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1 cally has not resulted in control of spotted wilt.
2 Ceratocystis fagacearum, causal agent of oak wilt.
3 liae, a fungal pathogen causing Verticillium wilt.
4 cular pathogens use many mechanisms to cause wilt.
5 the stems of tomato plants just beginning to wilt.
6 na rustica) stomata did not until the leaves wilted.
7 raulic failure and carbon starvation in tree wilting.
8 ite isoprene emission decreasing before leaf wilting.
9 pecies a visible sign of petal senescence is wilting.
11 ed drought tolerance demonstrated by delayed wilting after watering was ceased and quicker and better
13 of cotton genotypes tolerant to Verticillium wilt and was induced early and strongly by inoculation w
14 ense and SlFRK3-RNAi lines exhibited similar wilting and anatomical effects, confirming that these ef
18 examined further to determine the cause for wilting, and thus better understand how the anionic pero
22 lect melon cultivars to avoid melon Fusarium wilt, but also to monitor how quickly a Fom population c
26 zed the published data for stomatal closure, wilting, declines in hydraulic conductivity in the leave
27 c viruses, and on the incidence of bacterial wilt disease (a fatal disease vectored by cucumber beetl
29 Erwinia tracheiphila - which causes a fatal wilt disease - alters the foliar and floral volatile emi
31 ccurrence, severity, and symptoms of spotted wilt disease are highly variable from season to season,
33 in plant xylem vessels and causes bacterial wilt disease despite the low nutrient content of xylem s
38 8 y the number of plants dying from a sudden wilt disease has increased, leading to crop failure.
39 l-borne fungal pathogen that causes vascular wilt disease in many economically important crops worldw
43 lstonia solanacearum, which causes bacterial wilt disease of many plant species, produces several ext
44 quitous fungal pathogen that causes vascular wilt disease on a wide range of plant species and can pr
47 chus xylophilus, is the causal agent of pine wilt disease that has devastated pine forests in Asia.
49 od nematode and its vector beetle cause pine wilt disease, which threatens forest ecosystems world-wi
54 trains responsible for the various bacterial wilt diseases has in recent years led to the concept of
55 fungal pathogen responsible for devastating wilt diseases in many crops) cotton plants increase prod
56 s a multitude of strains that cause vascular wilt diseases of economically important crops throughout
59 d the hypothesis that oxalate induces foliar wilting during fungal infection by manipulating guard ce
61 to race 1 strains of the soil-borne vascular wilt fungi Verticillium dahliae and Verticillium albo-at
62 sequencing of a set of strains of the melon wilt fungus Fusarium oxysporum f. sp. melonis (Fom), bio
64 sequencing of a set of strains of the melon wilt fungus Fusarium oxysporum f.sp. melonis (Fom), bioi
66 he plant-infecting bunyavirus Tomato spotted wilt, Gc localizes at endoplasmic reticulum (ER) membran
68 leaf abscission zones when the leaves become wilted in response to limited water and HAE continues to
69 of cer9 are associated with delayed onset of wilting in plants experiencing water deficit, lower tran
71 genetic basis of resistance to Verticillium wilt is unknown in most crops, as are the subcellular si
72 than in control plants, thus indicating that wilting is a consequence of peroxidase expression in the
74 ed with glycinebetaine accumulation, include wilting, loss of chlorophyll, and increase in thiobarbit
83 icating that gene(s) important for bacterial wilt pathogenesis were interrupted by the IVET insertion
90 g the leaf and stem hydraulic traits and the wilting point, or turgor loss point, beyond those expect
92 cific suppression to four diseases, Fusarium wilts, potato scab, apple replant disease, and take-all,
93 se activity also decrease the rate of flower wilting, promote the rooting of cuttings, and facilitate
101 de novo genome and plastome assemblies for a wilt-resistant South African accession of Mentha longifo
104 nly of broad host-range pathogens that cause wilt, rot, and blackleg diseases on a wide range of plan
105 y, irNaMPK4 plants transpired more water and wilted sooner than did wild-type plants when they were d
109 th putrescine before inoculation accelerated wilt symptom development and R. solanacearum growth and
112 cess NH4+, low pH, salinity, osmotic stress, wilting) to induce substantial increases in Put in plant
114 production of several virulence factors and wilted tomato plants several days more slowly than the w
117 t overproduce the tobacco anionic peroxidase wilt upon reaching maturity, although having functional
118 its most important contribution to bacterial wilt virulence in the early stages of host plant invasio
119 nonstructural protein NSs of tomato spotted wilt virus (a plant-infecting bunyavirus), the interfero
120 es are described not only for Tomato spotted wilt virus (TSWV) in pepper and tomato but also for othe
121 baci is the primary vector of Tomato spotted wilt virus (TSWV) in some areas of the world, it is not
122 ilt caused by thrips-vectored tomato spotted wilt virus (TSWV) is a very serious problem in peanut (A
127 e we show that infection with Tomato spotted wilt virus (TSWV), type member of the only plant-infecti
128 yellow spot virus (IYSV) and Tomato spotted wilt virus (TSWV), were investigated for inter-virus int
129 The movement protein VP37 of broad bean wilt virus 2 (BBWV 2) forms tubules in the plasmodesmata
130 Y, tobacco mosaic virus, and tomato spotted wilt virus were mapped in two or more genera and did not
132 s of the osm1 mutant also were more prone to wilting when grown with limited soil moisture compared w
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