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1 st a lethal challenge dose of wild-type (WT) A. hydrophila.
2 cal activities of a diarrheal isolate SSU of A. hydrophila.
3 which could alter the virulence potential of A. hydrophila.
4 e wild-type (WT) and complemented strains of A. hydrophila.
5 )-positive or -negative background strain of A. hydrophila.
6 he detailed mechanisms of action of Act from A. hydrophila.
7 y play an important role in the virulence of A. hydrophila.
8 la and phage showed significant reduction of A. hydrophila.
9 actone at 10 microM in overnight cultures of A. hydrophila abolishes exoprotease production in azocas
10 egulates the most-potent virulence factor of A. hydrophila, Act.
11                               These data add A. hydrophila and A. salmonicida to the growing family o
12          Act is a potent virulence factor of A. hydrophila and has been shown to contribute significa
13            Fish and chicken meat spiked with A. hydrophila and phage showed significant reduction of
14 ng a correlation between the TTSS and Act of A. hydrophila and the production of lactones.
15 of the environmental isolate ATCC 7966(T) of A. hydrophila and the vacB gene of Shigella flexneri.
16 nd the T3SS from a diarrheal isolate, SSU of A. hydrophila, and defined the role of some regulatory g
17  of the TTSS translocon, from wild-type (WT) A. hydrophila as well as from a previously characterized
18 e was then introduced into the chromosome of A. hydrophila by using the suicide vector pJQ200SK, allo
19 d that only the full-length ACD of RtxA from A. hydrophila catalyzed the covalent cross-linking of th
20 her hand, the WT and complemented strains of A. hydrophila caused 80 to 90% of the mice to succumb to
21         Inactivation of the ahyI gene on the A. hydrophila chromosome abolishes C4-HSL production.
22 , a 2.6-kb SalI/HindIII DNA fragment from an A. hydrophila chromosome was cloned and sequenced.
23                  The exoprotease activity of A. hydrophila consists of both serine protease and metal
24 lled 100% of the animals inoculated with the A. hydrophila control strain.
25 ron regulation in the fur isogenic mutant of A. hydrophila could be restored by complementation.
26 rease in gidA and act gene expression in the A. hydrophila Dam-overproducing strain, and these data m
27 tion, we showed that animals challenged with A. hydrophila die because of kidney and liver damage, hy
28                             The tagA gene of A. hydrophila exhibited 60% identity with that of a rece
29              In contrast, the wild-type (WT) A. hydrophila exhibited significant growth at this low t
30 ementation experiments demonstrated that the A. hydrophila fur gene could restore iron regulation in
31 la is more similar to the X. campestris than A. hydrophila genes.
32                                    Thus, the A. hydrophila genome sequence provides valuable insights
33                                           An A. hydrophila genomic library was transferred into a P.
34 Members of the Aeromonas hydrophila complex (A. hydrophila, HG2, and A. salmonicida), a group that ha
35 table aspects of the metabolic repertoire of A. hydrophila include dissimilatory sulfate reduction an
36 established a role for three enterotoxins in A. hydrophila-induced gastroenteritis in a mouse model w
37 neutropenic animals were more susceptible to A. hydrophila infection than normal mice.
38 mice and enhances their survivability during A. hydrophila infection.
39 ted mice with the above AHLs prior to lethal A. hydrophila infection.
40 er predominant immune cells inflicted during A. hydrophila infections, such as murine macrophages, wh
41 sition of the S-layer on the cell surface in A. hydrophila is more similar to the X. campestris than
42                              In contrast, WT A. hydrophila killed 100% of the mice within 48 h.
43 y of the effector domains of V. cholerae and A. hydrophila MARTX toxins to elucidate the mechanism of
44          The biological activity of selected A. hydrophila mutants was restored after complementation
45 ulture supernatants from deletion mutants of A. hydrophila, namely, a Delta act mutant (a T2SS-associ
46 s of T6SS and ExoA in pathogenesis caused by A. hydrophila NF strains in both mouse peritonitis and N
47                                          The A. hydrophila pilD homologue, tapD, was identified by it
48 emonstrated for the first time that VgrG1 of A. hydrophila possessed actin ADPRT activity associated
49        Our study is the first to report that A. hydrophila possesses a functional RtxA having an ACD
50 ic activity against both vAh strains and the A. hydrophila reference strain ATCC 35654.
51             Overproduction of mutated Dam in A. hydrophila resulted in bacterial motility, hemolytic
52                                          The A. hydrophila RNase R-lacking strain was found to be les
53 rther, we showed that the full-length ACD of A. hydrophila RtxA disrupted the actin cytoskeleton of H
54 ed the DNA adenine methyltransferase gene of A. hydrophila SSU (dam(AhSSU)) in a T7 promoter-based ve
55 otentially be important for the viability of A. hydrophila SSU as we could delete the chromosomal cop
56 o acid residues ((252)FYDAEKKEY(260)) in the A. hydrophila SSU enolase involved in plasminogen bindin
57 ace-expressed enolase in the pathogenesis of A. hydrophila SSU infections and of any gram-negative ba
58 inished by 55% compared to that of a control A. hydrophila SSU strain harboring the pBAD vector alone
59 spectively, compared to those of the control A. hydrophila SSU strain.
60 am gene to be essential for the viability of A. hydrophila SSU, and, therefore, to better understand
61  the bacterium, and overproduction of Dam in A. hydrophila SSU, using an arabinose-inducible, P(BAD)
62 ence the expression of act and gidA genes in A. hydrophila SSU.
63  the act/aopB mutant, compared to that of WT A. hydrophila SSU.
64 generated a fur isogenic mutant of wild-type A. hydrophila SSU.
65 ns 343, 394, 420, 427, and 430 of enolase in A. hydrophila SSU; the mutated forms of enolase were hyp
66 ateral flagellum, that are reported in other A. hydrophila strains are not identified in the sequence
67 del was used, whereby either single or mixed A. hydrophila strains were injected intramuscularly.
68                                  We purified A. hydrophila TagA as a histidine-tagged fusion protein
69 Act was noted in the gidA isogenic mutant of A. hydrophila that was generated by marker exchange muta
70 luid secretion compared to that of wild-type A. hydrophila; the triple-knockout mutant failed to indu
71 toxic enterotoxin gene (act)-minus strain of A. hydrophila, thus generating aopB and act/aopB isogeni
72                  The tagA isogenic mutant of A. hydrophila, unlike its corresponding wild-type (WT) o
73                                          The A. hydrophila VacB protein contained 798 amino acid resi
74 n together, our data indicated alteration of A. hydrophila virulence by overproduction of Dam.
75 e of RNase R in modulating the expression of A. hydrophila virulence.
76                               The RNase R of A. hydrophila was a cold-shock protein and was required
77  as well as from the clinical isolate SSU of A. hydrophila, was exclusively expressed and produced du