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1 e PE and PI species are mostly absent in the yolk.
2 all carotenoid absorption via lipid-rich egg yolk.
3 memade matrix-matched standards based on egg yolk.
4 oncentration in albumen, but only for 13% in yolk.
5 unsaturated fatty acids (n-3 LC-PUFA) in egg yolk.
6 microstructure of granule fractions from egg yolk.
7 nfirmed in samples of SOD extracted from egg yolk.
8 ibutors to the antioxidant properties of egg yolk.
9 carotenoid, and malondialdehyde (MDA) in egg yolk.
10 x mobility in fresh and freeze-thawed gelled yolk.
11 e prepared with control yolks or soy control yolks.
12  fed breakfast doses of 0, 1, 2, 4, or 6 egg yolks.
13     Residues of Sudan I were detected in egg yolks (0.29+/-0.03microg/kg, mean+/-SD) only after the a
14 ses 1 and 2 (mug kg(-)(1)): 6521 and 7329 in yolk, 1370 and 1539 in white.
15 ghest SigmaOP concentration, followed by egg yolk (14.8+/-2.4 ng/g ww) approximately egg albumen (14.
16 tive to standard control and LA-rich control yolks, (2) compare the rheological properties of mayonna
17  both doses (mug kg(-)(1)): 5920 and 9453 in yolk; 4831 and 6050 in white, in doses 1 and 2, respecti
18 pical aspect directly contacting the oocytic yolk, acquire the ability to take up yolk macromolecules
19 ar role in the unwanted gel formation of egg yolk after conventional freezing.
20 MM dropped below the LOD (1.9 mug kg(-1)) in yolk after day 16 and 19 for doses 1 and 2.
21  extracellular space into oocytes along with yolk and accumulates in punctate structures within embry
22 (Larus argentatus; n=8) and the separate egg yolk and albumen of their entire clutches of eggs (n=16)
23 ealization of painted and gilded areas, i.e. yolk and albumen, respectively.
24 o different foodstuffs, sour cream, egg, egg yolk and chicken nuggets.
25 ction spend roughly half their body mass for yolk and egg production; following mass spawning, the pa
26 urated fats and cholesterol, like meats, egg yolk and high-fat dairy products, are associated with in
27                                          Egg yolk and its main component, low-density lipoproteins (L
28 e eye lens, with lower levels in the retina, yolk and other tissues.
29 les like phosvitin and lipovitellin from egg yolk and phospholipids/phosphopeptides from human serum.
30 o co-protein effect with soy glycinin or egg yolk and positive co-protein effects with bovine serum a
31 tting of Au, which is sandwiched between the yolk and shell, leads to the desired nanocup morphology.
32 ) showed an increased lipid oxidation in egg yolk and slight effects in liquid whole eggs; this was c
33 es in the SMM and TMP concentrations between yolk and white in post treatment period were found.
34 ethoxine (SMM) and trimethoprim (TMP) in egg yolk and white was measured during and after administrat
35                            Additionally, egg yolks and human milk appear to be bioavailable sources.
36 hoproteins, such as casein, nonfat milk, egg yolk, and human blood serum, are used to explore its pho
37           Carotenoids accumulated in the egg yolk are of importance for two reasons.
38 nted for 25% of the SigmaOP concentration in yolk, but was not detected in albumen.
39 elative yolk mass in larger eggs and reduced yolk carotenoid concentrations in early breeders.
40 der, poppy, sunflower and pumpkin seeds, egg yolk, carum, hazel nuts and amaranth) on the morphologic
41 sing maternal shRNA technology we found that yolk catabolism depends on the Tor pathway and on the au
42                                              Yolk catabolism initiates at cellularization in Drosophi
43 rved Tor metabolic sensing pathway regulates yolk catabolism, similar to Tor-dependent metabolic regu
44 n, autophagy was not required for initiating yolk catabolism.
45 ctin and microtubule cytoskeleton within the yolk cell and defects in the outer enveloping cell layer
46 ess in which the blastoderm spreads over the yolk cell.
47                                 Furthermore, yolk colour shifted from yellow to a more intense red co
48 ight, eggshell strength, eggshell thickness, yolk colour, albumen height) and performance of laying h
49 ngth, eggshell thickness, albumen height and yolk colour.
50 ika, used in animal husbandry to enhance egg yolk colour.
51 y, a non-destructive technique, can identify yolk components and detect changes in the matrix.
52                                              Yolk components endocytosed by LRP2-cubilin-amnionless,
53 ot significantly modified by the presence of yolk components.
54                            Average SigmaPBDE yolk concentrations in eggs from the Taizhou farms range
55 ion times of proton pools representing major yolk constituents.
56 m birds, reptiles and teleost fish whose egg yolk contain phosvitin.
57 ll type and not differences in pigmentation, yolk content, cell size, or position in the embryo.
58 eductions of ovary size, egg production, and yolk deposition in mature oocytes.
59            Together, these data suggest that yolk-derived E2 sets the ventral boundary of hemogenic v
60 eformed and newly formed lipid droplets, and yolk-derived very low density lipoprotein, shown to be e
61  and then on fresh and dried egg albumen and yolk down to 2.10(^4) and 1.10(^5) dilution factors, res
62           Changes in SOD activity of the egg yolk during its storage for 200days were also described.
63 aken up from the blood plasma, together with yolk, during oocyte growth and maturation, potentially c
64 on defects predominated, suggesting that the yolk expression of these genes rather than heart express
65                         Raman spectra of the yolk extracts were recorded in the range from 3100 to 99
66 rgic (71.3%): 29 reacted to CE, seven to egg yolk (EY) and 22 to egg white (EW) and 38 reacted to RE.
67                                  Egg weight, yolk fat content, yolk weight, yolk percentage and shape
68 ere measured above LOD (0.3 mug kg(-)(1)) in yolk for both doses on day 37 post-treatment.
69 ells are not protrusion-specific but reflect yolk-free cytoplasm and are quantitatively matched by th
70                             When the mass of yolk-free proteins in single blastomeres decreased from
71 ubule forces that probe blastomere shape and yolk gradients, biased by cortical polarity domains, may
72                          However, increasing yolk granule size did not slow their velocity, and the F
73 or detoxification and to growing oocytes for yolk granules and as a source of heme for embryo develop
74 e (1) compare the FA composition of CLA-rich yolk granules and plasma, relative to standard control a
75 of the embryo from the meiotic spindle while yolk granules are transported throughout the embryo by k
76 is study, phosvitin was first extracted from yolk granules using 10% NaCl, dialysed and then adjusted
77 ganelles were transported as a unit with the yolk granules, resulting in sperm DNA within 2 microm of
78 eir velocity, and the F-actin moved with the yolk granules.
79                  Cu,Zn-SOD isolated from egg yolk had an optimum at pH 6.
80              A stability test indicated that yolk had varied matrix mobility within the same sample a
81 d composition and cholesterol content in egg yolk has been evaluated during 13weeks of experiment.
82 shared genetic signals, maternally-deposited yolk hormones or other transient influences received pri
83 tides from casein variants, nonfat milk, egg yolk, human serum and HeLa cell extract.
84  PC molecular species are present inside the yolk in addition to the blastodisc, while PE and PI spec
85 Haugh unit, albumin pH, thick albumin ratio, yolk index, air cell size, and S-ovalbumin content of UC
86                                Catabolism of yolk is initiated by acidification of the yolk platelet,
87 ified as Phacellophora camtschatica (the egg-yolk jelly).
88       Concurrent with the appearance of "egg-yolk lesions," the OCT showed a cleft in the outer retin
89  and protons of lipids (i.e., protons of egg yolk lipids and amorphous lipid fraction of margarine) r
90 d 94% at 35 degrees C when compared with egg yolk lipids extract.
91                                          Egg yolk lipids were extracted with food grade ethanol and e
92 the CLnA concentrations, but also CLA in egg-yolk lipids.
93                 Egg preparations (egg white, yolk, liquid whole egg) were treated with UV-C (254 nm,
94 secondary structure of a newly developed egg yolk livetin formulation and its components alpha-liveti
95                                          Egg yolk low density lipoprotein (LDL)/polysaccharide nanoge
96 oocytic yolk, acquire the ability to take up yolk macromolecules during the vitellina-to-vasculosa tr
97 ased but different n-3 LC-PUFA levels in egg yolk, mainly docosahexaenoic acid enrichment.
98 f fat in winter resulted in smaller relative yolk mass in larger eggs and reduced yolk carotenoid con
99                                          CLA-yolk mayonnaise was more viscous, had greater storage mo
100 nd (3) compare the emulsion stability of CLA-yolk mayonnaise.
101  1200ngg(-1) in individual egg white and egg yolk, measured over 2days.
102 est demonstrated high repeatability of fresh yolk measurements, but significant differences (p<0.05)
103 d), and the efficacy of solid (Herrold's egg yolk medium [HEY]) and liquid (Bactec 12B and para-JEM)
104 uction of hspb7 or hspb12 was limited to the yolk, migration defects predominated, suggesting that th
105     Here we show that when injected into the yolk of a 2 day-post-fertilization (dpf) embryo of Tg (f
106                                              Yolks of 80 eggs were analysed as aliquots by the three
107  with a single injection of glucose into the yolk on day 0.
108 relative to mayonnaise prepared with control yolks or soy control yolks.
109 n egg presence in the extract, i.e. albumen, yolk, or their co-presence in the matrix.
110 osensor to simultaneously recognize albumen, yolk, or their mixtures in HVPD extracts.
111   Egg weight, yolk fat content, yolk weight, yolk percentage and shape index were not influenced by d
112 ch studied the enzymatic modification of egg yolk phospholipids and its effect on physicochemical pro
113                                          Egg yolk phosvitin is one of the most highly phosphorylated
114                                          Egg yolk phosvitin serves as a warehouse to provide metal io
115  However, the fatty acid (FA) composition of yolk plasma and granule fractions are unreported.
116 s between the forming basal membrane and the yolk plasmalemma.
117 stal tubular marker 4A6 and an impairment of yolk platelet degradation.
118 of yolk is initiated by acidification of the yolk platelet, leading to the activation of Cathepsin-li
119 cked into membrane-bound compartments called yolk platelets.
120 he parameters tested were: whole egg vs. egg yolk, polyunsaturated fatty acid (PUFA) enrichment, spra
121  oxidative stability of control and enriched yolk powders as revealed by a moderate increase of the q
122 ences between the odour of whole-egg and egg-yolk powders as well as between powders produced on an i
123 g temperature do not affect the odour of egg yolk powders.
124  In healthy colonies, the interaction of the yolk precursor protein, vitellogenin (Vg), and endocrine
125 rnesoate suppresses transcript levels of the yolk precursor Vitellogenin both in cell culture and in
126                        Vitellogenin, the egg yolk precursor, is a well-known biomarker of endocrine d
127 roteins include vitellogenins, which are egg yolk precursors and pathogen pattern recognition recepto
128 enormous size because of a massive uptake of yolk precursors such as very low density lipoprotein and
129 p- and down-regulation of female-specific Ds-Yolk protein 1 (Ds-Yp1) gene expression by temperature s
130  oocytes and the synthesis and deposition of yolk protein [10, 11].
131 ryo development cellularization and vitellin yolk protein degradation, processes that normally occur
132 reducing E2-dependent vitellogenin (VTG; egg yolk protein precursor) synthesis, (b) VTG-dependent egg
133 at body and subsequent accumulation of these yolk protein precursors by developing oocytes.
134 he receptor complex, competent for uptake of yolk proteins, is produced by EECs in the area vasculosa
135                                              Yolk provides an important source of nutrients during th
136 Y are selected as analytes for egg white and yolk recognition, respectively.
137 ally arrested larvae that produce sperm from yolk reserves.
138 denocarcinoma (PDAC) originate from both the yolk sac (YS) and bone marrow.
139 ro MF-depletion strategy and fate-mapping of yolk sac (YS) and fetal liver (FL) hematopoiesis.
140                               During chicken yolk sac (YS) growth, mesodermal cells in the area vascu
141 enitor/colony-forming cells of the embryonic yolk sac (YS), which are endowed with megakaryocytic pot
142 lose association in the blood islands of the yolk sac (YS).
143 cluding impaired vascular development in the yolk sac (YS).
144 of specific myeloid cell progenitors of both yolk sac and bone marrow origin.
145 rom erythromyeloid progenitors (EMPs) in the yolk sac and develop in the forming CNS.
146 CNS, originate during embryogenesis from the yolk sac and enter the CNS quite early (embryonic day 9.
147 ion of cancer cells after injection into the yolk sac and extravasation of cancer cells into tissues
148 sful establishment of nascent vasculature in yolk sac and in the developing embryos.
149 d extraembryonic ectoderm, as well as in the yolk sac and labyrinth tissues that form later.
150 types in normal embryos, as well as in E13.5 yolk sac and labyrinth.
151 ineage tracing revealed that the majority of yolk sac and many adult hematopoietic cells derive from
152 nd visualized at embryonic day (E)9.0 in the yolk sac and neuroectoderm; 2) at E10.5, CX3CR1 single-p
153 ainly from progenitor cells generated in the yolk sac and of 'passenger' or 'transitory' myeloid cell
154 delete YY1 from the visceral endoderm of the yolk sac and the definitive endoderm of the embryo.
155 extensive arterial morphogenesis both in the yolk sac and the embryo proper and disrupted arterial-ve
156 ath had markedly deformed vasculature of the yolk sac and the embryo, as well as poorly looped hearts
157   We demonstrate that DPFCs originate in the yolk sac and then rapidly migrate to other extra- and in
158                          EMPs develop in the yolk sac at embryonic day (E) 8.5, migrate and colonize
159 otential in vitro have been described in the yolk sac before emergence of HSCs, and fetal macrophages
160  the hemangioblast as the cell of origin for yolk sac blood and endothelium.
161        Primitive hematopoiesis occurs in the yolk sac blood islands during vertebrate embryogenesis,
162 sites of embryonic hematopoiesis such as the yolk sac by way of blood flow.
163 ion, bi-laminar disc formation, amniotic and yolk sac cavitation, and trophoblast diversification.
164 d Gpx3 in the same vesicles of d-13 visceral yolk sac cells, suggesting uptake by pinocytosis.
165  In differentiating mouse ES cells and mouse yolk sac cultures, addition of Indian Hh ligand increase
166                 cKO embryos display profound yolk sac defects at 9.5 days post coitum (dpc), includin
167 roglia in the brain that originates from the yolk sac during early development.
168 increase in the incidence of pericardial and yolk sac edema relative to controls.
169 n dilbit WAF-exposed embryonic zebrafish but yolk sac edema was similar in all exposures.
170 of toxicity included pericardial, ocular and yolk sac edema, nondepleted yolk, spinal curvature, tail
171 erm cell tumor predominantly consisting of a yolk sac element (Fig 1).
172  identify, in the fetal liver, a sequence of yolk sac EMP-derived and HSC-derived haematopoiesis, and
173 and HSC-derived haematopoiesis, and identify yolk sac EMPs as a common origin for tissue macrophages.
174 erize an ontogenic process of blood cell and yolk sac endothelial maturation that is required to disp
175 tissue-resident macrophages are derived from yolk sac erythromyeloid progenitors and fetal liver prog
176 ne-restricted potential originating from the yolk sac even before the emergence of the first hematopo
177 i, the exocoelomic cavity, and the secondary yolk sac function together as a physiological equivalent
178 OX7 is broadly expressed across the RUNX1(+) yolk sac HE population compared with SOX17.
179 ly well studied, the molecular regulation of yolk sac HE remains poorly understood.
180  specification in the dorsal aorta, enhanced yolk sac hematopoiesis, and exuberant cardiac blood isla
181 show that TRPM6 activity in the placenta and yolk sac is essential for embryonic development.
182                                          The yolk sac is phylogenetically the oldest of the extraembr
183 e species indicates that the human secondary yolk sac likely performs key functions early in developm
184                                 In a chicken yolk sac membrane model, under the same ultrasound param
185 feration were confined to the YY1-expressing yolk sac mesoderm indicating that loss of YY1 in the vis
186 escued angiogenesis and apoptosis in the cKO yolk sac mesoderm, but also restored the epithelial defe
187 ponsive paracrine signal, originating in the yolk sac mesoderm, is required to promote normal viscera
188 eral endoderm causes defects in the adjacent yolk sac mesoderm.
189     The vascular structure was absent in the yolk sac of Drosha homozygotes at E14.5.
190 and TC-(57)CoB12 accumulated in the visceral yolk sac of KO mice where megalin is expressed and provi
191  progenitors (EMPs) that first appear in the yolk sac of the early developing embryo.
192 (0)-resins can be carefully implanted in the yolk sac of zebrafish embryos and display excellent bioc
193 y give rise to chorio-allantoic and visceral yolk sac placentae, respectively.
194        Resident macrophages are derived from yolk sac precursors and seed the liver during embryogene
195 tiple progenitor pools, microglia arise from yolk sac progenitors and are widely considered to be equ
196 ng early embryogenesis, microglia arise from yolk sac progenitors that populate the developing centra
197  endoderm (PrE), which forms extra-embryonic yolk sac tissues.
198  associated with worse outcome, whereas pure yolk sac tumor (YST) was associated with better outcome,
199 s differentiated derivatives, teratoma (TE), yolk sac tumor (YST), and choriocarcinoma.
200 my, which revealed a 5-cm tumor that was 95% yolk sac tumor and 5% embryonal carcinoma, and retroperi
201 of Mexican-born mothers had a higher risk of yolk sac tumors (HR, 1.46; 95% CI, 0.99-2.17), while chi
202 d major dysplasia and malignant tumors, with yolk sac tumors and embryonal carcinomas positive for al
203 ial lipodystrophy and a history of childhood yolk sac tumour.
204        Several of these mutants also display yolk sac vascular defects, suggesting a role for thrombi
205 ial for normal growth and development of the yolk sac vasculature.
206                                  N629D/N629D yolk sac vessels and aorta consist of sinusoids without
207 e the ability to roll and adhere on inflamed yolk sac vessels during late fetal development, whereas
208 g, adhesion, and extravasation from inflamed yolk sac vessels is apparent late in development, but th
209 hen the blastoderm begins to spread over the yolk sac, a process involving coordinated epithelial sur
210 it is physiologically expressed in the fetal yolk sac, a tissue derived from the extraembryonic endod
211 n leads to vascular defects in the placenta, yolk sac, and embryo proper, as well as abnormal neural
212 o found in other embryonic niches (placenta, yolk sac, and extraembryonic vessels), attempts to detec
213 ascular abnormalities in the lung, placenta, yolk sac, and retina.
214 ion of target transcripts in placenta and/or yolk sac, and that some of these would be important for
215 evelop adjacent to blood vessel walls in the yolk sac, aorta-gonad-mesonephros region, embryonic live
216 ut the same in both WT and KO mouse visceral yolk sac, brain, and spinal column.
217 or erythro-myeloid progenitors (EMPs) in the yolk sac, but it decreased the expression of alpha4-inte
218                  Required histology included yolk sac, embryonal carcinoma, or choriocarcinoma.
219 primitive endoderm (PE), which will form the yolk sac, is a crucial developmental decision.
220 ptor 2(+) macrophages derived from primitive yolk sac, recombination activating gene 1(+) lymphomyelo
221 uced levels of VEGFA are observed in the cKO yolk sac, suggesting a cause for the angiogenesis defect
222 ells, identifying the earliest stages in the yolk sac, throughout embryonic development and in all ad
223  into the extra-embryonic region to form the yolk sac, umbilical cord and placenta.
224                   The human embryo retains a yolk sac, which goes through primary and secondary phase
225                                Functionally, yolk sac-derived chemokine (C-C motif) receptor 2(-) mac
226 ctor Runx1 is essential for the formation of yolk sac-derived erythroid/myeloid progenitors (EMPs) an
227 with different ontogenetic origins: prenatal yolk sac-derived Kupffer cells and peripheral blood mono
228 , red pulp macrophages, a discrete subset of yolk sac-derived macrophages, were found to be altered i
229 irculating adult monocytes or from primitive yolk sac-derived macrophages.
230 The recent paradigm shift that microglia are yolk sac-derived, not hematopoietic-derived, is reshapin
231 e made earlier, that displayed labyrinth and yolk sac-specific defects, but our findings extend those
232  protein is a rodent-specific, placenta- and yolk sac-specific member of the tristetraprolin (TTP) fa
233 he placenta, and the epithelial cells of the yolk sac.
234 lood vessel formation in the embryo body and yolk sac.
235 om Mesp1-Cre(+) cells in both the embryo and yolk sac.
236                    Predominant histology was yolk sac.
237 m which is derived the embryo proper and the yolk sac.
238 n both the labyrinth of the placenta and the yolk sac.
239 isceral endoderm defects observed in the cKO yolk sac.
240 y in trophoblast giant cells in the parietal yolk sac.
241 omic data for the coelomic fluid bathing the yolk sac.
242 xtensively self-renew and can be seeded from yolk sac/foetal liver progenitors with little input from
243           Genetic fate mapping revealed that yolk-sac and fetal monocyte progenitors gave rise to the
244 tic activity in mammalian development is the yolk-sac blood island, which originates from the hemangi
245           This study demonstrates that later yolk-sac Chinook larvae (before exogenous feeding) are e
246 cells that develop during organogenesis from yolk-sac erythro-myeloid progenitors (EMPs) distinct fro
247 We show that the previously reported lack of yolk-sac hematopoiesis and vascular development in Ldb1(
248 nadal macrophages are derived from primitive yolk-sac hematopoietic progenitors and exhibit hallmarks
249  in European sea bass (Dicentrarchus labrax) yolk-sac larvae was explored.
250 ty of organs in which substantial numbers of yolk-sac macrophages persisted in adulthood.
251 cing (RNA-seq) data for the human and murine yolk sacs and compare those data with data for the chick
252                     The defects in Osr1-null yolk sacs and embryos were virtually identical to those
253 Aggf1+/- KO caused defective angiogenesis in yolk sacs and embryos.
254 nt, have aberrant vasculogenesis in embryos, yolk sacs and placentas, and die between embryonic day 1
255                       In the absence of Eng, yolk sacs inappropriately express the cardiac marker, Nk
256 1-knock-out mice: no mature large vessels in yolk sacs, defective angiogenesis in the brain and inter
257 cripts, whereas there were few changes in KO yolk sacs.
258                                  For all egg yolk samples and all NaCl-containing LDL samples, the si
259 ) subtype avian influenza virus (AIV) in egg yolk samples, and 45% had antibodies against different A
260 ifferences (p<0.05) were found within gelled yolk samples.
261                                          Egg yolk seemed to play a double role on reactivity: protect
262  phase in oil-in-water emulsions, from which yolk-shell and dual-shell hollow SiNPs@C composites are
263                          Taken together, the yolk-shell Fe3 O4 @Au NPs can be regarded as an ideal ma
264                                          The yolk-shell material exhibited a stable capacity of 765 m
265 e demonstrate the design of a sulphur-TiO(2) yolk-shell nanoarchitecture with internal void space to
266                               Here by making yolk-shell nanocomposite of aluminium core (30 nm in dia
267 report the synthesis of a polyaniline-sulfur yolk-shell nanocomposite through a heating vulcanization
268                                              Yolk-shell nanostructures (YSNs) composed of a core with
269 fur-polyaniline core-shell counterparts, the yolk-shell nanostructures delivered much improved cyclab
270                                          The yolk-shell nanostructures possess the functions of nanop
271 such as porous solid spheres, core-shell and yolk-shell particles as well as single-shell and multi-s
272 ed by the cup-shaped void space created by a yolk-shell silica template that spontaneously takes an e
273                    To circumvent this issue, yolk-shell structured MPHNs comprising a Fe3 O4 core wit
274 ve than chemical leaching to prepare uniform yolk-shell structures.
275 he shell is continuous, but it transforms to yolk-shell, with neither sacrificial template nor core e
276        To address such issues, well-designed yolk-shelled carbon@Fe3 O4 (YSC@Fe3 O4 ) nanoboxes as hi
277 pective to design a carbon/metal-oxide-based yolk-shelled framework as a high sulfur-loading host for
278 framework-67/Ni-Co layered double hydroxides yolk-shelled structures are first synthesized and then t
279                          The presence of egg yolk slightly increased the susceptibility to hydrolysis
280 oyloleoylphosphatidylcholine (POPC)) and egg-yolk sphingomyelin (EYSM) lipids, and allowed us to extr
281 dial, ocular and yolk sac edema, nondepleted yolk, spinal curvature, tail malformations, and craniofa
282 quantitatively identify carotenoids from egg yolk such as spectrophotometric methods described by AOA
283             Here, we show that the zebrafish yolk syncytial layer (YSL) secretes a ventralizing signa
284           Targeted depletion of Hspb7 in the yolk syncytial layer is sufficient to disrupt KV morphol
285 an being attracted to their destination, the yolk syncytial layer, cells appear to migrate away from
286 ressed in the precardiac mesoderm and in the yolk syncytial layer, which supports the migration and f
287 vere YSL defects including clustering of the yolk syncytial nuclei and enhanced cell fusion, accompan
288 nfirming the capability to differentiate egg-yolk tempera from other kind of tempera binders as well
289 fat, which appears to derive from unconsumed yolk that was produced for reproduction.
290            Between the nuclear layer and the yolk, the cytoplasm was homogeneous and viscously domina
291    We used an N-glycan isolated from hen egg yolk together with the Nbz linker for Fmoc chemistry sol
292 ve tool to determine total carotenoid of egg yolk under laboratory-independent conditions with little
293 n of the developing YS with the induction of yolk uptake and lipoprotein secretion by EECs to ensure
294          Lay date, clutch size, egg mass and yolk vitamin E concentrations were not significantly aff
295                  Average SOD activity in egg yolk was 98.5+/-19.5U.g(-1) while in egg white reached 6
296 olk, whereas the hardness of hard-boiled egg yolks was not affected.
297  egg powder and its fractions (egg white and yolk) was developed by combining microwave-induced combu
298                             To determine egg yolk we focus here on the determination of immunoglobuli
299                Egg weight, yolk fat content, yolk weight, yolk percentage and shape index were not in
300 Cu,Zn-SOD derived from hen egg white and egg yolk were determined, and compared with those of enzymes
301 valbumin, egg white, whole egg, defatted egg yolk, wheat albumins and wheat globulins were detected.
302 d positive impact on the colour of the eggs' yolk, whereas the hardness of hard-boiled egg yolks was

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