ライフサイエンスコーパス: white blood cell

1 xy-2'-deoxyguanosine, plasma fibrinogen, and white blood cells).

2 rase (LE), an enzyme produced by leukocytes (white blood cells).

3 ion of disseminated cells with platelets and white blood cells.

4 e suggested that lipids increase activity of white blood cells.

5 of illegitimate transcripts from peripheral white blood cells.

6 resence of abdominal pain and an increase in white blood cells.

7 85) using a pyrosequencing assay in DNA from white blood cells.

8 gnetophoresis to remove magnetically-labeled white blood cells.

9 an in adjacent unaffected adrenal tissue and white blood cells.

10 ed tissue), and injection of (111)In-labeled white blood cells 24 h earlier.

11 macrophages engulf large numbers of red and white blood cells, a process called hemophagocytosis.

12 tudy binding of human platelets and platelet-white blood cell aggregation.

13 een reduction in platelet count and platelet-white blood cell aggregation.

14 e size, we observed significant reduction of white blood cell and absolute lymphocyte count up to 1 y

15 White blood cell and absolute lymphocyte count were sign

16 ads to substantial increases in the clinical white blood cell and granulocyte count and is a well-doc

17 White blood cell and neutrophil counts, serum or plasma

18 rmed at an outside hospital) showed elevated white blood cell and platelet counts but low hemoglobin

19 CSF in immunocompetent hosts with normal CSF white blood cell and protein levels (</=5 cells/mm(3) an

20 inine clearance, calcium level, below-normal white blood cell and/or platelet count, polychemotherapy

21 leukocytes, we achieve 3.8-log depletion of white blood cells and a 97% yield of rare cells with a s

22 find more than 4,000 protein-coding mRNAs in white blood cells and adipose tissue to have seasonal ex

23 4A3 transcript abundance was reduced in both white blood cells and mesenchymal cells of RCPS-affected

24 r aim was to determine the minimal number of white blood cells and the specific abilities of mononucl

25 DNA was isolated from peripheral blood white blood cells and TL was measured in DNA using the m

26 DNA was isolated from peripheral blood white blood cells and TL was measured in DNA using the m

27 pathogenesis; for example, they lyse red and white blood cells and trigger inflammatory responses.

28 otropy, being also associated with platelet, white blood cell, and lipid traits.

29 bundance cell types such as red blood cells, white blood cells, and platelets.

30 Leukocytes (white blood cells) are important participants at the var

31 hod to count red blood cells, platelets, and white blood cells, as well as to determine hemoglobin in

32 eads to a progressive increase in peripheral white blood cells, associated with increasing splenomega

33 for inferring changes in the distribution of white blood cells between different subpopulations (e.g.

34 lineages by Pitx1, Pitx2, Barhl2, and Lmx1a; white blood cells by Myb, Etv2, and Tbx6, and ovary by P

35 ted a vertical flow platform that quantifies white blood cells by trapping them in the paper.

36 ontinues to multiply is that a population of white blood cells called CD4 T cells that targets the vi

37 g dd-cfDNA to the proportion of donor DNA in white blood cells can differentiate between relapse and

38 ived from red blood cells (RBCs), platelets, white blood cells, cancer cells, and bacteria, exhibit p

39 ecovery of spiked cancer cells with very low white blood cell contamination (<1000).

40 cell count <13 x 10(9)/L) and proliferative (white blood cell count >/=13 x 10(9)/L) CMML.

41 r pseudomembranous colitis within 5 days; or white blood cell count >/=15 000 cells/microL within 1 d

42 te >90 bpm, mean arterial pressure <60 mmHg, white blood cell count >/=15 000 cells/mL, age >60 years

43 onvert to 109 per liter, multiply by 0.001); white blood cell count >/=15000/microL, 27% (95% CI, 18%

44 High-risk patients (those presenting with a white blood cell count >10 x 10(9) cells per L) could re

45 aboratory abnormality, commonly defined by a white blood cell count >100,000/microL, caused by leukem

46 ls/mm, a hemoglobin level </= 120 g/L, and a white blood cell count >11 g/L within 90 days before the

47 liferative chronic myelomonocytic leukaemia (white blood cell count <13 000/muL), and had anaemia wit

48 ed, CMML is stratified into myelodysplastic (white blood cell count <13 x 10(9)/L) and proliferative

49 OR, 3.9; 95% CI, 1.4-11.1), high peripheral white blood cell count (>10 x 10(9) cells/L; OR, 8.7; 95

50 Optimal thresholds for white blood cell count (11600/microL), absolute neutroph

51 most common grade 3 or 4 toxicities were low white blood cell count (14 [11%] in the CRT plus cetuxim

52 fusion (4.5% compared with 16.4%; P < 0.05), white blood cell count (14.4 +/- 3.3 compared with 15.6

53 ia (26 [29%]), anaemia (26 [29%]), decreased white blood cell count (17 [19%]), and decreased lymphoc

54 ients), leucopenia (27 [10%]), and decreased white blood cell count (21 [8%]).

55 g/dL), low glucose level (2 mg/dL), and high white blood cell count (330/mm(3); 28% lymphocytes, 56%

56 count (31 [10%] vs 41 [13%]), and decreased white blood cell count (39 [13%] vs 33 [11%]).

57 ucose level (124 vs 134 mg/dL, P = .03), and white blood cell count (6600/muL vs 17 200/muL, P < .001

58 normal range, 12-60 mg/dL), and an elevated white blood cell count (7/mm(3) [0.007 x10(9)/L] in tube

59 (4.9 [4.0-5.8] vs 4.5 [3.7-5.5] mg/dL), and white blood cell count (7000 [5900-8200] vs 6600 [5600-7

60 02), bruising (aOR, 3.17; P=.0059), abnormal white blood cell count (aOR, 0.52; P=.0100), and prior a

61 s significantly higher in patients with high white blood cell count (HR 2.45, p 0.011), raised serum

62 s: age (hazard ratio [HR], 3.299; P < .001), white blood cell count (HR, 1.910; P = .017), platelet c

63 ectiveness of four strategies for monitoring white blood cell count (national strategies used in the

64 04 [95% CI, .006-.23], P < .0001); and lower white blood cell count (OR = 0.93 [95% CI, .89-.97], P <

65 ent of Model for End-Stage Liver Disease and white blood cell count (OR, 4.68; 95% CI, 1.80-12.17; P

66 1), whereas it was inversely associated with white blood cell count (P < 0.0001).

67 cose level, hypertension (each P < .01), and white blood cell count (P = .04).

68 levels (multiple regression, P = 0.019) and white blood cell count (P = 0.032), whereas the number o

69 was significantly longer in patients with a white blood cell count (WBC) <50 Giga per liter (G/L) (P

70 a novel PheWAS using an individual's maximum white blood cell count (WBC) as a continuous measure.

71 eal inflammation, fluid, appendicoliths, and white blood cell count (WBC) were significantly correlat

72 White blood cell count also provides predictive informat

73 White blood cell count analysis after alpha-radioimmunot

74 ose episodes, 213 had data allowing complete white blood cell count analysis and were included in the

75 White blood cell count and albumin level are the most cl

76 re significantly predicted only by patients' white blood cell count and albumin level.

77 selective inhibitor tofacitinib reduced the white blood cell count and caused leukemic cell apoptosi

78 the mean value of several laboratory tests (white blood cell count and hepatic and lipid panels), ye

79 ence, waist-hip ratio, alanine transaminase, white blood cell count and lower high-density lipoprotei

80 e model uses repeatedly measured biomarkers (white blood cell count and lymphocyte percent) to predic

81 l fluid examination revealed mildly elevated white blood cell count and protein levels.

82 for sex, age (<10 years vs >/=10 years), and white blood cell count at diagnosis (<50 x 10(9)/L vs >/

83 y MRD result and balancing for sex, age, and white blood cell count at diagnosis by method of minimis

84 te respiratory, cardiac, and liver function, white blood cell count at least 3 x 10(9) cells per L, p

85 36) and OS (HR, 0.64; P = .02), with initial white blood cell count being the only factor significant

86 sition, increasing both neutrophil and total white blood cell count by 6 hours post-injection.

87 luorescent labeling or Coulter counting, the white blood cell count can be defined directly from a mi

88 atinib had a larger spleen size and a higher white blood cell count compared with those with BCR-ABL1

89 ssure, ultrafiltration rate, phosphorus, and white blood cell count declined (all P<0.001).

90 In this large cohort of APL patients, high white blood cell count emerged as an independent predict

91 With white blood cell count emerging as an important risk fac

92 ven percent of patients with CDI had a serum white blood cell count greater than 12 000 cells per muL

93 Conclusions and Relevance: White blood cell count greater than 20000 cells/microL a

94 al bilirubin level greater than 10 mg/dL and white blood cell count greater than 20000 cells/microL.

95 fectiveness of various strategies to monitor white blood cell count in adult patients with schizophre

96 The proposed platform enabled rapid white blood cell count in low resource settings with a s

97 far there has been no rapid test that allows white blood cell count in low-resource settings.

98 Existing strategies for monitoring white blood cell count in patients taking clozapine, bas

99 acterial infection with better accuracy than white blood cell count in the blood.

100 cholesterol in men, and with higher BMI and white blood cell count in women (differences 0.03-0.06 s

101 vein occlusion, n=1 each; placebo: vomiting, white blood cell count increased, n=1 each).

102 White blood cell count is an important indicator of each

103 Long-term monitoring of white blood cell count is compulsory in patients taking

104 Currently, white blood cell count is primarily conducted in central

105 plete blood cell count parameter thresholds: white blood cell count less than 5000/microL, 10% (95% C

106 ) OR (95% CI), 1.66 (1.21-2.29); P = 0.002], white blood cell count more than 16,000 [OR (95% CI), 1.

107 4 vs 0-2 and 5.20 (95% CI, 2.70-10.02) for a white blood cell count of >/=20 000/muL vs <20 000/muL.

108 He was found to have an elevated white blood cell count of 12.2 x 10(9)/L (reference rang

109 oratory analyses were notable for a complete white blood cell count of 17000/muL (31% blast cells), a

110 (n = 11), median age of 12 years, and median white blood cell count of 48.8 x 10(9)/L.

111 tologic laboratory investigations revealed a white blood cell count of 6.7 x 10(9), a C-reactive prot

112 Cerebrospinal fluid analysis showed a white blood cell count of 60/muL (to convert to x109 per

113 analysis of the validation cohort confirmed white blood cell count of more than 20000 cells/microL (

114 Therefore, monitoring white blood cell count on a regular basis can potentiall

115 globulins vs. late immunotherapy), and a low white blood cell count on the first cerebrospinal examin

116 factor positivity, and inflammatory markers (white blood cell count or cytokine level).

117 Fever, high white blood cell count or immature forms, low Glasgow co

118 halopathy, longer length of stay, and higher white blood cell count or MELD score at discharge.

119 r both for clinical examination findings and white blood cell count parameters compared with a valid

120 cases had higher median cerebrospinal fluid white blood cell count than noninfectious etiologies.

121 V patients were more likely to have a normal white blood cell count than the control group (82% vs 52

122 An abnormal white blood cell count usually results from an infection

123 The patient's white blood cell count was 6.8 x 10(9)/L.

124 The white blood cell count was determined by measuring the c

125 White blood cell count was normal, and there was no infl

126 l duration (QTc), deceleration capacity, and white blood cell count was not associated with UFP, AMP,

127 No significant differences in sex and white blood cell count were found.

128 superinfection, and elevated urea level and white blood cell count were independently associated wit

129 Her coagulation profile and white blood cell count were within normal limits.

130 elevated total protein and a mildly elevated white blood cell count with lymphocytic predominance.

131 atients with eosinophil counts (out of total white blood cell count) of 2% or greater (rate ratio 1.2

132 factor-alpha receptor 2, interleukin-6, and white blood cell count), oxidative stress (8-isoprostane

133 2 laboratory markers (C-reactive protein and white blood cell count).

134 /m(2) on day 1) added to high-risk patients (white blood cell count, >10 x 10(9)/L), as well as low-r

135 gic laboratory test results were as follows: white blood cell count, 11.2 x10(9)/L (normal range, [4.

136 Laboratory evaluation revealed leukocytosis (white blood cell count, 15.4 x 10(9)/L; normal range, [3

137 atase, 88.35 U/L (58.94-117.76 U/L); and for white blood cell count, 6890/microL (5700/microL-8030/mi

138 We tested the accuracy of the white blood cell count, absolute neutrophil count, and p

139 subset of patients with FLT3-ITD, only age, white blood cell count, and < 4-log reduction in PB-MRD,

140 ith VAC also have an abnormal temperature or white blood cell count, and be started on a new antimicr

141 er adjustment for age, sex, current smoking, white blood cell count, and fish consumption, each 1-SD

142 r pulse, higher waist-to-hip ratio, elevated white blood cell count, and heart failure.

143 Toxicity analysis included body weight, white blood cell count, and hematocrit.

144 patients had more severe neuropathy, higher white blood cell count, and lower endothelium-dependent

145 ere older, had a higher hemoglobin level and white blood cell count, and lower platelet count and ser

146 core, red blood cell transfusion dependency, white blood cell count, and marrow blasts retained indep

147 ng disease progression significantly reduced white blood cell count, blast cells, splenomegaly, lacta

148 ne were older; were hypertensive; had higher white blood cell count, blood glucose, D-dimer, and seru

149 cases had normal, the other two had elevated white blood cell count, but all of them had elevated CRP

150 ion and 8 other risk factors, including age, white blood cell count, cytogenetics, and gene mutations

151 rior descending CA, respectively), and lower white blood cell count, erythrocyte sedimentation rate,

152 Six biomarkers (white blood cell count, fibrinogen, D-dimer, troponin T,

153 th poor outcome (CSF culture positivity, CSF white blood cell count, hemoglobin, Glasgow Coma Scale,

154 performance status of two or more, increased white blood cell count, high-risk IPSS score, and higher

155 tors using tricuspid regurgitation velocity, white blood cell count, history of acute chest syndrome,

156 r pulse, higher waist-to-hip ratio, elevated white blood cell count, history of heart failure, diabet

157 the inflammatory markers C-reactive protein, white blood cell count, neopterin, and kynurenine:trypto

158 the known BT risk factors, such as age, sex, white blood cell count, percentage of blasts, IPSS progn

159 hypertension, C-reactive protein level, and white blood cell count, this association remained signif

160 Pertinent laboratory values, including white blood cell count, were normal.

161 ted prediction highlighted by our tool: that white blood cell count--a quantitative trait of the immu

162 d increased protein with a slightly elevated white blood cell count.

163 ore (age, creatinine clearance, haemoglobin, white-blood-cell count, and previous spontaneous bleedin

164 his approach had minimal toxicity with nadir white blood cell counts >2.7 K/microL 2 weeks after HSCT

165 Seventeen patients (42.5%) had CSF white blood cell counts >20/muL (mean, 57/muL), and 27 (

166 ssociated with older age (P < .0001), higher white blood cell counts (P < .0001), cytogenetically nor

167 This C. difficile variant elicited higher white blood cell counts and caused disease in younger pa

168 A content is also influenced by platelet and white blood cell counts and estroprogestogen intake.

169 AST and ALT, and negatively correlated with white blood cell counts and fibrinogen in free-ranging d

170 nosuppression, as monitored by reductions in white blood cell counts and lymphocyte proliferation act

171 We tested serum C-reactive protein and white blood cell counts as potential mediators of asthma

172 Raised white blood cell counts as well as peaks of serum levels

173 high-dose group had significantly lower mean white blood cell counts at months 5 and 6; however, prem

174 Red and white blood cell counts can also be performed on human b

175 the agranulocytosis was due to the lifelong white blood cell counts that are now required for clozap

176 White blood cell counts were elevated (>10 x 10(9)/L) in

177 come in PV, whereas response in platelet and white blood cell counts were predictive of less thromboh

178 d aldosterone), hepatobiliary enzyme levels, white blood cell counts, and iron homeostasis.

179 al use of G-CSF in these patients to support white blood cell counts, and suggest that direct targeti

180 the ICU including pancreatic stone protein, white blood cell counts, C-reactive protein, interleukin

181 ng Casp9 or its cofactor Apaf1 developed low white blood cell counts, decreased B-cell numbers, anemi

182 etry, blood samples analyzed for hemoglobin, white blood cell counts, eosinophil counts and total ser

183 etes and lower C-reactive protein levels and white blood cell counts.

184 suppressant FK506 (tacrolimus) decreases CSF white blood cell counts.

185 (30%) patients were afebrile and had normal white blood cell counts.

186 e were no significant changes in hemoglobin, white blood cells, creatinine, or tubular extraction rat

187 decreased (ten [16%] vs four [6%]; p=0.09), white blood cell decreased (15 [24%] vs seven [11%]; p=0

188 r depressed flows, endothelial inflammation, white blood cell-derived tissue factor, and ample red bl

189 flow (PF), spirometry, serum IgE levels and white blood cell differentials were recorded.

190 One white blood cell displayed the average esterolytic activ

191 enic exposure and gene-specific differential white blood cell DNA methylation, suggesting that epigen

192 l arsenic concentrations, and epigenome-wide white blood cell DNA methylation.

193 onger telomere length measured in peripheral white blood cell DNA was associated with increased risk

194 ed and fixed cells, such as red blood cells, white blood cells, DU-145 prostate cancer cells, MCF-7 b

195 Moreover, human white blood cells exposed to heat-killed Pseudomonas aer

196 pansion (GGGGCC) in patient cells, including white blood cells, fibroblasts, glia, and multiple neuro

197 e ratio of Gr1(+) granulocytes in peripheral white blood cells following bacteremia.

198 or future investigations when alterations in white blood cell fractions are unavailable.

199 reduced ubiquitination activity in vitro and white blood cells from affected individuals exhibited si

200 d then used in a meta-analysis of peripheral white blood cells from healthy control samples in two ex

201 nstrated improved enrichment and recovery of white blood cells from human blood.

202 visually differentiate the abnormal count of white blood cells from the normal count.

203 In multivariate analysis, risk group, white blood cell >100 x 10(9)/L at diagnosis, and monoso

204 atures derived from genome-wide profiling in white blood cells, identifying 26 expression probes and

205 also demonstrate that accurate estimates of white blood cell images can be recovered from extremely

206 Neutrophils are the predominant circulating white blood cell in humans, and contain an arsenal of to

207 device proved effective to retain >99.9% of white blood cells in 100 mul of WB without affecting pla

208 sing this platform, we were able to quantify white blood cells in 15 muL of blood, and visually diffe

209 ormed a genome-wide DNA methylation study of white blood cells in a population-based study (N = 717).

210 Neutrophils are the most abundant white blood cells in circulation, and patients with cong

211 For proof of principle, enumeration of the white blood cells in human blood samples on the RDM prov

212 fection marks neutrophils (the most abundant white blood cells in humans) as vital immune defenders.

213 n patterns of gene expression in circulating white blood cells in response to infection.

214 emia, these mice do not display elevation of white blood cells in the spleen or bone marrow; rather,

215 yte-macrophage colony-stimulating factor and white blood cells in transgenic sickle mice.

216 se cells types in vitro, and endothelial and white blood cells in vivo (ex ovo chick embryo model).

217 le cancer cell lines and 99.99% depletion of white blood cells in whole blood.

218 al fluid opening pressure of 28 mm H2O and 8 white blood cells, including 1 atypical plasma cell.

219 hypothalamic neurons, as well as a subset of white blood cells, including mast cells.

220 Observations noting the presence of white blood cell infiltrates within tumors date back mor

221 nduced increase in lactate:pyruvate ratio in white blood cells is alleviated.

222 In contrast to CD45(pos) white blood cells isolated and processed by the identica

223 mmune system through activation of a type of white blood cell known as natural killer T cell (NKT cel

224 he red blood cell, the neutrophil, and other white blood cell lineages.

225 helial cells/low-power field [lpf] and >/=25 white blood cells/lpf or a quality score [q-score] defin

226 Salsalate treatment decreased total white blood cell, lymphocyte, monocyte, and neutrophil c

227 ls with longer telomere length in peripheral white blood cells may have an increased risk of lung can

228 Cerebrospinal fluid (CSF) pleocytosis (>5 white blood cells/microL) was common (81%).

229 ffect was observed in the plasma (sHLA-G) or white blood cells (miRNA).

230 r examining the association between inferred white blood cell mixtures in infant cord blood and in ut

231 asts could also be distinguished from benign white blood cells (notably these also lacked MDR activit

232 White blood cells of the immune system--known as leukocy

233 rine culture) plus pyuria (ie, any number of white blood cells on urinalysis) assessed every 2 months

234 PA-b binding activity could be found in fish white blood cells or fish cell lines.

235 h lower global methylation in umbilical cord white blood cells (p = 0.05), but global methylation lev

236 e score included age, diagnosis, creatinine, white blood cells, platelets, albumin, use of vasopresso

237 in red blood cells, hemoglobin, hematocrit, white blood cells, platelets, and splenomegaly, deletion

238 adhesion between RBCs, and between RBCs and white blood cells, platelets, and the endothelium.

239 White blood cells play diverse roles in innate and adapt

240 jury are caused by the adhesion of a type of white blood cell--polymorphonuclear neutrophils--to the

241 Of note, neutrophils are the largest human white blood cell population.

242 reted by Staphylococcus aureus, which target white blood cells preferentially and consist of an S- an

243 regulated genes were linked to activation of white blood cells, production of cytokines, and inhibiti

244 mma-induced protein 10 (r = 0.39; P = .002), white blood cells (r = 0.32; P = .004), protein (r = 0.5

245 structure of a nucleotide exchange factor in white blood cells reveals an autoinhibitory mechanism th

246 rrelation between PPP1R11 and TLR2 levels in white blood cell samples isolated from patients with Sta

247 decarboxylase (HDC) gene directly from human white blood cells samples.

248 Interestingly, complete white blood cell sampling enabled determining an upper l

249 o compared with available bone scintigraphy, white blood cell scintigraphy, and (18)F-FDG PET/CT resu

250 Autoimmune diseases mediated by a type of white blood cell-T lymphocytes-are currently treated usi

251 nd educational factors, were associated with white blood cell telomere length at age 49-51 years.

252 Eosinophils are white blood cells that function in innate immunity and p

253 ay produce interacting epigenetic effects in white blood cells that influence immune function and hea

254 s (CACs) are an exercise-inducible subset of white blood cells that maintain vascular integrity.

255 sity of gold nanoparticles on the surface of white blood cells that were trapped in the paper mesh.

256 L and monocytes accounting for >/=10% of the white blood cells, this aging-associated disease combine

257 by computationally estimated proportions of white blood cell types.

258 vidual plasma proteins decreased, as did the white blood cell types.

259 ficient toxicological safety on normal human white blood cells was isolated and named HM-3A.

260 White blood cell (WBC) analysis provides rich informatio

261 age at time of cough onset, and higher peak white blood cell (WBC) and lymphocyte counts.

262 We investigated whether aortic and white blood cell (WBC) CoQ is programmed by suboptimal e

263 years (hazard ratio [HR] = 0.58, P = .002), white blood cell (WBC) count <10 x 10(9)/L (HR = 0.60, P

264 of CSF inflammation, including decreased CSF white blood cell (WBC) count (P < .001), interleukin (IL

265 hether there might be an interaction between white blood cell (WBC) count and bivalirudin for the ris

266 White blood cell (WBC) count appears to predict total mo

267 White blood cell (WBC) count is a common clinical measur

268 Elevated white blood cell (WBC) count is associated with increase

269 Average white blood cell (WBC) count was 37.7 x 10(9)/microL.

270 avy marijuana use and HIV disease markers or white blood cell (WBC) count were examined using mixed-e

271 say, C-reactive protein (CRP) concentration, white blood cell (WBC) count, and absolute neutrophil ce

272 reactive protein (hsCRP), lipid profile, and white blood cell (WBC) count, at baseline and 1, 3, and

273 hibited a significantly elevated circulating white blood cell (WBC) count, whereas animals injected w

274 cular volume (MCV), platelet count (PLT) and white blood cell (WBC) count.

275 es were the oncogenetic classifier, MRD, and white blood cell (WBC) count.

276 L) patients with >30% bone marrow blasts and white blood cell (WBC) counts </=15 x 109/L (AZA-AML-001

277 Circulating white blood cell (WBC) counts (neutrophils, monocytes, l

278 hemoglobin concentration, hematocrit levels, white blood cell (WBC) counts and platelet counts in 31,

279 Platelet (PLT) and white blood cell (WBC) counts are 2 markers of inflammat

280 lyses of phenotypic variation in circulating white blood cell (WBC) counts from large samples of othe

281 ariability (HRV) with electrocardiogram, and white blood cell (WBC) counts with hematology analyzer.

282 brary consisting of over one quarter-million white blood cell (WBC) nuclei together with CD15/CD16 pr

283 ous titer, the intravenous administration of white blood cells (WBC) resulted in efficient disease tr

284 from mammalian cells, more specifically from white blood cells (WBC).

285 ted that CyHV-3 becomes latent in peripheral white blood cells (WBC).

286 Conventional inflammatory markers (white blood cell [WBC] count, erythrocyte sedimentation

287 xamethylpropyleneamine oxime [HMPAO]-labeled white blood cells [WBC]).

288 of Treponema pallidum 16S RNA in CSF or CSF white blood cells (WBCs) >20/uL or a reactive CSF-Venere

289 rin, leukocyte function antigen-1 (LFA-1) on white blood cells (WBCs) and causes cell death.

290 White blood cells (WBCs) play an important role in host

291 l blood mononuclear cells (PBMCs) and of CSF white blood cells (WBCs) that were activated monocytes (

292 DNA methylation in white blood cells (WBCs) was associated with total IgE l

293 dium (Na(+)) channel mRNA splice variants in white blood cells (WBCs) with risk of arrhythmias in hea

294 o trigger an antimicrobial response in human white blood cells (WBCs).

295 White blood cells were tagged with gold nanoparticles, a

296 sidual disease negativity with CLL <1/10 000 white blood cells, which persisted even after ibrutinib

297 Eosinophils are rare white blood cells whose activities are primarily destruc

298 0% with a contamination of 0.6 +/- 0.1% from white blood cells with a 23.8 +/- 1.3-fold concentration

299 +/- 2.1% with 0.2 +/- 0.04% contamination of white blood cells with a 9.6 +/- 0.4-fold concentration

300 , and produce an ultrapure buffy coat (96.6% white blood cell yield, 0.0059% red blood cell carryover