ライフサイエンスコーパス: white spot

1 f melanocytes and the clinical appearance of white spots.

2 of me(v)/me(v) mice revealed numerous, small white spots.

3 abs were demineralized to produce artificial white spots.

4 ease of the skin that results in disfiguring white spots.

5 he retinal pigment epithelium (89.3%), small white spots (80.2%), and optic nerve atrophy (76.9%) wer

6 Saponins appeared as white spots against a pink background.

7 ption, and was marked by the appearance of a white spot and a dark ring, coincident with entry into m

8 mature leaves suffered from burnt edges and white spots as well as a reduction in photosynthetic pig

9 ith progressive piebaldism, exhibit dominant white spotting but show no evidence of progressive depig

10 ents with TSC develop hypomelanotic macules (white spots), but the molecular mechanisms underlying th

11 the survival of these cells in vivo, because white spotting (c-kit(W/W)) mice, carrying a natural ina

12 A low-caries group had no "white spot" caries (ws) in the selected dentition area;

13 cassava orthologous gene resulting in yellow-white spots characteristic of the inhibition of su expre

14 caries-free subjects (SRS), (3) plaque from "white spot" coronal lesions and sound coronal surfaces o

15 virus susceptibility gene 2 (Fv2), dominant white spotting gene (W), and Steel gene (Sl), regulate t

16 normal a-wave and b-wave activity, yellowish-white spots, hyperfluorescence, and reduced retinal thic

17 We have used the varied expressivity of white spotting (hypopigmentation) observed in intrasubsp

18 n, which are defined clinically as yellowish white spots in the outer retina, are cardinal features o

19 s generalized hypopigmentation and localized white-spotting in mice, with a lack of pigment on the be

20 es that differentiate this entity from other white spots, including acute placoid multifocal pigment

21 White spot lesions (WSL) due to enamel demineralization

22 White-spot lesions (WSL) associated with orthodontic app

23 Using enamel white-spot lesions, we hypothesize that the optical scat

24 appliances carrying an enamel slab cut from white-spot lesions.

25 e have a naturally occurring mutation in the white spotting locus that causes reduced c-Kit receptor

26 Adamts20 mutations cause the mouse white-spotting mutant belted (bt), whereas Adamts9 is es

27 nlight (mnlt), a second hypopigmentation and white-spotting mutation identified on the C57BL/6J backg

28 que from those described for other classical white-spotting mutations.

29 descriptions of a dilution of coat color and white spotting of the belly and extremities, suggesting

30 he silencing phenotype ranged from scattered white spots on the normal purple background to entirely

31 natal lethality, with viable mice exhibiting white spotting on their ventral surface.

32 e report that a point mutation, the dominant white spotting oncogene allele, Kit(W-42J), exacerbates

33 a pronounced forehead blaze, with additional white spots over the cervical region, as well as a very

34 Transgene induction in skin resulted in a white spotting phenotype due to somatic ORFeus-mediated

35 us that were exclusively present in white or white-spotted pigs, carrying the Dominant white, Patch,

36 traits with mendelian inheritance: the major white spotting (S) locus and the hair ridge in Rhodesian

37 nal detachment of the fovea accompanied with white spots surrounding the fovea in both eyes.

38 Exposure to virus (white spot syndrome baculovirus or M. rosenbergii nodovi

39 White spot syndrome virus (WSSV) is one of the major and

40 mmune defense toward a major viral pathogen, white spot syndrome virus (WSSV).

41 tance to infection by two unrelated viruses, white spot syndrome virus and Taura syndrome virus.

42 includes vertebrates, nonvertebrates, shrimp white spot syndrome virus, Streptococcus equi, and Bacil

43 retinal vasculitis, a characteristic retinal white spot syndrome, Bartonella retinitis, branch retina

44 diseases of the posterior fundus, including white spot syndromes and autoimmune, hereditary, paraneo

45 estruction of epidermal melanocytes produces white spots that can be repigmented by melanocyte precur

46 varian cancer risk using germ cell-deficient white-spotting variant (Wv) mice, incorporating oncogeni

47 us (IHHNV), a single-stranded DNA virus, and white spot virus (WSV), a double-stranded DNA (dsDNA) vi

48 clones from a hepatopancreas cDNA library of white spot virus (WSV)-infected shrimp provided a partia

49 lacking a SCF-responsive population of HPC [White spotted (W/WV) and Steel (SI/SId)] are unresponsiv

50 are encoded in close proximity at the murine white spotting (W) and patch (Ph) loci.

51 its receptor, c-kit, encoded by the dominant white spotting (W) gene.

52 Mice carrying certain mutations in the white spotting (W) locus (ie, c-kit) exhibit reduced c-k

53 Finally, mice having mutations in the white spotting (W) locus, which encodes the SCF-receptor

54 rtile mouse strains, Steel (Sl) and dominant white spotting (W), to determine if stem cells from an i

55 leading these mutants to be termed dominant White spotting (W).

56 the c-kit receptor tyrosine kinase (dominant white spotting, W).

57 nary night blindness characterized by yellow-white spots, which were classically described as subreti