ライフサイエンスコーパス: wind

1 here identified as the Central American Gap Winds.

2 s, corresponding to cell loss at the primary wound.

3 nd back of the cells on the two sides of the wound.

4 hold temperature associated with an infected wound.

5 ce of the Sun's hot atmosphere and the solar wind acceleration continues to be an outstanding problem

6 n the abilities to prevent infection of burn wound, aid healing, and an anti-inflammatory dressing ma

7 The variable and nondispatchable nature of wind and solar generation has been driving interest in e

8 Our multicriteria assessment of wind and solar potential for large regions of Africa sho

9 from this natural resource is comparable to wind and solar power, yet it does not suffer as much fro

10 ely purified from intact nerves and from the wound and distal regions of severed nerves.

11 in is compromised, bacteria can colonize the wound and impair wound healing.

12 field produces a magnetopause with the solar wind, and substantially increases their energy depositio

13 Given suitable temperature, humidity, wind, and water availability, HS-DAC can be largely pass

14 parture winds were predictive of future Gulf winds, and tested whether birds responded to predictabil

15 on cools tropical Africa and drives westerly wind anomalies in the Pacific favouring an El Nino respo

16 ure (SST) gradient and a mean easterly trade wind anomaly representing the multidecadal acceleration

17 g the multidecadal acceleration of the trade wind are both incorporated into the coupled model that e

18 roperties of cells surrounding a single-cell wound are investigated during closure of the defect.

19 lar matrix proteins are deposited within the wound area, resulting in persistent inflammation and res

20 est than at or above treeline because strong winds at high elevation reduced heating efficiency.

21 e from the low-level wind field via gradient wind balance.

22 nnective tissue matrix components within the wound beds compared to wounds treated with chitosan scaf

23 ch ES affects repair, microarray analysis of wound biopsy samples was performed on days 3, 7, 10, and

24 ults, and clinical outcome for foodborne and wound botulism patients confirmed by laboratory testing,

25 n favor of LC but with an increased perineal wound breakdown rate.

26 on that can result in delayed wound healing, wound breakdown, fistula formation, and compromised tiss

27 First, a stochastic parameterization of the wind bursts including both westerly and easterly winds i

28 temperature difference and further decrease winds by -0.06 (+/-0.05) m s(-1) averaged over eastern C

29 l fibroblasts are recruited into the clotted wound by a concentration gradient of platelet-derived gr

30 primarily driven by the Central American Gap Winds (CAGW), which intensify (weaken) during El Nino (L

31 The wind changes induce a subtropical North Pacific SST warm

32 tive variables (eg, age, comorbidities, ASA, wound classification), procedure type (eg, laparoscopic

33 ty, induced necroptosis, and delayed culture wound closing in three types of immortalized cancer cell

34 al precursor to collective cell migration in wound closure and cancer metastasis, respectively.

35 Here, we show that S. aureus inhibits wound closure and induces miR-15b-5p in acute human and

36 hCVAM) has been shown to effectively promote wound closure and reduce wound-related infections.

37 ble protein, leading to potent inhibition of wound closure following PMN-MP binding to IECs.

38 eting the fragment-5 region disrupted normal wound closure in both wild-type Hsp90alpha and Hsp90alph

39 Although rescue of epidermal wound closure in the absence of macrophages promotes bla

40 ng that CD301b-depleted mice exhibit delayed wound closure in vivo, which could be rescued by topical

41 After 20 weeks, wound closure occurred in 60 patients (48%) in the sucro

42 Although many reports suggest that wound closure rates depend on isolated cell speed and/or

43 Wound closure requires the activation of keratinocyte mi

44 Wound closure was clinically evaluated.

45 Delayed wound closure was, in part, attributable to damage of th

46 ffects of succinate-pretreated hMSC enhanced wound closure, vascularization and re-epithelialization

47 l innate immune contributor to IL-10-induced wound closure.

48 s during tissue repair, resulting in delayed wound closure.

49 was required for optimal intestinal mucosal wound closure.

50 Hsp90alpha as a potential driver for normal wound closure.

51 are developed with the aims to fight against wound colonization.

52 Superficial wound complication was the most common wound event, 2.24

53 5%, P = 0.26) rates favored LC with perineal wound complications (38.3% vs 50.0%, P = 0.26) in favor

54 Perineal wound complications frequently occur after eAPR with pre

55 ease in 30-day hospital readmission rates or wound complications when compared with discharge 1 or 2

56 and in response to this warming, the surface winds converge towards the subtropical North Pacific fro

57 Wind data derived from an assimilated meteorological dat

58 ge Mertk deficiency led to decreased cardiac wound debridement, increased infarct size, and depressed

59 ve either thunderstorm, hail and/or damaging wind decrease about 50% from 1961 to 2010.

60 meteorological conditions of wind speed and wind direction, data on geographic distance, mountains,

61 nificantly depending on the traffic rate and wind direction.

62 grees C) and cold (<10 degrees C) hours with wind directions parallel to and perpendicular downwind f

63 ungal spores transmitting the disease can be wind-dispersed over regions and even continents (1-11) .

64 done with a clear temporal or clear superior wound, does not affect intraocular pressure, bleb morpho

65 atio, 2.19; 95% CI, 1.00-4.82], performing a wound dressing [odds ratio, 8.35; 95% CI, 2.07-33.63] an

66 ctors of wound infection identified standard wound dressings as the only significant predictor of SSI

67 oward developing electronically controllable wound dressings that can deliver drugs with desired temp

68 of application, including tissue adhesives, wound dressings, and tissue repair.

69 high mass ( approximately 0.03 solar masses) wind-driven outflow with moderate electron fraction (Ye

70 o include patients of different gender, age, wound duration and type of surgery (general, vascular an

71 activation of Toll a few cell diameters from wound edges is reminiscent of local activation of Toll i

72 nvestigated the ability of components of the wound environment to modulate interactions between P. ae

73 requires dynamic cellular adaptation to the wound environment.

74 a2 in cultured keratinocytes in vitro and in wound epidermis in vivo.

75 ubtropical North Pacific SST warming through wind-evaporation-SST effect, and in response to this war

76 icial wound complication was the most common wound event, 2.24% in neoadjuvant-treated versus 2.45% i

77 .4 to 84.8% of sediment loss across all high wind events.

78 Here, we used a mouse wound excisional model to characterize the infection dyn

79 While no commercial-scale deep water wind farms yet exist, our results suggest that such tech

80 cit is highly predictable from the low-level wind field via gradient wind balance.

81 tes the rise of cytosolic calcium across the wound field.

82 malous surface-air temperature and low-level wind fields in the two polar regions that contribute to

83 TCPs of about 11 kDa are present in acute wound fluids as well as in fibrin sloughs from patients

84 grate from domain to domain with the carrier-wind force (electrical current).

85 ork suggests that current pulses and carrier-wind force could be external stimuli for domain engineer

86 position of the Southern Hemisphere westerly winds, forced deglaciation of this sector from at least

87 ow migrants to capitalise on more favourable winds further along the route, thus reducing energy expe

88 database are used here to estimate what the wind generated electricity in China would have been on a

89 ion of ocean stratification and near-surface winds, global warming contributes to an amplified surfac

90 Although the wound H2O2 gradient reaches deep into the tissue, it lik

91 with a concentration-dependent incidence of wound healing adverse events (WHAE).

92 Perineal wound healing after eAPR with preoperative radiotherapy

93 ferentiation, and migration and in epidermal wound healing and barrier repair.

94 ys, and cell behaviors to those activated in wound healing and identifies a repertoire of potential t

95 -derived exosomes may be involved in corneal wound healing and neovascularization, and thus, may serv

96 d for proteins involved in defense response, wound healing and protein phosphorylation when compared

97 his Review presents current understanding in wound healing and regeneration as two distinct aspects o

98 f FZD4 signaling on alveolar epithelial cell wound healing and repair, as well as on expression of el

99 ng axons, while in adult tissues they aid in wound healing and the maintenance of intestinal cell pop

100 anonical WNT-driven alveolar epithelial cell wound healing and transdifferentiation in vitro.

101 The wound healing assay reveals that inhibiting either BCL2L

102 All cell migration and wound healing assays are based on the inherent ability o

103 Furthermore, cell invasion and wound healing assays together with qRT-PCR determination

104 mputation-free survival by 18%, and improved wound healing by 59%, without affecting mortality.

105 sms of glucocorticoid (GC) downregulation of wound healing by interaction with the membrane bound GC

106 solving mediators of inflammation accelerate wound healing by preventing chronic inflammation and all

107 Wound healing complications were more prevalent in the b

108 point was the rate of uncomplicated perineal wound healing defined as a Southampton wound score of le

109 ay play a key role in tissue homeostasis and wound healing during Th2-mediated immune responses, such

110 Although the wound healing effects of nitric oxide (NO) are known, th

111 tions demonstrated clearance of bacteria and wound healing following repeated i.v. administration of

112 rosis and myofibroblast formation in corneal wound healing have not been fully elucidated.

113 gnized as a central clinical issue for RDEB, wound healing impairment has been only marginally invest

114 effects and mechanism of AS II on intestinal wound healing in both in vitro and in vivo models.

115 rove beneficial in the treatment of impaired wound healing in diabetes.

116 angiotensin system is implicated in abnormal wound healing in diabetic and older adults.

117 evaluating antiretroviral effects on genital wound healing in future clinical trials.

118 Here, using an in vivo model of cutaneous wound healing in mice, we provide evidence that GPNMB is

119 langiocyte cell line and demonstrated potent wound healing in mice.

120 blation of epidermal caspase-8 as a model of wound healing in Mus musculus, we analyzed the signaling

121 r occludin, down-regulation had no impact on wound healing in normal scratch assays, but after subjec

122 Compared to skin, wound healing in oral mucosa is faster and produces less

123 s the method can be readily applied to image wound healing in other injured or diseased tissues, or t

124 We concluded that corneal endothelial wound healing in rabbits has different outcomes dependin

125 elial cells (ATII) are instrumental in early wound healing in response to lung injury, restoring epit

126 c mucosa was sufficient to impair epithelial wound healing in vivo.

127 come was assessment of wound healing using a wound healing index (WHI).

128 Wound healing is a biological process directed towards r

129 Epithelial wound healing is an evolutionarily conserved process tha

130 IL-10-induced HA synthesis for regenerative wound healing is demonstrated by inhibition of HA synthe

131 hese findings are novel in understanding how wound healing is regulated.

132 Wound healing is significantly delayed in irradiated ski

133 ating TNFalpha and IL-1beta during the early wound healing phase and reduced inflammation by downregu

134 n by downregulating IL-1beta during the late wound healing phase.

135 othelial cellular traits associated with the wound healing process and may also be able to regulate t

136 s implies a potential regulatory role in the wound healing process and, thus highlights their potenti

137 the absence of LPA1 mitigates the epithelial wound healing process.

138 ion on CMR and increases in inflammation and wound healing proteins on post-MI day 7.

139 the mechanism by which NO modulates corneal wound healing remains unclear.

140 Lung fibrosis is an unabated wound healing response characterized by the loss and abe

141 and antiviral host defense during the normal wound healing response.

142 ted to stress responses and apoptosis at the wound healing stage, signaling pathways including Wnt an

143 Secondary outcome was assessment of wound healing using a wound healing index (WHI).

144 stress, which is normally present in wounds, wound healing was impaired.

145 ugh alpha3beta1 promotes this process during wound healing, alpha9beta1 has an inhibitory role, sugge

146 an impact allergic and autoimmune responses, wound healing, and anti-microbial defense.

147 elf-organization in biofilms, embryogenesis, wound healing, and cancer metastasis.

148 o been shown to favor embryonic development, wound healing, and even tumor growth, suggesting more co

149 Selective inhibition of cell adhesion, wound healing, and invasion are demonstrated; near-infra

150 T) is critical for embryonic development and wound healing, and occurs in fibrotic disease and carcin

151 revascularization are to relieve pain, help wound healing, and prevent limb loss.

152 During skin wound healing, CD26-positive cells accumulated over time

153 During the proliferative phase of cutaneous wound healing, dermal fibroblasts are recruited into the

154 through distinct, yet overlapping phases of wound healing, including hemostasis, inflammation, proli

155 on of gene expression, cellular homeostasis, wound healing, inflammation, allergy, autoimmunity, and

156 thy is characterized by proteins involved in wound healing, ongoing fibrosis, and inflammation.

157 ibrillogenesis, promoting corneal epithelial wound healing, regulating gene expression and maintainin

158 duction, the blinking reflex, and epithelial wound healing, resulting in loss of transparency and vis

159 cations, such as graft infection and delayed wound healing, were seen in 6 patients; 8 patients exper

160 HTS is the result of dysregulated wound healing, where excessive collagen and extracellula

161 whether Pseudomonas species directly impair wound healing, wild-type mice were infected with Pseudom

162 n resulted in significantly impaired scratch wound healing, with delayed migration and reduced prolif

163 ious complication that can result in delayed wound healing, wound breakdown, fistula formation, and c

164 (GFBLs) strongly regulated the expression of wound healing-related genes.

165 genesis of chronic inflammation, cancer, and wound healing.

166 s relevant to cancer, vascular diseases, and wound healing.

167 , bacteria can colonize the wound and impair wound healing.

168 Cre+) ) to determine the function of MLL1 in wound healing.

169 factors and conditions can lead to impaired wound healing.

170 host-pathogen defense, organ rejection, and wound healing.

171 did not interfere with skin development and wound healing.

172 erals (TM) play a role in skin integrity and wound healing.

173 lso reduced FPD development by promoting FPD wound healing.

174 are involved in developmental regulation and wound healing.

175 induced angiogenesis, especially in diabetic wound healing.

176 actor for osteoarthritis (OA) and diminished wound healing.

177 y a prolonged inflammatory phenotype in late wound healing.

178 in vivo for their ability to promote dermal wound healing.

179 the M2 polarization of macrophages in acute wound healing.

180 are shown to promote the corneal epithelial wound healing.

181 ation-related miRNAs with potential roles in wound healing.

182 ave the potential to induce angiogenesis and wound healing.

183 ession during papilloma induction and during wound healing.

184 ls and macrophages and play central roles in wound healing.

185 a key aspect of re-epithelialization during wound healing.

186 n regulating cellular traits associated with wound healing.

187 orly understood inputs for organogenesis and wound healing.

188 ion could promote fast and scarless gingival wound healing.

189 hort-lived, transplanted ASCs can accelerate wound-healing and reduce hair loss in acid-burn skin inj

190 sis and help delineate an altered profile in wound-healing markers during ONJ development.

191 ated to FHs promote tissue regeneration in a wound-healing model of mouse submandibular glands (mSMGs

192 The cutaneous wound-healing program is a product of a complex interpla

193 ent second messenger and master regulator of wound-healing programs, it is unknown what initiates the

194 lly, PTX3 mediated the hepatic stellate cell wound-healing response.

195 promote tissue regeneration in which classic wound-healing responses predominate.

196 ly, generating myofibroblasts and associated wound-healing responses.

197 (ASCs) accelerates the process of acid burn wound-healing.

198 ns of VOCs and O3 coincided with the surface wind, implying that the formation of O3 was impacted by

199 e same effect was observed for a single-cell wound induced by laser ablation and during closure of a

200 Robust activation of wound-induced transcription from ple and Ddc requires To

201 e a mouse model for investigating E faecalis wound infection determinants, and suggest that both immu

202 Analysis of predictors of wound infection identified standard wound dressings as t

203 Secondary endpoints were postoperative wound infection, intra-abdominal abscess, reoperation, l

204 ween the timing of surgery and postoperative wound infection, intra-abdominal abscess, reoperation, o

205 vent, prolonged hospital length of stay, and wound infection/dehiscence).

206 ith spontaneous chronic multi drug-resistant wound infections demonstrated clearance of bacteria and

207 lays an important role in sepsis, pneumonia, wound infections, and cystic fibrosis (CF), which is cau

208 coexist for long periods together in chronic wound infections.

209 re susceptible to horizontal transmission of wound infections.

210 spatial and temporal monitoring of potential wound infections.

211 hese two miRNAs are prominently expressed in wound-infiltrated neutrophils and macrophages and play c

212 ved to be the main driver to transport solar wind into the Earth's magnetosphere when the magnetopaus

213 bursts including both westerly and easterly winds is coupled to a simple ocean-atmosphere model that

214 n = 3; 6.8%), endophthalmitis (n = 1; 2.3%), wound leak (n = 1; 2.3%), and choroidal detachment (n =

215 of the epithelial NADPH oxidase Duox at the wound margin is required early during this response.

216 iers only within approximately 30 mum of the wound margin.

217 The loss of wound melanization in T. anophthalmus was an apomorphy a

218 The speed of wound melanization was uncorrelated with a difference in

219 smosis (LRO) and reverse osmosis (RO) spiral-wound membranes showed LRO membrane to be very efficient

220 his study provides evidence that the dynamic wound microbiome is indicative of clinical outcomes and

221 unreported temporal dynamics of the chronic wound microbiome.

222 ure to systemic antibiotics destabilized the wound microbiota, rather than altering overall diversity

223 ue, Scl1 adhesin specifically recognizes the wound microenvironment, promotes adhesion and biofilm fo

224 ed by inhibition of HA synthesis in a murine wound model by administering 4-methylumbelliferone.

225 Furthermore, in a skin excisional wound model, we found the effects of succinate-pretreate

226 nduces miR-15b-5p in acute human and porcine wound models and in chronic DFUs.

227 of non-healing wounds), in porcine and human wound models.

228 ite increasing knowledge about the role that wind, moisture availability and vegetation cover play in

229 direct relationship between the topological winding number of the spin texture and the polarized res

230 ical simulations to characterize the stellar wind of TRAPPIST-1 and the atmospheric ion escape rates

231 Natural sounds such as wind or rain, are characterized by the statistical occur

232 In contrast, external fertilisation and wind- or water-driven passive dispersal of gametes, or s

233 The correlation between PMGF and wind parameters was inconsistent in magnitude, direction

234 The magnetopause deflects the solar wind plasma and confines Earth's magnetic field.

235 The Poaceae family comprises over 12 000 wind-pollinated species, which release large amounts of

236 sustainable energy sources such as solar and wind power by storing the energy in liquid electrolytes.

237 ydropower-to another based on geothermal and wind power for approximately 90% of total capacity.

238 o provide flexibility to enable up to 50% of wind power penetration.

239 n account for the interannual variability of wind power, suggesting that advances in long-term foreca

240 entation of tissue-engineered constructs for wound regeneration, perhaps the most significant hurdle

241 a and in two slower (0.05 times light speed) wind regions.

242 effectively promote wound closure and reduce wound-related infections.

243 gulating macrophage-mediated inflammation in wound repair and identify a potential target for the tre

244 o cell adhesion regulation during intestinal wound repair and the development of IBD.

245 w this interaction influences the process of wound repair are not well understood.

246 versus pathogenic microbial interactions in wound repair is important.

247 oV pathogenesis, we have identified that the wound repair pathway, controlled by the epidermal growth

248 Skin wound repair requires a coordinated program of epithelia

249 The process of wound repair requires the coordinated participation of m

250 idelity" recreates a state akin to transient wound repair that persists to maintain uncontrolled grow

251 the role of each cell type in the process of wound repair, the nature of the dynamic interplay betwee

252 show that lineage plasticity is critical in wound repair, where it operates transiently to redirect

253 how they function in normal homeostasis and wound repair.

254 option of a morphogen-responsive function in wound repair.

255 linking innate immune activation to mucosal wound repair.

256 cellular responses are often required during wound repair.

257 s normally associated with tissue injury and wound repair.

258 ion in human acute wounds, thus accelerating wound repair.

259 n, can induce decorin expression and enhance wound repair.

260 iched for genes involved in inflammation and wound repair.

261 skeletal remodeling processes in single cell wound repair.

262 were found to exhibit a significant delay in wound repair.

263 rmittent energy sources, including solar and wind, require scalable, low-cost, multi-hour energy stor

264 arly in the morning, but they also depend on wind resources (i.e. uplifts) to subside flight which ar

265 , barrier repair requires activation of many wound response genes in epidermal cells surrounding woun

266 ility of prosystemin to trigger the systemic wound response in vivo.

267 during the activation of macrophage cells or wound response in vivo.

268 ineal wound healing defined as a Southampton wound score of less than 2 at 30 days postoperatively.

269 tion provides insight into the mechanisms of wound signaling in tomato.

270 ve distinct and specific regulatory roles in wound signalling and patterning to enable regeneration.

271 d repair, due to the paucity of cells at the wound site.

272 abscess formation at S. aureus-infected skin wound sites and were also more susceptible to horizontal

273 esponse genes in epidermal cells surrounding wound sites.

274 PUFAs were negatively correlated with OA and wound size, but positively correlated with adiponectin l

275 ayed calcium expansion event is predicted by wound size, indicating that a separate mechanism of calc

276 scales: the maximum azimuthal-mean azimuthal wind speed and half the product of the Coriolis paramete

277 information on meteorological conditions of wind speed and wind direction, data on geographic distan

278 between emission factors and aircraft-level wind speed or between methane and BC emission factors.

279 with distance and positively associated with wind speed, both of which were retained in models as a s

280 ogical information (pressure, precipitation, wind speed, dew point, and temperature) and self-monitor

281 l pressure deficit and the peak near-surface wind speed, is a long-standing problem in tropical meteo

282 imulations, that during years with decreased wind speed, large decreases in dust emissions (29%) mode

283 istorical economic damages than does maximum wind speed.

284 he electrical performance with variations in wind speed.

285 of precipitation, soil bareness, and surface winds speed.

286 sions of colocated or electrically proximate wind/storage and solar/storage facilities across the U.S

287 The current feedback on the surface wind stress modulates the air-sea transfer of momentum b

288 rface primary productivity, temperature, and wind stress.

289 ng photolysis and radiolysis of water, solar wind-surface interactions and gas-phase collisions.

290 We show that the galactic winds sustain turbulence in the 10-kiloparsec-scale envi

291 orbable (polydioxanone) suture material in a wound-suture ratio of minimum 1 : 4 was introduced in Ju

292 By using a stab wound telencephalic injury model, the impact of hypergly

293 enerative ability of adult brain, after stab wound telencephalic injury.

294 daptive wings for aircraft and drag-reducing wind turbines.

295 first quantitative estimate of global solar wind variations over the last 400 years.

296 ellite lidars measuring aerosol backscatter, wind vector, and CO2 concentration profiles may all prod

297 restoration strategies require knowledge of wind wave behavior in fetch and depth limited water as a

298 t model, calculated to what extent departure winds were predictive of future Gulf winds, and tested w

299 ly by a decrease in the strength of westerly winds, which has amplified the effects on temperature of

300 ds tend to associate with mild or supportive winds, whose speed and direction may change under global