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1 level of methionine in bacterial as well as yeast cell.
2 enotypes, inactivates mismatch repair in the yeast cell.
3 ion is closely guided by experiments in live yeast cells.
4 nodes and contractile rings in live fission yeast cells.
5 isappearance of mitochondria from the mutant yeast cells.
6 f NAD into peroxisomes against AMP in intact yeast cells.
7 ies of formaldehyde-cross-linking in budding yeast cells.
8 reams and lysis of Rhodosporidium toruloides yeast cells.
9 ges in senescing and post-senescent survivor yeast cells.
10 s cytoplasmic hydrogen ion concentrations in yeast cells.
11 near invaginations at the plasma membrane of yeast cells.
12 l2-L-13 induces mitophagy in Atg32-deficient yeast cells.
13 critical role in metabolic energy control in yeast cells.
14 he toxicity of human alpha-synuclein in live yeast cells.
15 m can be visualized on the surface of living yeast cells.
16 72 is able to inhibit S6K phosphorylation in yeast cells.
17 rion fusions to encode synthetic memories in yeast cells.
18 eisosomes in both fission yeast and budding yeast cells.
19 r redox regulation of calcium homeostasis in yeast cells.
20 al data, and tracking of low-signal mRNAs in yeast cells.
21 ibute to the softening of dough through dead yeast cells.
22 replication defect when expressed in budding yeast cells.
23 anslational modification found in animal and yeast cells.
24 olesterol pathway intermediates in human and yeast cells.
25 he GG-NER E3 ligase, promotes UV survival in yeast cells.
26 r lipid droplet (LD) biogenesis in human and yeast cells.
27 nations into detached vesicles in BAR mutant yeast cells.
28 ophil phagocytosis and subsequent killing of yeast cells.
29 e expression of a protein in a population of yeast cells.
30 fined into the mother compartment of budding yeast cells.
31 g the fidelity of start codon recognition in yeast cells.
32 porters are involved in sugar transport into yeast cells.
33 terologous expression in Xenopus oocytes and yeast cells.
34 on the function of the protein and effect on yeast cells.
35 to characterize Snf1-Mig1 dynamics in single yeast cells.
36 le of nontranslating ribosomes purified from yeast cells.
37 rfering with the cadmium accumulation by the yeast cells.
38 redominant form of mtDNA replication in rho+ yeast cells.
39 that mitophagy is perturbed in CL-deficient yeast cells.
40 accumulation, unlike the OsPCS2b transformed yeast cells.
41 in vitro, and blocked autophagy induction in yeast cells.
42 ggregate activity observed in living budding yeast cells.
43 f m(6)A-modified FAA1 transcripts in haploid yeast cells.
44 to monitor glutathione import into the ER of yeast cells.
45 e joint distributions of mRNA populations in yeast cells.
46 bacteria use lactic acid to communicate with yeast cells.
47 izations as well as phenotypes of expressing yeast cells.
48 artments and cell membrane when expressed in yeast cells.
50 found that when challenged with glucose, the yeast cells accumulate glycolytic intermediates and ATP,
52 val in response to sudden glucose depletion, yeast cells activate lipid-droplet (LD) consumption thro
56 isolated UPEC was subsequently determined by yeast cell agglutination and immunofluorescence microsco
57 ere, we show that in asymmetrically dividing yeast cells, aggregation of cytosolic misfolded proteins
58 d communities consisting of a basal layer of yeast cells and an upper layer of filamentous cells, tog
59 V2A in hexose transport-deficient EBY.VW4000 yeast cells and demonstrated that these cells are able t
60 , we measure gene network activity in single yeast cells and find that the activity of the compensate
64 s rapid screening of ADAR variants in single yeast cells and provides quantitative evaluation for enz
66 ies to track the replicative aging of single yeast cells and reveal that the temporal patterns of het
67 sensitive marker of increased ROS levels in yeast cells and suggest that changes in ribosomes may be
68 significant decrease in both phagocytosis of yeast cells and the frequency of nonlytic exocytosis.
69 e heat-stress response within populations of yeast cells and the presence of subpopulations that are
70 s approach, employing proteomics analysis in yeast cells and transcriptional analysis in human cells.
71 y in cultured mammalian cells, as well as in yeast cells and zebrafish embryos We disrupted murine bd
73 mplate for DNA double-strand break repair in yeast cells, and Rad52, a member of the homologous recom
77 previously observed that upon expression in yeast cells, bacterial beta-barrel proteins including th
78 As confer a competitive fitness advantage to yeast cells because expression of these non-coding molec
79 f molecular noise that is inevitable in tiny yeast cells, because mistakes in sequencing cell cycle e
80 scriptionally formed G4 DNA in vivo and that yeast cells become highly sensitivity to G4-stabilizing
81 urrent study we found that when expressed in yeast cells both the monomeric and trimeric forms of ful
86 MG inhibits the growth of glucose-fermenting yeast cells by inducing endocytosis and degradation of t
88 platform, we measure noise dynamics in aging yeast cells by tracking the generation-specific activity
89 ully folded bioactive cyclotides inside live yeast cells by using intracellular protein trans-splicin
92 stributes homogeneously in wild-type fission yeast cells, can be made to concentrate at cell ends by
98 duce rejuvenated daughters, dividing budding yeast cells confine aging factors, including protein agg
99 e presence of a proteasome inhibitor or when yeast cells contained mutations in the CDC48 or SSA1 gen
101 nal microscopic diagnosis, as characteristic yeast cells could be observed only in 14 pus samples.
107 we provide a stochastic model of the budding yeast cell cycle that accurately accounts for the variab
108 rom peripheral leukocytes, brain tissue, and yeast cell cycle, revealed novel marker genes that were
115 er investigations of the budding and fission yeast cell-cycle, we identify two generic dynamical rule
119 ast homologues are "not essential" proteins, yeast cells deficient in the homologue of PAF53 grow at
127 ined the range of proteins that aggregate in yeast cells during normal growth and after exposure to s
128 w that both vegetative and pheromone-treated yeast cells exhibit discrete and asynchronous Ca(2+) bur
130 the sizes of many mRNAs change when budding yeast cells exit mitosis and enter the meiotic different
136 alogs to thiophosphorylate its substrates in yeast cell extracts as well as when produced as recombin
137 re-RCs support replication of plasmid DNA in yeast cell extracts in a reaction that exhibits hallmark
139 molecules, have been proposed to explain how yeast cells filter fluctuations and detect shallow gradi
141 terized by emergence of a germ tube from the yeast cell followed by mold-like growth of branching hyp
147 biased proof that trehalose does not protect yeast cells from dying and that the stress-protecting ro
148 for the SWI/SNF complex in the transition of yeast cells from fermentative to respiratory modes of me
151 sterol biosynthesis in single living fission yeast cells grown in mixtures of normal and (13)C-labele
153 rformed a genome-wide expression analysis in yeast cells grown in the presence or absence of the drug
154 port vigorous and sustainable restoration of yeast cell growth by replacing missing protein ion trans
156 selected differently in haploid and diploid yeast cells: haploid cells bud in an axial manner, while
160 n early after telomerase inactivation (ETI), yeast cells have accelerated mother cell aging and mildl
161 ental conditions, employing basic catalysts, yeast cells have become the nucleation centers for a sil
162 Within a single generation time a growing yeast cell imports approximately 14 million ribosomal pr
165 ies revealed that PfPAT mediated survival of yeast cells in low pantothenate concentrations and resto
166 of Mediator subunits in wild-type and mutant yeast cells in which RNA polymerase II promoter escape i
167 function is required for sterol secretion in yeast cells, indicating that members of this superfamily
168 ch as triterpene sapogenins, from engineered yeast cells into the growth medium, thereby greatly enha
170 mice, secretion of the Cfp4 glycoprotein by yeast cells is consistent across clinical and laboratory
172 er-recombination phenotype of Top3-deficient yeast cells is partially a result of unprocessed D loops
173 yeast expression system and discovered that yeast cells lacking endogenous potassium channels could
178 e concentrations and restored sensitivity of yeast cells lacking pantothenate uptake to fenpropimorph
180 to cellular toxicity and cell cycle delay in yeast cells lacking PSH1, but not in cells lacking UBR1,
181 rnover as Cse4 degradation is compromised in yeast cells lacking RCY1 Excessive Cse4 accumulation in
190 of millions of individual RNAs isolated from yeasts, cell lines, Arabidopsis thaliana leaves, mouse l
191 and formation of a shmoo-like morphology in yeast cells, lower pheromone doses elicit elongated cell
192 purified a high quantity of mRNA from crude yeast cell lysate compared to a phenol/chloroform extrac
193 closporine A with cyclophilin A protein in a yeast cell lysate is successfully detected and quantifie
196 [GAR(+)] is advantageous to bacteria because yeast cells make less ethanol and is advantageous to yea
197 mimetic FUS reduces aggregation in human and yeast cell models, and can ameliorate FUS-associated cyt
198 XR2), deletion of mitochondrial MXR2 renders yeast cells more sensitive to oxidative stress than the
201 mely, the geometrical effect of the dividing yeast cell on the diffusion of protein aggregates, and t
202 at constitutive membrane anchoring of GIV in yeast cells or rapid membrane translocation in mammalian
203 ulation of stress tolerance and longevity of yeast cells, our data provide a model in which Sch9 regu
210 1R) ) in the FRB domain of Tor2 that renders yeast cells rapamycin resistant and temperature sensitiv
211 show that upon growth at higher temperature, yeast cells relax the retention of DNA circles, which ac
212 r fit, our model quantitatively predicts the yeast cell response to pheromone gradient providing an i
213 Nitrogen replenishment of nitrogen-starved yeast cells resulted in substantial transcriptome change
214 gnaling guided neutrophils to migrate to the yeast cells, resulting in optimal phagocytosis and subse
215 enically cooled biological sample--a budding yeast cell (Saccharomyces cerevisiae)--using hard (7.9 k
219 report comprehensive ribosome profiling of a yeast cell size series from the time of cell birth, to i
226 osition of both murine and human C3 onto the yeast cell surface, with M1g4 showing delayed activation
232 (GET) pathway was described in mammalian and yeast cells that serve as a blueprint of TA protein inse
234 H did not allow the complete inactivation of yeast cells; the treatment shall be optimized before win
240 used time-resolved reporter assays in living yeast cells to gain insights into the coordination of po
241 Transient exposure to lactic acid caused yeast cells to heritably circumvent glucose repression.
242 nthetic genetic array screen using taz1Delta yeast cells to identify genes whose deletion aggravated
243 tion localization microscopy of live fission yeast cells to improve the spatial resolution to approxi
244 tical ER to the plasma membrane in human and yeast cells to maintain ER morphology and stabilize ER-p
245 sensors by glucose concentration may enable yeast cells to maintain glucose sensing activity at the
246 activator of TORC1, is somehow required for yeast cells to recover efficiently from a period of trea
247 llular traffic of the Chs3 protein, allowing yeast cells to regulate morphogenesis, depending on envi
248 Here we characterized mechanisms that allow yeast cells to survive under conditions of thiol peroxid
251 d chromosomal loci during interphase in live yeast cells together with polymer models of chromatin ch
252 on on the nature of the encapsulation of the yeast cells, together with the architecture of the three
254 de enzymes produced by SSF were utilised for yeast cell treatment leading to simultaneous release of
255 t, we systematically monitored the growth of yeast cells under various frequencies of oscillating osm
256 shown that in response to pheromone, budding yeast cells undergo a rise of cytosolic Ca(2+) that is m
257 osed to a high dose of mating pheromone, the yeast cell undergoes growth arrest and forms a shmoo-lik
261 in-containing protein Mps3 on the NE in live yeast cells using fluorescence cross-correlation spectro
263 was found in glycolytic oscillations in real yeast cells, verifying that chronotaxicity could be used
264 n1, a DNA/RNA helicase that is essential for yeast cell viability and homologous to human senataxin,
265 small binders readily penetrate through the yeast cell wall and thus eliminate the requirement for i
268 esence of caffeine, a known modulator of the yeast cell wall integrity (CWI) mitogen-activated protei
272 Results showed that the sorption capacity by yeast cell walls for 4-EG was greater than that for 4-EP
273 tments (activated charcoal, bentonite, PVPP, yeast cell walls, potassium caseinate, zeolite and grape
281 er to monitor Fus3 and Kss1 activity in live yeast cells, we demonstrate that overall mating MAPK act
282 wild-type levels of mcm10-m2,3,4 in budding yeast cells, we observed a severe growth defect and a su
283 ing transcription at the GAL locus in living yeast cells, we show that antisense GAL10 ncRNA transcri
284 compaction of the chromatin fiber in living yeast cells, we show that chromosome condensation entail
287 oleaginous Cryptococcus curvatus VKM Y-3288 yeast cells were immobilized in a bimodal silica-organic
288 incidence of alphaSyn cytoplasmic foci, and yeast cells were rescued from alphaSyn-generated proteot
293 ntial to aid the enrichment of low-abundance yeast cells when filler volume fractions approximately 1
294 reas it displays internal staining of select yeast cells which also show propidium iodide staining, i
298 his issue, Dudin et al. image mating fission yeast cells with unprecedented spatiotemporal resolution
299 rowth and DNA replication defects in budding yeast cells, with diminished DDK phosphorylation of Mcm2
300 improved the ability to metabolize xylose of yeast cells without adaptive evolution, suggesting that
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