ライフサイエンスコーパス: whites

1 .001) more than the top 50% of non-Hispanic whites).

2 (Mexican Americans), and 6-10% (non-Hispanic whites).

3 s and odds ratio, 1.06; 95% CI, 0.80-1.41 in whites).

4 (for clinical history, n=126 blacks, n=1262 whites).

5 served among ethnic groups (Blacks>Hispanics>Whites).

6 n alcohol consumption traits in non-Hispanic whites.

7 3% (P trend=0.021) lower in 2013 relative to whites.

8 ms (SNPs) in 6 genes/loci for keratoconus in Whites.

9 ion to HCTZ or chlorthalidone BP response in whites.

10 spanics, and lower than what is reported for whites.

11 3% (P trend=0.040) lower in 2013 relative to whites.

12 (P trend=0.047) greater in 2013 relative to whites.

13 53 mutations and fewer PIK3CA mutations than whites.

14 greater (P trend=0.004) in 2013 relative to whites.

15 not differ significantly between blacks and whites.

16 ks had a significantly lower death risk than whites.

17 1) of being diagnosed with KCN compared with whites.

18 of mixed ethnicities, followed by blacks and whites.

19 stributions of risk and events in blacks and whites.

20 1) of being diagnosed with KCN compared with whites.

21 cially for blacks compared with non-Hispanic whites.

22 ) for low-risk blacks, and 3.9 (3.1-4.8) for whites.

23 e in African American men when compared with whites.

24 p were distinct but most similar to Hispanic whites.

25 ionately fewer racial/ethnic minorities than whites.

26 er inpatient mortality in blacks compared to whites.

27 y transplantations (LDKTs) than non-Hispanic whites.

28 likely to be treated with home dialysis than whites.

29 % CI, 1.07-1.51]) compared with non-Hispanic whites.

30 y treatment delay compared with non-Hispanic whites.

31 pressure, respectively, in GERA non-Hispanic whites.

32 22.2% for blacks and from 20.8% to 13.9% for whites.

33 lower risk of colorectal cancer-mostly among whites.

34 four-fold the same ratio among non-Hispanic whites.

35 ger age and because of different causes than whites.

36 eGFRcys decline over 9.3 years compared with whites.

37 ll racial/ethnic minority groups compared to whites.

38 , 0.57-1.18) was associated with CVD risk in whites.

39 hort was 54 years, with 61% females, and 56% whites.

40 s (RR = 0.63, 95% CI: 0.41, 0.97) than among whites.

41 r outcomes in hypertensive blacks but not in whites.

42 8 (95% CI, 2.01 to 5.05) in New Mexico among whites.

43 o-Asians with ALF was worse when compared to whites.

44 significantly higher in blacks compared with whites.

45 o compare the proportions between blacks and whites.

46 ng CVD deaths in non-Hispanic blacks than in whites.

47 rates of hypertension control compared with whites.

48 Americans, Japanese Americans, Latinos, and whites.

49 se, and Filipino) compared with non-Hispanic whites.

50 heart disease (CHD) mortality compared with whites.

51 blacks, Hispanics, and Asians compared with whites.

52 , African Americans have outcomes similar to Whites.

53 xperience greater poststroke disability than whites.

54 and 13 mg/dL (95% CI, 11-16) lower LDL-C in whites.

55 CI, 1.04 to 1.54) compared with non-Hispanic whites.

56 ported in a large population of asymptomatic whites.

57 2) in blacks and 0.82 (95% CI, 0.63-1.06) in whites.

58 nder patients had the highest, compared with whites.

59 ong nonwhites and 19 percentage points among whites.

60 ted with a survival advantage for Blacks and Whites.

61 ife expectancy between African Americans and whites.

62 s TOPS 0.21 [95% CI, -0.18 to 0.60] and PLRS whites 0.27 [95% CI, -0.04 to 0.58]).

63 ersus TOPS 0.23 [95% CI, 0.15-0.31] and PLRS whites 0.60 [95% CI, 0.33-0.87]).

64 Risk allele frequency in whites (0.43) was greater than in Chinese (0.24), transl

65 idence of type 2 diabetes among non-Hispanic whites (0.6%) was lower than that among non-Hispanic bla

66 , 95% CI [-0.28, -0.12], p < 0.001) than for whites (-0.09 SD/decade, 95% CI [-0.16, -0.02], p = 0.00

67 40%, 1 in 250) but varied by race/ethnicity (whites: 0.40%, 1 in 249; blacks: 0.47%, 1 in 211; Mexica

68 orbidity rates were higher in blacks than in whites (1.8% versus 2.5%, P<0.0001) and (13.6% versus 19

69 [1.48 (1.11, 1.98); P-trend = 0.002] but not whites [1.17 (0.96, 1.43); P-trend = 0.17] after adjustm

70 interval (CI): 1.15, 1.44) for non-Hispanic whites, 1.67 (95% CI: 1.20, 2.34) for non-Hispanic black

71 16 elderly renal transplant recipients (all whites; 10 men; mean age, 64 +/- 2 years (61-71 years),

72 red markedly by race (blacks=52/38/10 versus whites=10/44/46, P<0.001).

73 s sex balanced and included 39% non-Hispanic whites, 12% Chinese Americans, 28% African Americans, an

74 .4%; P<0.001), African Americans compared to whites (13.3% vs. 8.8%; P=0.010), and underinsured/ unin

75 d whole eggs (18 g protein, 17 g fat) or egg whites (18 g protein, 0 g fat).

76 nonischemic dilated cardiomyopathy in 27.5% (whites, 19.9%; P<0.001).

77 e United States, involving 2434 non-Hispanic whites, 1919 non-Hispanic blacks, and 1919 Mexican Ameri

78 th TMA, but nonwhites had more variants than whites (2.5 [range, 0-7] vs 0 [range, 0-2]; P < .0001).

79 e, among men, in Norway (2.9% per annum), US whites (2.6%), and Canada (2.4%) and, among women, in th

80 In whites, 2-fold higher TMAO associated with higher risk (

81 (1.0-2.2) ml/min per 1.73 m(2) per year for whites, 2.1 (1.4-3.1) ml/min per 1.73 m(2) per year for

82 ence was 5.0% in men, 4.4% in women, 5.6% in whites, 2.7% in Hispanics, 0.6% in blacks, and 2.1% in o

83 ricans (45 %) followed by Asians (22 %), and whites (20 %).

84 n than for men (10.4% vs. 3.7%), blacks than whites (21.7% vs. 6.9%), and, although associations were

85 [CI] 12.3%-19.0%) compared with non-Hispanic whites (22.3%, 95% CI 19.1%-25.7%) and Mexican Americans

86 296 decedents: 147 Blacks, 64 Hispanics, 49 Whites, 22 Asians, and 14 mixed ethnicities; 142 infants

87 ber of days from IPCC to death compared with whites (25 v 17 days; P = .006), and were more likely th

88 ticipants (mean age: 62+/-10; 52% women; 39% whites; 27% blacks; 20% Hispanics; 12% Chinese Americans

89 nts with AF, 390,590 (85%) were non-Hispanic whites, 31,702 (7%) were non-Hispanic African Americans,

90 0001) and Hispanics (46.9%, P<0.0001) versus whites (32.7%).

91 of MAN was 21.0% (95% CI, 18.4% to 23.9%) in whites, 32.2% (CI, 27.3% to 37.4%) in Chinese Americans,

92 Among normal-weight participants (n = 846 whites, 323 Chinese Americans, 334 African Americans, 25

93 se was the most common cause of cirrhosis in whites (38.2%), while hepatitis C virus was the most com

94 o 72%; P < .001 for trend), as well as among whites (39% to 86%; P < .001 for trend) and nonwhites (3

95 ater among Hispanics than among non-Hispanic whites (4.2% vs. 1.2%, P<0.001).

96 d a nonsignificantly higher rate of SMI than whites (4.45 versus 3.69 per 1000-person years; P=0.217)

97 d age were 21.4% in mixed ethnicities, 10.2% Whites, 4.5% Asians, 3.1% Hispanics, and 2% Blacks; 7.7%

98 .012) and in African Americans compared with whites (41.1% [215 of 518 participants] vs 32.4% [422 of

99 Analyses were restricted to blacks and whites 45 to 64 years of age without CHD who were not ta

100 have sex with men (58% to 69%), non-Hispanic whites (48% to 64%), and all age groups with the excepti

101 h incidence of early and late AMD highest in whites (5.3% and 4.1%, respectively), intermediate in Ch

102 30.6% for blacks and from 37.3% to 25.0% for whites; 5-year graft loss after LDKT improved from 37.4%

103 NoACE: 8.69% vs. 7.74%; p = 0.05) but not in whites (6.40% vs. 6.74%; p = 0.37).

104 LOS was 6.3 days and significantly longer in whites (6.5 d) than both blacks (5.4 d) and Hispanics (5

105 an Americans and Native Hawaiians to 4.1% in whites, 6.7% in Latinos, and 6.9% in Japanese.

106 ingestion of whole eggs (68% +/- 1%) and egg whites (66% +/- 2%), with no difference in whole-body ne

107 hree thousand three hundred six non-Hispanic whites (67%) were compared with 1612 non-Hispanic black

108 1) and for blacks compared with non-Hispanic whites (7.0% vs 3.0%; HR, 2.55 [95% CI, 2.17-3.01]; P <

109 ir mean age was 55, 61% were women, all were whites, 74% had previous treatment preference discussion

110 rtery was marginally lower in blacks than in whites (93.3% versus 92.2%, P<0.0001).

111 ICH risk in blacks and Hispanics relative to whites across all anatomic locations of ICH.

112 associated with lower metabolic syndrome for whites across all levels of childhood disadvantage.

113 ient mortality was lower for blacks than for whites (adj. OR = 0.50; 95 % CI = 0.32-0.78).

114 cs had similar mortality rates compared with whites (adjusted hazard ratio, 0.92; 95% CI, 0.76-1.11 a

115 had lower total 25(OH)D concentrations than whites [adjusted median: 20.3 ng/mL (95% CI: 16.2, 24.5

116 concentrations were lower in blacks than in whites [adjusted median: 4.5 ng/mL (95% CI: 3.7, 5.4 ng/

117 Compared with whites, African Americans had a greater likelihood of re

118 comes for African Americans as compared with Whites after 12 major surgical procedures across multipl

119 Hawaiians, Japanese Americans, Latinos, and whites aged 45 to 75 years at recruitment.

120 n Asians/Pacific Islanders than non-Hispanic whites (aIRR = 2.09); after induction immunosuppression

121 imilar associations in African Americans and whites (all ps < 0.03).

122 Compared with whites, all racial/ethnic minority groups had a statisti

123 However, in the subgroup of non-Hispanic whites, allele frequencies at the TMCO1 locus were stati

124 c minorities had a lower risk for death than whites; among individuals undergoing home HD, only black

125 dy (GWAS) of PSA in 28,503 Kaiser Permanente whites and 17,428 men from replication cohorts.

126 and for current smoking, 12% (7%-16%) among whites and 18% (11%-22%) among blacks.

127 ; for diabetes mellitus, 14% (10%-20%) among whites and 31% (28%-41%) among blacks; and for current s

128 DM).The longitudinal cohort consisted of 609 whites and 339 blacks who had BMI and fasting insulin me

129 onses (PEAR) (50 whites) and from PEAR-2 (50 whites and 50 blacks).

130 Among women with stage III disease, 74% of whites and 56% of blacks received trastuzumab.

131 ted MRSA incidence was 4.59 per 100000 among whites and 7.60 per 100000 among blacks (rate ratio [RR]

132 llows: for hypertension, 49% (45%-58%) among whites and 72% (68%-78%) among blacks; for diabetes mell

133 erall IPBR rate or disparity in IPBR between whites and African Americans (reduction of 1 percentage

134 adenocarcinoma rate rose among non-Hispanic whites and among black men.

135 Whites and Asians lived more years at lower BMI status l

136 ignificantly higher among Hispanics than for whites and blacks.

137 MetS severity seems highly heritable among whites and blacks.

138 TMAO concentration did not differ between whites and blacks.

139 d events are distributed among blacks versus whites and how interventions reduce racial disparities.

140 lent in the west Southeast and Midwest among whites and in the north among blacks.

141 This pattern contrasts starkly with whites and minorities from more privileged backgrounds,

142 0 degrees C, de novo formation dominates for whites and roses and MeSH.

143 with moderate to high melanoma incidence (US whites and the populations of the United Kingdom, Sweden

144 ion of Antihypertensive Responses (PEAR) (50 whites) and from PEAR-2 (50 whites and 50 blacks).

145 in American (Hispanics, African-American and Whites) and Mediterranean populations.

146 ase (851 events in blacks and 2662 events in whites) and stroke (523 events in blacks and 1660 events

147 re at >/=7.5% risk of CVD (30% versus 19% in whites), and an intervention that targeted multiple risk

148 2 times higher in black Africans compared to whites, and 1.4 times higher in males compared with fema

149 sians, 45.3% Hispanics, 44.20% Blacks, 36.7% Whites, and 14.3% in mixed ethnicities.

150 (both groups), 70% and 72% of patients were whites, and 19% were African American (both groups), res

151 is the most frequently implicated HPA among whites, and a single Leu33Pro amino acid polymorphism wi

152 Between 1982 and 2011, melanoma rates in US whites, and the populations of the United Kingdom, Swede

153 xplain 9.5% of PSA variation in non-Hispanic whites, and the remaining GWAS SNPs explain an additiona

154 ad poorer hypertension control compared with whites, and these differences were more pronounced in yo

155 70% lower risk of developing early AMD than whites, and this decreased only slightly to a 67% lower

156 In US whites, annual new cases will rise from around 70,000 in

157 ssociated with an increased risk of iGAS for whites (aRR = 3.47; 95% confidence interval [CI], 3.00-4

158 rom narrowing differences between blacks and whites as opposed to narrowing differences between black

159 s 112/69 mm Hg in blacks and 109/68 mm Hg in whites at Y0 and 117/77 mm Hg in blacks and 110/72 mm Hg

160 d 117/77 mm Hg in blacks and 110/72 mm Hg in whites at Y15.

161 wide significant association in non-Hispanic whites between the previously reported SNP rs1229984 in

162 In contrast to whites, black smokers prefer menthol cigarettes over non

163 Compared with whites, blacks (OR, 0.89; 95% CI, 0.86-0.92) and Latinos

164 Compared with non-Hispanic whites, blacks had a higher incidence of PTCL not otherw

165 Relative to whites, blacks undergoing coronary artery bypass surgery

166 Compared with whites, blacks were >6 years younger at the time of SCA

167 uring the study period, all 3 racial groups (whites, blacks, and Hispanics) experienced substantial i

168 multicenter, case-control study of ICH among whites, blacks, and Hispanics.

169 The cohort included non-Hispanic whites, blacks, Hispanics, and Asians.

170 of cadmium, lead, mercury, and arsenic than whites, blacks, Mexican Americans, and other Hispanics i

171 of cadmium, lead, mercury, and arsenic than whites, blacks, Mexican Americans, and other Hispanics i

172 -mortality hazard ratios similar to those of whites but higher hazard ratios for cardiovascular death

173 lent in the west and central Southeast among whites but in south Florida among blacks.

174 were higher than, or comparable to those of, whites, but differences were not significant.

175 ost prevalent in the central Southeast among whites, but in the west Southeast among blacks.

176 ly to withdraw life-supportive measures than whites, but that this disparity may be absent in patient

177 faster yearly eGFRcys decline compared with whites, but this difference was attenuated after adjustm

178 In whites, CaSR gene SNP rs1801725 was associated with seru

179 era of hepatitis C virus infection and among whites/Caucasians.

180 S, the new FSRP was a better predictor among whites compared with blacks.

181 n females, and was higher among non-Hispanic whites compared with non-Hispanic blacks or Hispanics.

182 tein synthesis than did the ingestion of egg whites, despite being matched for protein content in you

183 bility variants was higher in blacks than in whites (difference, 0.24; P = 2.3 x 10-5), while the est

184 risk score was much higher in blacks than in whites (difference, 0.48; P = 2.8 x 10-11).

185 after the consumption of whole eggs with egg whites during exercise recovery in young men.In crossove

186 ut biological differences between blacks and whites (e.g., "black people's skin is thicker than white

187 se (CHD) using data from 8,937 US blacks and whites enrolled during 2003-2007 in a prospective cohort

188 rimination salient increased the accuracy of Whites' estimates of Black-White economic equality, wher

189 Among whites, FFIs (change = 0.15; Ptrend < 0.0001) and NFIs (

190 was significantly higher for Hispanics than whites (fold increase = 1.25; 95 % CI = 1.01-1.49).

191 n Americans experienced poorer survival than whites for all cancers, and the racial difference decrea

192 in black and Asian females to match that in whites for vaccination; cervical screening in women who

193 tic regression models among 759 non-Hispanic whites from a case-control study of patients seen betwee

194 at 12% increased risk versus Hispanics, and whites had a 30% higher mortality versus non-white minor

195 us 3.69 per 1000-person years; P=0.217), but whites had higher rate of CMI than blacks (5.04 versus 3

196 Blacks, compared with whites, have an increased risk of progression to end-sta

197 s had a higher risk of tumor recurrence than whites (hazard ratio, 2.22; 95% CI, 1.05 to 4.67).

198 For whites, higher white segregation was associated with low

199 in four race/ethnicity groups: non-Hispanic whites, Hispanic/Latinos, East Asians and African Americ

200 verty across the neighborhoods where blacks, whites, Hispanics, and Asians reside.

201 tion with a vegan diet remained only for the whites (HR: 0.63; 95% CI: 0.46, 0.86), but the multivari

202 enrolled 5,115 healthy African Americans and whites in 1985 and 1986 (baseline).

203 tions remained 37% higher for nonwhites than whites in 2010 (40% higher in 2000), and nonwhites were

204 higher NO2 concentrations for nonwhites than whites in 2010.

205 omen, 240% (P trend=0.725) with reference to whites in 2013 with no significant change between 2002 a

206 d of receiving palliative care compared with whites in any hospital stratum, but the odds of palliati

207 dence and CHD case-fatality among blacks and whites in the Atherosclerosis Risk in Communities study

208 rvival outcome difference between blacks and whites in The Cancer Genome Atlas data set.

209 Compared with blacks in the same quintile, whites in the highest quintile for TMAO (>/=135 muM) had

210 Hawaiians, Japanese Americans, Latinos, and whites in the Multiethnic Cohort.

211 ) death rates are much higher in blacks than whites in the United States.

212 Hawaiians, Japanese Americans, Latinos, and whites in the US Multiethnic Cohort (MEC).

213 Ovalbumin, a major allergen in egg whites, is prone to aggregate upon heating.

214 Compared with whites, Latinos had a 46% decreased hazard of developing

215 and lowest NSES = 2.4 points; P = 0.004) and whites (mean difference = 0.7 points; P = 0.02) at basel

216 y Composition participants (48.5% men; 59.6% whites; mean age, 73.6+/-2.9 years), 111 developed incid

217 The 2 largest subgroups, non-Hispanic whites (n = 752; 70.7%) and blacks (n = 249, 23.7%), als

218 tore sources by race/ethnicity [non-Hispanic whites (NHWs), non-Hispanic blacks (NHBs), and Hispanic

219 Compared with non-Hispanic whites, non-Hispanic blacks had higher seroprevalence of

220 d the difference between low-risk blacks and whites (odds ratio 1.21, 95% confidence interval 0.86-1.

221 d higher odds of developing dysglycemia than whites (odds ratio [95% CI]: Blacks 1.24 [1.09-1.40]; Hi

222 nts had 21% lower odds of receiving DAA than whites (odds ratio [OR] = 0.79; 95% confidence interval

223 t risk factor across all locations of ICH in whites (odds ratio [OR], 1.57; 95% confidence interval [

224 Blacks were more likely to die than whites (odds ratio=1.33; 95% confidence interval: 1.21-1

225 any events in blacks [n = 140] compared with whites), of which 131 (87 in blacks) occurred after Y15.

226 weight, length, or head circumference among whites or Hispanics.

227 and anti-HEV seropositivity in non-Hispanic whites or Mexican Americans.

228 antly lower odds of receiving treatment than whites (OR = 0.74, 95% CI, .69-.79) in the previous trea

229 1.73, 2.24; P < 0.001) than in non-Hispanic whites (OR = 1.54, 95% CI: 1.25, 1.91; P < 0.001) and bl

230 n between myopia and POAG among non-Hispanic whites (OR, 1.12; 95% CI, 1.11-1.13) and NTG among Asian

231 R, 1.17; 95% CI, 1.15-1.20) and non-Hispanic whites (OR, 1.19; 95% CI, 1.15-1.22).

232 l/ethnic groups across all locations of ICH: whites (OR, 8.79; 95% CI, 5.66-13.66; P<0.0001), blacks

233 OR=5.90, CI=1.89-25.96) than in non-Hispanic whites (OR=2.10 CI= 0.69-7.13).

234 .8) at increased risk for regret relative to whites ( P = .001 across all categories).

235 ics were more likely to be hospitalized than whites (P < 0.001), adjusting for hospitalization did no

236 CCs that were p16+ (or ISH+) increased among whites (P = .04 for trend) but not among nonwhites (each

237 .05 (95% confidence interval: 0.73, 1.52) in whites (P for interaction = 0.08).

238 greater extent than did the ingestion of egg whites (P= 0.04).We show that the ingestion of whole egg

239 rall sample; P=0.27 and 0.05 in Non-Hispanic Whites) persisted after additional adjustment for change

240 receiving no glaucoma testing compared with whites possessing commercial health insurance (OR = 2.98

241 ls have a lower risk of hearing loss than do whites, possibly because of differences in cochlear mela

242 y from several smoking-related diseases than whites, raising the possibility that menthol cigarettes

243 es (rate ratio, 1.26; 95% CI, 1.25 to 1.27), whites (rate ratio, 1.72; 95% CI, 1.56 to 2.0), and pers

244 Since 1989, whites receive on average 36% more callbacks than Africa

245 re encountered between African Americans and Whites receiving surgery at hospitals administered by th

246 ion but similar mortality when compared with whites regardless of digoxin use.

247 00,000 in blacks, and 6.1 and 4.0/100,000 in whites, respectively.

248 .015) in the overall sample and Non-Hispanic Whites, respectively.

249 , and Spanish-speaking Latinas compared with whites, respectively.

250 f 219), and 5.5% (12 of 219) of non-Hispanic whites, respectively; 50.0% (2 of 4), 0 (0 of 4), and 0

251 f 4), 0 (0 of 4), and 0 (0 of 4) of Hispanic whites, respectively; and 38.5% (5 of 13), 38.5% (5 of 1

252 assessment of vitamin D status in blacks and whites should follow a single standard.

253 prior assessment in California non-Hispanic whites showed substantial increases in invasive melanoma

254 CRC incidence between African Americans and whites since the late 1980s.

255 er in racial/ethnic minorities compared with whites (survey linear regression, log beta [95% CI]: bla

256 were more rapid after the consumption of egg whites than after whole eggs (P = 0.01).

257 ds of choroidal nevus were 10-fold higher in whites than in blacks, 5-fold higher in Hispanics than i

258 others than in blacks, and 2-fold higher in whites than in Hispanics.

259 However, among Whites the association was linear and graded whereas amo

260 Among whites, the adjusted HR for diabetes corresponding to ea

261 ent MAN prevalence at a BMI of 25.0 kg/m2 in whites, the corresponding BMI values were 22.9 kg/m2 (CI

262 sk of glaucoma developing among non-Hispanic whites, the largest racial subgroup in the OHTS cohort,

263 Compared with whites, the odds ratio (95% confidence interval) for inc

264 In non-Hispanic whites, the presence of at least 1 perianal nevus was si

265 White economic equality, whereas encouraging Whites to anchor their estimates on their own circumstan

266 Blacks and Hispanics were more likely than whites to be hospitalized for circulatory system or endo

267 est cohort, blacks were 39% more likely than whites to experience 5-year graft loss (adjusted hazard

268 est cohort, blacks were 53% more likely than whites to experience 5-year graft loss (aHR, 1.53; 95% C

269 ncerting because blacks are more likely than whites to go blind from OAG and there are disproportiona

270 later life, blacks are also more likely than whites to have experienced the death of a child and of a

271 at blacks are significantly more likely than whites to have experienced the death of a mother, a fath

272 ut biological differences between blacks and whites to inform medical judgments, which may contribute

273 nd nonwhites were 2.5 times more likely than whites to live in a block group with an average NO2 conc

274 han Hispanics (59% v 41%; P = .006), but not whites, to be referred to hospice.

275 Compared with non-Hispanic whites, TP was more likely in Hispanics (adjusted [a]PR

276 ecologies, and individuals' inferences about whites track stereotypes of people from hopeful ecologie

277 rsus CT+CC=0.53+0.09, P=0.02) but evident in whites (TT=0.50++0.11; CT+CC=0.56+0.06, P=0.04).

278 edural outcomes in 11,697 blacks and 136,362 whites undergoing isolated coronary artery bypass surger

279 r others undergoing PD had a lower risk than whites undergoing PD.

280 cans, Asians/Pacific Islanders, and Hispanic whites was approximately three- to four-fold the same ra

281 s, the excess risk of death in blacks versus whites was nonsignificant and not affected by adjustment

282 justed hazard ratios comparing blacks versus whites were 2.61 (95% confidence interval, 1.57-4.34) an

283 Men and nonHispanic whites were most likely to have unfavorable response pro

284 reas disparities between low-risk blacks and whites were related to differences in traditional risk f

285 oke (523 events in blacks and 1660 events in whites) were calculated using pooled Mantel-Haenszel est

286 (blacks, Asians, Hispanics, and non-Hispanic whites) were targeted for recruitment.

287 adjusted odds of adherence than non-Hispanic whites when they initiated AET therapy with tamoxifen (O

288 ypertensive blacks have poorer outcomes than whites when treated with an ACE inhibitor-based regimen.

289 ests poorer outcomes in blacks compared with whites when treated with an angiotensin-converting enzym

290 nocarcinoma is more frequent in non-Hispanic whites, whereas esophageal squamous cell carcinoma with

291 dults younger than 65 years and non-Hispanic whites, whereas the prevalence of reduced GFR increased

292 much higher rates of ESRD than HIV-positive whites, which could be attributed to the APOL1 renal ris

293 sive tumor biology in African Americans than whites, which could contribute to racial disparity in br

294 Odds ratio 0.74 CI95% 0.58-0.94) compared to Whites whilst those with greater educational levels were

295 ticipants randomized, 1702 were non-Hispanic whites who completed the trial and had genotype data for

296 at least 1 Y142X or C679X variant and 31 306 whites with 955 (3.1%) having at least 1 R46L variant.

297 in eGFR over time among 1331 blacks and 1476 whites with CKD.

298 After adjustment for confounders, whites with OAG enrolled in Medicaid had 198% higher odd

299 icts black Americans, who when compared with whites with wild-type transthyretin amyloidosis, a pheno

300 uently arising on the head and neck in older whites, with a poorer prognosis.