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1 catalytic differences characterizing AOX and xanthine oxidoreductase.
2 d to be dependent on the enzymatic action of xanthine oxidoreductase.
3 hereas in cardiac homogenates, NTG inhibited xanthine oxidoreductase activity and scavenged NADPH oxi
4 increased reactive oxygen species (ROS) from xanthine oxidoreductase activity) and could be suppresse
5 d persistent basal and post-MI elevations in xanthine oxidoreductase activity.
6                    Established inhibitors of xanthine oxidoreductases (allopurinol oxypurinol, amflut
7 the NOS pathway is countered by induction of xanthine oxidoreductase, an enzyme capable of producing
8 itions occurring during ischemia, myocardial xanthine oxidoreductase and nitrate levels were determin
9 e is limited NO production from erythrocytic xanthine oxidoreductase and nitric-oxide synthase.
10                                              Xanthine oxidoreductase and nitrotyrosine levels were re
11 ant presence of the oxidative stress markers xanthine oxidoreductase and nitrotyrosine, findings that
12 ofile depends on endothelial NO synthase and xanthine oxidoreductase -catalyzed reduction of nitrite
13 tion in vivo via the generation, in part, of xanthine oxidoreductase-catalyzed NO production.
14                                              Xanthine oxidoreductase catalyzes the final two steps of
15 tural analogues of the oxidized sites in the xanthine oxidoreductase enzyme family.
16                                              Xanthine oxidoreductase from bovine milk can be prepared
17 t E-box and TATA-like elements repress human xanthine oxidoreductase gene (hXOR) expression.
18                                          The xanthine oxidoreductase gene (XOR) encodes an important
19         Xanthine oxidase (XO), also known as xanthine oxidoreductase, has long been considered an imp
20 xygen species arising from NADH oxidation by xanthine oxidoreductase in ischaemia-reperfusion injury
21                                              Xanthine oxidoreductase inhibition abolished the differe
22 ect that was attenuated significantly by the xanthine oxidoreductase inhibitor allopurinol.
23 n oxidation with ferricyanide but not by the xanthine oxidoreductase inhibitor allopurinol.
24  endothelial NO synthase or treated with the xanthine oxidoreductase inhibitor allopurinol.
25                                              Xanthine oxidoreductase is a ubiquitous cytoplasmic prot
26  bioactivation: reduction to nitric oxide by xanthine oxidoreductase, nonenzymatic disproportionation
27 ation is mediated by the enzymatic action of xanthine oxidoreductase, nonenzymatic disproportionation
28 wering uric acid in these mice by inhibiting xanthine oxidoreductase with allopurinol could improve t
29 eductants (ie, ascorbate); (4) inhibition of xanthine oxidoreductase with oxypurinol does not inhibit
30 e initiated to address the basis for the low xanthine oxidoreductase (XOR) activity in humans relativ
31                Butyrophilin 1A1 (BTN1A1) and xanthine oxidoreductase (XOR) are highly expressed in th
32 nd pharmacological studies further implicate xanthine oxidoreductase (XOR) as a source of reactive ox
33                                              Xanthine oxidoreductase (XOR) generates proinflammatory
34 d secretes milk lipid by this mechanism, and xanthine oxidoreductase (XOR) has long been thought to b
35 ive effects of pharmacological inhibitors of xanthine oxidoreductase (XOR) have implicated XOR in man
36 ctase activity) and could be suppressed with xanthine oxidoreductase (XOR) inhibition (i.e., allopuri
37                               Allopurinol, a xanthine oxidoreductase (XOR) inhibitor, abrogated the p
38                                           As xanthine oxidoreductase (XOR) is 1 of the major sources
39                                              Xanthine oxidoreductase (XOR) is a 300-kDa homodimer tha
40            Our previous studies suggest that xanthine oxidoreductase (XOR) is an important nitrite re
41                                              Xanthine oxidoreductase (XOR) is the enzyme responsible
42                                              Xanthine oxidoreductase (XOR) is the rate-limiting enzym
43                                              Xanthine oxidoreductase (XOR) modulates milk lipid secre
44                       Although inhibitors of xanthine oxidoreductase (XOR) or NOX2 NADPH oxidase caus
45                                              Xanthine oxidoreductase (XOR), a key enzyme of purine me
46 also was associated with increased levels of xanthine oxidoreductase (XOR), a known nitrite reductase
47                    Here, we demonstrate that xanthine oxidoreductase (XOR), a prototypic superoxide O
48 ystemic changes arising from inactivation of xanthine oxidoreductase (XOR), an enzyme that catalyzes
49                 Allopurinol, an inhibitor of xanthine oxidoreductase (XOR), blocks the oxidation of x
50  of AMP, and generation of uric acid through xanthine oxidoreductase (XOR).
51 ed hemoglobin, acidic disproportionation, or xanthine oxidoreductase (XOR).
52 re different from the well-studied mammalian xanthine oxidoreductases (XOR) and aldehyde oxidoreducta
53 amma (PPARgamma), and its upstream activator xanthine-oxidoreductase (XOR).

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