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1 taminergic itch, without impairing histamine-induced itch.
2 BB6F1-Kit(W/Wv) mice) was able to affect AYP-induced itch.
3 and to identify the molecular basis of IL-31-induced itch.
4 ically evoked persistent pain and pruritogen-induced itch.
5 nel acting together with TRPA1 to mediate CQ-induced itch.
6 reduces capsaicin-induced pain and histamine-induced itch.
7 ced by histamine but did not prevent cowhage-induced itch.
8 ch but did not significantly alter histamine-induced itch.
9 distinct from CMi fibers mediating histamine-induced itch.
10 of dry skin or compound 48/80- or Alternaria-induced itch.
11 el preferentially functions to antagonize CD-induced itch.
12 s are involved in broad-band UVB irradiation-induced itch.
13 ce that TRPV4 is a key mediator of serotonin-induced itch.
14 portant role of myelinated fibers in cowhage-induced itch: (1) a selective conduction block in myelin
15                                Spinal opioid-induced itch, a prevalent side effect of pain management
16 eater dlPFC and caudate activation to nocebo-induced itch also demonstrated greater dlPFC and caudate
17            Subjects reporting greater nocebo-induced itch also demonstrated greater placebo reduction
18 rphine and explain several aspects of opioid-induced itch and analgesia.
19                  Long persistence of cowhage-induced itch and diminished histamine-induced flare in n
20 unrecognized spinal circuit for mechanically-induced itch and elucidated a mechanism that keeps it in
21                                          BDL-induced itch and hypersensitivity involved a minor contr
22  in skin keratinocytes was essential for LPC-induced itch and itch in mice with cholestasis.
23    However, the mechanisms underlying opioid-induced itch are not fully understood, although an inter
24  the underlying mechanisms of UV irradiation-induced itch are poorly understood because of a lack of
25 kin with topical capsaicin abolished cowhage-induced itch but did not significantly alter histamine-i
26 PV1+ sensory neurons did not affect morphine-induced itch but impaired morphine-induced antinocicepti
27        Brain activity likely supports nocebo-induced itch, but is currently unknown.
28 he cellular mechanisms of intrathecal opioid-induced itch by conditional deletion of MOR-encoding Opr
29 mechanisms underlying cowhage- and histamine-induced itch differ.
30            Serpin E1 inhibitor reduced MC903-induced itch, epidermal hyperplasia, immunocyte infiltra
31                   The persistence of cowhage-induced itch for at least 30 min and a histamine-induced
32 ht to the brain mechanisms supporting nocebo-induced itch in AD, thus aiding our understanding of the
33 unication of compound 48/80- and chloroquine-induced itch in hairy skin.
34  scratching on skin blood flow and histamine-induced itch in healthy volunteers.
35 he kappa-opioid receptor alleviated morphine-induced itch in mice and nonhuman primates.
36 e both necessary and sufficient for morphine-induced itch in mice.
37                                     Morphine-induced itch is a very common and debilitating side effe
38     Together, these data suggest that opioid-induced itch is an active process concomitant with but i
39             These results indicate that 5-HT-induced itch is linked to TRPV4.
40      Providing potential insight into opioid-induced itch prevention, we demonstrate that molecular a
41 sial because the cellular basis for morphine-induced itch remains unclear.
42                    Squaric acid dibutylester-induced itch requires both nonhistaminergic and histamin
43 of bombesin-saporin, whereas intrathecal GRP-induced itch response remained intact in mice lacking Op
44 ly alleviated the broad-band UVB irradiation-induced itch response.
45                  Il31(-/-) mice lacked wound-induced itch responses.
46 itogens; IL-4 synergistically enhanced IL-13-induced itch, resulting in greater pruritic responses th
47  (SDH) is required selectively for histamine-induced itch sensation.
48 There was no significant difference in IL-31-induced itch start time, duration and intensity between
49                      The late onset of IL-31-induced itch supports the notion that IL-31 exerts its p
50 ely prescribed for the treatment of morphine-induced itch, their use is controversial because the cel
51                                     NMLTC(4)-induced itch was abrogated in mice deficient for Cysltr2
52                                     Allergen-induced itch was evaluated in 20 patients with AD after
53 itive neurons (pruriceptive fibers), and AYP-induced itch was reduced by the selective GRP receptor a
54                             Importantly, AYP-induced itch was reduced by treatment with either the se
55                                        IL-31-induced itch was significantly reduced in TRPV1-deficien