ライフサイエンスコーパス: white

1 Race (black or white).

2 of study, 53 [range, 8-83] years; 131 [93%] white).

3 n lead tested at age 11 years (54% male; 93% white).

4 s and odds ratio, 1.06; 95% CI, 0.80-1.41 in whites).

5 60 +/- 10 years; most patients were male and white.

6 ged 65+/-12 years, 61% were men and 65% were white.

7 (SD) age was 71.2 (5.7) years, and 81% were white.

8 (P trend=0.047) greater in 2013 relative to whites.

9 greater (P trend=0.004) in 2013 relative to whites.

10 pressure, respectively, in GERA non-Hispanic whites.

11 lower risk of colorectal cancer-mostly among whites.

12 , 0.57-1.18) was associated with CVD risk in whites.

13 , 95% CI [-0.28, -0.12], p < 0.001) than for whites (-0.09 SD/decade, 95% CI [-0.16, -0.02], p = 0.00

14 ]), and most participants self-identified as white (139 [90.2%]).

15 ) were black/African American, 11 (14%) were white, 16 (21%) were white Hispanic, and 26 (33%) were o

16 lled (race/ethnicity other than non-Hispanic white: 16%; no college degree: 35%; household income <$5

17 65 [20.0%]) youth compared with non-Hispanic white (17119 [23.1%]) youth (P < .001).

18 of 69.1 (11.5) years, and was predominantly white (18440 patients [80.8%] self-identified as white r

19 e, 31.0 [5.7] years), most were non-Hispanic white (1957 [73.7%]), 98 had panic disorder, 252 had GAD

20 was predominately female (23913 [66.1%]) and white (26091 [72.1%]).

21 of MAN was 21.0% (95% CI, 18.4% to 23.9%) in whites, 32.2% (CI, 27.3% to 37.4%) in Chinese Americans,

22 Among normal-weight participants (n = 846 whites, 323 Chinese Americans, 334 African Americans, 25

23 d age were 21.4% in mixed ethnicities, 10.2% Whites, 4.5% Asians, 3.1% Hispanics, and 2% Blacks; 7.7%

24 666 patients were assessed: 2,819 (77%) were white, 553 (15%) Hispanic, 256 (7%) black, and 38 (1%) o

25 (95% confidence interval [CI] 89.2-90.4) in white, 89.8% (95% CI 89.0-90.6) in black, 86.0% (95% CI

26 as 53 years (range, 28-77 years); 77.0% were white, 9.0% were black, and 10.7% were Asian; and the me

27 female; mean [SD] age, 69.4 [6.1] years; 611 white [94.9%]) seen by 31 primary eye care ophthalmologi

28 associated with lower metabolic syndrome for whites across all levels of childhood disadvantage.

29 f Egr1 identifies the molecular signature of white adipocyte browning downstream of Egr1 deletion and

30 chanism controlling the age-related beige-to-white adipocyte transition and identify Lsd1 as a regula

31 ing precursor fibroblasts differentiate into white adipocytes in the embryo.

32 ge-related transition of beige adipocytes to white adipocytes in vivo, whereas loss of Lsd1 precipita

33 geneous cell population consisting of mature white adipocytes, multipotent mesenchymal stem cells, co

34 ered energy storage capacity and browning of white adipocytes.

35 dams during pregnancy and lactation promoted white adipose browning and thermogenesis in offspring at

36 gene and UCP1 protein expression in inguinal white adipose tissue (iWAT), a common site for emergent

37 ereas preferential expansion of subcutaneous white adipose tissue (WAT) appears protective.

38 stromal vascular fraction from periprostatic white adipose tissue from obese HiMyc mice at 6 months o

39 scriptomic analysis of subcutaneous inguinal white adipose tissue in the absence of Egr1 identifies t

40 ellular energy metabolism, but their role in white adipose tissue physiology remains incompletely und

41 Induction of recall responses within white adipose tissue was associated with the collapse of

42 ereas sensitivity of the skeletal muscle and white adipose tissue was lower in HFHS than control dams

43 tes beige adipocyte development in offspring white adipose tissue.

44 ivate brown adipose tissue and by 'browning' white adipose tissue.

45 f the Ucp1 promoter in subcutaneous inguinal white adipose tissue.

46 nduced obesity and had significantly reduced white adipose tissue.

47 irect calorimetry was performed and visceral white adipose tissues (VWAT) were assessed for inflammat

48 cs had similar mortality rates compared with whites (adjusted hazard ratio, 0.92; 95% CI, 0.76-1.11 a

49 (n = 36) and in an independent population of white adult atopic asthmatic patients (n = 12) and contr

50 (ATWI) on electrocardiography (ECG) in young white adults raises the possibility of cardiomyopathy, s

51 or genomics between the African-American and White-American populations is also summarized.

52 cluded 38 nurses, 100% (n = 38) of whom were white and 92% (n = 35) of whom were female, and 100 chil

53 METHODS AND Participants were 27 078 white and black men and women enrolled during 2002 to 20

54 hifts in core demographic rates suggest that white and black older adults in the United States may ha

55 ce display abnormal fat accumulation in both white and brown adipose tissues, glucose intolerance and

56 White and brown champignons were found to be the richest

57 all expressed members of the KDM5 family in white and brown preadipocytes leads to deregulated gene

58 Using conditional knockout mice and derived white and brown preadipocytes, we show that endogenous K

59 The whole brain was segmented according to white and gray matter by using a dual-clustering algorit

60 magnetic resonance imaging (MRI) measures of white and grey matter in a large population-derived coho

61 ral cortex, in order to discriminate between white and grey matter location of contacts.

62 s noted in men and women, young and old, and white and non-white participants, and was not dependent

63 e the incidence of cutaneous disease between white and nonwhite OTRs.

64 he prognostic significance of blasts, and of white and red blood cells, in CSF samples at diagnosis o

65 n carried out using a total of 70 samples of white and red wines and two standard methods, the Ripper

66 f lead, cadmium, zinc, manganese and iron in white and wild rice samples.

67 onses (PEAR) (50 whites) and from PEAR-2 (50 whites and 50 blacks).

68 This pattern contrasts starkly with whites and minorities from more privileged backgrounds,

69 ion of Antihypertensive Responses (PEAR) (50 whites) and from PEAR-2 (50 whites and 50 blacks).

70 pants was 69 years, 76% of participants were white, and 90% were non-Hispanic.

71 is patients who were male, aged >/=40 years, white, and infected with human immunodeficiency virus.

72 enty patients were Hispanic; 13, African; 8, white; and 2, Native American.

73 hylation changes in nasal epithelia of adult white asthmatic patients.

74 Wheat bran, oat bran and white bean had a lower calcium:phosphate ratio than barl

75 l dataset of the network morphology within a white beetle wing scale.

76 -acting antivirals produce high SVR rates in white, black, Hispanic, and Asian/Pacific Islander/Ameri

77 Compared with whites, blacks (OR, 0.89; 95% CI, 0.86-0.92) and Latinos

78 es were the oncogenetic classifier, MRD, and white blood cell (WBC) count.

79 s: age (hazard ratio [HR], 3.299; P < .001), white blood cell count (HR, 1.910; P = .017), platelet c

80 eal inflammation, fluid, appendicoliths, and white blood cell count (WBC) were significantly correlat

81 4 vs 0-2 and 5.20 (95% CI, 2.70-10.02) for a white blood cell count of >/=20 000/muL vs <20 000/muL.

82 th poor outcome (CSF culture positivity, CSF white blood cell count, hemoglobin, Glasgow Coma Scale,

83 d aldosterone), hepatobiliary enzyme levels, white blood cell counts, and iron homeostasis.

84 nstrated improved enrichment and recovery of white blood cells from human blood.

85 ion of disseminated cells with platelets and white blood cells.

86 The AXs were added to 100g white bread formulae at dosages of 0.8-1.2% with flour r

87 At this dosage, AX addition in white bread holds both increased health and economic ben

88 Loaves of white bread were baked and stored at refrigeration, froz

89 o March 31, 2016, among 1657 participants of white British ancestry from the TwinsUK study cohort wit

90 Compared with white British men, the proportions of black Caribbean an

91 ean and 21.9% for black African vs 47.4% for white British) and the number of partners in the past 5

92 and 2 [1-5] for black African vs 1 [1-2] for white British), and although black Caribbean and black A

93 ean and 38.9% for black African vs 14.8% for white British), these differences were not significant a

94 2-aminothiophene was also observed in cooked white cabbage samples.

95 to complete the KT evaluation compared with white Canadians, and this association remained statistic

96 chanism for their survival and whether these white cells would use any metabolites as signal molecule

97 ion P = 0.025) and was stronger in Black vs. White children (OR = 0.30, 95% CI: 0.15, 0.61 vs. OR = 0

98 ildren had lower mortality than non-Hispanic white children.

99 inulin as a surfactant or stability agent on white compound chocolate sweetened with sucralose and St

100 ce dialysis survive longer than non-Hispanic white counterparts.

101 ctors remains prevailing in explaining black-white differences in birth outcomes, the individual cont

102 The white to white distance showed a statistically significant differ

103 cs of degenerate matter and lends support to white dwarf evolutionary theory.

104 -rotation radius) and then accretes onto the white dwarf, producing bursts powered by the release of

105 a main-sequence companion and ejecta from a white-dwarf explosion.

106 White dwarfs are often found in binary systems with orbi

107 ch a model may account for the explosions of white dwarfs in a mass range wider than previously suppo

108 ary electron (SE) microscopy, a SE spectrum (white electrons) associated with the reflectivity differ

109 addition, several of these DNA-CAs exhibited white emission in aqueous solution.

110 y included cross-sectional data from 119,859 white European adults, aged 37-73 y, participating in th

111 h medium-chain triglycerides (MCTs) plus egg white (EW) and was characterized by increased numbers of

112 with a skin prick test (SPT) response to egg white (EW) of less than 2 mm were randomized at age 4 mo

113 es brown fat-specific genes while repressing white fat and muscle-specific genes in adipocytes.

114 erature, and diminished browning of inguinal white fat.

115 g black female patients (44.1%), followed by white female patients (38.4%), black male patients (36.4

116 Furthermore, an intense white flash induced a small amplitude prolonged dark cur

117 new compounds being described in yellow and white-fleshed red pitayas, respectively.

118 The yellow seed and white flower phenotypes of the classical d mutation was

119 The mechanism underlying the evolution of white flowers in I. loxense differs from that uncovered

120 .6 % in 2015) students overall, non-Hispanic white (from 21.1 % in 2009 to 9.4% in 2015) and Hispanic

121 tion than wild-type plants in both brown and white grains.

122 0.932; 95% CI: 0.892-0.974) reduction in the white group (coefficients significantly different, p = 0

123 of 14 patients, or in black patches between white hairs in 1.

124 American, 11 (14%) were white, 16 (21%) were white Hispanic, and 26 (33%) were other/mixed race/ethni

125 in four race/ethnicity groups: non-Hispanic whites, Hispanic/Latinos, East Asians and African Americ

126 seven whisky brands: Red Label, Black Label, White Horse, Chivas Regal (12years), Ballantine's Finest

127 f primary care providers and their black and white hypertensive patients.

128 stem to encode the pixel values of black and white images and a short movie into the genomes of a pop

129 5 (9.3) years of age, 44.1% women, and 76.3% white In all 4 cohorts, there was a 3- to 5-letter impro

130 dence and CHD case-fatality among blacks and whites in the Atherosclerosis Risk in Communities study

131 Compared with blacks in the same quintile, whites in the highest quintile for TMAO (>/=135 muM) had

132 It is known that flying insects create "white" in extremely thin layers.

133 ating heroin use increased across time among white individuals (35.83% [SE, 6.03%] in 2001-2002 to 52

134 ruction was demonstrated in all non-Hispanic white individuals in the Pittsburgh Specialized Center o

135 ely to develop type 2 diabetes compared with white individuals, and these disparities are particularl

136 However, among white individuals, increased lifetime stress and posttra

137 .005) in Gabonese individuals compared with white individuals.

138 etes incidence between middle-aged black and white individuals.

139 ss-studied for the potential applications in white light emitting diodes, solar cells, optical codes,

140 on SOB3 mutants grown in moderate-intensity white light was reduced when polar auxin transport was i

141 ther treatments, individuals roosting in the white light were much more active at night.

142 Hypocotyls of SOB3 mutant seedlings grown in white light with a higher fluence rate also exhibited al

143 ed excited states that give efficient bluish white-light emissions with photoluminescence quantum eff

144 White lupin (Lupinus albus L.) is a valuable source of s

145 omodimeric receptors in complex with hen-egg white lysozyme demonstrate how nanomolar affinity bindin

146 ts (38.4%), black male patients (36.4%), and white male patients (30.2%, P<0.0001).

147 ficantly higher proportions of drinkers were white, male, and with higher levels of education compare

148 egmentation accuracy of a normal macula of a white man in his 60s as an emblematic example.

149 al cortex (-7.3%; P = .02), inferior frontal white matter (-11.4%; P < .001), and caudate (-10.6%; P

150 0.08, P = .03) temporal regions, subcortical white matter (beta = -0.13, P = .02), and occipital regi

151 d right: +10.8%; P = .01), superior temporal white matter (left: +14.6%; P = .003 and right: +9.5%; P

152 areas, that is, in areas of normal-appearing white matter (NAWM) and gray matter (GM).

153 ctivation in lesions and in normal-appearing white matter (NAWM) of multiple sclerosis (MS) patients

154 gions, respectively) and in normal-appearing white matter (P < .001 for both contrast-enhancing and n

155 particular advantage of sensitivity to both white matter (WM) and gray matter (GM) demyelination.

156 Patterns of gray matter (GM) and white matter (WM) atrophy at presentation were assessed

157 We report that the volume of white matter (WM) is disproportionately reduced in AS mi

158 structural brain alterations of the gray and white matter (WM).

159 oward understanding the relationship between white matter anatomy and the structural connectome.

160 is accompanied by preservation of myelinated white matter and motor neurons and an increase in axonal

161 amyloid pathologies significantly influence white matter and that these abnormalities may signify an

162 patients were also mapped at the grey matter-white matter boundary.

163 imaging, the fimbria-fornix, parahippocampal white matter bundle and uncinate fasciculus were reconst

164 on on a stereotypic connectome of converging white matter bundles (forceps minor, uncinate fasciculus

165 g data from 882 youth ages 8-22 to show that white matter connectivity becomes increasingly optimized

166 n the diseased microenvironment can underlie white matter defects.

167 troke can arise from cortical damage or from white matter disconnection.

168 t model and determine its relevance to human white matter disease.

169 oved regenerative medicine for patients with white matter disorders.

170 Normal-appearing white matter distribution volume ratio at baseline was c

171 odel including the baseline normal-appearing white matter distribution volume ratio, T2 lesion volume

172 tients showed decreased FA in multiple major white matter fiber bundles, which connect the fronto-par

173 Cerebral white matter from a patient with TUBB4A Asn414Lys mutati

174 es of network diffusion and nexus regions of white matter from edge density imaging, show converging

175 s for statistical inference and to study the white matter geometrical organization.

176 s12445022) was also associated with cerebral white matter hyperintensities (OR [95% CI] = 1.10 [1.05-

177 rofound in individuals with higher volume of white matter hyperintensities (P value for interaction=0

178 cclusion (17.8%), outcomes tended to vary by white matter hyperintensitiy volume (P = 0.10, Cochran-M

179 Among SVD markers white matter hyperintensity (WMH) score or volume were a

180 White matter hyperintensity volumes were stratified into

181 ophages and the ramified microglia of normal white matter in myelin disease models.

182 ments exist for preterm infants with diffuse white matter injury (DWMI) caused by hypoxia.

183 results indicate that the evolution of grey/white matter injury and blood-brain barrier disruption a

184 Subcortical white matter injury is often accompanied by orofacial mo

185 ubcortical grey matter volume (n = 1157) and white matter integrity (n = 1089) between depressed indi

186 nsity appear fundamental to abnormalities in white matter integrity in early psychosis.

187 campal grey matter volume in MDD and reduced white matter integrity in several brain regions.

188 n improvement, consistent with the increased white matter integrity in the corpus callosum connecting

189 frontal cortex, hippocampus, and cerebellum; white matter integrity in the uncinate fasciculus, ventr

190 Whole brain white matter integrity of HD-monkeys was examined longit

191 significant positive correlation between the white matter integrity of the pons and cerebellar gray m

192 Intact white matter integrity with decreased NAA/Cr levels sugg

193 The unique combination of white matter lesions, hypohomocysteinaemia and increased

194 ratio, T2 lesion volume and normal-appearing white matter magnetization transfer ratio for all of the

195 causal relationship, namely that changes in white matter microstructure impact cognition in part by

196 Hence, systematic evaluation of white matter microstructure in the normative brain is cr

197 We suggest that spatial locations of white matter modules overlap with cytoarchitecturally di

198 Connectome white matter organization measured through modularity an

199 ted to the integrity of the language-related white matter pathways in 40 adults (18 women).

200 lzheimer disease may benefit from the use of white matter reference regions.

201 wever, many studies indicate that failure of white matter repair goes beyond the intrinsic incapacity

202 However, many other potential causes of white matter signal abnormalities can mimic the changes

203 ere estimated and compared across the entire white matter skeleton between groups, and correlated wit

204 modify the effects of concussion history on white matter structure and neural recruitment.

205 Previous reports of altered grey and white matter structure in Major Depressive Disorder (MDD

206 can reveal dimensional relationships linking white matter structure to neurodevelopmental symptoms.

207 ble to demonstrate that epigenetic aging and white matter tract integrity also share common genetic i

208 llows performing anatomical manipulations on white matter tracts for statistical inference and to stu

209 haracteristics of preoperative temporal lobe white matter tracts known to be important in the generat

210 were variably sized, predominantly affected white matter tracts, and involved the middle cerebral ar

211 rative outcome and preoperative pathology of white matter tracts, which constitute crucial components

212 vity in centromedial amygdala-anterior vmPFC white matter was associated with greater anxiety/depress

213 n opposite directions across gray matter and white matter when MDD subjects were compared with contro

214 ain-like tau-containing neurites in gray and white matter with heaviest burden in basal ganglia.

215 r, MRI-based imaging studies have shown that white matter, primarily composed of myelinated axons, ca

216 most commonly employed metric for assessing white matter.

217 ompartment, the late myelinating superficial white matter.

218 orrelated with diffusion measures of frontal white matter.

219 White-matter (WM) microstructure in the optic radiation

220 subjects to show that signals from the human white-matter contain meaningful information.

221 tasets, and suggest a new way to explore the white-matter role in cognition and its disturbances in n

222 eoretic analysis showed that highly selected white-matter tracts were consistent across the group and

223 tworks composed of interacting long-distance white-matter tracts.

224 rected to the functional organization of the white-matter.

225 s ratio, 1.9; 95% CI, 1.2-2.8) compared with white men and the adjusted risk of MI was higher in mino

226 en were more likely to develop diabetes than white men and women (black women: HR, 2.86 [95% CI, 2.19

227 Black and white men and women from the observational Coronary Arte

228 ave similar risk for fatal CHD compared with white men and women, respectively.

229 tis (CAD) is classically described in older, white men, although increasing reports describe younger

230 fore ages 68, 84, and 88 years in black men, white men, and white women, respectively (ICAD prevalenc

231 , 1.38 (1.11-1.72), and 0.90 (0.73-1.12) for white men, black men, and black women compared with whit

232 Most patients were middle-aged white men.

233 fatal CHD risk may be lower for black versus white men.

234 ants borne by non-Hispanic black mothers and white mothers.

235 antially higher prevalence of infection than white MSM.

236 sample of US Black (n = 4,201), non-Hispanic White (n = 20,217) and Hispanic (n = 5,097) groups.

237 Using multielectrode recording methods and white noise stimuli, we recorded neural activity from en

238 in the absence of correlations, for Gaussian white noise, the conventional analysis leads to a strong

239 y age, sex, and race/ethnicity (non-Hispanic white, non-Hispanic black, Mexican American), and covari

240 enes in mesenchymal progenitors cells during white or brown adipogenesis.

241 irect information on tumor infiltration into white or gray brain matter based on N-acetylaspartate (N

242 Hispanic race/ethnicity versus non-Hispanic White (OR 1.10, 95% confidence interval (CI) 1.05-1.16),

243 This is the second of the two White Papers from the fourth UC Davis Cardiovascular Sym

244 and women, young and old, and white and non-white participants, and was not dependent on the method

245 3%] in the stent group; and 114 non-Hispanic white patients [65.9%] in the stoma group and 90 [52.3%]

246 alendar-year rates of survival for black and white patients after adjusting for baseline characterist

247 , and disparity in the referral of black and white patients as a secondary outcome.

248 Current comparative outcomes among black and white patients treated with percutaneous coronary interv

249 Relative to white patients, black patients were more likely to have

250 a 2-fold risk of SJS/TEN when compared with white patients.

251 associated with higher SVR rates compared to white patients.

252 argely by reductions for black compared with white patients.

253 104 black persons and 104 white persons aged 8 years or older who had had type 1 d

254 low-up was 7.1% in black persons and 5.8% in white persons.

255 ints with zinc oxide, one of the most common white pigments of the 20th century.

256 ted with worse outcomes in studies of mostly white populations, but its significance is not well esta

257 receiving no glaucoma testing compared with whites possessing commercial health insurance (OR = 2.98

258 ls have a lower risk of hearing loss than do whites, possibly because of differences in cochlear mela

259 ssive mutation that renders its plumage pure white, provides a unique opportunity to investigate mech

260 fe support, the majority of patients were of white race (56.9%) with black race (16.7%), Hispanic eth

261 ticipate was associated with self-identified white race, higher educational attainment, lower religio

262 White race/ethnicity was associated with lower diversity

263 e (18440 patients [80.8%] self-identified as white race/ethnicity).

264 genetic mapping approaches, we show that the white recessive allele is due to a splice donor site mut

265 The white recessive breed of the common canary (Serinus cana

266 ile their lowest amount was found in the egg-white recipe (3.1+/-0.1ngg(-1) for furan and 0.287+/-0.0

267 and ORAC methods)) of 30 grape cultivars of white, red and pink grape, as 28 interspecific hybrids a

268 like previous lignin biodegradation studies, white rot fungi were used to produce functional biopolym

269 The white sector cells containing undifferentiated chloropla

270 normal a-wave and b-wave activity, yellowish-white spots, hyperfluorescence, and reduced retinal thic

271 f melanocytes and the clinical appearance of white spots.

272 Although the current white spruce genome sequence remains highly fragmented,

273 Black stroke survivors were more likely than white stroke survivors to have a caregiver (62.5% versus

274 of approximately 11 more hours of care than white stroke survivors without substantial differences i

275 evere skin disease in a European and largely white study population.

276 xhibit eosinophilic airway inflammation than white subjects in the ICS+ group (odds ratio, 1.58; 95%

277 mmation differs between African American and white subjects is unclear.

278 the ICS+ group (248 African American and 674 white subjects) and 298 subjects in the ICS- group (49 A

279 the ICS- group (49 African American and 249 white subjects).

280 from chronic wasting disease (CWD)-infected white-tailed deer brain homogenates and found that lipid

281 served patients, half were male and all were white; the mean (SD) age was 69.7 (14.1) years.

282 The white to white distance showed a statistically significa

283 later life, blacks are also more likely than whites to have experienced the death of a child and of a

284 at blacks are significantly more likely than whites to have experienced the death of a mother, a fath

285 c phantoms with gray scales ranging from 0% (white) to 100% (black).

286 SK-like melanomas were the presence of blue-white veil, pseudopods or streaks, and pigment network.

287 Although the majority were non-Hispanic white, well educated, and economically well off, substan

288 justed hazard ratios comparing blacks versus whites were 2.61 (95% confidence interval, 1.57-4.34) an

289 nocarcinoma is more frequent in non-Hispanic whites, whereas esophageal squamous cell carcinoma with

290 much higher rates of ESRD than HIV-positive whites, which could be attributed to the APOL1 renal ris

291 7 (7.8) years of age, 65.9% women, and 97.6% white, while the DRCR.net participants were a mean (SD)

292 enols that occur during skin fermentation in white winemaking.

293 Treated white wines were analyzed for protein and phenolic conte

294 s have been developed for obtaining distinct white wines.

295 After adjustment for confounders, whites with OAG enrolled in Medicaid had 198% higher odd

296 Participants were 67% male, 84% non-Hispanic white, with mean age+/-SD 62+/-11 years; 16% (n=237) wer

297 ated with reduced Alzheimer disease risk for white women only (HR, 0.82, 95% CI, 0.70-0.95 and HR, 0.

298 nes identified in 41611 consecutively tested white women with breast cancer was estimated at 10.2%.

299 4, and 88 years in black men, white men, and white women, respectively (ICAD prevalence remained <50%

300 en, black men, and black women compared with white women.