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1  level of methionine in bacterial as well as yeast cell.
2 enotypes, inactivates mismatch repair in the yeast cell.
3 ion is closely guided by experiments in live yeast cells.
4  nodes and contractile rings in live fission yeast cells.
5 isappearance of mitochondria from the mutant yeast cells.
6 f NAD into peroxisomes against AMP in intact yeast cells.
7 ies of formaldehyde-cross-linking in budding yeast cells.
8 reams and lysis of Rhodosporidium toruloides yeast cells.
9 ges in senescing and post-senescent survivor yeast cells.
10 s cytoplasmic hydrogen ion concentrations in yeast cells.
11 near invaginations at the plasma membrane of yeast cells.
12 l2-L-13 induces mitophagy in Atg32-deficient yeast cells.
13 critical role in metabolic energy control in yeast cells.
14 he toxicity of human alpha-synuclein in live yeast cells.
15 m can be visualized on the surface of living yeast cells.
16 72 is able to inhibit S6K phosphorylation in yeast cells.
17 rion fusions to encode synthetic memories in yeast cells.
18  eisosomes in both fission yeast and budding yeast cells.
19 r redox regulation of calcium homeostasis in yeast cells.
20 al data, and tracking of low-signal mRNAs in yeast cells.
21 ibute to the softening of dough through dead yeast cells.
22 replication defect when expressed in budding yeast cells.
23 anslational modification found in animal and yeast cells.
24 olesterol pathway intermediates in human and yeast cells.
25 he GG-NER E3 ligase, promotes UV survival in yeast cells.
26 r lipid droplet (LD) biogenesis in human and yeast cells.
27 nations into detached vesicles in BAR mutant yeast cells.
28 ophil phagocytosis and subsequent killing of yeast cells.
29 e expression of a protein in a population of yeast cells.
30 fined into the mother compartment of budding yeast cells.
31 g the fidelity of start codon recognition in yeast cells.
32 porters are involved in sugar transport into yeast cells.
33 terologous expression in Xenopus oocytes and yeast cells.
34 on the function of the protein and effect on yeast cells.
35 to characterize Snf1-Mig1 dynamics in single yeast cells.
36 le of nontranslating ribosomes purified from yeast cells.
37 rfering with the cadmium accumulation by the yeast cells.
38 redominant form of mtDNA replication in rho+ yeast cells.
39  that mitophagy is perturbed in CL-deficient yeast cells.
40 accumulation, unlike the OsPCS2b transformed yeast cells.
41 in vitro, and blocked autophagy induction in yeast cells.
42 ggregate activity observed in living budding yeast cells.
43 f m(6)A-modified FAA1 transcripts in haploid yeast cells.
44 to monitor glutathione import into the ER of yeast cells.
45 e joint distributions of mRNA populations in yeast cells.
46 bacteria use lactic acid to communicate with yeast cells.
47 izations as well as phenotypes of expressing yeast cells.
48 artments and cell membrane when expressed in yeast cells.
49                                   In fission yeast cells, a microtubule-dependent network has been id
50 found that when challenged with glucose, the yeast cells accumulate glycolytic intermediates and ATP,
51                   In response to pheromones, yeast cells activate a MAPK pathway to direct processes
52 val in response to sudden glucose depletion, yeast cells activate lipid-droplet (LD) consumption thro
53                                              Yeast cells activate RNR in response to genotoxic stress
54                                         When yeast cells adapt to respiration the Snf1/4 glucose-sens
55                        These multifunctional yeast cells adhere to the gold sensor surface while simu
56 isolated UPEC was subsequently determined by yeast cell agglutination and immunofluorescence microsco
57 ere, we show that in asymmetrically dividing yeast cells, aggregation of cytosolic misfolded proteins
58 d communities consisting of a basal layer of yeast cells and an upper layer of filamentous cells, tog
59 V2A in hexose transport-deficient EBY.VW4000 yeast cells and demonstrated that these cells are able t
60 , we measure gene network activity in single yeast cells and find that the activity of the compensate
61                                              Yeast cells and hyphae recovered from the kidney of anti
62 n plant-infecting tombusvirus replication in yeast cells and in vitro using purified components.
63         Moreover, Becn1(+/-) mice as well as yeast cells and nematodes lacking the ortholog of human
64 s rapid screening of ADAR variants in single yeast cells and provides quantitative evaluation for enz
65                           Uptake assays with yeast cells and radiolabeled (32)P revealed that PvPht1;
66 ies to track the replicative aging of single yeast cells and reveal that the temporal patterns of het
67  sensitive marker of increased ROS levels in yeast cells and suggest that changes in ribosomes may be
68 significant decrease in both phagocytosis of yeast cells and the frequency of nonlytic exocytosis.
69 e heat-stress response within populations of yeast cells and the presence of subpopulations that are
70 s approach, employing proteomics analysis in yeast cells and transcriptional analysis in human cells.
71 y in cultured mammalian cells, as well as in yeast cells and zebrafish embryos We disrupted murine bd
72         Actin is critical for endocytosis in yeast cells, and also in mammalian cells under tension.
73 mplate for DNA double-strand break repair in yeast cells, and Rad52, a member of the homologous recom
74                                         When yeast cells are challenged by a fluctuating environment,
75                                 Fast growing yeast cells are predicted to perform significant amount
76 LM), we probed this question in live fission yeast cells at unprecedented resolution.
77  previously observed that upon expression in yeast cells, bacterial beta-barrel proteins including th
78 As confer a competitive fitness advantage to yeast cells because expression of these non-coding molec
79 f molecular noise that is inevitable in tiny yeast cells, because mistakes in sequencing cell cycle e
80 scriptionally formed G4 DNA in vivo and that yeast cells become highly sensitivity to G4-stabilizing
81 urrent study we found that when expressed in yeast cells both the monomeric and trimeric forms of ful
82 gnificant regulatory variation in individual yeast cells, both before and after stress.
83  (CPDs), increases survival of UV irradiated yeast cells but attenuates TCR.
84 Cdc42 activator, was essential for growth in yeast cells but not in established hyphae.
85 nd is toxic to wheat, tobacco, bacterial and yeast cells, but not to Z. tritici itself.
86 MG inhibits the growth of glucose-fermenting yeast cells by inducing endocytosis and degradation of t
87 ed for oxidative stress responses in fission yeast cells by promoting transcription initiation.
88 platform, we measure noise dynamics in aging yeast cells by tracking the generation-specific activity
89 ully folded bioactive cyclotides inside live yeast cells by using intracellular protein trans-splicin
90 mediate cell signaling events, ensuring that yeast cells can adapt and remain viable.
91                                Metabolism in yeast cells can be manipulated by supplying different ca
92 stributes homogeneously in wild-type fission yeast cells, can be made to concentrate at cell ends by
93                   Here, we show that fission yeast cells carrying a mutation in the DNA-binding prote
94                          Exposure of haploid yeast cells, carrying mating type "a," to "alpha pheromo
95        The major chitin synthase activity in yeast cells, Chs3, has become a paradigm in the study of
96  of alpha-synuclein was also demonstrated in yeast cells coexpressing TG2.
97  Au level was increased in COPT2, expressing yeast cells compared to vector transformed control.
98 duce rejuvenated daughters, dividing budding yeast cells confine aging factors, including protein agg
99 e presence of a proteasome inhibitor or when yeast cells contained mutations in the CDC48 or SSA1 gen
100                                              Yeast cells containing the intracellular beta-glucosidas
101 nal microscopic diagnosis, as characteristic yeast cells could be observed only in 14 pus samples.
102 two complex biological samples obtained from yeast cell cultures at the proteome level.
103                                Using fission yeast cell cycle as an example, we uncovered that the no
104                    Empirically, on synthetic yeast cell cycle data, CPchi(2) achieved much higher sta
105 ability, speed and robustness of the fission yeast cell cycle oscillations.
106                             In data from the Yeast cell cycle SW1PerS identifies genes not preferred
107 we provide a stochastic model of the budding yeast cell cycle that accurately accounts for the variab
108 rom peripheral leukocytes, brain tissue, and yeast cell cycle, revealed novel marker genes that were
109  expand a previous mathematical model of the yeast cell cycle.
110 ture are linked to ER inheritance during the yeast cell cycle.
111                                   In budding yeast, cell cycle progression and ribosome biogenesis ar
112 lity, as observed in the budding and fission yeast cell- cycle.
113                           Using a well-known yeast cell-cycle data set with 6,178 genes, we identifie
114                                  We consider yeast cell-cycle gene expression data, and show that the
115 er investigations of the budding and fission yeast cell-cycle, we identify two generic dynamical rule
116                       In fission and budding yeast, cell-cycle progression depends on cell size, alth
117 rus, F1L does not prevent caspase-9-mediated yeast cell death.
118                         We show that fission yeast cells deficient in ER-PM contacts exhibit aberrant
119 ast homologues are "not essential" proteins, yeast cells deficient in the homologue of PAF53 grow at
120                                              Yeast cells deficient in the Rieske iron-sulfur subunit
121                       Cytokinesis in fission yeast cells depends on conventional myosin-II (Myo2) to
122                       In the absence of LDs, yeast cells display alterations in their phospholipid co
123                                     However, yeast cells display polarized receptors.
124              We show that RNase H2-deficient yeast cells displayed elevated frequency of Rad52 foci,
125                   Finally, Yjr129c-deficient yeast cells displayed phenotypes related to eEF2 functio
126 r9 positions mitotic spindles during budding yeast cell division.
127 ined the range of proteins that aggregate in yeast cells during normal growth and after exposure to s
128 w that both vegetative and pheromone-treated yeast cells exhibit discrete and asynchronous Ca(2+) bur
129                                      Budding yeast cells exist in two mating types, a and alpha, whic
130  the sizes of many mRNAs change when budding yeast cells exit mitosis and enter the meiotic different
131      We study cell polarization when fission yeast cells exit starvation.
132                                Compared with yeast cells expressing Arabidopsis thaliana Pht1;5, cell
133 wth of hexose transport-deficient EBY.VW4000 yeast cells expressing human SV2A.
134                                 Infection of yeast cells expressing the reference SUP35 gene sequence
135 probe the effects of polyethylene glycol and yeast cell extract as crowding agents.
136 alogs to thiophosphorylate its substrates in yeast cell extracts as well as when produced as recombin
137 re-RCs support replication of plasmid DNA in yeast cell extracts in a reaction that exhibits hallmark
138                                              Yeast cell factories encounter physical and chemical str
139 molecules, have been proposed to explain how yeast cells filter fluctuations and detect shallow gradi
140                       In mitotically growing yeast cells, five septin subunits are expressed (Cdc3, C
141 terized by emergence of a germ tube from the yeast cell followed by mold-like growth of branching hyp
142                  Purification of Ty1-IN from yeast cells followed by mass spectrometry (MS) analysis
143 eplication of hundreds of individual fission yeast cells for over seventy-five generations.
144 cation of Tomato bushy stunt virus (TBSV) in yeast cell-free extracts and in plant extracts.
145 ngation-can be recapitulated in vitro with a yeast cell-free system.
146                                              Yeast cells from colonies or liquid cultures are lysed b
147 biased proof that trehalose does not protect yeast cells from dying and that the stress-protecting ro
148 for the SWI/SNF complex in the transition of yeast cells from fermentative to respiratory modes of me
149                Here we find DnaJB6-protected yeast cells from polyglutamine toxicity and cured yeast
150                           Rod-shaped fission yeast cells grow in a highly polarized manner, and genet
151 sterol biosynthesis in single living fission yeast cells grown in mixtures of normal and (13)C-labele
152                                          For yeast cells grown in synthetic defined (SD) medium, the
153 rformed a genome-wide expression analysis in yeast cells grown in the presence or absence of the drug
154 port vigorous and sustainable restoration of yeast cell growth by replacing missing protein ion trans
155 nment and that either K113E or E195K induces yeast cell growth defects rescued by E/K.
156  selected differently in haploid and diploid yeast cells: haploid cells bud in an axial manner, while
157             We demonstrate here that fission yeast cells harbouring a hyperactive Cdc2CDK1 mutation (
158                                      Budding yeast cells have a finite replicative life span; that is
159                                              Yeast cells have a single CL-specific phospholipase, Cld
160 n early after telomerase inactivation (ETI), yeast cells have accelerated mother cell aging and mildl
161 ental conditions, employing basic catalysts, yeast cells have become the nucleation centers for a sil
162    Within a single generation time a growing yeast cell imports approximately 14 million ribosomal pr
163  activity of cytochromes b and c in freezing yeast cells in a contactless, label-free manner.
164 d to improve the enrichment of low-abundance yeast cells in an iDEP channel.
165 ies revealed that PfPAT mediated survival of yeast cells in low pantothenate concentrations and resto
166 of Mediator subunits in wild-type and mutant yeast cells in which RNA polymerase II promoter escape i
167 function is required for sterol secretion in yeast cells, indicating that members of this superfamily
168 ch as triterpene sapogenins, from engineered yeast cells into the growth medium, thereby greatly enha
169                            Mating of budding yeast cells is a model system for studying cell-cell int
170  mice, secretion of the Cfp4 glycoprotein by yeast cells is consistent across clinical and laboratory
171                         Amino acid uptake in yeast cells is mediated by about 16 plasma membrane perm
172 er-recombination phenotype of Top3-deficient yeast cells is partially a result of unprocessed D loops
173  yeast expression system and discovered that yeast cells lacking endogenous potassium channels could
174 function of Dna2 in end resection in budding yeast cells lacking exonuclease 1.
175 itive phenotype that has long been noted for yeast cells lacking functional Get3.
176                                              Yeast cells lacking LDs are severely defective in PSM gr
177                                              Yeast cells lacking Msn2 and Msn4 exhibit prevalent repr
178 e concentrations and restored sensitivity of yeast cells lacking pantothenate uptake to fenpropimorph
179 ed, genome-wide synthetic lethal screen with yeast cells lacking profilin (pfy1Delta).
180 to cellular toxicity and cell cycle delay in yeast cells lacking PSH1, but not in cells lacking UBR1,
181 rnover as Cse4 degradation is compromised in yeast cells lacking RCY1 Excessive Cse4 accumulation in
182 of chromosomal rearrangements recovered from yeast cells lacking Sae2 or the Mre11 nuclease.
183                                              Yeast cells lacking Snf1 (AMP-activated protein kinase)
184                    In human somatic cells or yeast cells lacking telomerase, telomeres are shortened
185                                Using fission yeast cells lacking the debranching enzyme Dbr1, LaSSO n
186                                              Yeast cells lacking the ORF YCL047C/POF1 release conside
187   Expression of ORAOV1 restores viability to yeast cells lacking YNL260c.
188             Inhibition of Hsp104 function in yeast cells leads to a failure to generate new propagons
189                                              Yeast cell lines were genetically engineered to display
190 of millions of individual RNAs isolated from yeasts, cell lines, Arabidopsis thaliana leaves, mouse l
191  and formation of a shmoo-like morphology in yeast cells, lower pheromone doses elicit elongated cell
192  purified a high quantity of mRNA from crude yeast cell lysate compared to a phenol/chloroform extrac
193 closporine A with cyclophilin A protein in a yeast cell lysate is successfully detected and quantifie
194  for cross-linked protein-DNA complexes from yeast cell lysate.
195         Over the course of 17 d, immobilized yeast cells maintained >95% viability, whereas the viabi
196 [GAR(+)] is advantageous to bacteria because yeast cells make less ethanol and is advantageous to yea
197 mimetic FUS reduces aggregation in human and yeast cell models, and can ameliorate FUS-associated cyt
198 XR2), deletion of mitochondrial MXR2 renders yeast cells more sensitive to oxidative stress than the
199                               During mating, yeast cells must perforate their rigid cell walls at the
200  homeostasis of cation concentrations in the yeast cells of S. cerevisiae.
201 mely, the geometrical effect of the dividing yeast cell on the diffusion of protein aggregates, and t
202 at constitutive membrane anchoring of GIV in yeast cells or rapid membrane translocation in mammalian
203 ulation of stress tolerance and longevity of yeast cells, our data provide a model in which Sch9 regu
204 rvative replication in extracts from budding yeast cells overexpressing firing factors.
205 l tubes had increased adhesion compared with yeast cells ( P < 0.05).
206    Interestingly, in contrast with animal or yeast cells, plants contain a second nucleolin gene.
207                                       During yeast cell polarization localization of the small GTPase
208                                              Yeast cell populations harboring the same defined aneupl
209                                  In dividing yeast cells, protein aggregates that form under stress o
210 1R) ) in the FRB domain of Tor2 that renders yeast cells rapamycin resistant and temperature sensitiv
211 show that upon growth at higher temperature, yeast cells relax the retention of DNA circles, which ac
212 r fit, our model quantitatively predicts the yeast cell response to pheromone gradient providing an i
213   Nitrogen replenishment of nitrogen-starved yeast cells resulted in substantial transcriptome change
214 gnaling guided neutrophils to migrate to the yeast cells, resulting in optimal phagocytosis and subse
215 enically cooled biological sample--a budding yeast cell (Saccharomyces cerevisiae)--using hard (7.9 k
216                                              Yeast cells (Saccharomyces cerevisiae and Schizosaccharo
217                                      Haploid yeast cells secrete mating pheromones that are sensed by
218                       Prior work in HeLa and yeast cells showed that a decrease in ATAD5 protein leve
219 report comprehensive ribosome profiling of a yeast cell size series from the time of cell birth, to i
220                                   In budding yeast, cell size is thought to be controlled almost enti
221                                   In budding yeast, cell size primarily modulates the duration of the
222                                   In budding yeast cells, Slx5 resides in the nucleus, forms distinct
223 d abundantly and specifically by Histoplasma yeast cells, suggesting its role in pathogenesis.
224 nked to the end of DNA in RNase H2-deficient yeast cells, supporting this model.
225                                       We use yeast cell surface display to engineer E6AP to exclusive
226 osition of both murine and human C3 onto the yeast cell surface, with M1g4 showing delayed activation
227 g1 in removing exposed beta-glucans from the yeast cell surface.
228                                          The yeast cell-surface glucose sensors Rgt2 and Snf3 functio
229                                Similarly, in yeast cells that are defective for transcription termina
230          To answer this question, we monitor yeast cells that are genetically engineered to display e
231                                           In yeast cells that do not readily take up pyruvate, the ad
232 (GET) pathway was described in mammalian and yeast cells that serve as a blueprint of TA protein inse
233                                   In fission yeast cells, the formin Fus1, which nucleates linear act
234 H did not allow the complete inactivation of yeast cells; the treatment shall be optimized before win
235 of the replicative lifespan, and tracking of yeast cells throughout their entire lifespan.
236                             The ability of a yeast cell to propagate [PSI(+) ], the prion form of the
237                  Diverse bacteria can elicit yeast cells to acquire [GAR(+)], although the molecular
238 pression assays in Nicotiana benthamiana and yeast cells to examine its functionality.
239              We tested the platform to force yeast cells to express a desired fixed, or time-varying,
240 used time-resolved reporter assays in living yeast cells to gain insights into the coordination of po
241     Transient exposure to lactic acid caused yeast cells to heritably circumvent glucose repression.
242 nthetic genetic array screen using taz1Delta yeast cells to identify genes whose deletion aggravated
243 tion localization microscopy of live fission yeast cells to improve the spatial resolution to approxi
244 tical ER to the plasma membrane in human and yeast cells to maintain ER morphology and stabilize ER-p
245  sensors by glucose concentration may enable yeast cells to maintain glucose sensing activity at the
246  activator of TORC1, is somehow required for yeast cells to recover efficiently from a period of trea
247 llular traffic of the Chs3 protein, allowing yeast cells to regulate morphogenesis, depending on envi
248  Here we characterized mechanisms that allow yeast cells to survive under conditions of thiol peroxid
249 trehalose and/or the Tps1 protein, may serve yeast cells to withstand exposure to stress.
250  highly conserved from simple systems, e.g., yeast cells, to the much more complex human system.
251 d chromosomal loci during interphase in live yeast cells together with polymer models of chromatin ch
252 on on the nature of the encapsulation of the yeast cells, together with the architecture of the three
253                                              Yeast cells track gradients of pheromones to locate mati
254 de enzymes produced by SSF were utilised for yeast cell treatment leading to simultaneous release of
255 t, we systematically monitored the growth of yeast cells under various frequencies of oscillating osm
256 shown that in response to pheromone, budding yeast cells undergo a rise of cytosolic Ca(2+) that is m
257 osed to a high dose of mating pheromone, the yeast cell undergoes growth arrest and forms a shmoo-lik
258                                         In a yeast cell undergoing budding, cables are in constant dy
259                                      Fission yeast cells use Arp2/3 complex and formin to assemble di
260                  Ultrastructural analysis of yeast cells using electron microscopy reveals a signific
261 in-containing protein Mps3 on the NE in live yeast cells using fluorescence cross-correlation spectro
262 mmobilizing fungal laccase on the surface of yeast cells using synthetic biology techniques.
263 was found in glycolytic oscillations in real yeast cells, verifying that chronotaxicity could be used
264 n1, a DNA/RNA helicase that is essential for yeast cell viability and homologous to human senataxin,
265  small binders readily penetrate through the yeast cell wall and thus eliminate the requirement for i
266  chemokinesis, whose activity is enhanced by yeast cell wall components and aromatic alcohols.
267                    In the present study, the yeast cell wall fractionation process involving enzymati
268 esence of caffeine, a known modulator of the yeast cell wall integrity (CWI) mitogen-activated protei
269                                          The yeast cell wall integrity MAPK Slt2 mediates the transcr
270                                          The yeast cell wall of Saccharomyces cerevisiae is an import
271  bone-marrow-derived DCs stimulated with the yeast cell wall preparation zymosan.
272 Results showed that the sorption capacity by yeast cell walls for 4-EG was greater than that for 4-EP
273 tments (activated charcoal, bentonite, PVPP, yeast cell walls, potassium caseinate, zeolite and grape
274 ermodynamics of sorption of 4-EG and 4-EP by yeast cell walls, using a synthetic wine.
275  yield of 18.0% of the original ratio in the yeast cell walls.
276 ic process for the isolation of glucans from yeast cell walls.
277  the ability of adsorbing color molecules by yeasts' cell walls was assessed.
278 of an indirect diffusion of wood aromas from yeast cell-walls.
279 of polyphenols and volatile compounds to the yeast cell-walls.
280 the maximum accumulation of both ions in the yeast cells was observed.
281 er to monitor Fus3 and Kss1 activity in live yeast cells, we demonstrate that overall mating MAPK act
282  wild-type levels of mcm10-m2,3,4 in budding yeast cells, we observed a severe growth defect and a su
283 ing transcription at the GAL locus in living yeast cells, we show that antisense GAL10 ncRNA transcri
284  compaction of the chromatin fiber in living yeast cells, we show that chromosome condensation entail
285                                              Yeast cells were encapsulated in a matrix consisting of
286                                              Yeast cells were genetically modified to display both si
287  oleaginous Cryptococcus curvatus VKM Y-3288 yeast cells were immobilized in a bimodal silica-organic
288  incidence of alphaSyn cytoplasmic foci, and yeast cells were rescued from alphaSyn-generated proteot
289 one redox homeostasis were also monitored as yeast cells were subjected to oxidative stress.
290                                         When yeast cells were treated with hydroxyurea (HU) to block
291                       The surface engineered yeast cells were unaffected by the QDs present on their
292 silica capsule was found to form around each yeast cell when using 85 vol% MTES.
293 ntial to aid the enrichment of low-abundance yeast cells when filler volume fractions approximately 1
294 reas it displays internal staining of select yeast cells which also show propidium iodide staining, i
295                        This looping provides yeast cells with a transcriptional memory, enabling them
296                Third, treatment of wild-type yeast cells with E9591 or LMT generated cellular defects
297                                 Treatment of yeast cells with pyrodach-2 (D2) or pyrodach-4 (D4) reve
298 his issue, Dudin et al. image mating fission yeast cells with unprecedented spatiotemporal resolution
299 rowth and DNA replication defects in budding yeast cells, with diminished DDK phosphorylation of Mcm2
300 improved the ability to metabolize xylose of yeast cells without adaptive evolution, suggesting that

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