ライフサイエンスコーパス: white adipose tissue

1 f the Ucp1 promoter in subcutaneous inguinal white adipose tissue.

2 ivate brown adipose tissue and by 'browning' white adipose tissue.

3 id pools in lung, spleen, muscle, liver, and white adipose tissue.

4 increased expression of UCP1 and browning of white adipose tissue.

5 a process often referred to as "browning" of white adipose tissue.

6 ost, but not Ucp1 or Ppargamma expression in white adipose tissue.

7 ase in basal metabolic rate with browning of white adipose tissue.

8 ndocytosis, and lipid uptake in subcutaneous white adipose tissue.

9 s to diet and cold exposure and 'beiging' of white adipose tissue.

10 ow-density lipoprotein into Cxcr7-expressing white adipose tissue.

11 els, and reduces TNFalpha gene expression in white adipose tissue.

12 energy expenditure, and promoted browning of white adipose tissue.

13 utrophil and M1 macrophage infiltration into white adipose tissue.

14 exacerbate insulin resistance in muscle and white adipose tissue.

15 expression of brown fat-selective markers in white adipose tissue.

16 al biogenesis and energy expenditure, in the white adipose tissue.

17 s for the loss of FL-PGC-1alpha in brown and white adipose tissue.

18 ere was reduced browning of the subcutaneous white adipose tissue.

19 lucose uptake in skeletal muscle, heart, and white adipose tissue.

20 n adipose tissue, but not of intraperitoneal white adipose tissue.

21 nases attenuate beta-adrenergic signaling in white adipose tissue.

22 induced obesity, and elicits the browning of white adipose tissue.

23 uits modulate autonomic outflow to liver and white adipose tissue.

24 n adipose tissue-like depots of subcutaneous white adipose tissue.

25 adipocytokine production from the expanding white adipose tissue.

26 m is grossly expanded in the residual mutant white adipose tissue.

27 ET scans at the location of BAT, muscle, and white adipose tissue.

28 es are required for storage of energy in the white adipose tissue.

29 decreased malonyl CoA in skeletal muscle and white adipose tissue.

30 nduced obesity and had significantly reduced white adipose tissue.

31 r expression of Tfe3, Tf3b, and Ppargamma in white adipose tissue.

32 x43 expression was higher in BAT compared to white adipose tissue.

33 turnover in all organs, except the brain and white adipose tissue.

34 tes beige adipocyte development in offspring white adipose tissue.

35 s effects on Wnt signaling and metabolism in white adipose tissue.

36 mic alterations and inflammation in visceral white adipose tissue.

37 n within the same cells in classic brown and white adipose tissues.

38 1-dependent thermogenesis in mouse brown and white adipose tissues.

39 vs. 5.9 +/- 2.2 mL/100 g/min, P = 0.03) and white adipose tissue (7.2 +/- 3.4 vs. 5.7 +/- 2.3 mL/100

40 After infection, white adipose tissue accumulated large numbers of pathog

41 and relative expression of genes regulating white adipose tissue adipogenesis and Irx3.

42 act of CerS5-dependent ceramide synthesis in white adipose tissue after high fat diet feeding.

43 tively related to glucose uptake in visceral white adipose tissue, although glucose uptake in viscera

44 uced uncoupling protein 1 expression in both white adipose tissue and 3T3-L1 differentiated adipocyte

45 sufficient for the induction of lipolysis in white adipose tissue and are an efferent effector of lep

46 Human MSCs were isolated from white adipose tissue and bone marrow aspirates and were

47 ly 90% reduction in gonadal and subcutaneous white adipose tissue and brown adipose tissue, severe gr

48 ity associated with an increased browning of white adipose tissue and hypermetabolism.

49 hetic nervous system-dependent remodeling of white adipose tissue and increasing uncoupling protein 1

50 CYT1A-generated PC in the normal function of white adipose tissue and insulin action.

51 d visceral (0.49 +/- 0.24 SUVmean; P < 0.05) white adipose tissue and liver (0.95 +/- 0.28 SUVmean; P

52 ntified extravascular fibrin deposits within white adipose tissue and liver as distinct features of m

53 ment with RSG-NPs alleviated inflammation in white adipose tissue and liver but, unlike treatment wit

54 is up-regulated via Ppargamma activation in white adipose tissue and plasma following an acute treat

55 ociated immune cell responses predominate in white adipose tissue and protect against weight gain and

56 report that Epac1 null mutants have reduced white adipose tissue and reduced plasma leptin levels bu

57 he male-specific decrease of inflammation in white adipose tissue and skeletal muscle as well as a pa

58 ed lipolysis may be restricted to mesenteric white adipose tissue and that it contributes to hepatic

59 body energy expenditure, hyperplastic brown/white adipose tissues and larger hyperplastic hearts.

60 increasing glucose uptake in cardiac muscle, white adipose tissue, and brown adipose tissue through a

61 rvival, reduced steatohepatitis, browning of white adipose tissue, and improved lipid profile in an A

62 pling protein 1-positive beige adipocytes in white adipose tissue, and increased thermogenesis in mic

63 ner and impaired insulin signaling in liver, white adipose tissue, and skeletal muscle.

64 ut they show expansion of their subcutaneous white adipose tissue, as compared to wild-type (WT) mice

65 4LKO mice have reduced macrophage content in white adipose tissue, as well as decreased tissue and ci

66 mation with reduced macrophage counts within white adipose tissue, as well as near-complete protectio

67 mouse model with almost no visible brown and white adipose tissue at age 3 mo.

68 In obesity, inflammation of white adipose tissue (AT) is associated with diminished

69 ide exchange factor PDZ-RhoGEF (Arhgef11) in white adipose tissue biology.

70 White adipose tissue bridges body organs and plays a fun

71 In contrast to white adipose tissue, brown adipose tissue (BAT) is know

72 reduced mitochondrial oxygen consumption in white adipose tissue, brown adipose tissue, and hepatocy

73 h Hlx as a powerful regulator for systematic white adipose tissue browning and offer molecular insigh

74 ted with enhanced brown adipose function and white adipose tissue browning in HFD+RES compared with H

75 oltage-dependent potassium channel, promotes white adipose tissue browning, and protects mice against

76 , as a direct target gene of Rev-erbalpha in white adipose tissue but not liver.

77 Subcutaneous white adipose tissue can be induced to undergo "browning

78 fspring had lower thermogenesis in brown and white adipose tissues compared with CON offspring, which

79 ce by induction of beneficial changes to the white adipose tissue compartment.

80 that Lsd1 levels decrease in aging inguinal white adipose tissue concomitantly with beige fat cell d

81 Here, we have shown that at steady state, white adipose tissue contained abundant memory lymphocyt

82 It is unclear, however, how beige or white adipose tissue contributes to brown fat thermogeni

83 Within white adipose tissue, CTRP11 is primarily expressed by s

84 but exhibit increased weight gain, elevated white adipose tissue deposition, and diminished hypothal

85 glucose uptake in visceral and subcutaneous white adipose tissue depots was unchanged upon cold accl

86 Expansion of white adipose tissue-derived epididymal BK(L1/L1) preadi

87 hydrogels to support the differentiation of white adipose tissue-derived multipotent stem cells (ADM

88 omal versus adipogenic cell expansion during white adipose tissue development, with PDGFRalpha activi

89 White adipose tissue displays high plasticity.

90 ody fat, plasma hormone levels, and visceral white adipose tissue DNA methylome and transcriptome col

91 (SSc) is accompanied by attrition of dermal white adipose tissue (dWAT) and reduced levels of circul

92 ical of scleroderma is atrophy of the dermal white adipose tissue (DWAT).

93 nes that control a brown fat-like program in white adipose tissue, energy expenditure, and adiposity.

94 difference in the growth of their epididymal white adipose tissue (epiWAT) but they show expansion of

95 ipids extracted from mouse liver, epididymal white adipose tissue (eWAT) and subcutaneous white adipo

96 expressing Tnmd develop increased epididymal white adipose tissue (eWAT) mass, and preadipocytes deri

97 in cultured adipocytes and in the epididymal white adipose tissue (EWAT) of C57BL/6 mice.

98 In epididymal white adipose tissue (eWAT) of PDE3B KO mice on a SvJ129

99 nsulin singling in both liver and epididymal white adipose tissue (eWAT).

100 lysis of bioenergetics revealed thatNrf2(-/-)white adipose tissues exhibit greater oxygen consumption

101 ce with targeted deletion of EPO receptor in white adipose tissue exhibited sex-differential phenotyp

102 Memory T cells in white adipose tissue expressed a distinct metabolic prof

103 and key metabolic genes in adipocytes and in white adipose tissue from diet-induced obese wild-type m

104 White adipose tissue from MRTFA(-/-) mice contains more

105 stromal vascular fraction from periprostatic white adipose tissue from obese HiMyc mice at 6 months o

106 expressed a distinct metabolic profile, and white adipose tissue from previously infected mice was s

107 Consistent with this finding, white adipose tissue from S6K1-deficient mice exhibits n

108 stic of obesity and results from deregulated white adipose tissue function.

109 sitivity in Bscl2(-/-) mice by improving the white adipose tissue gene expression pattern.

110 STRA6 signaling in white adipose tissue has been shown to inhibit insulin r

111 White adipose tissue has emerged as a key determinant of

112 ated UCP1 expression in BAT and subcutaneous white adipose tissue, have increased BAT mass and higher

113 acrophages and dendritic cells (DCs) in lean white adipose tissue homeostasis have received little at

114 sis and oxidation in mouse brown, beige, and white adipose tissues; however, the cellular basis of th

115 brown adipocyte-specific gene expression in white adipose tissue in a murine model of obesity.

116 infiltration, except minimal infiltration in white adipose tissue in animals treated with the highest

117 trophin, a protein secreted by the liver and white adipose tissue in conditions of insulin resistance

118 in hypoxia, a serious comorbidity affecting white adipose tissue in obese individuals, and corrected

119 Hypoxia develops in white adipose tissue in obese mice, resulting in changes

120 A subset of UCP1+ adipocytes develops within white adipose tissue in response to physiological stimul

121 scriptomic analysis of subcutaneous inguinal white adipose tissue in the absence of Egr1 identifies t

122 itions, we were able to distinguish BAT from white adipose tissue in the cervical and supraclavicular

123 MI, MC progenitors originated primarily from white adipose tissue, infiltrated the heart, and differe

124 White adipose tissue inflammation (WATi) has been linked

125 intenance of glucose homeostasis and reduced white adipose tissue inflammation after high fat diet ch

126 These lead to systemic and visceral white adipose tissue inflammation in addition to altered

127 n features of this disorder, such as chronic white adipose tissue inflammation, adipocyte hypertrophy

128 s did not increase proliferation in inguinal white adipose tissue (ingWAT), the percentage of BAs, de

129 ersely, inducible expression of PGC-1beta in white adipose tissue is sufficient to induce beige fat g

130 Hypertrophic remodeling of white adipose tissues is associated with overexposure of

131 Ip6k1 in murine inguinal and retroperitoneal white adipose tissue (IWAT and RWAT) depots.

132 gene and UCP1 protein expression in inguinal white adipose tissue (iWAT), a common site for emergent

133 express UCP1 in beige adipocytes in inguinal white adipose tissue (iWAT), suggesting a role of this p

134 brown adipose tissue (BAT) and subcutaneous white adipose tissue (iWAT).

135 id production in liver and redistribution to white adipose tissue, leading to visceral obesity at 2 m

136 target genes were significantly elevated in white adipose tissues, leading to WAT energy expenditure

137 duction of a type 2 cellular response in the white adipose tissue leads to weight loss and improves g

138 formed postnatally in subcutaneous inguinal white adipose tissue lost thermogenic gene expression an

139 All three mutants showed increased white adipose tissue mass and adipocyte size.

140 overweight, mainly because of an increase in white adipose tissue mass and BAT whitening.

141 t decreased body weight, adipocyte size, and white adipose tissue mass, as assessed by magnetic reson

142 6 weeks after which metabolism, behavior and white adipose tissue morphology were analyzed together w

143 of pro-inflammatory markers was decreased in white adipose tissue of Cpt1b(m-/-) mice.

144 PGF2alphaEA levels are reduced in the white adipose tissue of high fat diet-fed mice where the

145 Furthermore, the white adipose tissue of iePPARgammaKO CR mice showed low

146 fects of FGF21 were markedly enhanced in the white adipose tissue of mice lacking Rev-erbalpha.

147 White adipose tissue of NASH mice was characterized by i

148 MARCH1 expression is increased in white adipose tissue of obese humans, suggesting that MA

149 cumulation of neutrophils and macrophages in white adipose tissue of wt and AhRR Tg mice.

150 expression was lower in inguinal and gonadal white adipose tissues of ESR1 total body knockout female

151 White adipose tissues of undernourished rats transferred

152 d glycerol for their release (in the case of white adipose tissue) or use by cells (in the case of ot

153 hat PDGFRalpha activation inhibits embryonic white adipose tissue organogenesis in a tissue-autonomou

154 hat SGBS adipocytes, which are considered of white adipose tissue origin can shift towards a brown/be

155 ellular energy metabolism, but their role in white adipose tissue physiology remains incompletely und

156 ed in wild-type female mice, suggesting that white adipose tissue plays an integral role in mediating

157 bited changes in liver, skeletal muscle, and white adipose tissue PPARdelta protein levels that may,

158 Beige adipocyte differentiation within white adipose tissue, referred to as browning, is seen a

159 approximately 80% in the liver and by 70% in white adipose tissue relative to control ASO-treated mic

160 showed that genotypic and dietary effects on white adipose tissue remodeling resulted in profound inc

161 Our results suggest that white adipose tissue represents a memory T cell reservoi

162 White adipose tissue samples from P2-null mice contain l

163 vements in glucose homeostasis, subcutaneous white adipose tissue (scWAT) from exercise-trained or se

164 Subcutaneous white adipose tissue (scWAT) is the major fat depot in h

165 ve energy balance reduces human subcutaneous white adipose tissue (scWAT) mass through the formation

166 ages recruited to cold-stressed subcutaneous white adipose tissue (scWAT) undergo alternative activat

167 White adipose tissue showed a >2-fold increase inUcp1gen

168 own adipocyte-specific genes and proteins in white adipose tissue, substantially increasing oxygen co

169 white adipose tissue (eWAT) and subcutaneous white adipose tissue (sWAT) were analyzed.

170 nd mitochondrial dysfunction in subcutaneous white adipose tissue (sWAT).

171 5(+)Cd11b(+)Cd11c(+)MHCII(+) F4/80(-) DCs in white adipose tissue than did wild-type controls.

172 In skeletal muscle and white adipose tissue, the abundance of GLUT4 protein, bu

173 s well as browning and lipid mobilization in white adipose tissue through stimulation of the sympathe

174 luation of MSCs from human bone marrow (BM), white adipose tissue, umbilical cord, and skin cultured

175 weight of various tissues but the brown and white adipose tissues underwent much more pronounced wei

176 ot show "browning" of abdominal subcutaneous white adipose tissue upon cold acclimation.

177 stem, whereas it increases fat deposition in white adipose tissue via the suppression of sympathetic

178 c function and AdipoQ expression in visceral white adipose tissue (VWAT) of offspring mice are unknow

179 irect calorimetry was performed and visceral white adipose tissues (VWAT) were assessed for inflammat

180 Induction of recall responses within white adipose tissue was associated with the collapse of

181 AO and lipolytic genes in FL-PGC-1alpha(-/-) white adipose tissue was highly correlated with decrease

182 ereas sensitivity of the skeletal muscle and white adipose tissue was lower in HFHS than control dams

183 ncoupling protein 1 competent brite cells in white adipose tissue was not influenced by presence or a

184 dified mice to define the roles of Chi3l1 in white adipose tissue (WAT) accumulation and Th2 inflamma

185 White adipose tissue (WAT) actively stores and releases

186 gh-fat diet (HFD)-induced M1-M2 imbalance in white adipose tissue (WAT) and blocked HFD-induced obesi

187 IGFRKO) had a approximately 25% reduction in white adipose tissue (WAT) and brown adipose tissue (BAT

188 ore, palmitate oxidation was elevated in the white adipose tissue (WAT) and brown adipose tissue of A

189 sue (BAT) and subcutaneous inguinal (SC Ing) white adipose tissue (WAT) and how it affects whole-body

190 TFAM in the subcutaneous and intra-abdominal white adipose tissue (WAT) and interscapular brown adipo

191 ughout the study and biochemical analyses of white adipose tissue (WAT) and liver were performed.

192 Col5a2 knockdown also led to loss of dermal white adipose tissue (WAT) and markedly decreased abdomi

193 recruitable BAT (rBAT), which resides within white adipose tissue (WAT) and skeletal muscle, and has

194 White adipose tissue (WAT) and the liver specifically ex

195 l model to investigate perivascular cells in white adipose tissue (WAT) and their potential to cause

196 hat endothelial production of PDGF-CC during white adipose tissue (WAT) angiogenesis regulates WAT br

197 ereas preferential expansion of subcutaneous white adipose tissue (WAT) appears protective.

198 Beige adipocytes in white adipose tissue (WAT) are similar to classical brow

199 on BA and the TGR5 receptor in subcutaneous white adipose tissue (WAT) are unknown.

200 nhanced Akt and AMPK signaling in muscle and white adipose tissue (WAT) as well as increased FoxO1 ph

201 We detected significant white adipose tissue (WAT) browning and improved systemi

202 fraction (SVF) under conditions that promote white adipose tissue (WAT) browning in mice.

203 fn expression in epididymal and subcutaneous white adipose tissue (WAT) but not in the liver or muscl

204 White adipose tissue (WAT) can undergo a phenotypic swit

205 Here, we discuss the role of white adipose tissue (WAT) cells and of related soluble

206 evated indicators of fatty acid oxidation in white adipose tissue (WAT) compared with control mice.

207 show that Sucnr1 is highly expressed in the white adipose tissue (WAT) compartment of mice and regul

208 Prenatal TBT exposure increased most white adipose tissue (WAT) depot weights, adipocyte size

209 uction of LCN2 expression and secretion from white adipose tissue (WAT) depots, the induction of LCN2

210 lated gene when comparing gene expression in white adipose tissue (WAT) from adipose-specific Glut4-k

211 We determined that white adipose tissue (WAT) from CDK4-deficient mice exhi

212 esity results from an excessive expansion of white adipose tissue (WAT) from hypertrophy of preexisti

213 White adipose tissue (WAT) functions as an energy reserv

214 LSD1 interacts with PRDM16 to repress select white adipose tissue (WAT) genes but also represses hydr

215 ity, the physiological functions of Epac1 in white adipose tissue (WAT) has not been explored.

216 The recent discovery of browning of white adipose tissue (WAT) has raised great research int

217 Persistent immune activation in white adipose tissue (WAT) impairs insulin sensitivity a

218 that IEX-1 expression was highly induced in white adipose tissue (WAT) in both epidydmal and subcuta

219 Tbx15 is also differentially expressed among white adipose tissue (WAT) in different body depots.

220 RK1/2 phosphatase, was induced in epididymal white adipose tissue (WAT) in response to diet-induced o

221 Human subcutaneous (SC) white adipose tissue (WAT) increases the expression of b

222 Obesity-induced white adipose tissue (WAT) inflammation and insulin resi

223 ajor risk factor for metabolic disease, with white adipose tissue (WAT) inflammation emerging as a ke

224 eleased Rosi promotes both transformation of white adipose tissue (WAT) into brown-like adipose tissu

225 In contrast, the transcriptional response in white adipose tissue (WAT) involved a depot-specific ind

226 White adipose tissue (WAT) is a complex organ with both

227 Promoting brown-like transformation in white adipose tissue (WAT) is a promising strategy for c

228 uring obesity, chronic inflammation of human white adipose tissue (WAT) is associated with metabolic

229 White adipose tissue (WAT) is essential for maintaining

230 e phenotype (CD45-CD34(+)) resident in human white adipose tissue (WAT) is known to promote the progr

231 White adipose tissue (WAT) is not only a lipogenic and f

232 The primary task of white adipose tissue (WAT) is the storage of lipids.

233 es chronic macrophage-driven inflammation in white adipose tissue (WAT) leading to insulin resistance

234 ic acetyl CoA by suppression of lipolysis in white adipose tissue (WAT) leading to reductions in pyru

235 Subsequent macrophage infiltration into white adipose tissue (WAT) leads to increased lipolysis,

236 r specifically in adipocytes, led to reduced white adipose tissue (WAT) mass, but resulted in an even

237 a high-fat diet (HFD) and that key liver and white adipose tissue (WAT) metabolic genes are altered i

238 as most significantly attenuated in visceral white adipose tissue (WAT) of DIO mice, and was coincide

239 ls and cytokines were decreased by 50-90% in white adipose tissue (WAT) of Mif(-/-) mice.

240 we show that RIPK3 is over expressed in the white adipose tissue (WAT) of obese mice fed with a chol

241 The critical influence of the white adipose tissue (WAT) on metabolism is well-appreci

242 White adipose tissue (WAT) overgrowth in obesity is link

243 White adipose tissue (WAT) primarily functions as an ene

244 ty lipoprotein-triglycerides (VLDL-TGs) into white adipose tissue (WAT) rather than oxidative tissues

245 pulation of beige adipocytes is increased in white adipose tissue (WAT) reflects a potential strategy

246 White adipose tissue (WAT) releases fatty acids from sto

247 Maintenance of white adipose tissue (WAT) requires the proliferation an

248 ntral to these pathologies is the fat depot: white adipose tissue (WAT) stores excess calories, and b

249 ed 56% less body weight and 74% less gonadal white adipose tissue (WAT) than WT mice.

250 Glucocorticoids promote lipolysis in white adipose tissue (WAT) to adapt to energy demands un

251 of rapamycin complex 2 (mTORC2) functions in white adipose tissue (WAT) to control expression of the

252 pose tissue (BAT) and discriminating it from white adipose tissue (WAT) using cross-validation via PE

253 "brown-like" adipocytes within subcutaneous white adipose tissue (WAT) via a mechanism that stimulat

254 ed attenuation of macrophage infiltration in white adipose tissue (WAT) was associated with reduced l

255 Here we observed that Egr-1 expression in white adipose tissue (WAT) was highly correlated with di

256 Sections of rat BAT and subcutaneous white adipose tissue (WAT) were stained for CB1 and unco

257 nses, and were recently identified in murine white adipose tissue (WAT) where they may act to limit t

258 lar brown tissue (iBAT) and those induced in white adipose tissue (WAT) with respect to their thermog

259 roduces a rapid and persistent remodeling of white adipose tissue (WAT), an increase in energy expend

260 glucose uptake in vivo into endogenous BAT, white adipose tissue (WAT), and heart muscle but, surpri

261 l BW), and inflammatory leukocyte content in white adipose tissue (WAT), despite comparable food inta

262 embrane protein that, upon overexpression in white adipose tissue (WAT), exerts a positive impact on

263 In white adipose tissue (WAT), FGF21 regulates aspects of g

264 Activation of inflammation in white adipose tissue (WAT), includes infiltration/expans

265 have long been known to cause adaptations to white adipose tissue (WAT), including decreases in cell

266 s to inhibition of lipogenesis in epididymal white adipose tissue (WAT), induction of browning in ing

267 sruption promotes the accumulation of TGs in white adipose tissue (WAT), leading to increased adiposi

268 induced obesity because of browning of their white adipose tissue (WAT), leading to increased whole b

269 f acyl-CoAs and histone acetylation in mouse white adipose tissue (WAT), liver, and pancreas.

270 BAT, white adipose tissue (WAT), muscle, liver, and heart wer

271 in the heart and skeletal muscle, but not in white adipose tissue (WAT), suggesting that lipasin supp

272 e tissue (BAT) thermogenesis and browning of white adipose tissue (WAT), which are both potential tar

273 n murine pregnancy, while there is a gain of white adipose tissue (WAT)-like features.

274 alcohol exposure on lipid homeostasis at the white adipose tissue (WAT)-liver axis in a mouse model o

275 and the number of brown-like/beige cells in white adipose tissue (WAT).

276 ige/brite adipocytes (so-called browning) in white adipose tissue (WAT).

277 ied by a decreased norepinephrine content in white adipose tissue (WAT).

278 hile enhancing thermogenesis and browning of white adipose tissue (WAT).

279 mportance of the enzyme for TG catabolism in white adipose tissue (WAT).

280 and human tissues reveals high enrichment in white adipose tissue (WAT).

281 ression in liver, TDAG51 was also present in white adipose tissue (WAT).

282 process of pre-adipocyte differentiation in white adipose tissue (WAT).

283 ol extract (CSEE) upon lipid accumulation in white adipose tissue (WAT).

284 ociations with changes in gene expression in white adipose tissue (WAT).

285 nesis and programming of beige adipocytes in white adipose tissue (WAT).

286 al co-activator Prdm16 regulates browning of white adipose tissue (WAT).

287 ar interest in thermogenesis and browning of white adipose tissue (WAT).

288 es in classic brown adipose tissue (BAT) and white adipose tissue (WAT).

289 triacylglycerol stored by adipocytes in the white adipose tissue (WAT).

290 nergy expenditure, partly due to browning of white adipose tissue (WAT).

291 exercise, brown fat cells also emerge in the white adipose tissue (WAT; also known as beige cells), a

292 Lipolysis in white adipose tissues (WAT) and lipolysis-induced blood

293 Beige/brite adipocytes are induced within white adipose tissues (WAT) and, when activated, consume

294 iver and visceral adipose tissue (epididymal white adipose tissue [WAT]), reduced WAT inflammation, e

295 rance of brown adipocytes (BAs) in brown and white adipose tissues (WATs) of adult mice.

296 White adipose tissue, where APN is produced and assemble

297 Here we report that FTO has a role in white adipose tissue which modifies its response to high

298 In contrast with white adipose tissue, which stores excess energy in the

299 of successful strategies to target brown and white adipose tissues will depend on investigations that

300 ed MPO expression and activity in epididymal white adipose tissue, with an increase in body weight ga