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1 0% by minimal, moderate, and severe alcohol withdrawal syndrome).
2 ic opiates has been implicated in the opiate withdrawal syndrome.
3 , which contributes to genesis of the opioid withdrawal syndrome.
4 l and autonomic manifestations of the opiate withdrawal syndrome.
5 rrupting PD treatment might lead to a severe withdrawal syndrome.
6 havioral features associated with the opiate withdrawal syndrome.
7 e with more severe manifestations of alcohol withdrawal syndrome.
8 ealthcare system risk factors for iatrogenic withdrawal syndrome.
9 ns, which constitute a severe symptom of the withdrawal syndrome.
10 c effect of FKBP5 on the severity of alcohol withdrawal syndrome.
11 drawal symptoms in ICU patients with alcohol withdrawal syndrome.
12 ail during the first 2 weeks of the nicotine withdrawal syndrome.
13 ccounts for some but not all aspects of this withdrawal syndrome.
14 ich are likely to contribute to beta-blocker withdrawal syndrome.
15 amide and is likely involved in bicalutamide withdrawal syndrome.
16 ng the validity and significance of cannabis withdrawal syndrome.
17 l will lead to a better understanding of the withdrawal syndrome.
18 antigen, which might result in antiandrogen withdrawal syndrome.
19 ted by a less severe antagonist-precipitated withdrawal syndrome.
20 ible pathway that might contribute to the HF withdrawal syndrome.
21 especially for people who have experienced a withdrawal syndrome.
22 osure, and increased frequency of iatrogenic withdrawal syndrome.
23 AR) agonists that may result in antiandrogen withdrawal syndrome.
24 ctivity 24 h later, suggestive of a caffeine withdrawal syndrome.
25 ons, and are therefore at increased risk for withdrawal syndrome.
26 trexone, substance dependence, and substance withdrawal syndrome.
27 on, and treatment of neonatal and iatrogenic withdrawal syndromes.
28 fe-threatening complications, dependence and withdrawal syndromes.
29 s appear similar to those of other substance withdrawal syndromes.
32 Chronic opioid-induced drug dependence and withdrawal syndrome after opioid cessation remain a seve
33 ng as criteria, adding cannabis and caffeine withdrawal syndromes, aligning tobacco use disorder crit
35 ission has long been implicated in the acute withdrawal syndrome and as a key signal for dependence-r
36 rrence, predictors, and prognosis of alcohol withdrawal syndrome and delirium tremens in patients wit
38 ity, validity, and time course of a cannabis withdrawal syndrome and have begun to explore the effect
39 Serious adverse events included the narcotic-withdrawal syndrome and sirolimus-associated pneumonitis
40 icant association between the development of withdrawal syndrome and the presence of ARDS (p = .017).
41 utcomes, pharmacologic treatment for alcohol withdrawal syndrome, and Clinical Institute Withdrawal A
42 sociated with congenital anomalies, neonatal withdrawal syndrome, and persistent pulmonary hypertensi
43 ed alcohol use leading to tolerance, alcohol withdrawal syndrome, and physical and psychological depe
45 olism, models for specific factors, like the withdrawal syndrome, are useful for identifying potentia
48 nergic agonist clonidine triggers a powerful withdrawal syndrome associated with massive CNS expressi
49 IE is validated as a model for human alcohol withdrawal syndrome (AWS) by demonstrating increased lev
52 ons into either brain region induced a quasi-withdrawal syndrome, but the observed behaviors differed
53 e of GHB or its analogs is associated with a withdrawal syndrome characterized by autonomic excitatio
54 ry characteristics, risk factors for alcohol withdrawal syndrome, clinical outcomes, pharmacologic tr
56 tate tumors may confer the development of HF withdrawal syndrome, commonly diagnosed in patients with
57 gth of stay, ICU length of stay, and alcohol withdrawal syndrome complications differed significantly
58 doses of opioids, several complications like withdrawal syndrome, delirium, mental status changes, an
63 pecific issues included possible addition of withdrawal syndromes for several substances, alignment o
67 o possible that individual components of the withdrawal syndrome have individual and unique rate limi
72 ional or alternative strategies for managing withdrawal syndromes in ICU patients should therefore be
74 siology, diagnosis, and treatment of alcohol withdrawal syndromes in the intensive care unit as well
76 alyses, significant predictors of iatrogenic withdrawal syndrome included younger age, preexisting co
77 of control over intake and the presence of a withdrawal syndrome, including both motivational and phy
78 f control over intake, and the presence of a withdrawal syndrome, including both motivational and phy
80 H (1 microM) displayed an abstinence-induced withdrawal syndrome, indicative of the development of ph
82 2-4-week period associated with the nicotine withdrawal syndrome is indicated when abstinence is atte
83 he hormone refractory stage to battle the HF withdrawal syndrome may become an alternative strategy t
84 aspirin is held preoperatively, the aspirin withdrawal syndrome may significantly increase the risk
87 e (10 mg/kg) to induce naloxone-precipitated withdrawal syndrome on the final day of the experiment (
88 zodiazepines, results in the production of a withdrawal syndrome, one feature of which is increased s
91 bstinence syndrome (NAS) is a postnatal drug withdrawal syndrome primarily caused by maternal opiate
93 ratory and clinical studies indicates that a withdrawal syndrome reliably follows discontinuation of
96 plications differed significantly by alcohol withdrawal syndrome severity and were worse with more se
97 ality also significantly differed by alcohol withdrawal syndrome severity but was only greater in pat
100 overdosed in the intensive care unit, though withdrawal syndromes should be prevented, and communicat
101 eously providing relief from the craving and withdrawal syndrome that accompanies cessation attempts.
102 ated prostate tumor growth, the antiandrogen withdrawal syndrome that allows antiandrogens to stimula
103 in chronic opiate abusers produces a severe withdrawal syndrome that is highly aversive, and avoidan
104 of the neonatal abstinence syndrome, a drug-withdrawal syndrome that most commonly occurs after in u
105 eported by patients given pregabalin, and no withdrawal syndrome was associated with pregabalin treat
109 ne receptor is relevant for the onset of the withdrawal syndrome, we used a mouse model of nicotine w
110 syndrome, combined with the observation that withdrawal syndromes were also associated with the use o
111 uscular blockers, pain assessments, and drug withdrawal syndromes were gathered during the first 28 d
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