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1 try, and bronchoconstriction was assessed by whole body plethysmography.
2 HR to methacholine challenge was measured by whole-body plethysmography.
3 in 7- to 9-week-old unanaesthetized rats via whole-body plethysmography.
4 a, and severe airway hyperreactivity through whole-body plethysmography.
5 Minute ventilation (VE) was measured using whole-body plethysmography.
6 Ventilation was measured by whole-body plethysmography.
7 exposures by monitoring their sniffing with whole-body plethysmography.
8 medullary respiratory circuits, we performed whole-body plethysmography and electrophysiological reco
10 -old intact, freely behaving rat pups, using whole-body plethysmography during breathing of room air
12 respiratory frequency (RF) was monitored by whole-body plethysmography immediately after the 4th cha
13 olume, and minute volume were measured using whole-body plethysmography in rats administered GHB.
14 of AR to inhaled methacholine by barometric whole-body plethysmography is a valid indicator of airwa
17 ompared with airway resistance measured with whole-body plethysmography (Raw-p) in 10 of the 16 infan
20 yper-reactivity was measured by non-invasive whole-body plethysmography, Th2 response and airway infl
21 ext]e was recorded in newborn rat pups using whole-body plethysmography under normoxic and hypoxic co
22 s to inhaled methacholine were determined by whole body plethysmography using changes in enhanced pau
23 ventilation) or noninvasive techniques, like whole body plethysmography (WBP), assesses the severity
24 e models of pathology is the need to combine whole-body plethysmography (WBP) to measure respiration
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