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1 d cells (down to 3 out of approximately 1000 wild-type cells).
2 increase in vancomycin resistance, even in a wild-type cell.
3 strain germinated significantly faster than wild type cells.
4 ted to the airways by the chemokine CXCL2 as wild type cells.
5 but the CL/MLCL ratio is similar to that of wild type cells.
6 ayed greatly reduced migration compared with wild type cells.
7 ontrast to the atheroprotective phenotype of wild type cells.
8 in the tsetse flies at rates comparable with wild-type cells.
9 bose) polymerase (PARP) 1(-/-) compared with wild-type cells.
10 inflammatory cytokine production compared to wild-type cells.
11 rway epithelia with varying ratios of CF and wild-type cells.
12 in the smc6 mutant is similar to that in the wild-type cells.
13 pensable for robust H3 eviction in otherwise wild-type cells.
14 have longer glycans in their PG relative to wild-type cells.
15 -catenin inhibition blocked this response in wild-type cells.
16 5 degrees C that is essentially identical to wild-type cells.
17 lipid mutant sta1 from a mixture of sta1 and wild-type cells.
18 n are coupled events during SPB formation in wild-type cells.
19 in nup116 mutants and increased longevity in wild-type cells.
20 telomere sequence was different from that of wild-type cells.
21 only 1% of the mutant cells in a mixture of wild-type cells.
22 sis and cell death not seen in KRAS and BRAF wild-type cells.
23 y in PTEN-deficient cells in comparison with wild-type cells.
24 more sensitive to MET inhibitor SU11274 than wild-type cells.
25 and had reduced proliferation compared with wild-type cells.
26 amma-glutamylcysteine synthetase relative to wild-type cells.
27 ses H3K9 methylation from tethering sites in wild-type cells.
28 ion start sites becomes broader than that in wild-type cells.
29 ecreased levels of p65 protein compared with wild-type cells.
30 d accumulation of SERCA levels compared with wild-type cells.
31 ATPase proton transport in inositol-deprived wild-type cells.
32 ndogenous Ags were more readily deleted than wild-type cells.
33 kines in response to TLR stimulation than do wild-type cells.
34 R)-induced, mutations than similarly treated wild-type cells.
35 more acidic phagolysosomal compartments than wild-type cells.
36 crete more proinflammatory cytokines than do wild-type cells.
37 in of Num1, an event that is not observed in wild-type cells.
38 nd breaks, and cell death compared with IDH1 wild-type cells.
39 nse granules in Ctr2(-/-) mast cells than in wild-type cells.
40 tivate the Th2 pathway in vitro than similar wild-type cells.
41 mediators in response to infection than did wild-type cells.
42 s with the timing of DraRnl replenishment in wild-type cells.
43 ore ROS in the presence of DOX compared with wild-type cells.
44 ing the fine and mesh-like MT network in the wild-type cells.
45 rexpression of the Arg/N-end rule pathway in wild-type cells.
46 on similar to those seen with virus-infected wild-type cells.
47 ndent introns are splicing-defective also in wild-type cells.
48 fungal load after fluconazole challenge than wild-type cells.
49 nesis deficiency when 3xAsp was expressed in wild-type cells.
50 depth of ingression of the endocytic pit in wild-type cells.
51 Ubp3 have a shorter life span than isogenic wild-type cells.
52 -2, Blimp-1 induction, and SLEC formation in wild-type cells.
53 Wnt5a perturbed polarization of neighboring wild-type cells.
54 mPtch1 was similarly induced in both mes and wild-type cells.
55 otations at a higher level than transplanted wild-type cells.
56 us proliferation signal from the neighboring wild-type cells.
57 oalesce into stable clusters, in contrast to wild-type cells.
58 the absence of HELLS are similar to those in wild-type cells.
59 mation, and restored growth close to that of wild-type cells.
60 nt LDLR shed from HEK293 SimpleCells and CHO wild-type cells.
61 izes to subdistal appendages by immuno-EM in wild-type cells.
62 Bs and chromosomal aberrations compared with wild-type cells.
63 roduced less IFN-gamma, IL-5, and IL-13 than wild-type cells.
64 phagocytosis of this pathogen compared with wild-type cells.
65 s prematurely in G1 in mutant cells, but not wild-type cells.
66 resent in supernatants from stationary-phase wild-type cells.
67 ells glided more than twice as frequently as wild-type cells.
68 er cells compared with those of the parental wild-type cells.
69 pidly in cells lacking Cdc7 function than in wild-type cells.
70 responses that are elevated in comparison to wild-type cells.
71 derwent reduced apoptosis in comparison with wild-type cells.
72 rvations in pH taxis for various mutants and wild-type cells.
73 produce almost the same diastolic INCX as in wild-type cells.
74 ow large deviations from what is observed in wild-type cells.
75 n and caspase activation induced in adjacent wild-type cells.
76 levels of AngII in the mutant compared with wild-type cells.
77 n2 is constantly localized to the nucleus in wild-type cells.
78 tical to keeping incorporation levels low in wild-type cells.
79 arable to levels exhibited by ST-246-treated wild-type cells.
80 Rgamma expression in knock-out cells than in wild-type cells.
81 PY2R undergo signal-dependent ectocytosis in wild-type cells.
82 sion of Rec8 is sufficient to trigger UPD in wild-type cells.
83 increased median elastic modulus compared to wild-type cells.
84 SMARCA4/BRG1 mutant but not of SMARCA4/BRG1 wild-type cells.
85 n, similar to those induced by DNA damage in wild-type cells.
86 herichia coli and is essential for growth in wild-type cells.
87 4me2/3 levels observed at their promoters in wild-type cells.
88 Mek1 is excluded from synapsed homologues in wild-type cells.
89 R22beta(-/-) Sertoli cells moved faster than wild-type cells.
90 in expression to levels similar to analogous wild-type cells.
91 er apoptosis in Tsc2-deficient cells but not wild-type cells.
92 pported more efficient SVNI replication than wild-type cells.
93 of the N3 mutant was comparable with that of wild-type cells.
94 ivated protein kinase (MAPK) pathway in BRAF wild-type cells.
95 in under shear flow conditions compared with wild-type cells.
96 in the rate of exchange across the locus in wild-type cells.
97 ling was 50% lower in HD cells compared with wild-type cells.
98 ) BMDM long after they were downregulated in wild-type cells.
99 he surface before departing was the same for wild-type cells (12 s) and pilus-minus mutant cells (13
101 Moreover, reversible adhesion events in wild-type cells (6.8 events/min) occur twice as frequent
102 in TbRFT1 null parasites when compared with wild-type cells, a defect that is corrected by expressin
105 ller in deletion mutants for Tol-Pal than in wild-type cells, although it is still larger than would
106 equency, in competition with matrix mutants, wild-type cells always increase in relative abundance in
107 increased in mutant Hdh cells compared with wild type cells and in 3-nitropropionic acid-treated pri
108 ecifically, we assume that fitness values of wild type cells and mutants at different locations come
109 ion of galactose residues on the exterior of wild type cells and their absence in the DeltaepsE mutan
110 herwise lethal disease more efficiently than wild-type cells and bypassed the requirement for interle
111 by comparing genome-wide DNA repair rates in wild-type cells and cells defective in the global genome
112 acterize the magnetosome arrangement in both wild-type cells and DeltamamJ mutants, which exhibit dif
113 y in an HA-expressing cell line, it infected wild-type cells and expressed both viral proteins and Ps
114 ely accounts for the kinetics of recovery of wild-type cells and for the nature and severity of the e
115 cent D-amino acids (FDAAs) were performed in wild-type cells and in cells in which Pbp2x activity was
116 Interestingly, enhancement occurs both in wild-type cells and in cells lacking either the p53 tumo
117 y pericentromere stretch during metaphase in wild-type cells and in mutants with disrupted chromosome
118 tely accounts for the variable phenotypes of wild-type cells and more than 20 mutant yeast strains si
119 ells, thiol levels were similar to untreated wild-type cells and not significantly depleted by AS-HK0
120 false alarm and miss error probabilities in wild-type cells and provide a formulation which shows ho
121 nothione were identified in AS-HK014-exposed wild-type cells and reproduced by chemical reaction.
122 icked by the fusion inhibitor chloroquine in wild-type cells and rescued by expression of Munc13-4 bu
123 largest 102-kbp genomic island was lethal to wild-type cells and resulted in a reduction of up to 2.5
124 m hole and ring are circular and centered in wild-type cells and that in the absence of a functional
125 ustained higher levels of WNV infection than wild-type cells and that this difference was greater und
126 eshold to predetermine methylation levels in wild-type cells and the magnitude of methylation reducti
127 eted strain exhibits more robust growth than wild-type cells and the shift in translation from polyso
128 proteins TraK and TraB were extremely low in wild-type cells and were undetectable by Western blottin
129 can be significantly affected, compared to a wild-type cell, and the method is able to model and meas
130 cerevisiae and found that 1-3% of all ECs in wild-type cells, and 5-7% of all ECs in cells lacking pr
131 ed p53-dependent G1 cell-cycle arrest in p53 wild-type cells, and a p53-independent pathway impaired
132 Fe(II) efflux is physiologically relevant in wild-type cells, and null mutants accumulate elevated le
133 ial drug susceptibilities of KRAS mutant and wild-type cells, and predict relapse based on increased
134 ast (MEF) cells as compared with the control wild-type cells, and the proliferative advantage of the
135 nt to UVB-triggered cell death compared with wild-type cells, and tumor necrosis factor-alpha release
137 amycin do not fully inhibit proliferation of wild-type cells, and we found that the residual prolifer
138 h was found at both enhancer and promoter in wild-type cells, appeared to have been replaced by H3K27
140 evidence that most spontaneous SCE events in wild-type cells are not due to the repair of DNA double-
143 ransporters (i.e. xCT, LAT1, and y(+)LAT2 in wild-type cells) are crucial to control reactive oxygen
144 ich are normally not substrates for PAP I in wild-type cells, are rapidly polyadenylated as PAP I lev
145 exhibit a strongly impaired SOCE compared to wild-type cells as a result of reduced calcium release a
146 mitotic defects (and to the same extent) in wild-type cells as observed in unchallenged HR(-) cells.
147 hemotaxing Dictyostelium cells, and examined wild-type cells as well as mutants with defects in contr
150 in rings at approximately 90% of the rate of wild-type cells at 30 degrees C and 36 degrees C, sugges
151 icroglia lacking CR3 are more efficient than wild-type cells at degrading extracellular Abeta by secr
154 re normally weakly coupled to one another in wild-type cells become strongly synchronized following a
155 ate and bile, and in competition assays with wild-type cells both in vitro and in the infant mouse sm
156 both Tcon and Treg cell function compared to wild-type cells but disproportionally affected Treg cell
157 d phosphorylated eEF-2 cycle in abundance in wild-type cells but not in cells deleted for OS-2 or the
159 ed as that caused by extensive DNA damage in wild-type cells but showed genetic dependencies distinct
160 of Hsp90, was degraded relatively slowly in wild-type cells but was rapidly destroyed in naa10Delta
161 of NF-kappaB dynamics that is unaltered from wild-type cells, but activation of the TNFalpha promoter
162 (IGF-1R) are suppressed by excess glucose in wild-type cells, but increased in GM3S (-/-) KCs with su
163 at SeV PI can arise from infection of normal wild-type cells, but only if they can find a way to impa
164 that TLR4 activation inhibits growth of TP53 wild-type cells, but promotes growth of TP53 mutant brea
165 hat peroxisomes form de novo continuously in wild-type cells by heterotypic fusion of endoplasmic ret
166 , both of which are dynamically modulated in wild-type cells by TOR kinase activity and the presence
169 nsitive to compaction, that interaction with wild-type cells causes their compaction and that crowdin
170 cells elicited a dominant-negative effect in wild-type cells, causing paralyzed short flagella with h
172 eater in acetate-grown versus methanol-grown wild-type cells, consistent with the previously publishe
176 we also find that tRNA thiolation levels in wild-type cells decrease when cells are grown at elevate
178 Gpc4 hypomorphic cell grafts, in contrast to wild-type cells, did not generate teratomas in the host
179 gly, isogenic lines of Ctip-S326A mutant and wild-type cells displayed comparable levels of HDR funct
180 ng IgV R-loops by RNase HI overexpression in wild-type cells does not affect IgV diversification, sho
182 se of Ssb from ribosomes is also observed in wild-type cells during growth in poor synthetic medium.
183 that H2O2 exposure caused bacteriostasis in wild-type cells during which time SCVs appeared spontane
185 d it is Gaussian-like for immotile bacteria, wild-type cells exhibit anomalous non-Gaussian deviation
186 o osteoclasts was similar in TRAF6[L74H] and wild-type cells, explaining why the bone structure and t
189 e but migrate significantly more slowly than wild-type cells; expressing Flag-AbpG in mutant cells el
190 S-deprived cells and dies more rapidly than wild-type cells following exposure to S-, phosphorus-, o
191 information about endocytic actin patches in wild-type cells from measurements of the fluorescence of
194 reduced levels similar to those observed in wild-type cells grown planktonically rather than as biof
197 educed survival in neutrophils compared with wild type cells, highlighting the importance of D-lactat
198 CP2-HepG2 cells compared with those found in wild-type cells in addition to the transport data indica
200 st-translational modification, we first grew wild-type cells in buffered tryptone broth with glucose
201 In support of this model, resuspension of wild-type cells in conditioned medium from DeltabsrA cul
204 r vesicles that did not differ from those of wild-type cells in quantity, surface molecule expression
205 ll cells transform phenotypes of neighboring wild-type cells in vivo in such manner that they become
206 had enhanced effector function compared with wild-type cells, including increased production of IL-2
207 in CypA knockdown chondrogenic cells than in wild-type cells, indicating that CypA plays a functional
208 d lipid accumulation similar to those of the wild-type cells, indicating that DEX-bound GR accelerate
209 markably enhanced adipogenesis compared with wild-type cells, indicating that FABP4 regulates adipoge
210 er cisternae per Golgi apparatus relative to wild-type cells, indicative of protein trafficking defec
214 Inhibition of let-7a using anti-miRNA in wild type cells is sufficient to enhance the expression
215 5alpha is present within the mitochondria in wild-type cells, it is instead located mostly outside in
216 ed a perinuclear pattern in undifferentiated wild-type cells, it predominantly localized to the nucle
219 romatid cohesion in STAG2 mutated but not in wild-type cells leading to mitotic catastrophe, defectiv
220 complex presenilin 1 from Tsc1-null cells to wild-type cells leading to the activation of Notch and R
221 athways in the stem cell compartment and how wild-type cells limit the proliferation of mutant cells
222 subsequent reanalysis of the remaining TP53 wild type cell lines clearly demonstrated that unfortuna
225 cell lines would be more sensitive than BRAF wild-type cell lines to three MEK1/2 inhibitors tested.
229 th STAT1 and STAT3 were activated by mOSM in wild type cells or by mLIF/hOSM in wild type and Osmr(-/
231 n active hippo pathway signaling compared to wild-type cells or cells lacking both Mst1 and Mst2.
233 lamydomonas reinhardtii that, in contrast to wild-type cells placed under conditions of nitrogen depr
234 se regulations within the heterogeneity of a wild-type cell population growing in optimal conditions.
236 Finally, overexpressing the RQ-mutant in wild type cells produced no effect on either docking or
237 emporal expression of this locus observed in wild-type cells promotes initiation of early biofilm for
238 nt romaine lettuce, the proportion of viable wild-type cells recovered from plants relative to that o
239 r show that KRAS-mutated cells, but not KRAS wild-type cells, rely on the alt-NHEJ repair pathway on
240 K induction, the isogenic KRAS mutant versus wild-type cells remained resistant to GDC-0623-induced a
243 trated that the smaller realignment angle of wild-type cells results in the higher directional persis
245 tion rates in TSC1/2-deficient cells, unlike wild-type cells, sensitizes these cells to endoplasmic r
246 on in lamin-A/C-deficient cells, whereas the wild-type cells show much less plastic deformation.
247 rved in smtA and smtB mutants, DeltasmtA and wild-type cells showed a similar 9,Me-GlcCer content, re
248 as effective in cisplatin-resistant cells as wild-type cells, signifying that they circumvent cisplat
251 erties of pRb-deficient cells are similar to wild-type cells suggesting there may be processes that c
252 longest cell-cell communication distance in wild-type cells, suggesting that criticality provides an
253 a level equivalent to or even higher than in wild-type cells, suggesting that the four hypersensitive
254 and reach a higher maximum cell density than wild type cells, tend to be multinucleate, accumulate no
256 cells was indistinguishable from that of the wild-type cells, the mutant did exhibit reduced chemotax
259 reintroduced by CRISPR-Cas9 gene editing of wild-type cells, these mutations reversed both ISRIB-med
260 an AML cells were more sensitive than IDH1/2 wild-type cells to ABT-199, a highly specific BCL-2 inhi
264 in-mutated cells required higher forces than wild-type cells to reach high indentation depths, where
265 ners, and we propose that this enables E1 in wild-type cells to selectively activate ubiquitin protei
266 ut did slow the replication fork movement of wild-type cells to the same level than in HR(-) cells.
268 , endogenous ATF6 was markedly stabilized in wild-type cells treated with kifunensine, an inhibitor o
274 were higher in L110R MM-1alpha cells than in wild-type cells under normal conditions and were increas
276 but only the PKBR1 activity was increased in wild-type cells under the equivalent conditions, indicat
279 bolomics revealed a broad metabolic shift in wild type cells upon biotin withdrawal which was blunted
281 more intense unfolded protein response than wild-type cells upon treatment with inducers of this pat
282 units co-immunoprecipitated with V-ATPase in wild-type cells; upon deletion of one subunit, the other
283 eated Ewing sarcoma cells were compared with wild-type cells using an isobaric mass-tag quantitative
287 more abundant in dcl3 mutant strains than in wild-type cells were not due to sRNA-targeted RNA degrad
288 ls showed a dependency on PDHK4 whereas KRAS wild-type cells were significantly resistant to PDHK4 kn
289 m hypocotyls with reduced CP are longer than wild-type cells, whereas CP OX lines have shorter cells.
290 ent B cells egress from PPs more slowly than wild-type cells, whereas CXCR5-deficient cells egress mo
291 creased sensitivity to ER stress relative to wild-type cells, which could be restored by proline or t
294 lided at approximately half the frequency of wild-type cells, while prpC mutant cells glided more tha
295 VACV-infected TRAF2(-/-) MEFs, treatment of wild-type cells with a JNK inhibitor did not affect viru
297 tive cell realignments of about 90 degrees , wild-type cells with secondary filaments exhibited a ran
299 in vivo cassette-inversion method that marks wild-type cells with the endogenous EGFP-tagged protein,
300 t of genes that were essential for growth in wild-type cells yet dispensable when pbp1a was deleted.
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