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1 erexpressing H441 non-small cell lung cancer xenograft.
2 th survivin siRNA, inhibited prostate cancer xenograft.
3 educed primate immune response in pig kidney xenograft.
4 alize the tumor in male mice bearing a SKOV3 xenograft.
5 ivation was also seen in a colorectal cancer xenograft.
6 [Y881F] is severely attenuated in human skin xenografts.
7 iferation in vitro, as well as in orthotopic xenografts.
8 pedestals were demonstrated in all infected xenografts.
9 ssion), or Calu-1 (no HER3 expression) tumor xenografts.
10 pe (p < 0.001) and drug-resistant (p < 0.05) xenografts.
11 ration both in vitro and in vivo as leukemia xenografts.
12 egression of all treated A2780 ovarian tumor xenografts.
13 owth in vivo, including TNBC patient-derived xenografts.
14 gnificantly prolonged survival in murine AML xenografts.
15 llular carcinoma cells in vitro and in mouse xenografts.
16 s retained abnormal SHH signaling like tumor xenografts.
17 vastatin caused regression of Ovcar-4 tumour xenografts.
18 reduction in the growth of MDA-MB-231 tumor xenografts.
19 s in approximately 50% of infected human gut xenografts.
20 sitive CWR22Rv1 and PSMA-negative PC-3 tumor xenografts.
21 combinations tested in patient-derived tumor xenografts.
22 ctal cancer cells in vitro and in vivo tumor xenografts.
23 gnancies and of human colorectal cancer cell xenografts.
24 l compared with untreated GSCs in orthotopic xenografts.
25 in KRAS(MUT) colorectal and pancreatic tumor xenografts.
26 th in HT-29 and SW480 cells and in nude mice xenografts.
27 ll migration and reduced the growth of tumor xenografts.
28 Multimodality imaging was performed in AR42J xenografts.
29 combined immunodeficiency mice bearing PC-3 xenografts.
30 asis of intravenous and intraprostatic tumor xenografts.
31 treatment of cells from PPR patient-derived xenografts.
32 d macrophages and the growth of colon cancer xenografts.
33 tumors in nude mice bearing dual-flank tumor xenografts.
34 er cells as well as impaired growth of tumor xenografts.
35 th in cell culture and mouse rhadomyosarcoma xenografts.
36 as in HDM201-resistant patient-derived tumor xenografts.
37 e bearing subcutaneous GPA33-positive SW1222 xenografts.
38 s and 15 models of cell- and patient-derived xenografts.
39 patient-derived GD2-expressing neuroblastoma xenografts.
41 ivation but infiltrated a Burkitt's lymphoma xenograft and efficiently inhibited tumor growth followi
44 phages infiltrate a human Burkitt's lymphoma xenograft and inhibit tumor growth, generating complete
45 s of lung cancer, including orthotopic human xenograft and Kras(LSL/G12D) mouse models of lung cancer
46 nhibited tumor growth and cancer invasion in xenograft and orthotopic mouse models, respectively.
47 f OTUD1 increases metastasis in intracardial xenograft and orthotopic transplantation models, and cor
48 toxicity and potent activity in both murine xenograft and patient-derived breast tumor explant model
49 results were confirmed in various cell line xenograft and patient-derived xenograft mouse models in
51 cell lung cancers, including patient-derived xenograft and the genetically engineered mutant KRAS-dri
53 ly suppresses development of intraperitoneal xenografts and prolongs the survival of ovarian cancer-b
55 le clonogens in some SiHa cervical carcinoma xenografts, and in combination with gemcitabine using a
56 c cancer cell lines, primary patient-derived xenografts, and pancreatic controls and revealed strikin
58 on of subcutaneous and orthotopic SCLC tumor xenografts as well as distant organ metastases with high
64 L) and diffuse large B-cell lymphoma (DLBCL) xenografts blocked tumor growth, both when delivered in
65 e sites using flapless surgery combined with xenograft blocks provided complete formation of the bucc
67 3D triple-negative cultures and intraductal xenografts by sustaining expression of E-cadherin and in
68 genesis on rigid substrates, with pretreated xenografts calcifying in vivo to a similar extent as nat
69 ally over an hour in a large volume of mouse xenograft colon tumor, and 3) determine the impact of th
71 ne responses to neonatal porcine islet (NPI) xenografts compared with rhesus islet allografts at 1 ho
72 r, our results suggest that zebrafish larvae xenografts constitute a promising fast assay for precisi
73 dency, experiments in cell culture and mouse xenografts demonstrated that inhibition of CHK1 selectiv
75 ion-media derived from bevacizumab-resistant xenograft-derived cells, while recombinant MIF drove M1
77 ls showed enhanced intratumoral branching in xenografted E2f7/8-deficient neoplasms compared with E2f
78 neuroblastoma cell lines and patient-derived xenografts engraft and adopt a metastatic program in chi
79 reast tumor growth in ERalpha-negative mouse xenografts, especially when combined with TAM treatment.
80 ly grown castration-resistant VCaP (CR-VCaP) xenografts express high levels of AR and retain intratum
84 timize ChIP-Seq protocols on patient-derived xenografts from human papillomavirus-related (HPV(+)) he
88 enic capacity in vitro, and suppressed tumor xenograft growth in severe combined immunodeficiency mic
92 e per gram [%ID/g]), whereas uptake in MKN45 xenografts (HGF-negative) was 5.0 +/- 1.3 %ID/g and a co
93 DFO-AMG102 at 120 h after injection in U87MG xenografts (HGF-positive) was high (36.8 +/- 7.8 percent
94 ers to identify radioresistant breast cancer xenografts highly amenable to sensitization by cotreatme
96 ppresses the growth of human colon carcinoma xenograft in nude mice in an RXRalpha-dependent manner.
97 ncer cells in vitro and cervical cancer cell xenograft in vivo in nude mice, and suppress cervical ca
99 colonized tenascin-C-coated trabecular bone xenografts in a novel system that employed chorioallanto
103 using human cancer cells and patient-derived xenografts in mice, we show that the cyclin D3-CDK6 kina
106 growth of WT and mutant ER-expressing tumor xenografts in NOD/SCID-gamma mice after oral or subcutan
108 IVIS spectrum imaging also visualized PC-3 xenografts in vivo and ex vivo with a high-contrast rati
111 of decorin for treatment of breast carcinoma xenografts induces paternally expressed gene 3 (Peg3), a
113 ophage polarization in bevacizumab-resistant xenografts is the source of their aggressive biology and
114 erapy in treating mice with intracranial GBM xenograft markedly slows tumor growth and provides a sig
116 evels (p<0.05) in mice bearing ectopic human xenograft MIA PaCa-2 pancreatic tumours with an average
124 formed phenotype, it slows tumor growth in a xenograft model and correlates with prolonged survival i
125 e imaged using (68)Ga-PSMA-11 PET in a mouse xenograft model and in a patient with castration-sensiti
127 umors and lung metastases in a breast cancer xenograft model as well as extravasation following injec
129 When evaluated for the efficacy in A549 xenograft model in mice, both the liposomes demonstrated
136 more likely by systemic insults, a humanized xenograft model of FSGS resulted in an expansion of Gr-1
142 from both human cancer cell lines and mouse xenograft model showed that cancer cells carrying the un
144 We found that cimetidine treatment in a xenograft model using A549 lung adenocarcinoma cells res
145 se-expressing ES-2 (ES-2-luc) ovarian cancer xenograft model, single i.p. injections of g-E and g-EAR
170 axel based chemotherapies in patient-derived xenograft models (PDX) with RSPO3 fusions and in tumors
172 delayed tumor growth in all rodent pediatric xenograft models and extended animal survival while demo
175 We treated 10 childhood ALL patient-derived xenograft models harboring various Ph-like genomic alter
176 elastic modulus in colorectal adenocarcinoma xenograft models in vivo and investigate any correlation
177 of aneuploidy, and genetic heterogeneity in xenograft models likely through modulation of Wnt signal
178 in AR variants-expressing CRPC cell line and xenograft models markedly reduces tumor growth through i
182 netically engineered mouse models and murine xenograft models of human MIBC, we demonstrate that tumo
186 n 4 different gastric cancer patient-derived xenograft models showed low uptake of (89)Zr-DFO-AMG102
188 atin ADCs imparted activity in cell line and xenograft models that are refractory to ADCs comprised o
189 In this study, we evaluated in human tumor xenograft models the proinflammatory properties of an on
191 vitro and led to tumor growth regression in xenograft models with a KRAS, NRAS or BRAF mutation at t
192 roblast (CAF)-derived EVs (from patients and xenograft models) laden with whole genomic mtDNA as a me
193 cers, facilitation of tumor growth in murine xenograft models, and centrosomal amplification induced
195 cancer cells and polyclonal patient-derived xenograft models, including tumours resistant to PARP in
204 SMA uptake increased 1.5- to 2.0-fold in the xenograft mouse model after treatment with both orchiect
205 We confirmed using a human-derived tumor xenograft mouse model that bicalutamide pre-treatment is
212 d kinetics of (18)F-FETrp in patient-derived xenograft mouse models and compared them with (11)C-AMT
213 improved ovarian cancer treatment in SKOV-3 xenograft mouse models in comparison with free drugs and
217 he knockdown of LAMB1 or K19 in subcutaneous xenograft mouse models resulted in significant loss of c
220 human NSCLC tumour samples, patient-derived xenografts, murine model of NSCLC, NSCLC cell lines and
223 , resulting in aggressive tumor formation in xenograft nude mice, which could be suppressed by combin
224 ution properties, SPECT and CT scans of HT29-xenografted nude mice injected with (177)Lu-3BP-227 were
226 tting-in several nonprostatic cell lines and xenografts of melanoma and small cell lung cancer (SCLC)
229 ution studies were performed in mice bearing xenografts of the same cell lines, comparing (68)Ga-THP-
230 F ELISA experiments were performed on murine xenografts of U87MG (HGF-positive, MET-positive) and MKN
233 was applied to a panel of 12 patient-derived xenograft (PDX) models of glioblastoma to predict gliobl
235 ssion, we used two different patient-derived xenograft (PDX) models: Patient 17(CXCR4-low) and P15(CX
236 n breast cancer patients and patient-derived xenograft (PDX) samples to predict pathway activity, and
240 atient avatars; for example, patient-derived xenografts (PDXs) established in mice and used for drug
242 ncogene (BRAF(amp)) in patient-derived tumor xenografts (PDXs) that were treated with a direct inhibi
243 one metastasis specimens and patient-derived xenografts (PDXs) were found to co-express endothelial m
244 Inhibiting HER2 expression in bone tumor xenografts reduced proliferation and RANK expression whi
245 ation in human MDA-MB-231 mammary tumor cell xenografts reduced the sizes of both the primary tumor a
246 inhibitors to orthotopic AML patient-derived xenografts reduced tumor burden and prolonged overall su
247 o mice engrafted with a PTCL patient-derived xenograft resulted in a shift among tumor-associated mac
248 nally, treatment of mice bearing GSC-derived xenografts resulted in significant inhibition of tumor p
249 ing a human cancer cell line panel and mouse xenografts revealed that 6OTD exhibits antitumor activit
250 earing contralateral flank UM-UC-3 and RT112 xenografts selectively arrested tumor growth in UM-UC-3
255 kedly prolonged survival of donor swine skin xenografts that may be applicable to clinical solid orga
257 ce in vivo using a series of patient-derived xenografts to generate paired chemosensitive and chemore
258 of the antiangiogenic response of 786-0 RCC xenografts to sunitinib, which revealed that pretreatmen
260 polarization, whereas bevacizumab-resistant xenografts transduced to upregulate MIF exhibited the op
261 ation and cell migration in vitro as well as xenograft tumor growth and metastasis in an orthotopic m
262 nks of severe combined immunodeficient mice; xenograft tumor growth and metastasis were assessed.
263 C cell proliferation in vitro and to abolish xenograft tumor growth in vivo Taken together, our findi
266 in a set of patient-derived prostate cancer xenograft tumor lines, we identified miR-100-5p as one o
269 FLT uptake were also observed for MDA-MB-231 xenografts (tumor-to-muscle ratio, 7.2 +/- 0.9 for femal
270 an breast carcinoma and IHC analysis of mice xenograft tumors demonstrated that DACH1 inversely relat
272 tor canertinib; these drugs slowed growth of xenograft tumors from MAN2A1-FER cells and prevented the
274 g) liposome treatment revealed regression of xenograft tumors in both wild type (p < 0.001) and drug-
275 P90 inhibitors, in culture and when grown as xenograft tumors in mice, depended on expression of miRN
276 and invasiveness and formed larger (>2-fold) xenograft tumors in mice, with more metastases, than cel
282 ells with MAN2A1-FER knockout formed smaller xenograft tumors, with fewer metastases, than control HU
283 vironment, we performed in vivo treatment of xenografted tumors with FPS-ZM1 (1 mg/kg, two times per
288 ted NPs was evaluated in mice bearing LS174T-xenografts using magnetic resonance (MR) imaging and flu
289 13)Bi and (177)Lu for PRIT of CEA-expressing xenografts, using the bispecific monoclonal antibody TF2
290 highly diverse, clonal composition in serial xenografts was highly similar between recipients of the
291 observed complete eradication of solid tumor xenografts, we conclude that targeted alpha-therapy regi
292 jects bearing reporter-modified intracranial xenografts, we quantitatively assessed MGMT knockdown by
293 Regardless of carbidopa premedication, the xenografts were characterized by an early increase in (1
294 eukemia, we demonstrate that patient-derived xenografts were highly polyclonal, consisting of tens to
295 selectively arrested tumor growth in UM-UC-3 xenografts, which had reduced tumor size, reduced Ki-67,
297 ed venetoclax treatment of MCL and ABC-DLBCL xenografts with a pretargeted RIT (PRIT) system directed
299 e harboring platinum-resistant ovarian tumor xenografts with pHLIP-PNA constructs suppressed HOTAIR a
300 amples to generate zebrafish patient-derived xenografts (zPDX) and provide proof-of-concept experimen
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