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1 e time points (first grade, adolescence, and young adulthood).
2 inct age groups (childhood, adolescence, and young adulthood).
3 tly predicted preclinical atherosclerosis in young adulthood.
4 s transition to a relatively stable state by young adulthood.
5 sk of developing depression and psychosis in young adulthood.
6 ipation of monetary reward from childhood to young adulthood.
7 ring adolescence compared with childhood and young adulthood.
8 annabis use are wide ranging and extend into young adulthood.
9 ed to juvenile monkeys upregulates SERT into young adulthood.
10 ce exposed to CAPs, and it persisted through young adulthood.
11 ys on a long-term ~30% restricted diet since young adulthood.
12 he induction of EAE during the age window of young adulthood.
13 s that FFA is associated with IR starting in young adulthood.
14 ood, and internalizing symptoms, assessed in young adulthood.
15  prospectively followed up from childhood to young adulthood.
16 nction plays a major part in adolescence and young adulthood.
17 upwardly mobile minorities is compromised in young adulthood.
18 e exacerbated high-frequency hearing loss in young adulthood.
19  caused behavioral deficits that manifest in young adulthood.
20 veloping bipolar disorder in adolescence and young adulthood.
21 d reduced quality of life in adolescence and young adulthood.
22 urse of self-harm from middle adolescence to young adulthood.
23 h in early childhood and body composition in young adulthood.
24 isk of Hodgkin lymphoma in childhood through young adulthood.
25 ancy (<3 months) with any outcome measure in young adulthood.
26 ns during memory retrieval also develop into young adulthood.
27 cy was associated with insulin resistance in young adulthood.
28 stems in the human brain from childhood into young adulthood.
29 t periodontal outcomes in adolescence and in young adulthood.
30 chronic illness are reaching adolescence and young adulthood.
31 h increased mortality in early childhood and young adulthood.
32 d elevated blood pressure in adolescence and young adulthood.
33 r (OUD) frequently begins in adolescence and young adulthood.
34 in girls with and without ADHD followed into young adulthood.
35 t is often discovered during adolescence and young adulthood.
36 at ages 9, 11, and 13 and followed them into young adulthood.
37 des (>or=1.70 mmol/L [>or=150 mg/dL]) during young adulthood.
38 urance coverage for survivorship services in young adulthood.
39 avior that often develops in adolescence and young adulthood.
40 nd to have subsequent experiences of rape by young adulthood.
41  unclear whether these problems persist into young adulthood.
42 llowed longitudinally from childhood through young adulthood.
43 on and coherence dynamics that continue into young adulthood.
44 on-based reciprocity from early childhood to young adulthood.
45  development of DSM-IV alcohol dependence in young adulthood.
46 sive myelination dynamics that continue into young adulthood.
47 ng across life stages from childhood through young adulthood.
48 lopmentally dynamic from middle childhood to young adulthood.
49 en and adolescents with ADHD when they reach young adulthood.
50 sive myelination dynamics that continue into young adulthood.
51 cell aging during late fetal development and young adulthood.
52 and gradually declined in importance through young adulthood.
53 ough the periadolescent period and even into young adulthood.
54 D events and deaths is projected to occur in young adulthood.
55 ence interval [CI], 1.06-2.14; P< or=.05) in young adulthood.
56 ype as children progress from childhood into young adulthood.
57 hosocial work environment encountered during young adulthood.
58 bone fragility or osteoporosis recognized by young adulthood.
59 ne accretion among females from childhood to young adulthood.
60 ostnatal mouse cerebral cortex, lasting into young adulthood.
61 lescence (age 15-21) and psychoticism during young adulthood.
62 erythropoiesis were transient, correcting by young adulthood.
63 is primarily concentrated in adolescence and young adulthood.
64 d-stage renal disease (ESRD) in childhood or young adulthood.
65  among women who performed shift work during young adulthood.
66 n sibling loss during childhood and death in young adulthood.
67 uced myopia, particularly in adolescence and young adulthood.
68 ntal and mental health outcomes persist into young adulthood.
69 ges in dental caries from early childhood to young adulthood.
70 2 weeks of gestation and HF in childhood and young adulthood.
71 rly childhood, but also into adolescence and young adulthood.
72 ff enough in youth to alter BP levels during young adulthood.
73 n childhood and DSM-5 diagnostic criteria in young adulthood.
74 e increases substantially in adolescence and young adulthood.
75 nificant precursors of crime and violence in young adulthood.
76 demic performance throughout adolescence and young adulthood.
77 permits protein expression and survival into young adulthood.
78 me physical activity in late adolescence and young adulthood.
79  levels of depressive symptoms, persist into young adulthood.
80  Reactions to insect venom were seen more in young adulthood.
81 an important determinant of LTL, at least by young adulthood.
82 slipidemia and markers of atherosclerosis in young adulthood.
83 ngly healthy level of MVPA from childhood to young adulthood.
84 ompetence (eg, academic or interpersonal) in young adulthood.
85 stress, which often emerge in adolescence or young adulthood.
86 ril 1, 2002, through January 31, 2006) until young adulthood.
87 e development of asthma through childhood to young adulthood.
88 12 years), adolescence (12 to 18 years), and young adulthood (18 to 24 years).
89 ars old), adolescence (13-17 years old), and young adulthood (18-24 years old).
90 73 participants were assessed 3 times during young adulthood (19, 21, and 24-26 years of age, for a t
91 ion, or a suicide attempt), were assessed in young adulthood (19, 21, and 24-26 years) by use of stru
92 ctively examined the relationships of BMI in young adulthood (21 years of age) and adult obesity traj
93  childhood (ages 6, 11, and 15 years) and in young adulthood (22-25 years).
94 learly associated with incident self-harm in young adulthood (5.9, 2.2-16).
95  time periods: adolescence (27-46 d old) and young adulthood (60-79 d old).
96                                           In young adulthood, a genetic internalizing factor influenc
97 d the association between history of acne in young adulthood, a marker of hormone activity, and cause
98 ly during the transition from adolescence to young adulthood, a period characterised by a sharp rise
99 for body mass index (BMI) in adolescence and young adulthood, a period of high risk for weight gain,
100 age for depression and metabolic syndrome in young adulthood, across race/ethnicity.
101 re consistently obese during adolescence and young adulthood (adjusted odds ratio = 1.97, 95% confide
102 set in childhood (age 18 years or before) or young adulthood (age 19-45 years) was associated with al
103 ds ratios (OR) for overweight and obesity in young adulthood (age 20-29) were calculated using logist
104 rs for Hodgkin lymphoma in childhood through young adulthood (ages 0-37 years).
105                   Criminal offense status in young adulthood (ages 16 to 21) was ascertained through
106 across four distinct epochs of adulthood: 1) young adulthood (ages 18-25 years), adulthood (ages 26-4
107 , 0.95-1.03; P = .64) and then reappeared in young adulthood (ages 18-36 years: aHR, 0.96; 95% CI, 0.
108 and Adolescent Psychiatric Assessment and in young adulthood (ages 19 and 21 years) with the Young Ad
109 vering childhood/adolescence (ages 9-16) and young adulthood (ages 19 and 21).
110 tatus, sexual behavior, and mental health in young adulthood (ages 19 and 21).
111 ths) for symptoms of DMDD and three times in young adulthood (ages 19, 21, and 24-26 years; 3,215 obs
112  strong predictors of asthma incidence up to young adulthood (ages 20-27 years).
113 s maternal educational immunity such that by young adulthood, all immune cells responding to a foster
114           Asthma with onset in childhood and young adulthood also was associated with a greater %LAA
115  and behavioral problems from adolescence to young adulthood among individuals with and without food
116  smoking during pregnancy and tobacco use in young adulthood, an association that was largely concent
117 s shown in this paper and elsewhere, between young adulthood and >70 y of age the effective clonal di
118                   Persistence of tics beyond young adulthood and a previous suicide attempt were the
119 amine the association between body weight in young adulthood and achieved fertility in later life.
120 es that evaluated the associations of BMI in young adulthood and adult weight trajectory with mortali
121 cular health from birth through childhood to young adulthood and beyond.
122 intake of fruits and vegetables (F/V) during young adulthood and coronary atherosclerosis later in li
123 and parietal cortices during adolescence and young adulthood and discrepantly accelerated age-related
124 ng heritability from early childhood through young adulthood and females exhibiting decreasing herita
125 re commonly associated with poor outcomes in young adulthood and in midlife, were largely unrelated t
126 red strain begins at high frequencies during young adulthood and is severe by 12 months (middle age).
127               Ideal cardiovascular health in young adulthood and its maintenance to middle age is ass
128 evaluate the association between early life, young adulthood and later adulthood SEP with each inflam
129  link between regional aerobic glycolysis in young adulthood and later development of Alzheimer patho
130                        Overweight/obesity in young adulthood and middle age has long-term adverse con
131 er number of ideal cardiovascular metrics in young adulthood and middle age were independently associ
132 actors that powerfully influence outcomes in young adulthood and midlife, this long-term prospective
133 mice to methamphetamine neurotoxicity during young adulthood and possibly increase age-related deteri
134 nce of airborne allergen sensitization up to young adulthood and risk factors for early and late onse
135 een cumulative exposure to hyperlipidemia in young adulthood and subsequent CHD risk (adjusted hazard
136 se HBsAg seroprevalence in childhood through young adulthood and subsequently reduce the risk of PLC
137 different phenotypic clusters persisted into young adulthood and that these clinical differences were
138 at human cognitive function improves through young adulthood and then declines across the later life
139 gratification improves between childhood and young adulthood and this improvement is predicted by the
140 pressure (BP) trajectories from childhood to young adulthood and to examine whether this relation is
141  between undernutrition during childhood and young adulthood and type 2 diabetes in adulthood.
142 rtality risk of being overweight or obese in young adulthood and underweight or obese in later life.
143 -onset depression and development of T2DM in young adulthood and whether the association differs by s
144 ing mortality (during infancy and throughout young adulthood) and psychiatric (psychotic or bipolar d
145 n institution (n=67 at ages 6 years; n=50 at young adulthood) and UK controls (n=52 at age 6 years; n
146  night vision in adolescence, side vision in young adulthood, and central vision in later life becaus
147 d swelling and rapid neurological decline in young adulthood, and had marked hydrocephalus of the lat
148 s that affect development in adolescence and young adulthood, and influence educational attainment an
149 onnectivity during an attention bias task in young adulthood, and internalizing symptoms, assessed in
150 sed over a wide age range from birth through young adulthood, and older age at diagnosis is associate
151 worsen as pediatric patients transition into young adulthood, and that SES does not explain all of th
152  in reporting their weight as they move into young adulthood, and we examined differences in self-rep
153             Healthy lifestyle changes during young adulthood are associated with decreased risk and u
154 ther lower energy and higher PUFA intakes in young adulthood are associated with shorter LTL in cross
155 mal levels of LDL and HDL cholesterol during young adulthood are independently associated with corona
156 ut the consequences of lipid exposure during young adulthood are unclear.
157  consequences of low-level elevations during young adulthood are unknown.
158 e stories begin to emerge in adolescence and young adulthood, as the person authors a narrative ident
159 15, 2.99) adolescent girls who were obese by young adulthood, as well as young women who were consist
160    Because bone mass accrual is completed by young adulthood, assessing stimulant effects on bone den
161 nces at various ages, including migration in young adulthood associated with entering and leaving col
162 icantly in CNS through early development and young adulthood before stabilizing.
163        Common, mild disorders often remit in young adulthood, but more severe disorders can become ch
164 may relate to aspects of physical fitness in young adulthood, but replication of these findings is re
165 was recently shown that increasing age after young adulthood can cause hypothalamic microinflammation
166                         Late adolescence and young adulthood coincide with greater mortality hazard a
167 r at five points in adolescence and three in young adulthood, commencing at a mean age of 15.5 years
168 isorder, were more likely to be depressed in young adulthood compared to their counterparts.
169 smaller effect on BMI during adolescence and young adulthood compared with older adults (P < 0.05).
170 displayed a greater increase in BP levels in young adulthood compared with their counterparts without
171 ent and tracking of lung function throughout young adulthood could be used to provide an early warnin
172 ted that depressive symptoms were highest in young adulthood, decreased across middle adulthood, and
173 ith increased odds of depressive symptoms in young adulthood, especially in young men.
174 h was associated with increased mortality in young adulthood even among individuals born late preterm
175 eparation examined during postnatal life and young adulthood exhibited enhanced hippocampal neurogene
176 in childhood and healthier lifestyles during young adulthood explained most of the association betwee
177 es a lethal phenotype consisting of death in young adulthood, extensive damage to the intestine, impa
178 problems were wide ranging but diminished by young adulthood for all but a small group.
179 tes during adolescence and the transition to young adulthood for patients with type 1 diabetes.
180 The mortality risk was 2-fold higher through young adulthood for persons with ASD than for persons wi
181           How these associations evolve from young adulthood forward is unknown.
182  additional adjustment for maximum weight in young adulthood further attenuated this association (mul
183                                           In young adulthood, gestational age at birth had the strong
184                                           By young adulthood, girls with ADHD were at high risk for a
185 ho lost a parent to suicide in childhood and young adulthood had earlier onset of hospitalization for
186 n institution (n=98 at ages 6 years; n=72 at young adulthood) had persistently higher rates than UK c
187 nd UK controls (n=52 at age 6 years; n=39 at young adulthood) had similarly low levels of symptoms ac
188 ss index (BMI; weight (kg)/height (m)(2)) in young adulthood have been associated with glioma risk, b
189 al obesity and hypo-alpha-lipoproteinemia in young adulthood, hypertension and hypertriglyceridemia i
190 lipoproteinemia, and hypertriglyceridemia in young adulthood, hypertension in middle age, and diabete
191 cation are maintained across adolescence and young adulthood, implicating developmentally dissociable
192 of achieving the developmental milestones of young adulthood, important work remains to be done in th
193 ctural impairments may be normalizing during young adulthood in ASD.
194 authors analyzed outcomes in adolescence and young adulthood in girls in a longitudinal study.
195 th aging suppresses disease onset after late young adulthood in mice.
196 ted were at increased risk for depression in young adulthood in models adjusted for sex and family ha
197 nce, and the possibility of its emergence in young adulthood in nonclinical populations.
198 tacks in adolescence and psychoticism during young adulthood in the community.
199 in cerebellar volume between adolescence and young adulthood in VPT individuals, which is correlated
200 y matter density increases from childhood to young adulthood, in contrast with gray matter volume and
201                     However, mice survive to young adulthood, in part from Snf2l compensation that re
202  is strongly associated with hypertension in young adulthood, including an increased risk among those
203 e-like patterns of excessive drinking during young adulthood increase the propensity for alcohol use
204 unburns, particularly during adolescence and young adulthood, increase the risk of developing skin ca
205     Cumulative exposure to hyperlipidemia in young adulthood increases the subsequent risk of CHD in
206                The incidence of IBD peaks in young adulthood indicating that pediatric environmental
207  the association of body mass index (BMI) in young adulthood, into middle age, and through late life
208 ce of anti-double-stranded DNA antibodies in young adulthood, intravascular deposition of immunoglobu
209             Heavy alcohol consumption during young adulthood is a risk factor for the development of
210 y cortical thickness, during adolescence and young adulthood is also aberrant.
211 ze that higher intake of these B vitamins in young adulthood is associated with better cognition late
212        These results suggest that obesity in young adulthood is associated with higher mortality risk
213 ther adopting a healthy lifestyle throughout young adulthood is associated with the presence of the l
214                       Prehypertension during young adulthood is common and is associated with coronar
215                                     Although young adulthood is often characterized by rapid intellec
216   Maintaining a healthy lifestyle throughout young adulthood is strongly associated with a low cardio
217 t (kg)/height (m)(2)) and premature death in young adulthood is very limited, especially for specific
218 nol but not continuous ethanol or water from young adulthood leads to an increase in DOP-R activity i
219           Greater cardiopulmonary fitness in young adulthood, less decline in fitness from young adul
220     Results suggest that being overweight in young adulthood may be associated with a lower prevalenc
221  that the process of synaptic pruning during young adulthood may be reflected in decreased rCPS.
222     Maintaining high activity levels through young adulthood may lessen weight gain as young adults t
223 or at least delaying the onset of obesity in young adulthood may lower the risk of developing atheros
224  that smoking, especially in adolescence and young adulthood, may increase risk of moderate to severe
225 ciated with similarity in weight change from young adulthood (mean age: 20 y) to middle age (mean age
226 tic city in the mid-1980s were followed into young adulthood (mean age=21).
227 comparison baseline (mean age=9.89 years) to young adulthood (mean age=24.40 years).
228  responses to everyday stressors manifest in young adulthood, months before tumor development, and pe
229                                           In young adulthood, mortality rates (per 1000 person-years)
230                                           By young adulthood, most of these differences had attenuate
231 ximately 75% participated in an interview at young adulthood (n = 1692).
232 and the frequent emergence in their teens or young adulthood of myelodysplastic syndrome (MDS) and ac
233 he symptomatic and neuroanatomic effects, in young adulthood, of exposure to peer verbal abuse during
234 tial consequences of asthma in childhood and young adulthood on lung structure in older adults have n
235 to tobacco dependence syndrome in smokers by young adulthood: one nondependent class of smokers (50%
236     Participants who reported being obese in young adulthood only or in both young and middle adultho
237 ding which blood pressure (BP) components in young adulthood optimally determine cardiovascular disea
238 ce of polycystic ovary syndrome when used in young adulthood or adolescence.
239 d conditions that may have an onset early in young adulthood or to measurements that can fluctuate ov
240  symptoms that persisted from adolescence to young adulthood [OR = 2.05 (1.04, 4.03)].
241 o resided in the southern geographic tier in young adulthood (P for interaction = .07).
242 years (p=0.17), and then marked increases by young adulthood (p=0.0005), with similar effects seen fo
243 ompared with UK controls, to normal rates at young adulthood (p=0.76).
244 l, which ranged from postnatal day 0 (P0) to young adulthood (P45).
245 t of cardiovascular risk factors measured in young adulthood, particularly favorable (low-risk) profi
246  in cognitive performance in adolescence and young adulthood, particularly in verbal ability, is asso
247  and inattention and overactivity through to young adulthood (pooled p<0.0001 for all).
248 , respectively) and all anxiety subscales in young adulthood (r = 0.06-0.19).
249 excess risk of becoming tobacco-dependent by young adulthood (risk ratio = 1.6, 95% confidence interv
250 ical, and health outcomes in adolescence and young adulthood; role transitions, and later health and
251                                           In young adulthood, SBP in black individuals and DBP in whi
252  gene have functional brain abnormalities in young adulthood, several decades before the possible ons
253                                           By young adulthood, significant effects remained at metacar
254 nal age, not only in childhood but also into young adulthood, suggesting that growth factor pathways
255         The peak onset of these disorders in young adulthood suggests that epigenetic changes normall
256 r likelihood of having ongoing disorder into young adulthood than did those without such a background
257 tly more likely to develop severe obesity in young adulthood than normal-weight or overweight adolesc
258 vior changes that increase mortality through young adulthood that are stronger than the protective ef
259 f sudden death in childhood, adolescence and young adulthood that presents during fetal life, but is
260 djustment for modifiable risk factors during young adulthood, the disparity was no longer statistical
261                                           By young adulthood, the most highly-affected regions (hippo
262 individuals progressed through childhood and young adulthood, they occurred at similar rates for indi
263                                         From young adulthood through later life, blacks are also more
264  in increasing the risk of hypertension from young adulthood through middle age and into late life.
265 f the DOP-R in high ethanol consumption from young adulthood through to late adulthood by measuring D
266 defined by the American Heart Association in young adulthood to middle age and cognitive function in
267 le age, and achieving increased fitness from young adulthood to middle age are associated with less d
268 cumulative exposure to BP over 25 years from young adulthood to middle age is associated with incipie
269 oung adulthood, less decline in fitness from young adulthood to middle age, and achieving increased f
270 l LV hypertrophy and geometry increased from young adulthood to middle age.
271 rse regarding the natural history of ER from young adulthood to middle age.
272 ), we documented profound aging effects from young adulthood to old age (18-83 y) on neurocognitive a
273 same genes, but to differential levels, from young adulthood to old age.
274       Blood pressure trajectories throughout young adulthood vary, and higher BP trajectories were as
275 eported famine exposure during childhood and young adulthood was associated with an increased type 2
276 cores.Higher intake of B vitamins throughout young adulthood was associated with better cognitive fun
277           Asthma with onset in childhood and young adulthood was associated with increased mean airwa
278           Asthma with onset in childhood and young adulthood was associated with large decrements in
279 al cohort study, higher intake of F/V during young adulthood was associated with lower odds of preval
280            Asthma with onset in childhood or young adulthood was associated with reduced lung functio
281               Greater time spent outdoors in young adulthood was associated with risk of exfoliation
282                      Higher folate intake in young adulthood was longitudinally associated with a low
283               Obesity (BMI >/=30 kg/m(2)) in young adulthood was strongly associated with incident hy
284 ack-years and smoking during adolescence and young adulthood were also independently associated with
285 ring who lost a parent during adolescence or young adulthood were at greatest risk within 1 to 2 year
286 ange their properties during late puberty to young adulthood, when bone growth and accrual decelerate
287 ntiation of muscarinic excitation emerges in young adulthood, when executive function typically reach
288  risk for albuminuria and eGFRcys decline in young adulthood, whereas disparities between low-risk bl
289 ship networks are linked to risk for SV into young adulthood, which suggests that network-focused SV
290 omyelitis (EAE) during adolescence and early young adulthood, while an increase in immunological tole
291 al, socioeconomic, and behavioral factors in young adulthood with the observed racial disparity in di
292 ract most often diagnosed in adolescence and young adulthood, with a rising incidence in pediatric po
293 adolescence reported no further self-harm in young adulthood, with a stronger continuity in girls (13
294 idence of acne occurs during adolescence and young adulthood, with approximately 80% of persons aged
295 rder symptoms in childhood, adolescence, and young adulthood, with depression and anxiety becoming mo
296 prove symptomatically during adolescence and young adulthood, with less frequent remissions and incre
297 ntally dynamic from middle childhood through young adulthood, with males and females displaying diffe
298 -/-)) deficient in IL-18 were hyperphagic by young adulthood, with null mutants then becoming overwei
299  methylation (DNAm) in inflammatory genes in young adulthood would be predicted by early life nutriti
300 clerosis risk and change in risk assessed in young adulthood years before subclinical atherosclerosis

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