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1 th published maps revealed a hole in the MIT/Whitehead and CEPH YAC maps that includes the immediate
2 zed CACNB4 to the 1.3-Mb YAC clone 952F10 in Whitehead contig WC861, along with the polymorphic marke
3 ed YAC chimerism and placed three additional Whitehead contigs (WC952, WC799, WC436) within the integ
4 urther narrowed down to a 2 cM (based on the Whitehead genetic map) or 0.36 megabase (based on gdb ma
6 amples from chromosome 17, obtained from the Whitehead Institute Center for Genome Research (WICGR) p
7 ulation and empirically generated data sets (Whitehead Institute's inflammatory bowel disease data se
8 markers D1S117 and D1S466 by analysis of the Whitehead Institute's recently described chromosome 1 co
11 ap of mouse genes, a combined project of the Whitehead Institute/Massachusetts Institute of Technolog
12 re we report the results of a project at the Whitehead Institute/MIT Center for Genome Research to co
13 y screening 154 of the STSs published by the Whitehead Institute/MIT Genome Center, we have identifie
14 t ends from YACs in the pRML vector from the Whitehead Institute/MIT-820 mouse YAC library and from B
15 g 619 loci (with 121 loci in common with the Whitehead map and 498 additional loci), providing especi
16 pply this method to the leukemia data of the Whitehead/MIT group that attempts to differentially diag
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