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1 ed NMR parameters in the presence of heme or zinc protoporphyrin.
2 or; (4) 3 mmol/L L-arginine plus 650 mumol/L zinc protoporphyrin; (5) 15 mumol/L methylene blue, a cG
3                  Moreover, adjunctive use of zinc protoporphyrin, a competitive HO-1 inhibitor, has s
4 rginine, a precursor for NO; (3) 650 mumol/L zinc protoporphyrin, a heme oxygenase inhibitor; (4) 3 m
5                          Treating VSMCs with zinc protoporphyrin, an HO-1 inhibitor, or HO-1 small in
6 ion pressure was observed in the presence of zinc protoporphyrin, an inhibitor of HO.
7 creased levels of free protoporphyrin IX and zinc protoporphyrin are generated in IRP2-/- erythroid c
8 induced apoptosis, whereas pretreatment with zinc protoporphyrin attenuated morphine-induced macropha
9 On the other hand, pretreatment of MRCs with zinc protoporphyrin attenuated the effect of morphine on
10  of HO-1 or inhibition of HO-1 activity with zinc protoporphyrin blocked these effects of gAcrp.
11 roups in transferrin saturation, erythrocyte zinc protoporphyrin concentration, hemoglobin concentrat
12 esis as evidenced by an elevated erythrocyte zinc protoporphyrin concentration.
13 microcytic anemia, with an elevated red cell zinc protoporphyrin, consistent with functional erythroi
14                                              Zinc protoporphyrin did not change in either group.
15 ral fitting is used to distinguish the faint zinc protoporphyrin fluorescence from the much greater t
16 erized biochemically by a high proportion of zinc-protoporphyrin in erythrocytes, in which a mismatch
17 hed indicator of iron status, red blood cell zinc protoporphyrin, in the microcirculation of the lowe
18                  We have shown that heme and zinc protoporphyrin inhibit both human immunodeficiency
19 iles capable of binding the metalloporphyrin zinc protoporphyrin IX ((PPIX)Zn) have been synthesized.
20 of animals with the heme oxygenase inhibitor zinc protoporphyrin IX (50 micromol/kg IP) markedly decr
21 ate that cobalt protoporphyrin IX (CoPP) and zinc protoporphyrin IX (ZnPP) are ligands that bind dire
22                                              Zinc protoporphyrin IX (ZnPP), an endogenous heme analog
23 as ablated when HO-1 activity was blocked by zinc protoporphyrin IX (ZnPPIX).
24            Treatment with the HO-1 inhibitor zinc protoporphyrin IX exacerbated the inflammatory resp
25                    Mixed-metal hybrids, with zinc protoporphyrin IX in place of heme on both alpha or
26                                 Furthermore, zinc protoporphyrin IX, an inhibitor of HO activity, abr
27 r the alpha or the beta subunits replaced by zinc protoporphyrin IX, which is unable to bind a ligand
28 retreatment of ganglia with the HO inhibitor zinc protoporphyrin-IX (ZnPP) (10 microm) completely and
29 s associated with a decreased red blood cell zinc protoporphyrin to heme ratio, indicative of porphyr
30          Rapid application of HO inhibitors (zinc protoporphyrin (ZnPP) and tin protoporphyrin (SnPP)
31                                              Zinc protoporphyrin (ZnPP) and zinc mesoporphyrin (ZnMP)
32 lt protoporphyrin (CoPP) as HO-1 inducer and zinc protoporphyrin (ZnPP) as HO-1 inhibitor.
33 mal RBCs, combined with exogenous prooxidant zinc protoporphyrin (ZnPP) induce a potent tumoricidal r
34                                              Zinc protoporphyrin (ZnPP), a naturally occurring molecu
35  or those pretreated with the HO-1 inhibitor zinc protoporphyrin (ZnPP), upregulation of HO-1 by West
36 O-1 enzymatic activity, by administration of zinc protoporphyrin (ZnPPIX) at the time of transplantat
37 e transferrin receptor (TfR; > 8.3 mg/L), or zinc protoporphyrin (ZP; > 80 mumol/mol) concentrations
38 ildren with CM or SMA, as well as 35 CC, had zinc protoporphyrin (ZPP) concentrations >/=80 mumol/mol

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