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1 for screening of androgenic compounds in the zebrafish embryo.
2 enes at the single cell level in whole-mount zebrafish embryo.
3 tic stem and progenitor cells (HSPCs) in the zebrafish embryo.
4 nd organizer specification in the developing zebrafish embryo.
5 collectively migrate along the trunk of the zebrafish embryo.
6 genes have unique expression patterns in the zebrafish embryo.
7 ures, that migrates from head to tail in the zebrafish embryo.
8 ce, was expressed in isolated neurons of the zebrafish embryo.
9 neurons in the developing spinal cord of the zebrafish embryo.
10 dividing cells and the neuromast organ of a zebrafish embryo.
11 g of hindbrain segments (rhombomeres) in the zebrafish embryo.
12 ditis elegans embryo and in the gastrulating zebrafish embryo.
13 genesis, both in vitro and in the developing zebrafish embryo.
14 xplain the enantioselectivity of fipronil to zebrafish embryos.
15 he use of human hematopoietic stem cells and zebrafish embryos.
16 and mitotic dysfunction compared to wildtype zebrafish embryos.
17 ed pesticide ziram is synuclein-dependent in zebrafish embryos.
18 markably decreased in ORF119L-overexpressing zebrafish embryos.
19 ivilege and transparent nature of developing zebrafish embryos.
20 is sufficient to expand definitive HSPCs in zebrafish embryos.
21 of fluorescently tagged BMP2b and Chordin in zebrafish embryos.
22 with pronephric cysts and microphthalmia in zebrafish embryos.
23 its overexpression results in excess PGCs in zebrafish embryos.
24 ng of sialylated glycoconjugates within live zebrafish embryos.
25 lyphenol compounds in a red wine extract and zebrafish embryos.
26 ells, xenografts of mice, budding yeast, and zebrafish embryos.
27 em and progenitor cell (HSPC) development in zebrafish embryos.
28 human ECs and during vascular development in zebrafish embryos.
29 y inducing a series of in-frame deletions in zebrafish embryos.
30 ific nuclear proteins in mammalian cells and zebrafish embryos.
31 n is that of primordial germ cells (PGCs) in zebrafish embryos.
32 hed in cytosolic puncta in ciliated cells in zebrafish embryos.
33 measuring the frequency of HR events in live zebrafish embryos.
34 both substrates for N-linked fucosylation in zebrafish embryos.
35 cdc80, as validated by functional studies in zebrafish embryos.
36 ffect was also stronger in ZFL cells than in zebrafish embryos.
37 l model and prevented CUG repeat toxicity in zebrafish embryos.
38 oderm accompanied by endodermal expansion in zebrafish embryos.
39 ells, cultured mouse embryos, and developing zebrafish embryos.
40 en Gata4 protein is depleted from developing zebrafish embryos.
41 y using stage-matched WT and eif3ha morphant zebrafish embryos.
42 al repressor for neural crest development in zebrafish embryos.
43 4(+) hematopoietic stem cells and in vivo in zebrafish embryos.
44 y neurons within the olfactory epithelium of zebrafish embryos.
45 elopment was impaired in MitoBloCK-6-exposed zebrafish embryos.
46 LLp terminates prematurely in lef1-deficient zebrafish embryos.
47 e proliferation of renal progenitor cells in zebrafish embryos.
48 imposed to trapped red blood cells of living zebrafish embryos.
49 ted with bioavailable silver ions in exposed zebrafish embryos.
50 oth systemic and localized infections within zebrafish embryos.
51 nositol (PI), in newly fertilized individual zebrafish embryos.
52 even signals in time-lapse imaging of living zebrafish embryos.
53 -like cells and corneal endothelium of early zebrafish embryos.
54 de larvae and embryos, Drosophila brain, and zebrafish embryos.
55 scale expression screening of human cDNAs in zebrafish embryos.
56 ein we report such a method using developing zebrafish embryos.
57 unction of the GOF TRPP2 was investigated in zebrafish embryos.
58 al cells, and analyzed expression pattern in zebrafish embryos.
59 tumor suppression in basal keratinocytes of zebrafish embryos.
60 ed transient gene knockdown was performed in zebrafish embryos.
61 ion, and led to potent angiogenic defects in zebrafish embryos.
62 xpression analysis and time-lapse imaging of zebrafish embryos.
63 ription factors in real-time in gastrulating zebrafish embryos.
64 an AKAP, recruits PKA RI to primary cilia in zebrafish embryos.
65 FISH method that is suitable for whole-mount zebrafish embryo, a popular vertebrate model organism fo
67 ile manipulation of protein function in live zebrafish embryos, a transparent and commonly used model
68 uantifies spontaneous activity within single zebrafish embryos after exposure to test chemicals in a
74 lso occurs in vivo in two different systems: zebrafish embryos and adult rats, indicating that this N
75 tant role for zebrafish Mate transporters in zebrafish embryos and adults and provide a basis for det
79 expression profile analysis in human cells, zebrafish embryos and C. elegans that were individually
80 ide transcriptional analysis of MeHg-exposed zebrafish embryos and combined this with a whole-mount i
81 of-function effect by functional analyses in zebrafish embryos and cultured hippocampal neurons from
82 an be carefully implanted in the yolk sac of zebrafish embryos and display excellent biocompatibility
83 e required to maintain Warburg metabolism in zebrafish embryos and in both primary and malignant mamm
84 ivate a typical human oncogene, kRASG12V, in zebrafish embryos and investigate the developmental and
86 Here we report genetic code expansion in zebrafish embryos and its application to the optogenetic
87 metry has been applied for the first time to zebrafish embryos and larvae to study five neurotransmit
88 mouse embryos, and excess BMP2 signaling in zebrafish embryos and mouse embryonic stem cell-derived
89 ding the ability to form fibrotic lesions in zebrafish embryos and mouse lungs, and a transcriptional
90 ergence and extension movements in wild-type zebrafish embryos and mutants for the Wnt/PCP core compo
91 in zebrafish liver cells (ZFL) cells and in zebrafish embryos and novel insights into their molecula
93 increased MG concentrations in tg(fli:EGFP) zebrafish embryos and rapidly induced several additional
94 a set of genes that are highly expressed in zebrafish embryos and systematically analyzed for enrich
95 these advances to deliver BE3 RNPs into both zebrafish embryos and the inner ear of live mice to achi
96 -EGFP in cerebellar cells of live transgenic zebrafish embryos and the role of palmitoylation in its
97 ocal imaging, parabiotic surgical pairing of zebrafish embryos, and blastula transplantation assays,
98 ls lacking BBS1, BBS4, and OFD1, in morphant zebrafish embryos, and in induced neurons from Ofd1-defi
99 ubcellular behaviours in live C. elegans and zebrafish embryos, and show how TLS-SPIM can facilitate
100 ecting mRNAs in cell culture and whole-mount zebrafish embryos, and that combined with SPIM and PACT
101 tic chemical probes has been demonstrated in zebrafish embryos, and these reagents have been employed
103 d platform that integrates an innovative LOC zebrafish embryo array technology with an electronic int
106 ose V2 neural progenitor cells in developing zebrafish embryo as their successive shape changes can b
107 rgence of hematopoietic stem cells (HSCs) in zebrafish embryos as a model to investigate the role of
109 propionate (CLO), cortisol and cortisone in zebrafish embryos as single compounds and binary mixture
110 ts on liver versus pancreas specification in zebrafish embryos as well as mouse endodermal progenitor
111 ockdown of Kapbeta2 affected Hh signaling in zebrafish embryos, as well as in vitro cultured cerebell
112 osterior lateral and ventral mesoderm of the zebrafish embryo at the gastrula stage, by directly inte
115 ss-correlation analysis to the blood flow in Zebrafish embryos at 4 days after fertilization, measuri
116 teractome capture, we identified 227 RBPs in zebrafish embryos before and during ZGA, hereby named th
117 the glycosyltransferase domain Afp18(G) into zebrafish embryos blocks cytokinesis, actin-dependent mo
118 as an early marker of HSC commitment in the zebrafish embryo, but recent studies have suggested that
119 simulate cardiac hemodynamics in developing zebrafish embryos by coupling 4-D light sheet imaging wi
120 f Nodal ligands and Lefty inhibitors in live zebrafish embryos by fluorescence correlation spectrosco
122 nd provide proof of concept that a screen in zebrafish embryos can identify therapeutic candidates fo
127 ression of nesprin-1alpha2 WT and mutants in zebrafish embryos caused heart developmental defects tha
131 is restricted to fast-twitch myocytes in the zebrafish embryo; consistent with this, fro mutant embry
132 e identified a number of key roles of GCs in zebrafish embryos contributing to adaptive physiological
133 ydrolysis in eukaryotic cell cultures and in zebrafish embryos; crucially, the biliverdin chromophore
136 o silver in nano-, bulk-, and ionic forms on zebrafish embryos (Danio rerio) using a Next Generation
141 ties, recapitulating some of the features of zebrafish embryos deficient in the glaucoma-related gene
142 b function in Tg(kdrl:EGFP)(s843) transgenic zebrafish embryos delayed the angiogenesis of intersegme
143 Loss- and gain-of-function experiments in zebrafish embryos demonstrate that col15a1b expression a
144 coupled with immunofluorescence performed on zebrafish embryos demonstrate that enox1 message and tra
149 active in an in vivo overexpression assay in zebrafish embryos demonstrating that the HP1 interaction
150 also track migrating cells in the developing zebrafish embryo, demonstrating the utility of this syst
156 ese compounds show no off-target toxicity in zebrafish embryos, do not cause haematological, biochemi
157 ization, culture and treatment of developing zebrafish embryos during fish embryo toxicity (FET) biot
159 stein, from 28-52 hours postfertilization in zebrafish embryos enhanced Hepcidin transcript levels, a
160 tified putative androgen-responsive genes in zebrafish embryos exposed to 0.05-5000 nM 11-ketotestost
163 eduction of sox9b expression in TCDD-exposed zebrafish embryos has been shown to contribute to heart
166 recovery after photobleaching experiments in zebrafish embryos identified a pool of dynamic F-actin w
167 lex sensory organ, the inner ear, by imaging zebrafish embryos in vivo over an extended timespan, com
170 or CRISPR-mediated deletions of sec61al2 in zebrafish embryos induced convolution defects of the pro
171 harboring any of three patient mutations in zebrafish embryos induced defects in axon guidance, conf
173 s involved in the bystander response between zebrafish embryos induced through X-ray irradiation.
176 x gangliosides in patient fibroblasts and in zebrafish embryos injected with antisense morpholinos th
177 ted from the medium that had conditioned the zebrafish embryos irradiated at 5 hpf with 4-Gy X-ray un
179 nd that overexpression of hand2 in the early zebrafish embryo is able to enhance cardiomyocyte produc
180 e neurodevelopmental toxicity of BMAA in the zebrafish embryo is presented in relation to the potenti
181 shment of the BMP activity gradient in early zebrafish embryos is determined by graded expression of
182 edgehog and Wingless pathways in cichlid and zebrafish embryos is sufficient to mimic differences bet
184 y in wild-type but also in immunocompromised zebrafish embryos lacking either macrophages or neutroph
185 actor Tal1 in endocardial tube formation: in zebrafish embryos lacking Tal1, endocardial cells form a
186 of the bisphenols BPA, BPS, BPF, and BPAF in zebrafish embryo-larvae and an assessment on their estro
187 s were confirmed by experimental analysis of zebrafish embryo LC50 according to OECD guideline 236.
188 e evidence that the deregulation of EGFL7 in zebrafish embryos leads to a severe integrin-dependent m
189 loss of function in both Xenopus laevis and zebrafish embryos leads to a significant reduction in re
190 y human and mouse kidney cells as well as in zebrafish embryos leads to enhanced DNA damage signaling
191 f wild-type human VEGFC in the floorplate of zebrafish embryos leads to excessive sprouting in neighb
195 ticoids (GCs) are known to be present in the zebrafish embryo, little is known about their physiologi
198 in vivo and single-myosin detection to study zebrafish embryo models of human muscle disease is a mul
201 aled the induction of cellular senescence in zebrafish embryos overexpressing mutant, but not wild-ty
204 by injection of an antisense morpholino into zebrafish embryos prevented photoreceptor-driven migrati
205 d anthocyanin metabolites were determined in zebrafish embryos previously exposed to the red wine ext
208 developmental periods to limit CM number in zebrafish embryos: prior to gastrulation and after the i
209 b does not compensate for Ssrp1a loss in the zebrafish embryo, probably owing to insufficient express
213 th knockdown and genome editing of znhit3 in zebrafish embryos recapitulate the patients' cerebellar
214 ssion or CRISPR-mediated deletion of brf1 in zebrafish embryos recapitulated key neurodevelopmental p
215 Expression of the histone H4 mutants in zebrafish embryos recapitulates the developmental anomal
216 panied by reduced numbers of thrombocytes in zebrafish embryos, recapitulating key aspects of Stormor
217 ting yielded red fluorescent erythrocytes in zebrafish embryos, recapitulating the phenotype observed
218 rotoxicity, whereas MMP-13 overexpression in zebrafish embryos rendered the skin vulnerable to injury
219 Constitutive expression of these mutants in zebrafish embryos resulted in a heart failure phenotype
222 of dominant-negative ror2 (ror2-TM) mRNA in zebrafish embryos resulted in convergence and extension
224 ound that inactivation of bdh2 in developing zebrafish embryo results in heme deficiency and delays e
231 cell cycle of single cells in culture and in zebrafish embryos showed that the total surface increase
232 time measurements of genetic oscillations in zebrafish embryos showing that their time scale is not s
235 of Enox1 in Tg(fli1-eGFP) and Tg(flk1-eGFP) zebrafish embryos significantly impairs the development
236 We show that sphk2 maternal-zygotic mutant zebrafish embryos (sphk2(MZ)) display early developmenta
239 significantly retarded granule formation in zebrafish embryos, suggesting that any combination of at
240 rm differentiation upon forced expression in zebrafish embryos, suggesting that they have dominant-ne
241 ading and immobilization of large numbers of zebrafish embryos suspended in a continuous microfluidic
242 ino or the coexpression of ror2 and wnt11 in zebrafish embryos synergetically induced more severe con
248 and rapid framework that relies on the early zebrafish embryo to assess mutational effects on a commo
249 , by exploiting the unique properties of the zebrafish embryo to capture the dynamics of signaling an
250 ty of Pdots were evaluated on HeLa cells and zebrafish embryos to demonstrate their great biocompatib
252 e conducted a small-scale chemical screen in zebrafish embryos to identify small molecules that modul
254 ng the week-long experiments, students raise zebrafish embryos to learn principles of development and
255 gentle mechanical deformation of developing zebrafish embryos to probe the role of physical forces i
256 can supplement existing methods, such as the Zebrafish Embryo Toxicity assay (OECD TG236), with molec
257 b-on-a-Chip technology for automation of the zebrafish embryo toxicity test common in aquatic ecotoxi
260 tial toxicity to a fish cell line (BF-2) and zebrafish embryos under dark and Simulated Solar Light (
264 k dynamics in the anterior PSM in developing zebrafish embryos using an in vivo clock reporter, her1:
265 ole in vascular development was validated in zebrafish embryos using morpholino oligonucleotides.
268 Delivery of 8-oxo-dGTP and 2-OH-dATP to zebrafish embryos was highly toxic in the absence of MTH
269 mmalian cells, as well as in yeast cells and zebrafish embryos We disrupted murine bdh2 by homologous
270 indbrain regions by sensory afferents in the zebrafish embryo, we mapped the fine-grained topographic
271 Using live imaging and transplantation in zebrafish embryos, we additionally reveal that axon init
272 e cardiomyocyte proliferation events in live zebrafish embryos, we generated transgenic zebrafish lin
274 netically encoded calcium indicators in live zebrafish embryos, we show that ICOs depend on Pkd2 and
275 mbination of in vitro and in vivo studies in zebrafish embryos, we show that PAK3(N389) escapes its p
276 ying a multi-scalar morphometric analysis in zebrafish embryos, we show that posterior body elongatio
277 es (HaCaT), zebrafish liver cells (ZFL), and zebrafish embryos were also used to study the toxicity o
278 d interleukin (Il)10(-/-) mice and germ-free zebrafish embryos were colonized with specific pathogen-
280 cts of EETs were conserved in the developing zebrafish embryo, where 11,12-EET promoted HSPC specific
281 Wnt3 and subsequently, its secretion in live zebrafish embryos, where chemical inhibition of Porcupin
282 es, in part, dorsoventral axis patterning in zebrafish embryos, whereas BMP signaling through Smad3 f
283 nockdown abolishes nodal signalling in early zebrafish embryos, whereas overexpression of rab5ab mRNA
284 the region where hematopoiesis occurs in the zebrafish embryo, which recapitulates a BM-like niche.
286 ed secreted signaling protein, Wnt3, in live zebrafish embryos, which is necessary for the investigat
287 expressed in wild-type or dmd(ta222a/ta222a) zebrafish embryos, which lack Dystrophin, and in Gt(dmd-
289 ing to assess the functional architecture of zebrafish embryos with a retrospective cardiac synchroni
291 to induce targeted genetic modifications in zebrafish embryos with efficiencies similar to those obt
296 re physiological relevance, we microinjected zebrafish embryos with the same oligonucleotides, as a s
298 halts SRC-associated neuromast migration in zebrafish embryos without inducing life-threatening hear
300 ) with a diversity of functional groups into zebrafish embryos (ZFE) was studied over 96 h of exposur
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