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1 y of CWD infection in natural populations of white-tailed deer.
2 d States after the hunters had field-dressed white-tailed deer.
3 in sheep, elk, and small numbers of mule and white-tailed deer.
4 s an alternative natural reservoir, possibly white-tailed deer.
5 described malignant catarrhal fever virus of white-tailed deer.
6 oir system consisting of lone star ticks and white-tailed deer.
7 ed saliva and urine samples from CWD-exposed white-tailed deer.
8 ly related species (Anaplasma marginale, the white-tailed deer agent, and additional E. chaffeensis-p
9 assay for anaplasma matched sequences of the white-tailed deer agent.
10 ) PrP(c) We demonstrate that the presence of white-tailed deer and bovine NTDs hindered seeded conver
11               As a result, a confirmed 1,000 white-tailed deer and pronghorn antelope died over the c
12           We used bioassay studies of native white-tailed deer and transgenic cervidized mice to dete
13  found seasonal patterns in selection by the white-tailed deer and were able to link use of conifer f
14  of North American cervids, i.e., mule deer, white-tailed deer, and elk (wapiti).
15      Therefore, CWD isolates from mule deer, white-tailed deer, and elk were inoculated intracerebral
16         Brain samples from CWD-positive elk, white-tailed deer, and mule deer produced disease in Tg(
17  from chronic wasting disease (CWD)-infected white-tailed deer brain homogenates and found that lipid
18 est that CWD prions from elk, mule deer, and white-tailed deer can be readily transmitted among these
19                                  Thirty-five white-tailed deer collected from three Amblyomma america
20                                       In the white-tailed deer examples, a recently developed index f
21 ting disease (CWD) and from 210 free-ranging white-tailed deer harvested from an area in Wisconsin wh
22  and from EDTA-blood specimens obtained from white-tailed deer in Maryland.
23 ctly from I. scapularis ticks collected from white-tailed deer in Minnesota and represent the first i
24 phagocytophilum-like organisms obtained from white-tailed deer in the United States.
25 rsection) and genetic relatedness for female white-tailed deer in Wisconsin's area of highest CWD pre
26 in all mule deer sampled but was absent from white-tailed deer, indicating that this virus originally
27 laris, is a vector of the HGE agent, and the white-tailed deer is the primary host for adult Ixodes t
28  the native ungulate Odocoileus virginianus (white-tailed deer) is overabundant and Alliaria petiolat
29    This virus, which causes classical MCF in white-tailed deer, is a newly recognized agent belonging
30 hough A. phagocytophilum-like organisms from white-tailed deer may be closely related to A. phagocyto
31 ,243 retropharyngeal lymph node samples from white-tailed deer, mule deer, and moose, collected in th
32 ush samples collected antemortem from farmed white-tailed deer (n= 409).
33  of habitat use that varied seasonally for a white-tailed deer (Odocoileus virginianus) and coyote (C
34                         We demonstrated that white-tailed deer (Odocoileus virginianus) and elk (Cerv
35 wo lines of Tg mice expressing PrP common to white-tailed deer (Odocoileus virginianus) and mule deer
36 e N. caninum oocysts in their feces and that white-tailed deer (Odocoileus virginianus) are natural i
37 eld and experimental studies have implicated white-tailed deer (Odocoileus virginianus) as probable r
38  to an empirical telemetry data set of three white-tailed deer (Odocoileus virginianus) dyads.
39 linical signs and lesions in five out of six white-tailed deer (Odocoileus virginianus) in a North Am
40        Louis, Missouri region, we found that white-tailed deer (Odocoileus virginianus), a preeminent
41 llus), gray squirrel (Sciurus carolinensis), white-tailed deer (Odocoileus virginianus), and guinea p
42 vement in analyses of first-passage time for white-tailed deer (Odocoileus virginianus).
43  a sable antelope (Hippotragus niger), and a white-tailed deer (Odocoileus virginianus).
44 the prevalence of chronic wasting disease in white-tailed deer (Odocoileus virginianus).
45 , were characterized for E. chaffeensis from white-tailed deer (Odocoileus virginianus).
46  pulverized and inoculated intranasally into white-tailed deer once a week for 6 weeks.
47 (c), hinders seeded conversion of bovine and white-tailed deer PrP(c)s to the prion forms, but it fac
48 examined RAMALT collected postmortem from 76 white-tailed deer removed from a farm in Wisconsin known
49 more efficient seed for feline rPrP than for white-tailed deer rPrP; (iii) conversely, FSE more effic
50 blood from codon 96 glycine/glycine, captive white-tailed deer that were analyzed for prion infection
51 f choice for use for the diagnosis of CWD in white-tailed deer, the results of the present study furt
52 ge gap in early CWD pathogenesis, we exposed white-tailed deer to CWD prions by mucosal routes and pe
53 from the sambar deer, the waterbuck, and the white-tailed deer were collected during winter dysentery
54 een 1998 and 2001, tissues from four captive white-tailed deer were observed to have histologic lesio
55 and orally infected preclinical and infected white-tailed deer with clinical chronic wasting disease
56 e a bacterium previously referred to as the "white-tailed deer (WTD) agent" from two captive fawns in

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