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1 during the dark in both the Crx(-/-) and the wild type mouse.
2 c, NPS 2143, inhibited secretion only in the wild-type mouse.
3 hereas a blunted response is observed in the wild-type mouse.
4 en fluorescent protein (GFP) (delta neo) and wild-type mouse.
5 gp91(phox-/-)) mice as much as it was in the wild-type mouse.
6 that are between 9 and 40% of the Gclm(+/+) wild-type mouse.
7 t mouse was approximately double that of the wild-type mouse.
8 conditions in which it had no effect in the wild-type mouse.
9 0-fold lower in the mutant mouse than in the wild-type mouse.
10 the ADG to the levels found in the HG of the wild-type mouse.
11 from Osx-expressing cells isolated from the wild-type mouse.
12 m-operated site and the sites in the injured wild-type mouse.
13 ansplantation of skin from a transgenic to a wild-type mouse.
14 hages decreased with age at 18 months in the wild-type mouse.
16 tants ( approximately 14 fold) over those of wild-type mouse AChE, indicating that these residues may
18 GAD67-GFP (Deltaneo) mouse compared with the wild type mouse after prolonged exposure to kainate.
20 eral lines of transgenic mice overexpressing wild-type mouse amyloid precursor protein (moAPP) either
22 oteolytically inactive thrombin can activate wild-type mouse and human platelets after treatment with
23 tiserum to the C-terminus (CT-2) labels both wild-type mouse and human Purkinje cell nuclei, but not
25 G1alpha disulfide formation was increased in wild-type mouse aortas by in vivo nitroglycerin treatmen
27 iated with homogeneous sized alpha-HDL, when wild-type mouse apoA-I was present (in TgDelta6 +/+ and
29 cess, we determined the crystal structure of wild-type mouse apoE, which, like apoE4, forms a four-he
30 dy used a beta(3) integrin (-/-) mouse and a wild-type mouse as a control for in vivo PO by transvers
35 0.5 microg/ml human G117H BChE, 2 microg/ml wild-type mouse BChE, and 0.06 microg/ml wild-type mouse
37 on cathepsin K expression in the MOCP-5 and wild-type mouse bone marrow co-culture systems by Northe
38 duce the formation of PrP(Sc) molecules from wild type mouse brain homogenate substrate in serial pro
41 nerating regions of pcd mouse brain, but not wild-type mouse brain, show elevated autophagy, which ca
42 these peptides, designated WE-15, exists in wild-type mouse brain, thus validating the approach to i
46 mceph mouse brains are enlarged and - unlike wild-type mouse brains - they keep growing throughout ad
47 significantly decreased in BACE1-null versus wild-type mouse brains, remaining unchanged in BACE1-het
51 mensional structure of myosin filaments from wild-type mouse cardiac muscle and from a MyBP-C knockou
54 yo fibroblasts from Fbxw7-deficient mice, or wild-type mouse cells expressing Fbxw7 small interfering
55 d3(-/-) mouse cells migrate more slowly than wild-type mouse cells, a change in cell behaviour that p
57 roblasts with kinetics equivalent to that of wild-type mouse CMV, did not negatively affect replicati
59 ation of Angptl4 and Adrp mRNA expression in wild-type mouse colon but not in Pparbeta/delta-null mou
60 expression patterns for Cdx-2 and PTEN along wild-type mouse colon, as well as in colon tumors occurr
61 napA strain showed normal (like that of the wild type) mouse colonization efficiency in the conventi
63 alization and enhanced cell proliferation of wild-type mouse corneal epithelial cells in an organ cul
65 lantation and naked MT1-MMP DNA injection in wild-type mouse corneas compared with either bFGF pellet
66 l electrophoresis were present in mutant and wild-type mouse cortex and hypothalamus at comparable le
67 the only two sites basally phosphorylated in wild-type mouse cTnI with full sequence coverage, which
69 ouse-derived brown adipocytes, compared with wild-type mouse-derived brown adipocytes, displayed an i
71 rs analysed DNA binding affinity data of the wild-type mouse EGR1 protein and four variants differing
72 for such cleavage, it is shown that, whereas wild type mouse embryo fibroblasts (MEFs) produce cleave
73 a superfamily) induce p19(Arf) expression in wild type mouse embryo fibroblasts (MEFs), and they enha
74 1,25(OH)(2)D(3) induces p27 accumulation in wild type mouse embryo fibroblasts and arrests wild type
75 essing of endogenous pro-alpha1(V) chains by wild type mouse embryo fibroblasts and by fibroblasts de
76 B1 is similar to that occurring in infected wild-type mouse embryo fibroblast cells but the levels o
77 UV light inhibits translation of IkappaB in wild-type mouse embryo fibroblasts (MEF(S/S)) and that t
80 results in accelerated TRAF2 degradation in wild-type mouse embryo fibroblasts (MEFs), as compared w
83 induced nuclear Nrf2 and HO-1 effectively in wild-type mouse embryo fibroblasts (wt MEFs) and in B-Ra
85 roteins, DNA, and mRNA and virus yields from wild-type mouse embryo fibroblasts and sibling cells lac
88 p53 knockout mice, which was not the case in wild-type mouse embryo fibroblasts, suggesting that p53
93 overexpression of miR-199 or miR-214 in the wild-type mouse embryonic brains was sufficient to distu
95 properties of 26S proteasomes purified from wild-type mouse embryonic fibroblast cells and those lac
97 ssion in tuberous sclerosis complex 2 (TSC2) wild-type mouse embryonic fibroblasts (MEF) but not in T
98 s virus led to higher levels of autophagy in wild-type mouse embryonic fibroblasts (MEF) than in RNas
102 hat veliparib enhanced the effects of CPT in wild-type mouse embryonic fibroblasts (MEFs) but not Par
107 in human umbilical vein endothelial cells or wild-type mouse embryonic fibroblasts because of an inte
108 AP 2, Bcl-x(L), Bfl-1/A1, TRAF1, and FLIP in wild-type mouse embryonic fibroblasts but not in GSK-3be
109 minantly to the nuclear compartment, whereas wild-type mouse embryonic fibroblasts localize survivin
111 ormation and anchorage-independent growth in wild-type mouse embryonic fibroblasts, and that these an
112 paB activation as measured by DNA binding in wild-type mouse embryonic fibroblasts, but deletion of G
114 NF-kappaB and JNK are activated by LIGHT in wild-type mouse embryonic fibroblasts, no activation of
117 re of cystogenesis was assessed in explanted wild-type mouse embryonic metanephroi, using 8-Br-cAMP a
122 Finally, palatal shelves from prefusion wild-type mouse embryos cultured in the presence of a sy
126 rom endothelin receptor A (EdnrA) mutant and wild-type mouse embryos, we identified Dlx6, a member of
128 to alphaAsp-206-Arg-231, reversibly inhibits wild-type mouse ENaCs expressed in Xenopus oocytes, as w
129 vo, miR-exon4 was most highly amplified from wild-type mouse enamel organs at the secretory stage.
130 Interestingly, increasing cytosolic GSH in wild-type mouse endothelial cells decreased constitutive
131 CR1 on their erythrocytes to the results for wild-type mouse erythrocytes that do not express CR1.
133 Thus, 5-aza-dC induces Dnmt1 degradation in wild-type mouse ES cells, but not in Dnmt [3a(-/-), 3b(-
136 y in situ hybridization using wholemounts of wild-type mouse eyes and by immunofluorescence staining
137 eal injection of TRPM1-positive MAR IgG into wild-type mouse eyes, and the appearance of the IgG in t
138 Using cultured neurospheres from PPT1-KO and wild-type mouse fetuses, we further demonstrate that the
140 was observed in p53-deficient but not in p53 wild-type mouse fibroblasts treated with the DNA demethy
144 rrelated with FLVCR1 expression, we examined wild-type mouse Flvcr1 mRNA levels in the posterior colu
148 rticle tracking to study the mobility of the wild-type mouse H-2L(d) class I MHC molecule and of seve
149 derwent transplantation heterotopically with wild-type mouse heart expressing alpha-gal epitopes and
151 -ND hearts was at a level similar to that of wild type mouse hearts under beta-adrenergic stimulation
152 omyocyte telomere length decrease sharply in wild-type mouse hearts after birth, resulting in cardiom
153 e performed RNA Seq analysis of AE3-null and wild-type mouse hearts and evaluated the data with respe
155 iac damage and impaired function relative to wild-type mouse hearts following ischemia reperfusion.
156 g a 20 min ischemic period in transgenic and wild-type mouse hearts was assessed using an ex vivo wor
160 Furthermore, human apoE was resecreted from wild-type mouse hepatocytes after a pulse with human VLD
161 mma in vitro inhibited cell proliferation of wild-type mouse hepatocytes, but not STAT1(-/-) hepatocy
163 alpha (PGC1alpha), to the Adam10 promoter in wild-type mouse hippocampal neurons and shifted APP proc
166 gh dose of recombinant human ADAMTS13 into a wild-type mouse immediately before reperfusion reduces i
167 transplantation of a D(5)(-/-) kidney into a wild-type mouse increased the expression of both, sugges
172 Glucose normally does not produce TDP in wild-type mouse islets except under forced intracellular
173 ibited and compared with the TDP response in wild-type mouse islets with and without forced intracell
179 CS expression were analyzed in transgenic or wild-type mouse lens and lens epithelial cells stimulate
180 Appropriate controls were used, including wild-type mouse lens, scrambled oligonucleotides, and a
184 use hepatocytes were incubated (pulsed) with wild-type mouse lipoproteins, and cells and media were c
186 as well as in Smad4, and their reduction to wild-type mouse liver levels in AHR -/- mice fed the ret
187 ibody inhibited RA 4-hydroxylase activity of wild-type mouse liver microsomes to the levels of AHR-nu
189 Livers of SPTBN1(+/-) mice, compared to wild-type mouse livers, display a significant increase i
191 hallenge, Siglec-F ligands were increased in wild-type mouse lungs but less so in St3gal3 mutants, wh
195 affects mammary tumorigenesis, we generated wild-type mouse mammary tumor virus/polyoma middle-T (WT
196 e found that a primary stable cell line from wild-type mouse marrow cells expressing BCR-ABL caused s
198 rotein-PACSIN2 were concentrated in midstage wild-type mouse MKs in a well-defined invagination of th
199 ATP-facilitated sIPSCs were also recorded in wild-type mouse MNTB neurones, but were absent in the MN
200 ing oligomers and the mThy-1 alpha-synuclein wild-type mouse model (Line 61), which accumulates vario
201 duced gastric intraepithelial neoplasia in a wild-type mouse model and show no additive effect of hig
209 We found that prior AICAR stimulation of wild-type mouse muscle increases insulin sensitivity to
210 of mutated human MYOC prevented secretion of wild-type mouse Myoc but did not dramatically affect sec
213 study, Calhm1 knockout, Panx1 knockout, and wild-type mouse nasal septal epithelial cells were grown
216 to the kinetochores of polar chromosomes of wild-type mouse oocytes, in which polar chromosomes are
217 ely restored after the addition of exogenous wild-type mouse or human alpha-synuclein, but not by A30
218 34-deficient platelets, ex vivo treatment of wild-type mouse or human platelets with the Vps34-specif
220 rative transcriptomic analysis of mutant and wild-type mouse organs revealed lipid metabolic changes
222 hepatocytes, we transplanted suspensions of wild-type mouse pancreatic cells into syngeneic recipien
223 rhodopsin in native membranes obtained from wild-type mouse photoreceptors and opsin isolated from p
224 he 50 known targets from the knockout versus wild-type mouse plasma, class-specific precursor/fragmen
226 LTC4 induced surface expression of CD62P by wild-type mouse platelets in platelet-rich plasma (PRP)
228 Deposition patterns of untreated human and wild-type mouse platelets were consistent with random di
235 iPS-RPE cells compared with that of isolated wild-type mouse primary RPE (mpRPE) cells in vitro and i
239 tion (mouse- equivalent) abolished FRET with wild-type mouse PrP, whereas mutant PrP-P101L displayed
243 zol-induced seizures in postnatal day 7 (P7) wild-type mouse pups and that transgenic knock-in mice h
244 cles containing the mouse opsin promoter and wild-type mouse Rds gene were injected subretinally into
245 mmary gland explants into cleared fat pad of wild type mouse recipients indicates that the observed a
247 oft agar can be reverted by re-expression of wild-type mouse RelA, but not by expression of RelA muta
253 of the total photoreceptor population in the wild-type mouse retina, we generated mouse lines with CN
254 of the total photoreceptor population in the wild-type mouse retina, we used Cngb3(-/-)/Nrl(-/-) mice
256 ARPE-19 and primary RPE cells isolated from wild type mouse retinas from H(2)O(2)-induced cell death
257 e pik3r1 KO mice was slower than that in the wild-type mouse retinas at 5 minutes of exposure to ligh
258 sgene in a line that was hemizygous null for wild-type mouse rhodopsin (mrho(+/-)), and the eyes of t
259 emission, 457 or 540 nm) appears in frog and wild-type mouse rod outer segments reaching a maximum in
262 th a time constant of approximately 40 ms in wild-type mouse rods, much faster than previous estimate
264 RyR2 cDNA were constructed, one encoding the wild type mouse RyR2 (RyR2(wt)) and the other encoding m
266 ferocytosis) is bolstered in the presence of wild-type mouse serum, through the C3 deposition on the
268 was suppressed in TNF-alpha(-/-) compared to wild-type mouse skin in response to the tumour promotor
269 (VD3) and/or all-trans retinoic acid (RA) on wild-type mouse skin induces a human atopic dermatitis-l
270 ted PKCalpha and other kinases were lower in wild-type mouse skin treated with 12-O-tetradecanoylphor
271 nflammation in both PPAR-gamma-deficient and wild-type mouse skin, indicating that the inhibition of
272 ditional pairs of highly branched SGs, or in wild-type mouse skin, K6a expression consistently coinci
278 rimary cortical neurons from mice expressing wild-type mouse SNCA, wild-type human SNCA, or mutant A5
280 ntiation of monocyte-derived fibrocytes from wild-type mouse spleen cells, but not from SIGN-R1(-/-)
281 he vast majority of exons harboring m(6)A in wild-type mouse stem cells is spliced the same in cells
282 c segregation of afferents, we now show in a wild-type mouse strain that the visual pathway can be su
284 Here we report the observed changes for two wild type mouse strains commonly used in transgenic stud
288 ven by a BAC transgene containing the entire wild-type mouse tau locus, including the endogenous prom
290 n of the axons in the corpus callosum from a wild-type mouse to a knockout mouse showed that myelin i
292 nd RT-PCR experiments of NKCC1-deficient and wild type mouse transcriptomes, we confirmed the absence
295 e determine the plasma clearance rate in the wild-type mouse, using time-resolved fluorescence on a s
297 essed in the basolateral membrane of the RPE wild-type mouse, was expressed at very low levels in bot
298 of target transcripts in RNA extracted from wild-type mouse whole eyes or retinas between embryonic
299 ssion profile of the rod-dominated retina of wild type mouse with that of the cone-only retina of Nrl
300 HCN1/HCN2/HCN3/HCN4 = 9:9:1:89 in OC of the wild-type mouse, with HCN4 protein primarily attributabl
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