ライフサイエンスコーパス: 17beta-estradiol

1 ge from 0 (no activity) to 1 (bioactivity of 17beta-estradiol).

2 emale immunodeficient mice supplemented with 17beta-estradiol.

3 estrogen receptor alpha (ERalpha) to that of 17beta-estradiol.

4 t of ERalpha-AF2 in the metabolic actions of 17beta-estradiol.

5 ignaling mechanisms from ERalpha, ERbeta, or 17beta-estradiol.

6 rs (ER) are activated by the steroid hormone 17beta-estradiol.

7 M site for steroid hormone estrogens such as 17beta-estradiol.

8 indicate that this transporter is induced by 17beta-estradiol.

9 oved growth of xenografts in the presence of 17beta-Estradiol.

10 ESR1) in the presence of its cognate ligand, 17beta-estradiol.

11 onal regulation of the SNAT2 transporter via 17beta-estradiol.

12 , and its protective effect is stronger than 17beta-estradiol.

13 ment for NO/cGMP/PKG signaling downstream of 17beta-estradiol.

14 enotype by antagonizing the growth effect of 17beta-estradiol.

15 y prevented when cells were pre-treated with 17beta-estradiol.

16 apeutic effect of post-SCI administration of 17beta-estradiol.

17 (ER) alpha and beta differ from that used by 17beta-estradiol.

18 aptamer, which highly specifically binds to 17beta-estradiol.

19 nted conductive polymer-coated electrodes to 17beta-estradiol.

20 aring CIN lesions were treated with MPA plus 17beta-estradiol.

21 eported 35-mer aptamer for a small molecule, 17beta-estradiol.

22 wine samples, as well as cocaine (1 nM) and 17beta-estradiol (0.2 nM) in spiked synthetic urine and

23 were 0.045 ng/L for estrone, 0.086 ng/L for 17beta-estradiol, 0.030 ng/L for estriol, 0.049 ng/L for

24 ere randomly assigned to receive either oral 17beta-estradiol (1 mg per day, plus progesterone [45 mg

25 al, as shown by vesicular transport of [(3)H]17beta-estradiol-17-beta-(D-glucuronide) and doxorubicin

26 sformation of two natural steroid estrogens [17beta-estradiol (17beta-E2) and estrone (E1)] and two s

27 ne hormones, 17alpha-estradiol (17alpha-E2), 17beta-estradiol (17beta-E2), and estrone (E1), are rout

28 Addition of 17beta-estradiol (17betaE2) protected cultured neurons a

29 PET with the ER ligand 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) and to evaluate whether tra

30 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) is a PET tracer for ER with

31 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) is an estrogen receptor (ER

32 ression with the tracer 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) may be valuable to select o

33 16alpha-(18)F-fluoro-17beta-estradiol ((18)F-FES) with PET is a noninvasive t

34 reated for 5 weeks with cyclic injections of 17beta-estradiol-3-benzoate (EB, 10 mug) or oil vehicle.

35 ed with a (14)C labeled prototype conjugate, 17beta-estradiol-3-glucuronide (E2-3G).

36 emical transformation from parent compounds (17beta-estradiol, 4-nonylphenolpolyethoxylates, and 4-no

37 Accordingly, 17beta-estradiol administration regulated key metabolic

38 lso exerts a stronger protective effect than 17beta-estradiol against kanic acid-induced hippocampal

39 ently found that the protection conferred by 17beta-estradiol against obesity and insulin resistance

40 recursor prodrug of the main human estrogen, 17beta-estradiol, alleviates hot flushes in rat models o

41 Unlike control mice treated with 17beta-estradiol alone, cervical cancer was absent in th

42 sensitive and highly selective detection of 17beta-estradiol, an EDC that is frequently detected in

43 n, some methods reported false positives for 17beta-estradiol and 17alpha-ethynylestradiol in unspike

44 In these studies, we assessed the ability of 17beta-estradiol and equol to regulate markers of hippoc

45 Similarly, 17beta-estradiol and equol treatment had no effect on mi

46 endocrine-disrupting chemicals, particularly 17beta-estradiol and estrone, and fish exposed to the po

47 These data indicate that 17beta-estradiol and GPER independently regulate hippoca

48 17beta-estradiol and IL-6 may act synergistically to pro

49 hondrial damage using synergistic effects of 17beta-estradiol and methylene blue, previously shown by

50 Chronic treatment with 17beta-estradiol and tamoxifen elicited differential gen

51 Here we show that physiologic estrogen (17beta-estradiol) and progesterone reciprocally regulate

52 nts were respectively theophylline, digoxin, 17beta-estradiol, and aldrin.

53 In WTP influents, estrogens (estrone, 17beta-estradiol, and estriol), androgens (androstenedio

54 ted the fate and transport of 4-nonylphenol, 17beta-estradiol, and estrone in a 10-km reach of the Re

55 ural estrogenic hormones (17alpha-estradiol, 17beta-estradiol, and estrone) in aqueous solutions blen

56 of the initial amounts of 17alpha-estradiol, 17beta-estradiol, and estrone, respectively.

57 tain florfenicol, pyrimethamine, estrone and 17beta-estradiol at levels from 0.095 to 2.7 mug/kg.

58 anure excreted by implanted (40 mg TBA, 8 mg 17beta-estradiol) beef cattle.

59 n terms of research and monitoring have been 17beta-estradiol (beta-E2) and 17alpha-ethinylestradiol,

60 f Fe(3+)-saturated montmorillonite catalyzed 17beta-estradiol (betaE2) transformation was investigate

61 he alpha4beta2-selective positive modulators 17beta-estradiol (betaEST) and desformylflustrabromine (

62 BPA and 17beta-estradiol bind to ERs in a similar fashion, where

63 This effect was associated with increased 17beta-estradiol but not with insulin-like growth factor

64 UM-SCC-11A and UM-SCC-12 both produce 17beta-estradiol, but only UM-SCC-12, not UM-SCC-11A, xe

65 Across women, lower 17beta-estradiol concentrations were related to more pro

66 creened eel serum samples to determine their 17beta-estradiol concentrations, which were found to be

67 17beta-estradiol-D5 was used as internal standard.

68 We found that post-SCI administration of 17beta-estradiol decreased neuronal loss in the ventral

69 Neutrophils treated with 17beta-estradiol demonstrated an enhanced oxidative burs

70 that in human breast cancer cells, the acute 17beta-estradiol-dependent activation of functional enha

71 yo implantation is coordinately regulated by 17beta-estradiol (E(2)) and progesterone (P(4)).

72 We report that 17beta-estradiol (E(2)) decreased osteoclast number by p

73 as safer estrogenic candidates compared with 17beta-estradiol (E(2)) for the treatment of endocrine-r

74 l studies indicates that the gonadal hormone 17beta-estradiol (E(2)) impacts the structure and functi

75 agonist G-1 mimics the beneficial effects of 17beta-estradiol (E(2)) on hippocampal CA1 spine density

76 (PKC) signaling can be activated rapidly by 17beta-estradiol (E(2)) via nontraditional signaling in

77 ablation blocked the reduction of T(SKIN) by 17beta-estradiol (E(2)), which occurred in the environme

78 17beta-Estradiol (E(2))-mediated ER activation stabilize

79 in part owing to a reduction in circulating 17beta-estradiol (E(2)).

80 plex by different types of estrogens-planar [17beta-estradiol (E(2))] and angular triphenylethylene (

81 nal epithelium were treated with either 4 nM 17beta-estradiol (E) for seven days, 50 ng/ml E.coli fla

82 -enhancing effects of the modulatory hormone 17beta-estradiol E2.

83 17beta-estradiol (E2 or estrogen) is an endogenous stero

84 mug L(-1)), while that of the native hormone 17beta-estradiol (E2) (1 muM, i.e., 272 mug L(-1)) was 1

85 alpha-containing cancer cells, the estrogen, 17beta-estradiol (E2) activates the UPR through a phosph

86 We found recently that 17beta-estradiol (E2) acutely suppresses GABAergic inhib

87 We report that 17beta-estradiol (E2) affects the behavior and the epige

88 17beta-estradiol (E2) also influences hypothalamic progr

89 fluctuations and systemic administration of 17beta-estradiol (E2) alter spine density in the dorsal

90 s often found in treated and natural waters, 17beta-estradiol (E2) and 17alpha-ethynylestradiol (EE).

91 The response of ovariectomized adults to 17beta-estradiol (E2) and artificial decidualization wer

92 We investigated the potential of 17beta-estradiol (E2) and estrogen receptor (ER) signali

93 ch were transformed into the free estrogens, 17beta-estradiol (E2) and estrone (E1), respectively.

94 owever, few studies have assessed both serum 17beta-estradiol (E2) and exogenous hormone therapy (HT)

95 to analogues similar to progesterone such as 17beta-estradiol (E2) and norethisterone (NET).

96 e sought to Determine the mechanism by which 17beta-estradiol (E2) and progesterone (P4) increase IL-

97 We demonstrated that 17beta-estradiol (E2) and the GPER-selective ligand G-1

98 tudy, we demonstrate for the first time that 17beta-estradiol (E2) and the selective GPER ligand G-1

99 The neuroprotective effects of 17beta-estradiol (E2) and three synthetic nonfeminizing

100 nse-response relationships describing plasma 17beta-estradiol (E2) as a function of plasma fadrozole,

101 ator of an inflammatory microenvironment and 17beta-Estradiol (E2) as an agonist of Estrogen Receptor

102 using thyroid stimulating hormone (TSH) and 17beta-estradiol (E2) as model analytes, respectively.

103 -ethylbenzthiazoline-6-sulfonate) (ABTS) and 17beta-estradiol (E2) as the probing substrates.

104 We previously observed that 17beta-estradiol (E2) augments ischemic borderzone vascu

105 study examined whether the steroid hormone, 17beta-estradiol (E2) can exert long-lasting beneficial

106 this study was to investigate if exposure to 17beta-estradiol (E2) causes abnormal development in lar

107 to affect TMJ-responsive neurons, we applied 17beta-estradiol (E2) directly at the spinomedullary (Vc

108 Exposure of zebrafish embryos to 17beta-estradiol (E2) during liver development significa

109 Our laboratory has demonstrated that 17beta-estradiol (E2) enhances hippocampal memory consol

110 monstrated by an immunospecific detection of 17beta-Estradiol (E2) following the competitive inhibiti

111 ensor for the rapid, label-free detection of 17beta-estradiol (E2) from femtomolar to micromolar leve

112 Although chronic 17beta-estradiol (E2) has been shown to be a cognition-p

113 17beta-Estradiol (E2) has been shown to regulate GM-CSF-

114 17beta-estradiol (E2) has broad tropism within the CNS,

115 The hormone 17beta-estradiol (E2) has shown vascular protective effe

116 e molecular and cellular mechanisms by which 17beta-estradiol (E2) impacts the microenvironment and m

117 Suwannee River NOM-sensitized photolysis of 17beta-estradiol (E2) in freshwater and saline media.

118 ptor alpha (ERalpha) mediates the effects of 17beta-estradiol (E2) in normal mammary gland, and it is

119 ing ultrasensitive colorimetric detection of 17beta-estradiol (E2) in water and urine samples using D

120 Exposure to 17beta-estradiol (E2) increased proliferation of hepatoc

121 Although systemically injected 17beta-estradiol (E2) increases CA1 dendritic spine dens

122 Intriguingly, recent reports have shown that 17beta-estradiol (E2) induces Noxa expression, although

123 Here, we report that 17beta-estradiol (E2) influences production of runx1+ HS

124 We found 17beta-estradiol (E2) inhibited hepatic gluconeogenic ge

125 Previously, we reported that 17beta-estradiol (E2) inhibits azoxymethane/dextran sulf

126 The uterotropic response of the uterus to 17beta-estradiol (E2) is genetically controlled, with ma

127 pressed during estrous cycle stages in which 17beta-estradiol (E2) is naturally high (e.g., proestrus

128 process by demonstrating that neuron-derived 17beta-estradiol (E2) is neuroprotective and critical fo

129 17beta-Estradiol (E2) is produced from androgens via the

130 The steroid 17beta-estradiol (E2) is well known to influence hippoca

131 of BSEP was inversely correlated with serum 17beta-estradiol (E2) levels before, during, and after g

132 In cystic fibrosis, 17beta-estradiol (E2) may inhibit store-operated Ca(2+)

133 The estrogen 17beta-estradiol (E2) modulates dendritic spine plastici

134 This study investigated the effects of 17beta-estradiol (E2) on gene regulation in human cardia

135 The effect of 17beta-estradiol (E2) on IL-13-induced signaling pathway

136 fed high-fat diet (HFD) that received either 17beta-Estradiol (E2) or vehicle implants.

137 t stimulation of ERalpha-positive cells with 17beta-estradiol (E2) promotes global citrullination of

138 ive and positive feedback effects of ovarian 17beta-estradiol (E2) regulating release of gonadotropin

139 d subsequently explored the distribution and 17beta-estradiol (E2) regulation of kisspeptin mRNA (Kis

140 Given that chronic 17beta-estradiol (E2) replacement of ovariectomized rats

141 We previously found that 17beta-estradiol (E2) stimulates apolipoprotein A-IV (ap

142 Treatment with 17beta-estradiol (E2) synergized with DAC to reduce prol

143 Expression of the 17beta-estradiol (E2) synthesis enzyme aromatase is high

144 The ability of 17beta-estradiol (E2) to enhance hippocampal object reco

145 We have previously reported that 17beta-estradiol (E2) treatment activates Notch signalin

146 f rodents, and this reduction is reversed by 17beta-estradiol (E2) treatment in a model of human estr

147 17beta-Estradiol (E2) treatment limits the pathology ass

148 Here we show that 17beta-estradiol (E2) up-regulates total CaM level in en

149 (s) of D3 action and compare it with that of 17beta-estradiol (E2) using both in vitro and in vivo ut

150 ductase (5alpha-R) or to the active estrogen 17beta-estradiol (E2) via the aromatase enzyme.

151 An aptamer capable of binding to our target 17beta-estradiol (E2) was isolated by SELEX with dissoci

152 The classic estrogen 17beta-estradiol (E2) was recently identified as a novel

153 After 2 weeks of exposure, levels of plasma 17beta-estradiol (E2) were significantly elevated in hig

154 gned and synthesized nine new derivatives of 17beta-estradiol (E2) with a bulky side chain attached t

155 17beta-estradiol (E2), acting through ERalpha, and pro-i

156 tes express the enzyme aromatase and produce 17beta-estradiol (E2), although the precise role of astr

157 case for the transformation of estrone (E1), 17beta-estradiol (E2), and 17alpha-ethinylestradiol (EE2

158 maceuticals, 17alpha-ethinylestradiol (EE2), 17beta-estradiol (E2), and diclofenac throughout Europea

159 t ovarian sex-steroid hormones, particularly 17beta-estradiol (E2), are important trophic factors tha

160 Estrogens, particularly 17beta-estradiol (E2), are powerful neuroprotective agen

161 l cells, DNA damage following treatment with 17beta-estradiol (E2), BP-3, and PP was determined by im

162 AROM), the enzyme converting testosterone to 17beta-estradiol (E2), contributes to the regulation of

163 G protein-coupled receptor reported to bind 17beta-estradiol (E2), couple to the G proteins Gs and G

164 termination of four estrogens [estrone (E1), 17beta-estradiol (E2), estriol (E3), and 17alpha-ethinyl

165 Estrogen, 17beta-estradiol (E2), is a powerful therapeutic agent t

166 iated by chemical neuromodulators, including 17beta-estradiol (E2), or patterns of synaptic activatio

167 epithelial cells (NHBE) were pretreated with 17beta-estradiol (E2), propyl-pyrazole-triol (PPT, ER-al

168 tablished, recipients were supplemented with 17beta-estradiol (E2), tamoxifen, or left untreated.

169 derivatizaton dehyroepiandrosterone (DHEA), 17beta-estradiol (E2), testosterone (T), and their sulfa

170 spensable for the atheroprotective action of 17beta-estradiol (E2), the main ligand of ERs.

171 roles in reproductive tissues--specifically, 17beta-estradiol (E2), the primary estrogen, which is se

172 decreases the conversion of testosterone to 17beta-estradiol (E2), thereby reducing E2-dependent vit

173 zed into 5alpha-dihydrotestosterone (DHT) or 17beta-estradiol (E2), which activate different hormonal

174 nism to facilitate auditory consolidation is 17beta-estradiol (E2), which is associated with human sp

175 least 17 CoRs from nuclear extracts bound to 17beta-estradiol (E2)-liganded estrogen receptor-alpha o

176 was decreased in bone in SPI-fed, but not in 17beta-estradiol (E2)-treated rats.

177 AR-dependent synaptic plasticity and memory, 17beta-estradiol (E2).

178 mouse responsive to long-term treatment with 17beta-estradiol (E2).

179 ironmental fate of steroid hormones, such as 17beta-estradiol (E2).

180 gnificantly correlated with plasma levels of 17beta-estradiol (E2).

181 -1)) sensitivity for the endocrine disruptor 17beta-Estradiol (E2).

182 l aromatase, which produces the neurosteroid 17beta-estradiol (E2).

183 ontributes to the neuroprotective effects of 17beta-estradiol (E2); however, the mechanisms associate

184 eak proestrus levels of the primary estrogen 17beta-estradiol (E2; 10 mug/kg, i.p., 1-h pretreatment)

185 tics), genistein (GEN; found in soybean), or 17beta-estradiol (E2; an endogenous estrogen).

186 erator-activated receptor gamma agonist) and 17beta-estradiol (E2; an estrogen receptor alpha agonist

187 distinct cellular signaling pathways (i.e., 17beta-estradiol [E2] and TNF-alpha).

188 Elevations in estrogen (17beta-estradiol, E2) are associated with increased alco

189 We have shown that estrogen (17beta-estradiol, E2) inhibits expression of these genes

190 pes) after exposure to two concentrations of 17beta-estradiol (E2beta; 2 ng/L and 50 ng/L) during fou

191 ively at an average concentration of 10 ng/l 17beta-estradiol equivalent (EEQ), 26 ng/l testosterone

192 Results showed that 17beta-estradiol equivalent levels were higher than thos

193 ptor activities, with 13 surface waters with 17beta-estradiol-equivalent (E2Eq) activities greater th

194 ore the WWTP replacement, in vitro ER (24 ng 17beta-estradiol equivalents/L)-, GR (60 ng dexamethason

195 Sex steroid hormones such as 17beta-estradiol (estradiol) regulate neuronal function

196 e estrogens included for study were estrone, 17beta-estradiol, estriol, 17alpha-ethinylestradiol, and

197 Signaling of 17beta-estradiol (estrogen) through its two nuclear rece

198 Four steroidal estrogens (17beta-estradiol, estrone, estriol, and 17alpha-estradio

199 Although 17beta-estradiol has long been known to regulate memory

200 17beta-Estradiol improved microcirculatory perfusion and

201 Additionally, post-SCI administration of 17beta-estradiol improved skilled forelimb function and

202 Long-term cyclic administration of 17beta-estradiol improves working memory, and restores h

203 e selective detection of the water pollutant 17beta-estradiol in buffer and tap water.

204 me inexpensive and easy-to-use monitoring of 17beta-estradiol in environmental samples such as efflue

205 , whereas it mimics the protective action of 17beta-estradiol in other tissues such as arteries.

206 , showing the ability of detecting traces of 17beta-estradiol in serum at concentrations lower than t

207 We here report on the immunodetection of 17beta-estradiol in serum by antibody-immobilized microc

208 n castrated male mice, and IL-6 induction by 17beta-estradiol in splenocytes from naive female mice (

209 construct a voltammperometric biosensor for 17beta-estradiol in the 0.9-11 pM range.

210 This study revealed that 17beta-estradiol in the brain mediated the physiological

211 et of seizures by promoting the synthesis of 17beta-estradiol in the brain.

212 ot only the expression of P450arom, but also 17beta-estradiol in the cerebral cortex.

213 s genomic/transcriptional) demonstrated that 17beta-estradiol-induced acceleration of endothelial hea

214 type 16 E6 and E7, short-term treatment with 17beta-estradiol induces CINs that progress to cervical

215 17beta-Estradiol is a multi-active steroid that imparts

216 4+CD25+FoxP3+ regulatory T-cells (Tregs) and 17beta-estradiol is crucial in the pathogenesis of sex b

217 lower threshold for synaptic plasticity when 17beta-estradiol is elevated.

218 The steroid 17beta-estradiol is known to acutely potentiate glutamat

219 thinyl estradiol (EE), a synthetic analog of 17beta-estradiol, is prescribed commonly and found in or

220 Attenuation of 17beta-estradiol (k(stream) = -3.2 +/- 1.0 day(-1)) was

221 ensor surface, and a higher concentration of 17beta-estradiol leads to less fluorescence-labeled DNA

222 17beta-estradiol led to a significant upregulation in pe

223 utcomes were percent change from baseline in 17beta-estradiol levels (E2) and tricuspid annular plane

224 A P450arom inhibitor, letrozole, reduced 17beta-estradiol levels and completely suppressed the el

225 eceptor-dependent inhibition in females when 17beta-estradiol levels are elevated.

226 tolerance, and suggest approaches to restore 17beta-estradiol levels as a novel treatment option for

227 onbreeding conditions by manipulating plasma 17beta-estradiol levels in wild-caught female Gambel's w

228 n Cyp19a1 suppression, decreased circulating 17beta-estradiol levels, abnormal fat accumulation, and

229 elopment, and photoproducts alter whole-body 17beta-estradiol levels.

230 n in males, was positively related to plasma 17beta-estradiol levels.

231 (Cyp19a1) expression and reduced circulating 17beta-estradiol levels.

232 ial to influence the human daily exposure to 17beta-estradiol like activity in various risk groups wi

233 that treatment of postmenopausal women with 17beta-estradiol markedly enhances TLR-7- and TLR-9-depe

234 Thus, the anti-oxidative effects of 17beta-estradiol may be involved in the prevention of se

235 om naive female mice (p<0.05) suggested that 17beta-estradiol may enhance sex bias through IL-6 induc

236 Osteocalcin and 17beta-estradiol mediate their effects through G protein

237 17beta-Estradiol-mediated enhancement of ethanol-induced

238 17beta-Estradiol mediates the sensitivity to pain and is

239 vitro culture of isolated uterine ILC2s with 17beta-estradiol modified expression of a number of gene

240 about the molecular mechanisms through which 17beta-estradiol modulates hippocampal memory.

241 The acute vasodilatory effects of 17beta-estradiol (non-specific estrogen receptor (ER) ag

242 itment to the cell cycle following strain or 17beta-estradiol occurs within 30 min, as determined by

243 stration of dexamethasone or prednisolone or 17beta-estradiol on Charolais bulls.

244 receptor (GPER) activation mimics effects of 17beta-estradiol on hippocampal memory consolidation.

245 characterize the effects of DHEA, prolactin, 17beta-estradiol on insulin-growth factor-1 and -2 (IGF-

246 to determine the molecular level effects of 17beta-estradiol on single MCF-7 cells using Fourier tra

247 DC lineage abrogated the enhancing effect of 17beta-estradiol on their TLR-mediated production of IFN

248 Delivery of 17beta-estradiol or an estrogen receptor (ER)-alpha (but

249 ranscriptional activity include the agonists 17beta-estradiol or conjugated estrogens with the antago

250 on proestrous morning, when serum levels of 17beta-estradiol peak, the nonspecific opioid receptor a

251 women with unopposed estrogen, we implanted 17beta-estradiol pellets in adult female Pten heterozygo

252 ng/ml E.coli flagellin (F) for 12 h, or 4 nM 17beta-estradiol plus 50 ng/ml flagellin (E + F(12 h)).

253 mice synchronized into estrus by delivery of 17beta-estradiol prior to intravaginal challenge with wi

254 17beta-estradiol production by LSCC cell lines UM-SCC-11

255 data suggest that post-SCI administration of 17beta-estradiol protected both the gray and white matte

256 In conclusion, 17beta-estradiol protects osteocytes against apoptosis b

257 study, we elucidated the mechanism by which 17beta-estradiol regulates the transcription of SNAT2.

258 17beta-estradiol replacement in SERT (-/-) mice reversed

259 that the anti-apoptotic effects of cGMP and 17beta-estradiol required BAD phosphorylation on Ser(136

260 Akt and ERK1/2 activation by 17beta-estradiol required PKG II, and cGMP mimicked the

261 CC cultures and xenografts were examined for 17beta-estradiol responsiveness in vivo.

262 The study provides evidence that 17beta-estradiol restores the tetramer-to-monomer ratio

263 Coinjection with 17beta-estradiol resulted in a decrease in (18)F-FES upt

264 A structure of the ancestor with 17beta-estradiol revealed only one molecule in the activ

265 that maximal binding occurred at the highest 17beta-estradiol serum concentration.

266 In contrast, 17beta-estradiol significantly increased the abundance o

267 DHEA-sulfate (DHEA-S), DHEA, and 17beta-estradiol stimulated keratinocyte and fibroblast

268 g of each of these proteins nearly abolished 17beta-estradiol-stimulated SNAT2 promoter activity.

269 ith estrogen receptor alpha (ER-alpha) after 17beta-estradiol stimulation.

270 Ovariectomized females supplemented with 17beta-estradiol succumbed to P. aeruginosa challenge ea

271 sses seizures, focusing on the regulation of 17beta-estradiol synthesis in the brain.

272 estrogens (o-CEE), 0.45 mg/d, or transdermal 17beta-estradiol (t-E2), 50 mcg/d, each with 200 mg of o

273 Here we demonstrate that 17beta-estradiol, tamoxifen, and fulvestrant induce nucl

274 tes with PMCA with or without treatment with 17beta-estradiol, thapsigargin, or G-1.

275 In animals, 17beta-estradiol (the major estrogen in most mammals, re

276 When treated with 17beta-estradiol, the lipid utilization in cancer cells

277 with potential carcinogenic effects such as 17beta-estradiol, the most powerful substance with estro

278 Like 17beta-estradiol, the non-steroid Br-PBTC only requires

279 HED for the development of a brain-selective 17beta-estradiol therapy to relieve hot flushes without

280 and that transcript levels are modulated by 17beta-estradiol through the estrogen receptor (ER)alpha

281 how that a single intracerebral injection of 17beta-estradiol to ovariectomized female rats immediate

282 hoeae following treatment with water-soluble 17beta-estradiol to promote long-term gonococcal infecti

283 tion of VMH PI3K activity blocked effects of 17beta-estradiol to stimulate energy expenditure, but di

284 scriptional coregulator that is recruited by 17beta-estradiol to the promoters of estrogen target gen

285 17beta-Estradiol-treated ovariectomized female mice demo

286 17beta-Estradiol treatment was associated with 2-fold in

287 grow larger in vivo in response to systemic 17beta-estradiol treatments.

288 vertebra (C5) followed by administration of 17beta-estradiol via a slow release pellet (0.5 or 5.0 m

289 l squamous cell carcinoma (LSCC) responds to 17beta-estradiol via estrogen-receptor (ER, transcribed

290 ation reaction between 17alpha-estradiol and 17beta-estradiol via estrone was observed in aqueous sol

291 17beta-estradiol was detected below its quantitation lim

292 trone was detected in all of the samples and 17beta-estradiol was detected in one.

293 17beta-Estradiol was effective in improving mesenteric p

294 The dose-response curve of 17beta-estradiol was established and a detection limit w

295 lipid synthesis in cancer cells treated with 17beta-estradiol was increased by 42%.

296 Coinjection of (18)F-FES with 17beta-estradiol was performed to determine whether trac

297 For comparison, estrone and 17beta-estradiol were modeled and are likely capable of

298 mples containing different concentrations of 17beta-estradiol were premixed with a given concentratio

299 d as vitellogenesis-related and regulated by 17beta-estradiol were significantly enriched among those

300 These profound differences are influenced by 17beta-estradiol, which contributes both to T cell activ