198 Wheezing in the last 12 months was reported by 10.7% of

199 intake during pregnancy may protect against wheezing in the offspring, but the preventive effect of

200 rgic rhinitis increased, whereas the rate of wheezing in the past 12 months decreased from 2006 to 20

201 The incidence of asthma and recurrent wheezing in their children at age 3 years was lower by 6

202 egnancy is associated with increased odds of wheezing in their children during the first 6 years of l

203 tress during pregnancy had increased odds of wheezing in their children from 1 to 4 years of life (ov

204 eczema, allergic sensitization, or recurrent wheezing in young children.

205 associated with increased risk of atopy and wheezing, in this study only among females.

206 risk factors facilitating severe asthma and wheezing, including airborne viruses, smoke, indoor damp

207 B. breve was more abundant in the wheezing infants (P = 0.02).

208 ontroversies in the diagnostic evaluation of wheezing infants.

209 ild cohort LINA for whom maternal stress and wheezing information was available (n = 443).

210 Infantile wheezing is a common problem, but there are no guideline

211 Rhinovirus-induced wheezing is an important risk factor for recurrent wheez

212 Wheezing is common in childhood.

213 Wheezing is common in SCD and in some individuals repres

214 Childhood wheezing is common particularly in children under the ag

215 Early childhood wheezing is common, but predicting who will remit or hav

216 Although infantile wheezing is common, there is a paucity of evidence to gu

217 ciation of breastfeeding with reduced asthma/wheezing is supported by the combined evidence of existi

218 = 1.2; 95% CI: 1.0, 1.5, respectively), and wheezing lasting >/= 2 days, resulting in a doctor visit

219 tically deregulated and could be linked with wheezing later in children's life.

220 more likely to experience severe attacks of wheezing limiting speech, 10.14-fold (1.27-81.21) more l

221 Variable patterns of childhood wheezing might indicate differences in the cause and pro

222 noea (n=513 [84%]), cough (n=500 [81%]), and wheezing (n=427 [70%]); 294 (48%) patients had fever.

223 f boiled jellyfish, he experienced erythema, wheezing, nausea, and abdominal pain.

224 CI], 0.06 [0.01-0.12]) and increased risk of wheezing (odds ratio [95% CI], 1.07 [1.00-1.14], per Z s

225 gative ARI, were more likely to present with wheezing (odds ratio [OR], 1.7; 95% CI, 1.23-2.35; P < .

226 sociations seen for PM2.5 with prevalence of wheezing {odds ratio (OR)=1.16 per 5mug/m(3) [95% confid

227 toms, children infected with RV enrolled for wheezing or acute rhinitis had similar viral loads in th

228 of the prevalence and severity of recurrent wheezing or asthma in children aged up to 6 years.

229 associated with offspring hayfever, eczema, wheezing or asthma.

230 cy, cardiac disease, or previous episodes of wheezing or inhaled bronchodilator use.

231 chronic bronchitis or COPD, and a history of wheezing or use of respiratory inhalers in the last 12 m

232 e inverse relationships with both asthma and wheezing (OR = 0.88; 95% CI: 0.74, 1.04, and OR = 0.83;

233 ure were significantly more likely to report wheezing (OR = 1.92; 95% CI: 1.32, 2.79); headaches (OR

234 zziness (OR = 4.21; 95% CI: 2.69, 6.58), and wheezing (OR = 4.20; 95% CI: 2.86, 6.17).

235 1.1-1.9), and 26% versus 17% reported recent wheezing (OR, 1.7; 95% CI, 1.3-2.4).

236 a (OR, 1.06 [95% CI, 0.99-1.14]; n = 14438), wheezing, or allergy.

237 with asthma symptoms; and days of coughing, wheezing, or chest tightness) across 6, 9, and 12 months

238 ever (OR, 1.4; 95% CI, 1.05-1.8) and recent wheezing over the last 12 months than full-term control

239 was significantly associated with recurrent wheezing (P </= 0.01).

240 aire symptom scores (p=0.037), and increased wheezing (p=0.018), but no evidence of an association wi

241 of children aged 1 to 5 years with episodic wheezing participating in clinical trials.

242 ations with the number of wheezing episodes, wheezing patterns, and physician-diagnosed asthma at 6 y

243 Childhood wheezing phenotype was related to 1989, 1995, 2001, and

244 ssociated with both transient and persistent wheezing phenotypes (adjusted odds ratio [aOR] = 1.64; 9

245 g was assessed at each age, and longitudinal wheezing phenotypes (early-transient, late-onset, persis

246 21 locus are specific to asthma and specific wheezing phenotypes and are not explained by association

247 Associations with wheezing phenotypes and asthma at 8 years of age were an

248 nship between TRAP exposure and longitudinal wheezing phenotypes and asthma at age 7 years.

249 l milk fatty acid composition with childhood wheezing phenotypes and asthma up to age 13 years using

250 The relationship between TRAP exposure and wheezing phenotypes and asthma was examined.

251 C), we analyzed associations of longitudinal wheezing phenotypes and asthma with single nucleotide po

252 Adjusted risk ratios with parent-reported wheezing phenotypes and doctor-diagnosed asthma were com

253 new insights into the physiology underlying wheezing phenotypes based on age of onset and duration o

254 We characterized childhood wheezing phenotypes from infancy to adolescence and thei

255 COPSAC study or with asthma or virus-induced wheezing phenotypes in the COAST study.

256 Six wheezing phenotypes were identified: never/infrequent, p

257 ween 34 and 36 Mb on chromosome 17 and early wheezing phenotypes, doctor-diagnosed asthma and atopy a

258 isms are associated with asthma and specific wheezing phenotypes; that is, most SNPs are associated w

259 sociated with an increased risk of preschool wheezing (pooled odds ratio [pOR], 1.34; 95% CI, 1.25-1.

260 for gestational age at birth with preschool wheezing (pOR, 1.10; 95% CI, 1.00-1.21) and school-age a

261 A reliable asthma diagnosis is difficult in wheezing preschool children.

262 ed to derive phenotypes based on patterns of wheezing recorded at up to 14 time points from birth to

263 he development of allergic sensitization and wheezing respiratory tract illnesses caused by viruses a

264 hma (RR 0.99 [95% CI: 0.77-1.27], P = 0.95), wheezing (RR 1.02 [95% CI: 0.89-1.17], P = 0.76) or rhin

265 isodic nonproductive cough with intermittent wheezing since birth.

266 Wheezing status, Ascaris lumbricoides and Trichuris tric

267 analysis was undertaken among 309 currently wheezing subjects at 18 years in the Isle of Wight birth

268 out cold (OR 0.45, 95% CI 0.19-1.09) and any wheezing symptom (OR 0.52, 95% CI 0.27-1.02).

269 ibodies (year 1, 4.5, 6), atopic dermatitis, wheezing symptoms, and asthma (year 1, 1.5, 2, 3, 4, 5,

270 ripheral eosinophil counts (P = .03) but not wheezing symptoms, baseline spirometric indices, or resp

271 ere not associated with an asthma diagnosis, wheezing symptoms, lung function measures, or prior sick

272 tion of infants with recurrent or persistent wheezing that is not relieved or prevented by standard t

273 Early childhood-onset wheezing that persists into adolescence represents the c

274 nfant biomarkers to the history of recurrent wheezing, the Asthma Predictive Index and its subsequent

275 Among children with wheezing, those in the complete mite sensitization traje

276 s improved newborn PFT results and decreased wheezing through 1 year in the offspring.

277 t of physician-diagnosed asthma or recurrent wheezing through 3 years of age and (2) third trimester

278 zed to vitamin C had significantly decreased wheezing through age 1 year (15/70 [21%] vs 31/77 [40%];

279 Secondary outcomes included incidence of wheezing through age 1 year and PFT results at age 1 yea

280 ldren were assigned to wheeze phenotypes (no wheezing, transient, late-onset, and persistent) and ato

281 Asthma and recurrent wheezing until age 3 years were recorded.

282 ants were followed up for medically attended wheezing until they reached age 3 years.

283 Parental-reported wheezing was assessed at each age, and longitudinal whee

284 Follow-up assessment including wheezing was assessed through age 1 year, and PFTs were

285 TSLP to allergic sensitization and recurrent wheezing was conducted in the birth cohort from the Urba

286 A physician's diagnosis of asthma/recurrent wheezing was noted in 67%, and 51% were receiving regula

287 ohort of infants and toddlers with recurrent wheezing, we compared predictive values of single-breath

288 Hispanic ethnicity, and diagnosed asthma or wheezing were also predictors of obesity; common infecti

289 d serum samples from 121 children with acute wheezing were analyzed by means of serology.

290 The odds for wheezing were increased if children who tested positive

291 Only higher scores on wheezing were significantly associated with higher value

292 it inflammation have efficacy for RV-induced wheezing, whereas the anti-RSV mAb palivizumab decreases

293 ren have high rates of allergic diseases and wheezing, which are diseases associated with type 2-bias

294 infection is a common trigger for childhood wheezing, which is a risk factor for subsequent asthma d

295 , aged 1 to 16 years, admitted for asthma or wheezing who identified as African American (n = 441) or

296 roup of children with persistent troublesome wheezing, who have markedly different outcomes compared

297 1), eczema (adjusted OR, 4.8; P = .014), and wheezing with rhinovirus (adjusted OR, 5.0; P = .035).

298 When adjusted for all viral etiologies, wheezing with RV (odds ratio = 3.3; 95% CI, 1.5-7.1), bu

299 se associations between alpha tocopherol and wheezing without cold (OR 0.45, 95% CI 0.19-1.09) and an

300 ht be useful in disease modification for the wheezing young child at risk of persistent asthma.