ライフサイエンスコーパス: ABR

1 ABR exhibited a moderate HOMO-LUMO gap (1.827 eV) and an

2 ABR gene profiles were maintained in recipients for up t

3 ABR latency findings showed a significantly greater incr

4 ABR shifts after combination treatment were similar to t

5 ABR thresholds develop rapidly in normal, euthyroid anim

6 ABR thresholds differed between 129S-Cdh23(c.753A) SNV a

7 ABR thresholds increased maximally immediately after exp

8 ABR-217620 was well tolerated with evidence of immunolog

9 ABRs in AQP4 null CD1 mice measured in response to tone

10 ABRs were obtained by using 4-, 8-, 16-, and 32-kHz tone

11 ata demonstrate a critical role for the ZO-1 ABR in barrier function and suggest that MLCK-dependent

12 The identification of BCL-6 ABR provides new tools for the diagnosis of lymphomas ca

13 In this study, we investigated abrocitinib (ABR), a selective JAK1 inhibitor, as a candidate for rep

14 our of eight Coch(G88E/G88E) mice had absent ABRs at all frequencies tested and two of three Coch(G88

15 two of three Coch(G88E)(/+) mice had absent ABRs at three of four frequencies tested.

16 in the same Coch(G88E/G88E) mice with absent ABRs.

17 essment of tinnitus, in this study, acoustic ABR (aABR) and optoacoustic ABR (oABR) were compared in

18 In addition, ABR-001 achieves a high level of genome editing when del

19 st-hoc efficacy analyses, including adjusted ABR, factor IX activity, participants within factor IX r

20 therapy to the lead-in period, mean adjusted ABR significantly reduced from 4.18 to 1.51 (p=0.0002) f

21 ve focused on CS strategies designed against ABR mediated by chromosomal mutations.

22 These changes are associated with altered ABR latency during development.

23 sed estimates of GPP over the North American ABR.

24 An ABR was obtained after removal of the probe to assess lo

25 er loci in Atp6v1b1vtx/vtx mice, we analysed ABR thresholds of progeny from a backcross segregating M

26 ion of auditory brainstem response analyses (ABR) and electron microscopy, we identified an unexpecte

27 ADPHd expression increased with both age and ABR thresholds in the medial superior olive but not in e

28 ry test battery that included audiometry and ABR measures.

29 holds had recovered, but synaptic counts and ABR amplitudes at high frequencies were reduced by up to

30 DPOAE and ABR exhibited an increasing threshold at high frequencie

31 had slightly, but reliably, lower DPOAEs and ABR wave V amplitudes than HIV-negative controls.

32 nificant changes in Aqp4 gene expression and ABR and DPOAE hearing status in the cochlea and auditory

33 MRE prevented the inflammation and ABR by reducing TNF-alpha and IL-1beta, preventing the o

34 While acoustic PPI remained intact and ABR thresholds recovered, the ABR wave P1-N1 amplitude r

35 ssion of immediately adjacent genes, such as ABR, and could be a common mechanism in the causation of

36 n the OP to 1.39 (0.85-2.29) during the ATP (ABR ratio 0.07 [0.042-0.118]; 2-sided P<.0001).

37 received the 9 x 10(11) vg dose, the average ABR threshold was improved from greater than 95 dB at ba

38 eceived 1.5 x 10(12) AAV1-hOTOF, the average ABR thresholds changed from greater than 95 dB at baseli

39 5-week-old mice in a CD1 genetic background, ABR thresholds in response to a click stimulus were rema

40 ods applied to evaluate arterial baroreflex (ABR) function provide the same information as the modell

41 Model-based ABR for treated bleeds was 0.4 (95% confidence interval,

42 um prestin was significantly elevated before ABR threshold shifts in mice exposed to cisplatin compar

43 We found significant correlations between ABR thresholds and the loss of outer hair cells and spir

44 reflects the difference between the binaural ABR and the sum of the monaural ABRs (i.e., binaural - (

45 on, treated with the specific S100A9 blocker ABR-238901 for 7 or 21 days.

46 from noise-induced hearing loss (NIHL): both ABR threshold shifts and hair cell death were significan

47 Both ABRs and IPM-FRs were observed most clearly from similar

48 At high frequencies, both ABRs and DPOAEs were attenuated by loss of M2 and/or M4,

49 ignificant increase in the threshold of both ABRs and a significant decrease in the amplitude of wave

50 tem response (ABR) threshold, the tone-burst ABR threshold and the auditory steady-state response (10

51 On an individual level, click and tone-burst ABR thresholds, but not the auditory steady-state respon

52 xposure in a mouse model system, measured by ABR.

53 engineered Type V-I CRISPR system (Cas12i), ABR-001, which utilizes a tracr-less guide RNA.

54 Although all subjects displayed clear ABRs, the BIC was not reliably observed.

55 rs and bacteria should be explored to combat ABR and propose more tailored solutions in a specific re

56 We compared ABR thresholds and cochlear pathologies of these SNV mic

57 associated with the acquisition of complete ABR plasmids, including the clinically important carbape

58 7620 followed by docetaxel in 21-day cycles (ABR-217620 dose escalation combination with docetaxel [C

59 Older adults had delayed ABRs, especially in response to the rapidly changing for

60 ely 70% of Clrn1(-/-) mice had no detectable ABR and by P30 these mice were deaf.

61 d acoustically based, yielded very different ABRs between the two sound classes.

62 with similar ABR threshold, and 6) elevated ABR thresholds and deceased wave I amplitudes at high fr

63 normal VsEPs with normal to mildly elevated ABR thresholds.

64 In Mcu(+/-) mice, significantly elevated ABR thresholds were observed at 6 months and 9 months of

65 ean (95% confidence interval [CI]) estimated ABR declined from 19.78 (16.12-24.27) in the OP to 1.39

66 rve and brainstem nuclei, and a click-evoked ABR typically shows three positive peaks ("waves") withi

67 /55 dB sound pressure level and click-evoked ABR using a slow (21.1/s) and fast (61.1/s) click rate.

68 at this locus (genotype ahl2/ahl2) exhibited ABR thresholds that were on average 26 decibels above th

69 hese effects manifested as larger and faster ABRs to deviant stimuli, with the strongest responses oc

70 19; noninhibitor, n = 46) were eligible for ABR analyses.

71 Interventions for ABR typically target antibiotic use, overlooking the wid

72 Patients who opted for ABR had significantly higher satisfaction with their bre

73 The free ABR also prevented increases in barrier function followi

74 Moreover, the free ABR interfered with full-length ZO-1 exchange and reduce

75 ake no longer than a few minutes, apart from ABR testing which takes upto 3.5 h per mouse.

76 nd GN-ARB reservoirs, is crucial to curve GN-ABR transmission.

77 Bayesian regularized cardiac strain imaging (ABR-CSI) algorithm for in vivo murine myocardial functio

78 t/rapamycin-based maintenance immunotherapy (ABR) prevents kidney allograft rejection and specificall

79 ss of ribbons in both regions and changes in ABR sensitivity and dynamic responsiveness.

80 sticity that matched commensurate changes in ABR wave 4.

81 he more robust ABR wave-V mirrors changes in ABR wave-I amplitude.

82 with ABRs during development, a decrease in ABR thresholds between P13 and P15, and anatomical resul

83 adult CBA/CaJ mice prevented the decrease in ABR wave-I amplitude and the synaptopathy relative to in

84 t VsEPs) with mild to moderate elevations in ABR thresholds.

85 creased noise vulnerability was seen only in ABRs, consistent with a role for dopaminergic signaling

86 fluoroquinolone, and antibiotic inactivation ABR genes were increased in RCDI patients, although dono

87 PNE also increased ABR thresholds and reduced ABR peak amplitudes, indicati

88 T-dfw2J/ + heterozygotes displayed increased ABR thresholds, suggesting that a second locus, controll

89 etermined by showing significantly increased ABR thresholds 6 weeks after adoptive transfer of peptid

90 Individual ABR waves showed a series of distinct spatial topographi

91 We investigated ABR S. aureus nasal carriage prevalence among adults wit

92 e overall median (interquartile range [IQR]) ABR was 1.49 (0.00-4.40) for subjects on variable prophy

93 ts included spontaneous ABR (AsBR) and joint ABR (AjBR).

94 nnual bleeding rate (ABR), spontaneous joint ABR, the number of infusions and dose required to resolv

95 y and in the control of bleeding events, low ABR in patients on prophylaxis, and a favorable safety p

96 fe compared with rFVIII, and resulted in low ABRs when dosed prophylactically 1 to 2 times per week.

97 nd 0.28 muV (95% CI, 0.01 to 0.33 muV) lower ABR wave V amplitudes in the right ear.

98 stablish normative values of forward-masking ABRs and investigate the development of auditory tempora

99 Estimated mean ABRs were substantially reduced with fitusiran by 79.7%

100 The median ABR was 2.0 (0.0-3.1) overall and 0.0 (0.0-0.0) for spon

101 Median ABRs observed in arms 1, 2, and 3 were 1.6, 3.6, and 33.

102 Observed median ABRs were 6.5 (interquartile range [IQR], 2.2-19.6)/4.4

103 atment strategies to combat plasmid-mediated ABR.

104 the binaural ABR and the sum of the monaural ABRs (i.e., binaural - (left + right)).

105 oves the anatomical specificity of the mouse ABR as a noninvasive marker in biomedical mouse models.

106 The orthodontic tooth movement-ABR model was used to move a molar into the regenerated

107 Nevertheless, ABRs show longer latencies in central waves (II-IV) that

108 This QTL is responsible for 57 and 43% of ABR threshold variance, respectively, in each strain com

109 ed statistically significant associations of ABR thresholds with markers on chromosome 5, with a peak

110 Two phase I studies were conducted of ABR-217620 alone or in combination with docetaxel.

111 tly with a decreased number and diversity of ABR genes and increased Bacteroidetes and Firmicutes wit

112 tients had a greater number and diversity of ABR genes compared with donors and healthy controls.

113 NSCLC received 4 daily, escalating doses of ABR-217620 followed by docetaxel in 21-day cycles (ABR-2

114 However, one of the main drivers of ABR in clinically relevant bacteria is the horizontal tr

115 iotic-resistant organisms and elimination of ABR genes.

116 The extent of ABR S. aureus exposure in IHO workers and children livin

117 Although frequency of ABR genes remained stable, other accessory genes, especi

118 ably, despite its importance, the origins of ABR waves specific to mice remain poorly identified.

119 n of Spns2 led to less effective recovery of ABR thresholds, suggesting that there is a critical peri

120 evant bacteria is the horizontal transfer of ABR genes mediated by plasmids.

121 n humans, this study highlights the value of ABR recordings with excellent temporal resolution in inv

122 est that measures of the effects of noise on ABR wave-V latency can be used to diagnose cochlear syna

123 study, acoustic ABR (aABR) and optoacoustic ABR (oABR) were compared in the control and tinnitus gro

124 BREAST-Q scores from IBR or ABR patients at a tertiary center were prospectively col

125 product otoacoustic emission thresholds, or ABR wave I amplitudes.

126 ve treatment with the Q compound paquinimod (ABR-215757) during the first 5 days after induction of e

127 troduced by the American Board of Radiology (ABR) allows international medical graduates (IMGs) who h

128 Division of the American Board of Radiology (ABR) approved the studies (human subjects and HIPAA comp

129 ce to that of 4 American Board of Radiology (ABR) certified radiologists on an independent test set o

130 The American Board of Radiology (ABR) has provided certification for diagnostic radiologi

131 ncology and the American Board of Radiology (ABR) Written Radiation Oncology Board Examination.

132 ry endpoints included annualized bleed rate (ABR).

133 y end points included annualized bleed rate (ABR): treated, all, treated spontaneous, and treated joi

134 inferiority of the annualised bleeding rate (ABR) during the 52 weeks following stable factor IX expr

135 mary end point was annualized bleeding rate (ABR) in the BPA/CFC prophylaxis and fitusiran efficacy p

136 ary endpoints were annualized bleeding rate (ABR) of treated bleeds and safety.

137 imary endpoint was annualized bleeding rate (ABR) of treated spontaneous BEs (sABR) during rVWF proph

138 the median overall annualized bleeding rate (ABR) was 1.14 (Q1, Q3: 0.0, 4.2).

139 nd points included annualized bleeding rate (ABR), inhibitor development, and adverse events.

140 were pharmacokinetics, annual bleeding rate (ABR), spontaneous joint ABR, the number of infusions and

141 o-beta ratio (TBR), and alpha-to-beta ratio (ABR) responses) benefits.

142 t carotid, the arterial-wall-to-blood ratio (ABR) 4.5 h post-infusion was 2.13 +/- 0.35 in patients v

143 To assess this hypothesis, we recorded ABRs to the speech syllable /da/ in normal hearing young

144 NE also increased ABR thresholds and reduced ABR peak amplitudes, indicating impaired central auditor

145 bons correlates with the presence of reduced ABR peak one amplitudes in both levels of noise exposure

146 Tempol alone significantly reduced ABR threshold shifts on day 10 but not on day 1.

147 nisms underlying alveolar bone regeneration (ABR) and orthodontic tooth movement into bovine bone (BB

148 ing a similar N-terminal APC binding region (ABR) and Src-homology 3 (SH3) domain.

149 lts indicate that this actin-binding region (ABR) is both necessary and sufficient for binding to F-a

150 Substitutions in the YFV Ag-binding region (ABR) occur at four of the eight highly conserved residue

151 itro, ZO-1 lacking the actin binding region (ABR) was not stabilized by MLCK inhibition, either in th

152 d cover changes in the Arctic-Boreal region (ABR) have been poorly characterized.

153 In the Arctic and Boreal region (ABR) where warming is especially pronounced, the increas

154 ly identified alternative breakpoint region (ABR), showed a rearrangement.

155 we called the alternative breakpoint region (ABR), were found to be recurrent in lymphomas carrying t

156 anted use leads to antibacterial resistance (ABR) and causes serious health problems.

157 The emergence of antibacterial resistance (ABR) is an urgent and complex public health challenge wo

158 l health crisis of antibacterial resistance (ABR) poses a particular threat in low-resource settings

159 ity, and frequency of antibiotic resistance (ABR) and accessory genes.

160 oarray containing 354 antibiotic resistance (ABR) genes.

161 the rising problem of antibiotic resistance (ABR).

162 oods which accelerate antibiotic resistance (ABR).

163 pport the emergence of antibiotic-resistant (ABR) Staphylococcus aureus.

164 d its influence on alveolar bone resorption (ABR) in rats.

165 blink activity, auditory brainstem response (ABR) amplitudes, and tail-flick latency.

166 es, such as the auditory brainstem response (ABR) and distortion product otoacoustic emissions (DPOAE

167 measurements of auditory brainstem response (ABR) and distortion product otoacoustic emissions (DPOAE

168 neration of the auditory brainstem response (ABR) and receives direct innervation from the cochlea, i

169 suprathreshold auditory brainstem response (ABR) and reduced number of synapses between sensory hair

170 The auditory-evoked brainstem response (ABR) in Thrb-/- mice, although greatly diminished, displ

171 The auditory brainstem response (ABR) is a critical tool for assessing auditory brainstem

172 The auditory brainstem response (ABR) is an acoustically evoked EEG potential that is an

173 as assessed by auditory brainstem response (ABR) measurements, and their otolithic organs appeared n

174 sured using the auditory brainstem response (ABR) or distortion product otoacoustic emissions (DPOAE)

175 In the auditory brainstem response (ABR) test, auditory potentials can be evoked by acoustic

176 e average click auditory brainstem response (ABR) threshold, the tone-burst ABR threshold and the aud

177 elevated auditory-evoked brainstem response (ABR) thresholds and prolonged peak and interpeak latenci

178 in the average auditory brainstem response (ABR) thresholds at 0.5-4.0 kHz.

179 ferences in the auditory brainstem response (ABR) thresholds between mutant rats and WT controls.

180 ion (DPOAE) and auditory brainstem response (ABR) thresholds during and after middle ear infusion of

181 GLUT3 delivery, auditory brainstem response (ABR) thresholds normalize, along with partial rescue of

182 Auditory brainstem response (ABR) thresholds of Coch(G88E/G88E) mice were substantial

183 study that used auditory brainstem response (ABR) thresholds to identify QTLs affecting HFHL.

184 FME and auditory brainstem response (ABR) thresholds were closely aligned after noise-induced

185 Auditory brainstem response (ABR) thresholds were compared to VsEP thresholds for 14

186 essed by any of auditory brainstem response (ABR) thresholds, distortion product otoacoustic emission

187 Auditory brainstem response (ABR) thresholds, peak latencies, and amplitude growth we

188 dB had similar auditory brainstem response (ABR) thresholds, wave-1 amplitudes, and latencies.

189 TL analysis for auditory brainstem response (ABR) thresholds, which indicates hearing ability, was pe

190 neural output [auditory brainstem response (ABR) wave 1] and in outer hair cell function [distortion

191 icantly delayed auditory brainstem response (ABR) wave I latencies at low and middle frequencies comp

192 er latencies of auditory brainstem response (ABR) wave I, lower distortion product otoacoustic emissi

193 a reduction in auditory brainstem response (ABR) wave-I amplitude.

194 he amplitude of auditory brainstem response (ABR) wave-I.

195 was analyzed by auditory brainstem response (ABR) which confirmed that severe age-related hearing los

196 rived the human auditory brainstem response (ABR), a measure of subcortical encoding, to recorded mus

197 The auditory brainstem response (ABR), a scalp-recorded electrical potential, is known fo

198 resholds for an auditory brainstem response (ABR), at least at low to mid stimulus frequencies.

199 pecifically the Auditory Brainstem Response (ABR), provides objective measures of auditory processing

200 changes in the auditory brainstem response (ABR).

201 gical test, the auditory brainstem response (ABR).

202 ed based on the auditory brainstem response (ABR).

203 ssay and acoustic brainstem evoked response (ABR) threshold analysis were carried out to evaluate the

204 le in hearing, auditory brain stem response (ABR) thresholds were compared in wild-type mice and tran

205 re measured by auditory brain stem response (ABR).

206 nd frequency (auditory brainstem response -- ABR thresholds, and distortion-product otoacoustic emiss

207 ts (measured by auditory brainstem response, ABR) of up to 73 dB (16 kHz) on day 1, and up to 50 dB (

208 ectorin 71-90, auditory brainstem responses (ABR) showed significant hearing loss at all frequencies

209 he analysis of auditory brainstem responses (ABR) showed that mtl and bsd homozygotes are deaf, where

210 no discernible auditory brainstem responses (ABR) to sound pressure level stimuli up to 100 dB, indic

211 Mouse auditory brainstem responses (ABR), inner ear histology, perilymph cisplatin sampling,

212 as measured by auditory brainstem responses (ABR).

213 POAEs) and the auditory brainstem responses (ABRs) across the entire range of tested frequencies (4-4

214 amplitudes in auditory brainstem responses (ABRs) and decreased distortion product otoacoustic emiss

215 , we collected auditory brainstem responses (ABRs) and determined cochlear hair cell loss in 13-month

216 Auditory brainstem responses (ABRs) and distortion product otoacoustic emissions (DPOA

217 is we obtained auditory brainstem responses (ABRs) and micro-CT x-ray scans to measure changes in the

218 , we collected auditory brainstem responses (ABRs) from rhesus monkeys spanning in age from 10 to 35

219 ively recorded auditory brainstem responses (ABRs) to investigate deviance detection at population le

220 s by employing auditory brainstem responses (ABRs) to investigate the earliest neural correlates of d

221 Auditory brainstem responses (ABRs) were obtained in anesthetized animals for click an

222 present study, auditory brainstem responses (ABRs) were recorded in a forward-masking paradigm in hea

223 howed improved auditory brainstem responses (ABRs) with smaller latencies and lower thresholds.

224 y by recording auditory brainstem responses (ABRs)-the BIC reflects the difference between the binaur

225 rmally delayed auditory brainstem responses (ABRs).

226 hold shifts in auditory brainstem responses (ABRs).

227 ve V (central) auditory brainstem responses (ABRs).

228 tion (PPI) and auditory brainstem responses (ABRs).

229 emissions and auditory brainstem responses (ABRs).

230 Auditory brain stem responses (ABRs) from subjects reporting nontinnitus (controls), oc

231 , we measured auditory brain stem responses (ABRs), hair cell loss, and free radical activity in the

232 king noise on the latency of the more robust ABR wave-V mirrors changes in ABR wave-I amplitude.

233 estern Canada to characterize regional-scale ABR land cover changes.

234 cies compared with control mice with similar ABR threshold, and 6) elevated ABR thresholds and deceas

235 e-treated subjects had significantly smaller ABR threshold shifts on day 1 and on day 10.

236 onstruction to reevaluate the mouse-specific ABR origins.

237 Secondary end points included spontaneous ABR (AsBR) and joint ABR (AjBR).

238 ow that the device is capable of stimulating ABRs in vivo with latencies and growth functions compara

239 tection against noise-induced suprathreshold ABR wave-I amplitude reduction.

240 This suggests that interventions to tackle ABR need to take account of intersectional socio-environ

241 orouracil and capecitabine demonstrated that ABR achieved more favorable binding energies and greater

242 Regression analysis demonstrated that ABR patients had greater postoperative satisfaction with

243 The ABR from occasional tinnitus was indistinguishable from

244 The ABR is located 200-270 kb telomeric to MBR.

245 The ABR model was based on osseous defects filled with BB.

246 The ABR originates from the response sequence of auditory ne

247 The ABR V/I amplitude ratio was examined as a measure of cen

248 estingly, IEM-1460 itself did not affect the ABR threshold, presumably because GluA2-containing AMPAR

249 A primary tool used by the ABR in fulfilling this responsibility is the secure proc

250 t proposes actions that could facilitate the ABR certification process and help integrate these highl

251 Based on our findings, the ABR test has the potential to be used in the assessment

252 For the ABR MOC examinations, 1280 x 1024 displays should be use

253 omen, 1.95:1) had an initial sitting for the ABR written examination.

254 Furthermore, the ABR is required for localization to a novel actin-rich p

255 urements), and explains in each case how the ABR implements that standard.

256 ed the strength of deviance detection in the ABR.

257 eric CO(2) SCA amplification observed in the ABR.

258 Interestingly, the ABR in CBy-dfw2J/+ heterozygotes is also abnormal, showi

259 Translocations involving the ABR may juxtapose BCL-6 to distantly acting, heterologou

260 VSVG-tagged transgenes that lack the ABR still accumulate at both early and late cell-cell co

261 ever, accumulation of constructs lacking the ABR is markedly reduced at tight junctions in confluent

262 Moreover, the ABR of Asef2 did not function independently but acted in

263 edian +/- SE; range from 0.01% to 1%) of the ABR amplitude.

264 The top two CpGs annotated upstream of the ABR gene (cg01912040, cg10003262 ).

265 EEG can vary widely, and that wave II of the ABR is shaped by NM units.

266 However, an assignment of the waves of the ABR to specific sources is difficult, and a quantificati

267 ted peak coincided best with the peak of the ABR wave II, independent of the click sound level.

268 Transport of the ABR-anchored exchangeable pool is regulated by MLCK.

269 rther confirmed the dynamic stability of the ABR-PDZ1 complex.

270 In patients, the ABR correlated with the TBR of the corresponding vessel

271 effect was evident in patients receiving the ABR in that the repopulation of CD8+CD57+PD1- TEM cells

272 ned intact and ABR thresholds recovered, the ABR wave P1-N1 amplitude reduction persisted in all blas

273 ions in confluent cells, suggesting that the ABR does play an important role in the localization of Z

274 contacts in MDCK cells, suggesting that the ABR is not required for tight junction localization.

275 Our request to the ABR to permit us to study performance on their examinati

276 An average single-neuron contribution to the ABR was predicted by convolving the STA with the cell's

277 and a quantification of contributions to the ABR waves is not available.

278 to estimate single-cell contributions to the ABR.

279 In order to use the ABR test in the objective assessment of tinnitus, in thi

280 ecipients who underwent transplant using the ABR regimen were studied longitudinally.

281 lymphomas carrying rearrangements within the ABR showed that the breakpoints cluster within a 20-kb d

282 do not significantly differ for any of these ABR waves.

283 This ABR specialization indicates that even though YFV is pol

284 valence and potential sources of exposure to ABR S. aureus among children living with IHO workers.

285 Wistar rats were subjected to ABR by ligature with nylon thread in the second upper-le

286 ively, elevations in 4-, 8- and 20 kHz tonal ABR thresholds and reduced dynamic output at higher inte

287 loci account for more than 70% of the total ABR threshold variation among the backcross mice at all

288 Results were consistent by hemophilia type (ABR ratio 0.05 [hemophilia A, n=40]; 0.13 [hemophilia B,

289 patients were included, with 336 undergoing ABR and 2932 undergoing IBR.

290 Unfortunately, ABR wave-I is difficult to measure in humans, limiting i

291 The engineered variant, ABR-001, exhibits broad genome editing capability in hum

292 red with wild-type mice (p < 0.001), whereas ABR thresholds were not affected by AQP1, AQP3, or AQP5

293 nearly all AQP4 null mice were deaf, whereas ABRs could be elicited in wild-type controls.

294 While ABR wave latencies and interpeak intervals decreased ear

295 Conversely, PV expression correlated with ABR amplitudes and outer hair cell loss in the cochlea,

296 The strongest correlation with ABR thresholds was the number of different pathologies p

297 und highly significant linkage of Mass1 with ABR threshold variation among mice from two backcrosses

298 Regression analyses of these values with ABR properties and cochlear histopathologies revealed re

299 ng changes in the percentage of animals with ABRs during development, a decrease in ABR thresholds be

300 , it is unlikely that Ag is bound in the YFV ABR in the manner typical of class Ia molecules.