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3 ntity of shedding were compared between strains and seasons with adjustment for age and prior LAIV (n = 436), inactivated
5 azard ratios of autism according to MMR vaccination status, with adjustment for age, birth year, sex, other childhood vac
6 e regression models were used to estimate odds ratios (ORs) with adjustment for age, gender, smoking, education, setting,
7 iptions was estimated using conditional logistic regression with adjustment for age, marital status, prescription drug mo
8 to examine the association between AMH and CRP without and with adjustment for age, race, body mass index (BMI), smoking
9 vertime (1.93 and 1.34 times higher, respectively; P<.001), with adjustment for age, sex, and HBV DNA.
10 y using logistic regression under an additive genetic model with adjustment for age, sex, body mass index, smoking status
11 least 0, 5, 10, and 15 years) prior to dementia diagnosis, with adjustments for age, sex, education, apolipoprotein E (A
12 at 12 weeks (n = 710) and 24 weeks (n = 662) were analysed with adjustment for confounders including maternal age, compl
13 nce intervals for associations between drugs and infection, with adjustment for confounders, were estimated by means of l
16 n analyses were used to assess predictor of upgrade status, with adjustment for demographic covariates jointly.
18 pwind UOGD well count and the downwind gross-beta radiation with adjustment for environmental factors governing the natur
19 teeth, and gum disease-cross-sectionally and prospectively, with adjustment for established periodontal disease risk fact
20 and without type 1 diabetes from a linear regression model with adjustment for grade, test topic, and year was 0.24 (95%
21 n 78.8% of those in the short-regimen group - a difference, with adjustment for human immunodeficiency virus status, of 1
23 walking time as measures of physical function) without and with adjustment for inflammatory and cardiac markers.
24 eity across histotypes was reduced [P-heterogeneity = 0.15] with adjustment for LOC components [1.08 serous, 1.11 endomet
25 ociations at 720,077 cytosine-guanine dinucleotides (CpGs), with adjustment for maternal age, gravidity, smoking, BMI, ch
27 Rs) were estimated in each propensity score-matched cohort, with adjustment for more than 90 baseline characteristics.
28 n A1c (HbA1c); and cognition and patient-reported outcomes, with adjustment for multiple comparisons to control for false
29 utcomes, CGM glucose metrics, and patient-reported outcomes with adjustment for multiple comparisons to control for the f
30 istorical (1960s, 1970s) and monitoring (1985 to 2015) data with adjustments for nutrient loadings and climatic effects;
31 of animal foods with risk were examined with Cox regression with adjustment for other animal foods and relevant covariate
32 A competing risks regression analysis, with adjustment for patient and tumor characteristics, estima
34 of life-EQ-5D-5L on a 0 to 1 scale-using linear regression with adjustment for patient, tumor, and treatment-related fac
36 linear regression, and PACC score using linear regression, with adjustment for potential confounders.
37 d these immune cells in a large cohort with atopic children with adjustment for prenatal and postnatal confounders.
38 ta for quantitative results of QFT-GIT, T-SPOT.TB, and TST (with adjustment for prior bacillus Calmette-Guerin [BCG] vacc
40 ness (VE) was estimated by conditional logistic regression, with adjustment for reported contact with children and area o
42 ry (PCS) scores, respectively, over 1-7 years post-surgery, with adjustment for sex, age, race, pre-surgery body mass ind
43 sociations between burn size and LIBRE Profile scale scores with adjustments for sex, current work status, burns to criti
44 models were used to calculate hazard ratios and odds ratios with adjustment for sociodemographic and clinical variables.
45 -regression was performed for the 5-year mortality outcomes with adjustment for the covariates age, physical status, tumo
46 roke was maintained among the subgroup of male smokers even with adjustment for the depth and amount of cigarette smoking
47 disorder-specific PRS were estimated using Cox regressions with adjustment for the other two PRSs.
48 ) as a continuous variable, using a restricted cubic spline with adjustment for the same covariates as in the primary ana
50 scribed best by a model based on a human development index, with adjustments for urbanization and age.