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1 each aphids) and Bemisia tabaci (silver leaf whitefly).
2 nto virions and transmitted as an episome by whiteflies.
3 storical tritrophic relationships in Bemisia whiteflies.
4 ed structural constituent of cuticle in male whiteflies.
5 GS-2 episomes were also found in virions and whiteflies.
6 primary endoparasitoids of scale insects and whiteflies.
7 ptera, such as scale insects, mealybugs, and whiteflies.
9 Hemiptera), including aphids, planthoppers, whiteflies and stink bugs, present one of the greatest c
10 t population values of 26.3 and 39.2 mug per whitefly and 23.1 and 35.2 mug per aphid for BGL-1 and u
11 ther species, females are endoparasitoids of whiteflies, and males are primary endoparasitoids of egg
14 d performance and sex-ratio bias of infected whiteflies are sufficient to explain the spread of Ricke
16 a PGRP gene, BtPGRP, encodes a PGRP from the whitefly Bemisia tabaci (MEAM1) that binds and kills bac
17 dy, the gender difference in the sweetpotato whitefly Bemisia tabaci was investigated using Illumina-
18 um of whiteflies, including the sweet potato whitefly Bemisia tabaci, and provides essential nutrient
19 development of the phloem-feeding silverleaf whitefly (Bemisia tabaci type B; SLWF) on Arabidopsis (A
21 roxyfen in 84 populations of the sweetpotato whitefly (Bemisia tabaci) from cotton fields in central
23 ly driven by two so-called supervectors: the whitefly, Bemisia tabaci, and the Western flower thrips,
24 transmitted specifically by the sweetpotato whitefly, Bemisia tabaci, in a semipersistent manner.
27 formation indicating an involvement of SA in whitefly-derived plant defence against Agrobacterium.
29 mutant CPm genotypes were not transmitted by whiteflies, even though virion concentrations were above
32 ns or recombinant virus capsid components to whiteflies, followed by feeding them antibodies to the v
33 ing RNA interference (RNAi) to down regulate whitefly genes by expressing their homologous double str
36 resistance and the transmission of SEGS-2 by whiteflies has major implications for the long-term dura
37 y immunity and provides insight into how the whitefly immunity acts in complex mechanisms of Begomovi
38 ork addresses a visible gap in understanding whitefly immunity and provides insight into how the whit
39 obligate primary endosymbiotic bacterium of whiteflies, including the sweet potato whitefly Bemisia
44 ana leaves were exposed to a sucking insect (whitefly) infestation and benzothiadiazole (BTH) for 7 d
47 acterium-mediated transformation of roots of whitefly-infested plants was clearly inhibited when comp
50 ing of virus transmission by aphids, thrips, whiteflies, leafhoppers, planthoppers, treehoppers, mite
52 by real time quantitative PCR indicated that whitefly mortality was attributable to the down-regulati
57 used worldwide for the biological control of whiteflies on vegetables and ornamental plants grown in
58 immature E. formosa, population dynamics of whitefly-parasitoid interactions, and commercial use in
60 , has potential as a bio-pesticide to reduce whitefly population size and thereby decrease virus spre
61 h uninfected whiteflies, Rickettsia-infected whiteflies produced more offspring, had higher survival
63 fed to whitefly vectors, significantly more whiteflies retained the recombinant minor coat protein (
72 at controlling pests such as the greenhouse whitefly Trialeurodes vaporariorum (Westwood) which are
73 zed in the anterior foregut or cibarium of a whitefly vector biotype but not within those of a whitef
76 strated previously and in the present study, whitefly vectors failed to transmit virions preincubated
77 g host range, semipersistent transmission by whitefly vectors, and impact on diverse cropping systems
78 V capsid components were individually fed to whitefly vectors, significantly more whiteflies retained
81 it on samples from fission yeast, mouse and whitefly, whose reference genome is not yet available.
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