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1 ings were taken at 3 h intervals starting at zeitgeber time 0 (ZT0) and stomach content was measured.
5 ted at noon (Zeitgeber time 5) and midnight (Zeitgeber time 17) and identified a subgroup of 12 miRNA
7 lipogenic genes only under fed conditions at Zeitgeber time 22 (ZT22) but not under fasting condition
8 d to cold fare considerably better at 05:00 (Zeitgeber time 22) when Rev-erbalpha is barely expressed
9 sucrose was given once daily for 5-6 min at Zeitgeber Time 4 (ZT 4) for 17-18 days to 8 food-deprive
10 of the ovarian cycle, whether hours before [zeitgeber time 4 (ZT4)-ZT6] or just before (ZT10) the ex
11 in sham and SCN-lesioned (SCNx) rats at ZT4 (Zeitgeber time 4, 4 h after lights-on) or ZT20 (8 h afte
13 rofiling with retinal RNA harvested at noon (Zeitgeber time 5) and midnight (Zeitgeber time 17) and i
15 at SCN during the light-dark cycle (peaks at Zeitgeber time 6 and 18) and also in constant darkness (
18 e and less sleep time than control mice in a zeitgeber time- and sleep deprivation-dependent manner.
20 of SG files was constantly high at different Zeitgeber times, the in situ signals in BG and XLG files
21 each action potential against the respective zeitgeber time, we describe oscillations of spike activi
22 12-h light-dark cycle with lights on between zeitgeber time (ZT) 0 to ZT12 fed 60% of normal calories
23 m animals sacrificed during the day, between zeitgeber time (ZT) 0430 and 0530, were incubated, and t
24 ein kinase A (PKA) inhibitor onto the SCN at Zeitgeber time (ZT) 10 on the first day in vitro phase d
25 of bright GFP(+) NOS(+) cells was greater at Zeitgeber time (ZT) 10 than at 22, and this pattern pers
26 Expression of Hmgb1 was least at night at Zeitgeber time (ZT) 18 and maximal in the middle of the
27 mice: TTVO varied from 24.6+/-2.7 minutes at zeitgeber time (ZT) 2 to 40.3+/-4.3 minutes at ZT8, 24.3
28 sed at a time when the animals are inactive (zeitgeber time (ZT) 20) or at a time when they are awake
30 se-dependent manner: advances are induced at zeitgeber time (ZT) 6 and delays are induced at ZT 22.
31 e-dependent manner: advances were induced at zeitgeber time (ZT) 6, and delays were induced at ZT 22.
32 gamma regulate these genes directly and in a Zeitgeber time (ZT)-dependent manner through these ROREs
35 to ischemia at the sleep-to-wake transition (zeitgeber time [ZT]12) resulted in 3.5-fold increases in
36 ior to the onset of either the active phase (zeitgeber time [ZT]12: Experiment 1) or the inactive pha
37 ox or the vehicle two hours after lights on (zeitgeber time (ZT2), or two hours after lights off (ZT1
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