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1 ation of polyPs in the acidocalcisomes using 4',6-diamidino-2-phenylindole.
2 hese embryos were dispersed and treated with 4',6-diamidino-2-phenylindole.
3 rmed by visualization of nuclei stained with 4,6'-diamidino-2-phenylindole.
4 ization of polyP in the dense granules using 4',6-diamidino-2-phenylindole and by its release togethe
5 taining with anti-alpha-tubulin antibody and 4',6-diamidino-2-phenylindole and flow cytometric analys
6 ibe activation of the channel by diminazene, 4',6-diamidino-2-phenylindole, and pentamidine, examine
7 rns after combined propidium iodide (PI) and 4', 6-diamidino-2-phenylindole (DAPI) staining.
8 pected for intracapsid steric restriction of 4',6-diamidino-2-phenylindole (DAPI) binding to packaged
9                                Staining with 4',6-diamidino-2-phenylindole (DAPI) indicated that poly
10 , we show that the ICD of minor-groove-bound 4',6-diamidino-2-phenylindole (DAPI) originates from an
11 ing magnetic resonance imaging, mCherry with 4',6-diamidino-2-phenylindole (DAPI) staining and green
12 tending toxin (Cdt), connective tissue (CT), 4',6-diamidino-2-phenylindole (DAPI), human gingival epi
13           cytolethal distending toxin (Cdt), 4',6-diamidino-2-phenylindole (DAPI), human gingival epi
14 tron microscope after labeling in vitro with 4',6-diamidino-2-phenylindole (DAPI), intracellular inje
15                                              4',6-diamidino-2-phenylindole (DAPI), netropsin, and pen
16  nuclei frequently do not stain equally with 4',6-diamidino-2-phenylindole (DAPI), suggesting that by
17 ex affinity exhibited by 5PTB, netropsin and 4',6-diamidino-2-phenylindole (DAPI), two AT-specific mi
18 nds, ibuprofen (IBU), fluorescein (FLU), and 4',6-diamidino-2-phenylindole (DAPI), was achieved with
19 surement of changes in the fluorescence of a 4',6-diamidino-2-phenylindole (DAPI)-dsDNA complex upon
20 ssay based on changes in the fluorescence of 4',6-diamidino-2-phenylindole (DAPI)-dsDNA complexes.
21 ty of a fluorescent DNA minor groove binder, 4',6-diamidino-2-phenylindole (DAPI).
22 en bond with the minor groove binding ligand 4',6-diamidino-2-phenylindole (DAPI).
23 de macrophages were stained with the DNA dye 4',6-diamidino-2-phenylindole (DAPI).
24 yed in the method: TO-PRO-3 iodide (TP3) and 4',6-diamidino-2-phenylindole (DAPI).
25 t methods: staining intact chloroplasts with 4',6-diamidino-2-phenylindole (DAPI); staining at the si
26 ely charged dyes, propidium iodide (PrI) and 4'-6-diamidino-2-phenylindole (DAPI).
27 le chromosome probes, single-copy genes, and 4'-6-diamidino-2-phenylindole (DAPI-) and G-banded chrom
28 ick-end labeling (TUNEL) in conjunction with 4'6'-diamidino-2-phenylindole (DAPI) staining and by flu
29 es by assessment of nuclear fragmentation by 4, 6-diamidino-2-phenylindole (DAPI) staining.
30 nted in this investigation clearly show that 4,6-diamidino-2 phenylindole (DAPI) is superior to both
31 scent images of the chromosomes stained with 4,6-diamidino-2-phenylindole (DAPI) [5].
32 inor groove binding compounds, netropsin and 4,6-diamidino-2-phenylindole (DAPI), and a DNA hairpin h
33 of cell extracts, and staining of cells with 4,6-diamidino-2-phenylindole (DAPI), and the polyP-bindi
34         Two types of foci have been defined: 4,6-diamidino-2-phenylindole (DAPI)-sensitive foci that
35 s, the peptide gramicidin, and a DNA-ligand (4',6-diamidino-2-phenylindole, DAPI) system in this work
36   Cytochemical staining of cellular DNA with 4,6-diamidino-2-phenylindole demonstrated TNF-alpha-indu
37 ensitive fluorescent reagents SYPRO Ruby and 4',6-diamidino-2-phenylindole dihydrochloride (DAPI), to
38                   Apoptosis was evaluated by 4',6-diamidino-2-phenylindole dihydrochloride and termin
39 /PMS kit and of dead (apoptotic) cells using 4',6-diamidino-2-phenylindole dihydrochloride nuclear st
40 uorescent dye molecules per CPMV using DAPI (4',6-diamidino-2-phenylindole dihydrochloride), propidiu
41                  Microbes were enumerated by 4',6-diamidino-2-phenylindole, dihydrochloride (DAPI) st
42 NA was stained with propidium iodide (PI) or 4',6-diamidino-2-phenylindole, dihydrochloride (DAPI).
43  Furthermore, microtubule immunostaining and 4,6-diamidino-2-phenylindole DNA staining demonstrated t
44 in condensation, as observed with the use of 4,6-diamidino-2-phenylindole-fluorescent staining, and b
45          These cells were later stained with 4', 6-diamidino-2-phenylindole for simultaneous DNA anal
46  cell level using double immunostaining plus 4,6-diamidino-2-phenylindole (for nuclei) counterstainin
47 3), leading to H3.3 deposition on H3K9me3(+)/4',6-diamidino-2-phenylindole(+) heterochromatin nuclear
48                                        DAPI (4,6-diamidino-2-phenylindole) inhibited the assembly of
49                   It involves segmenting the 4',6-diamidino-2-phenylindole-labelled image into cells
50 unctate nuclear localization correlated with 4,6-diamidino-2-phenylindole light regions and is exclud
51 niformly; locally high levels accumulated in 4',6-diamidino-2-phenylindole-negative zones containing
52 assessed by trypan blue exclusion assays and 4',6-diamidino-2-phenylindole nuclear staining.
53             Using the nucleus-specific stain 4',6-diamidino-2-phenylindole, nuclear fragmentation was
54  of fixed permeabilized red blood cells with 4',6'-diamidino-2-phenylindole or YOYO-1, a sensitive nu
55 ite-specific probes, in addition to inverted 4,6-diamidino-2-phenylindole or conventional G-banding a
56  fluorescent foci that colocalize with DAPI (4',6'-diamidino-2-phenylindole)-positive material and fo
57 ence contrast and fluorescence microscopy of 4',6-diamidino-2-phenylindole-stained log phase cells.
58 undergoing apoptosis verifies that the FD of 4',6-diamidino-2-phenylindole-stained nuclear structures
59 ense dot-like signal always colocalized with 4',6-diamidino-2-phenylindole-stained nuclei.
60 distribution of the actin cytoskeleton, DNA (4', 6-diamidino-2-phenylindole staining), and calcofluor
61 e-contrast microscopy, and in selected cases 4',6'-diamidino-2-phenylindole staining and DNA fragment
62                                              4',6'-diamidino-2-phenylindole staining demonstrated tha
63 poptotic, as assessed by Annexin V staining, 4',6'-diamidino-2-phenylindole staining, and DNA fragmen
64                                 We also used 4',6-diamidino-2-phenylindole staining of isolated plast
65 enome was developed based on the patterns of 4',6-diamidino-2-phenylindole staining of the Nipponbare
66 ase-mediated dUTP nick-end labeling (TUNEL), 4',6-diamidino-2-phenylindole staining, and immunohistoc
67 ristic apoptotic changes, as demonstrated by 4',6-diamidino-2-phenylindole staining, lack of either D
68 also localized to the contractile vacuole by 4',6-diamidino-2-phenylindole staining, suggesting, with
69 ured by caspase-3 activity assay, TUNEL, and 4',6-diamidino-2-phenylindole staining.
70 ing a combination of chromosome painting and 4',6-diamidino-2-phenylindole staining.
71 titanium surfaces were measured according to 4'-6-diamidino-2-phenylindole staining.
72 romatin fragmentation by acridine orange and 4'6-diamidino-2-phenylindole staining, and (3) agarose g
73 nd changes in nuclear morphology detected by 4,6-diamidino-2-phenylindole staining, DNA fragmentation
74                                        DAPI (4',6'-diamidino-2-phenylindole) staining of the arrested
75                                  Also, DAPI (4',6'-diamidino-2-phenylindole) staining revealed that c
76 mal fragmentation of cellular DNA, and DAPI (4',6-diamidino-2-phenylindole) staining revealed condens
77 deoxyuridine (BrdU) pulse-labeling and DAPI (4',6-diamidino-2-phenylindole) staining, which precisely
78 sitive for cytokeratins 8, 18, and/or 19 and 4',6-diamidino-2-phenylindole were considered to be CTCs