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1 e STAT5-regulated milk genes casein beta and whey acidic protein.
2 land and regulated similar to the endogenous whey acidic protein.
4 ferentiation (eg, beta-casein, kappa-casein, whey acidic protein) and induced morphological different
5 decreased expression of alpha-lactoalbumin, whey acidic protein, and beta-casein, possibly because o
7 helial cells expressed abundant beta-casein, whey acidic protein, and WDNM1 mRNA, indicating a relati
8 , phosphorylation of STAT5 and expression of whey acidic protein are significantly reduced in the mam
10 ecreased postlactational apoptosis, elevated whey acidic protein expression and aberrant pErk2 activa
14 owever, targeted expression of maspin by the whey acidic protein gene promoter inhibits the developme
17 expression of transmembrane ErbB-2 from the whey acidic protein-Her-2 cassette and its up-regulation
18 rminal, cysteine-rich (Cys-box) domain and a whey acidic protein-like (WAP) domain, followed by four
19 terminus of anosmin-1 (cysteine-rich region, whey acidic protein-like domain and the first fibronecti
23 ession following pregnancy and involution in whey acidic protein promoter (WAP)-Cre/Rosa26-flox-stop-
25 jected cells and transient activation of the whey acidic protein promoter-Cre gene during pregnancy a
27 mmary cancer were investigated utilizing the whey acidic protein promoter-T antigen transgenic mouse
34 is of AIDS for three distinct members of the whey acidic protein (WAP) family, secretory leukocyte pr
36 e distal region (-830 to -720 bp) of the rat whey acidic protein (WAP) gene contains a composite resp
38 nsgene under control of the mammary-specific whey acidic protein (WAP) gene promoter or the mouse mam
39 e of interleukin-2, under the control of the whey acidic protein (WAP) gene promoter, exhibit aberran
42 ss we developed a transgenic model using the whey acidic protein (WAP) gene to direct expression of r
43 lular domain (Int3) under the control of the whey acidic protein (WAP) or mouse mammary tumor virus-l
44 targeted to mammary epithelial cells by the Whey Acidic Protein (WAP) promoter for overexpression.
45 this idea, the CRD-BP was expressed from the whey acidic protein (WAP) promoter in mammary epithelial
46 n CR-1 transgene under the regulation of the whey acidic protein (WAP) promoter in the FVB/N mouse ba
47 e established, in which the transgene is the Whey acidic protein (WAP) promoter linked to h-Int3sh.
48 targeted to the mammary gland by means of a whey acidic protein (WAP) promoter were characterized as
49 dings, truncated Int3 was expressed from the whey acidic protein (WAP) promoter, the activity of whic
50 ession of Bcl-2 in the mammary gland using a whey acidic protein (WAP) promoter-driven Bcl-2 transgen
54 its major components, alpha/beta-casein and whey acidic protein (WAP), is significantly reduced due
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