ライフサイエンスコーパス: AMPA-type

1 AMPA-type (alpha-amino-3-hydroxy-5-methyl-4-isoxazolepro

2 how conformation regulated interactions with AMPA-type and NMDA-type glutamate receptors (AMPARs/NMDA

3 Clusters of PSD-95 and subunits of AMPA-type and NMDA-type glutamate receptors accumulate i

4 ing were mimicked by intra-vmPFC blockade of AMPA-type but not NMDA-type glutamate receptors.

5 was markedly attenuated by a Ca2+ permeable AMPA-type (Ca-AMPA) glutamate channel blocker, or by a n

6 single subunit is sufficient to desensitize AMPA-type channels and that receptors with one to four g

7 ion with a Poisson train of fast excitatory (AMPA-type) conductance transients, to simulate independe

8 in, mediates homeostatic synaptic scaling of AMPA type glutamate receptors (AMPARs) via its ability t

9 We observe that antagonism of NMDA and AMPA type glutamate receptors protects neurons from cond

10 sporter 2 (KCC2) and the excitatory NMDA and AMPA type glutamate receptors.

11 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) type glutamate receptors mediate most fast synapti

12 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptor desensitization.

13 alpha-amino-3-hydroxy-5-methyl-4-isoxazole (AMPA)-type glutamate receptor has recently been demonstr

14 hydroxy-5-methyl-4-isoxazole-propionic acid (AMPA)-type glutamate receptor.

15 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPA-Rs), which mediate

16 3-hydroxy-5-methyl-4-isoxazolepropionic acid(AMPA)-type glutamate receptors (AMPARs) are the predomin

17 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) at Schaffer coll

18 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (AMPARs) mediate excitato

19 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) mediate the majo

20 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (AMPARs) to synapses is a

21 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (but not by blockade of N

22 ydroxy-5-methyl-4-isoaxazole propionic acid (AMPA)-type glutamate receptors (GluR1 and GluR2/3) durin

23 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors and the function of synap

24 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors and the stabilization of

25 ve IDRA 21 and other positive modulators of (AMPA)-type glutamate receptors are considered potential

26 o-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA)-type glutamate receptors cause the enhanced respon

27 hydroxy-5-methyl-4-isoxazoleproprionic acid (AMPA)-type glutamate receptors during long-term potentia

28 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors have distinct roles in co

29 no 3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA)-type glutamate receptors in rat brain and to test

30 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors mediate the majority of e

31 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors were studied using equili

32 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors, and is implicated in mul

33 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors, and thereby enhance fast

34 -hydroxy-5-methylisoxazole-4-propionic acid (AMPA)-type glutamate receptors, which become phosphoryla

35 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.

36 o-3-hydroxyl-5-methyl-4-isoxazolepropionate (AMPA)-type glutamate receptors.

37 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.

38 -hydroxy-5-methylisoxazole-4-propionic acid (AMPA)-type glutamate receptors.

39 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.

40 to spines, reduced the synaptic targeting of AMPA-type glutamate (GluR1) receptors, and decreased AMP

41 Modulation of AMPA-type glutamate channels is important for synaptic p

42 Inhibitors of AMPA-type glutamate ion channels are useful as biochemic

43 taneous synaptic currents mediated by either AMPA-type glutamate or nicotinic acetylcholine receptors

44 on at synapses, which are mediated by either AMPA-type glutamate or nicotinic acetylcholine receptors

45 Cornichon homologs (CNIHs) are AMPA-type glutamate receptor (AMPAR) auxiliary subunits

46 The C terminus of AMPA-type glutamate receptor (AMPAR) GluA1 subunits cont

47 NMDA-type glutamate receptor (NMDAR) but not AMPA-type glutamate receptor (AMPAR) mediated currents.

48 Regulation of AMPA-type glutamate receptor (AMPAR) number at synapses

49 synaptic strength in brain are dependent on AMPA-type glutamate receptor (AMPAR) recycling, which is

50 ances have been made in our understanding of AMPA-type glutamate receptor (AMPAR) regulation by trans

51 The AMPA-type glutamate receptor (AMPAR) subunit composition

52 e that the specific intracellular domains of AMPA-type glutamate receptor (AMPAR) subunits are critic

53 AMPA-type glutamate receptor (AMPAR) trafficking is esse

54 that are regulated by phosphorylation of the AMPA-type glutamate receptor (AMPAR).

55 in the central nervous system relies on the AMPA-type glutamate receptor (AMPAR).

56 ansmission is mediated primarily through the AMPA-type glutamate receptor (AMPAR); the regulation of

57 units for a very different ion channel - the AMPA-type glutamate receptor - prominently regulating ea

58 th factor I but not N-methyl-D-aspartate- or AMPA-type glutamate receptor antagonists.

59 tamate input is necessary for clustering the AMPA-type glutamate receptor but not for clustering the

60 some antigen 1 (EEA1), a protein involved in AMPA-type glutamate receptor endocytosis.

61 AMPK targets both the AMPA-type glutamate receptor GLR-1 and the metabotropic

62 Following ER exit, the AMPA-type glutamate receptor GluA1 and neuroligin 1 unde

63 We demonstrate that EphB2 controls AMPA-type glutamate receptor localization through PDZ (p

64 piny neurons as a primary site of persistent AMPA-type glutamate receptor plasticity by two widely us

65 et neurons of ALa in dorsal pallidum possess AMPA-type glutamate receptor profiles resembling those o

66 ill training induces an increase of synaptic AMPA-type glutamate receptor subunit 1 (GluA1), there is

67 -4-isoxazolepropionic acid receptor (AMPAR) [AMPA-type glutamate receptor subunit 1 (GluR1 subunit)],

68 ing both processes to a single molecule: the AMPA-type glutamate receptor subunit 1 (GluR1).

69 PICK1 protein interacts in neurons with the AMPA-type glutamate receptor subunit 2 (GluR2) and with

70 AMPA receptor complexes that contain the AMPA-type glutamate receptor subunit 2 (GluR2) are respo

71 h correlates with a significant reduction of AMPA-type glutamate receptor subunit 2 (GluR2) at the sy

72 similar to the decrease in the number of the AMPA-type glutamate receptor subunit 2/3-immunoreactive

73 KAP5 is important for phosphorylation of the AMPA-type glutamate receptor subunit GluA1 on Ser-845 by

74 sociated with enhanced surface levels of the AMPA-type glutamate receptor subunit GluA2, an effect th

75 ctive effect of EphB2 may be mediated by the AMPA-type glutamate receptor subunit GluA2, which can be

76 ever, silenced neurons could not recruit the AMPA-type glutamate receptor subunit GluR1 as efficientl

77 of the presynaptic marker synaptophysin, the AMPA-type glutamate receptor subunit GluR1, and the puta

78 , and PKA form a signalling complex with the AMPA-type glutamate receptor subunit GluR1, which is lin

79 estradiol, DPN, and PPT increased PSD-95 and AMPA-type glutamate receptor subunit GluR1.

80 Moreover, loss of the GluA2 AMPA-type glutamate receptor subunit, which decreased p(

81 bridization, we show that mRNAs encoding the AMPA-type glutamate receptor subunits (GluRs) 1 and 2 ar

82 Drugs of abuse alter expression of AMPA-type glutamate receptor subunits (GluRs) in the nuc

83 rc protein has been demonstrated to regulate AMPA-type glutamate receptor trafficking by recruiting e

84 chanisms have focused mainly on postsynaptic AMPA-type glutamate receptor trafficking.

85 display a dramatic reduction in frequency of AMPA-type glutamate receptor-mediated miniature excitato

86 ls exhibit a large and selective decrease in AMPA-type glutamate receptor-mediated synaptic transmiss

87 tic peptide that prevents internalization of AMPA-type glutamate receptor.

88 tire human P2X receptor family and the human AMPA-type glutamate receptor.

89 In particular, AMPA-type glutamate receptors (AMPA receptors) reach exc

90 AMPA-type glutamate receptors (AMPA-Rs) mediate a majori

91 Positive allosteric modulators of AMPA-type glutamate receptors (ampakines) have been show

92 excitatory synapses where it associates with AMPA-type glutamate receptors (AMPAR) and enhances synap

93 molecules to synapses and in endocytosis of AMPA-type glutamate receptors (AMPAR) in the dendrites o

94 KII and destabilized for TARPs, which anchor AMPA-type glutamate receptors (AMPAR).

95 Alternative splicing of AMPA-type glutamate receptors (AMPARs) and allosteric mo

96 The interplay between AMPA-type glutamate receptors (AMPARs) and major histoco

97 Postsynaptic AMPA-type glutamate receptors (AMPARs) are among the maj

98 zation, number, and function of postsynaptic AMPA-type glutamate receptors (AMPARs) are crucial for s

99 ity is the regulated addition and removal of AMPA-type glutamate receptors (AMPARs) at excitatory syn

100 rength of neurotransmission is the number of AMPA-type glutamate receptors (AMPARs) at synapses.

101 Postsynaptic AMPA-type glutamate receptors (AMPARs) can be inserted i

102 Although the properties and trafficking of AMPA-type glutamate receptors (AMPARs) depend critically

103 In the brain, AMPA-type glutamate receptors (AMPARs) form complexes wi

104 While AMPA-type glutamate receptors (AMPARs) found at principa

105 The regulated trafficking of AMPA-type glutamate receptors (AMPARs) from dendritic co

106 ates endocytosis of GluR2 subunit-containing AMPA-type glutamate receptors (AMPARs) in an ATPase-depe

107 ), which activates postsynaptic synthesis of AMPA-type glutamate receptors (AMPARs) in dendrites and

108 /Arg3.1 selectively modulates trafficking of AMPA-type glutamate receptors (AMPARs) in neurons by acc

109 We studied the dynamics of newly synthesized AMPA-type glutamate receptors (AMPARs) induced with lear

110 The assembly of AMPA-type glutamate receptors (AMPARs) into distinct ion

111 Regulated membrane trafficking of AMPA-type glutamate receptors (AMPARs) is a key mechanis

112 The synaptic insertion of GluR1-containing AMPA-type glutamate receptors (AMPARs) is critical for s

113 Synaptic transmission mediated by AMPA-type glutamate receptors (AMPARs) is regulated by s

114 Abnormal influx of Ca(2+) through AMPA-type glutamate receptors (AMPARs) is thought to con

115 AMPA-type glutamate receptors (AMPARs) lacking an edited

116 AMPA-type glutamate receptors (AMPARs) mediate excitator

117 AMPA-type glutamate receptors (AMPARs) mediate fast exci

118 AMPA-type glutamate receptors (AMPARs) mediate fast exci

119 AMPA-type glutamate receptors (AMPARs) mediate fast exci

120 Postsynaptic AMPA-type glutamate receptors (AMPARs) mediate most fast

121 AMPA-type glutamate receptors (AMPARs) mediate most fast

122 AMPA-type glutamate receptors (AMPARs) mediate rapid sig

123 AMPA-type glutamate receptors (AMPARs) mediate the major

124 AMPA-type glutamate receptors (AMPARs) mediate the major

125 AMPA-type glutamate receptors (AMPARs) play a critical r

126 AMPA-type glutamate receptors (AMPARs) play a major role

127 Current influx through AMPA-type glutamate receptors (AMPARs) provides the depo

128 The GluA2 subunit of AMPA-type glutamate receptors (AMPARs) regulates excitat

129 naptic density protein-95 (PSD-95) localizes AMPA-type glutamate receptors (AMPARs) to postsynaptic s

130 The regulated delivery of AMPA-type glutamate receptors (AMPARs) to synapses is an

131 xocytic fusion events mediating insertion of AMPA-type glutamate receptors (AMPARs) to the somatodend

132 The regulated transport of AMPA-type glutamate receptors (AMPARs) to the synaptic m

133 ynthesis alters endocytosis and recycling of AMPA-type glutamate receptors (AMPARs), implicating PI(3

134 tic strength through changes in postsynaptic AMPA-type glutamate receptors (AMPARs), suggesting the e

135 AMPA-type glutamate receptors (AMPARs), which are centra

136 AMPA-type glutamate receptors (AMPARs), which mediate fa

137 affinity tags for labeling and manipulating AMPA-type glutamate receptors (AMPARs), which mediate ne

138 ransmission in the CNS is mediated mainly by AMPA-type glutamate receptors (AMPARs), whose biophysica

139 ron synapses were dominated by GluA2-lacking AMPA-type glutamate receptors (AMPARs), with little cont

140 utamatergic synapses often lack postsynaptic AMPA-type glutamate receptors (AMPARs).

141 ses to its release predominantly mediated by AMPA-type glutamate receptors (AMPARs).

142 e amplitude of synaptic currents mediated by AMPA-type glutamate receptors (AMPARs).

143 ission in the mammalian brain is mediated by AMPA-type glutamate receptors (AMPARs).

144 itatory synaptic transmission is mediated by AMPA-type glutamate receptors (AMPARs).

145 as an increase in the number of postsynaptic AMPA-type glutamate receptors (AMPARs).

146 that modulate the pharmacology and gating of AMPA-type glutamate receptors (AMPARs).

147 n the number and the spatial distribution of AMPA-type glutamate receptors (AMPARs).

148 turation by recruitment of calcium-permeable AMPA-type glutamate receptors (CP-AMPARs) after drug wit

149 quires opening of calcium (Ca(2+))-permeable AMPA-type glutamate receptors (CP-AMPARs) and signaling

150 Calcium-permeable AMPA-type glutamate receptors (CP-AMPARs) contribute to

151 Calcium-permeable AMPA-type glutamate receptors (CP-AMPARs) contribute to

152 hannels, including that of calcium-permeable AMPA-type glutamate receptors (CP-AMPARs).

153 y mediating the action of calcium-permeable, AMPA-type glutamate receptors (CP-AMPARs).

154 p) has been implicated in the aggregation of AMPA-type glutamate receptors (GluR) at excitatory synap

155 Most AMPA-type glutamate receptors (GluRs) exhibit rapid and

156 AMPA-type glutamate receptors (GluRs) mediate most excit

157 AMPA-type glutamate receptors (GluRs) play major roles i

158 city that converge on regulation of NMDA and AMPA-type glutamate receptors (NMDAR, AMPAR), including

159 d of synaptic levels of the GluA1 subunit of AMPA-type glutamate receptors after 48 h silencing with

160 wo glycine receptors, one GABA receptor, two AMPA-type glutamate receptors and one purinergic recepto

161 phrenia, RNA editing sites in genes encoding AMPA-type glutamate receptors and postsynaptic density p

162 tentiation was expressed postsynaptically by AMPA-type glutamate receptors and required calmodulin-de

163 oning induce similar changes in postsynaptic AMPA-type glutamate receptors and that occluding these c

164 These results suggest an interaction between AMPA-type glutamate receptors and the gap junction prote

165 ic data implicates Arc in the endocytosis of AMPA-type glutamate receptors and the weakening of synap

166 EAAT2 buffers basal glutamate activation of AMPA-type glutamate receptors and therefore decreases ba

167 AMPA-type glutamate receptors are ligand-gated cation ch

168 AMPA-type glutamate receptors are tetrameric ion channel

169 AMPA-type glutamate receptors are the predominant excita

170 Several studies have implicated a change in AMPA-type glutamate receptors as being responsible for t

171 These results identify DARPP-32 and AMPA-type glutamate receptors as likely essential cellul

172 orylation cascades that alter the density of AMPA-type glutamate receptors at excitatory synapses; ho

173 ely increases the level of GluA1 subunits of AMPA-type glutamate receptors at the synapses of the nuc

174 then the present data suggest that forebrain AMPA-type glutamate receptors can be classified into a l

175 s in the subunit composition of postsynaptic AMPA-type glutamate receptors can be induced at CNS syna

176 were used to test if positive modulators of AMPA-type glutamate receptors have regionally differenti

177 selectively reduces postsynaptic function of AMPA-type glutamate receptors in a dose-dependent manner

178 Prolonged blockade of AMPA-type glutamate receptors in hippocampal neuron cult

179 ion molecule linked to autism, in organizing AMPA-type glutamate receptors in the calyx of Held synap

180 AMPA-type glutamate receptors in the nucleus tractus sol

181 igate the relationship between the number of AMPA-type glutamate receptors in the PSD and synaptic st

182 azole chemistry enables covalent labeling of AMPA-type glutamate receptors in the same brain regions.

183 investigated whether positive modulators of AMPA-type glutamate receptors influence neurotrophin exp

184 Here we report that the transport of AMPA-type glutamate receptors into synapses occurs in tw

185 idal cells, TNFalpha drives the insertion of AMPA-type glutamate receptors into synapses, and contrib

186 Here we report that fear conditioning drives AMPA-type glutamate receptors into the synapse of a larg

187 AMPA-type glutamate receptors mediate fast excitatory tr

188 AMPA-type glutamate receptors mediate fast excitatory tr

189 AMPA-type glutamate receptors mediate most excitatory po

190 AMPA-type glutamate receptors mediate the majority of fa

191 d hippocampal neurons to aggregate NMDA- and AMPA-type glutamate receptors on each other as a way of

192 eurons results in clusters of both NMDA- and AMPA-type glutamate receptors on hippocampal interneuron

193 cally and probably involves up-regulation of AMPA-type glutamate receptors on hypocretin neurons.

194 Spinal axons, which normally cluster only AMPA-type glutamate receptors on other spinal neurons, c

195 ells; however, fast transmission mediated by AMPA-type glutamate receptors remains unaffected.

196 Dynamic regulation of AMPA-type glutamate receptors represents a primary mecha

197 Although modulation of AMPA-type glutamate receptors shows promise for the trea

198 Type 1 astrocytes express AMPA-type glutamate receptors that are unmasked by reduc

199 changes is the remodeling of the ionotropic AMPA-type glutamate receptors that underlie fast excitat

200 pses in the mammalian cortex lack sufficient AMPA-type glutamate receptors to mediate neurotransmissi

201 d Proteins (TARPs), which mediate binding of AMPA-type glutamate receptors to PSD-95, was increased i

202 he postsynaptic density, tethering NMDA- and AMPA-type glutamate receptors to signaling proteins and

203 easing the ubiquitination and degradation of AMPA-type glutamate receptors via a mechanism depending

204 Synaptic transmission mediated by AMPA-type glutamate receptors was potentiated in the NAc

205 the responses other than those generated by AMPA-type glutamate receptors were blocked.

206 ertension alters dendritic spines containing AMPA-type glutamate receptors within NTS, suggesting tha

207 In contrast, blocking AMPA-type glutamate receptors within the Acb shell (the

208 sent study tested if a positive modulator of AMPA-type glutamate receptors would counteract the behav

209 the subunit that limits Ca2+ permeability of AMPA-type glutamate receptors) was markedly and specific

210 se EPSCs were abolished by the antagonist of AMPA-type glutamate receptors, 6-cyano-7-nitro-quinoxali

211 the cell surface expression of NMDA-type and AMPA-type glutamate receptors, along with prominent func

212 eds, wave initiation depends increasingly on AMPA-type glutamate receptors, and an ever increasing fr

213 s onto FSIs, which are mediated primarily by AMPA-type glutamate receptors, glutamate release by chol

214 ), an agent used to block desensitization of AMPA-type glutamate receptors, on heterologously express

215 eversibly modifies the kinetic properties of AMPA-type glutamate receptors, on synaptic responses is

216 e, we show that membrane proteins, including AMPA-type glutamate receptors, rapidly diffuse within th

217 ls with an ampakine, a positive modulator of AMPA-type glutamate receptors, rescues plasticity and re

218 n is of particular importance with regard to AMPA-type glutamate receptors, the multimeric complexes

219 striatum is mediated, in part, by ionotropic AMPA-type glutamate receptors, which are heteromers comp

220 ction in the level of synaptically localized AMPA-type glutamate receptors.

221 NMDA receptors (NMDARs) and Ca2+-impermeable AMPA-type glutamate receptors.

222 ation that enhanced the expression levels of AMPA-type glutamate receptors.

223 al and activate the AIB interneurons through AMPA-type glutamate receptors.

224 control synaptic targeting and insertion of AMPA-type glutamate receptors.

225 y involves activity-dependent trafficking of AMPA-type glutamate receptors.

226 l pentraxin domains mediate association with AMPA-type glutamate receptors.

227 of glutamate receptor type 1 subunits of the AMPA-type glutamate receptors.

228 ell characterized as a negative modulator of AMPA-type glutamate receptors.

229 nase regulates the physiological activity of AMPA-type glutamate receptors.

230 fficient in promoting synaptic clustering of AMPA-type glutamate receptors.

231 excitatory synaptic transmission mediated by AMPA-type glutamate receptors.

232 gh removal and dephosphorylation of synaptic AMPA-type glutamate receptors.

233 ration of a positive allosteric modulator of AMPA-type glutamate receptors.

234 stitutive, internalization of both NMDA- and AMPA-type glutamate receptors.

235 al activity- and PDZ-dependent regulation of AMPA-type glutamate receptors.

236 ptic activity to postsynaptic endocytosis of AMPA-type glutamate receptors.

237 ths of age, which involves calcium-permeable AMPA-type glutamate receptors.

238 c input from bipolar cells through NMDA- and AMPA-type glutamate receptors.

239 via its regulatory effect on trafficking of AMPA-type glutamate receptors.

240 ayed release, a large quantal size, and fast AMPA-type glutamate receptors.

241 s, we studied the distributions of NMDA- and AMPA-type glutamate receptors; the NMDA receptor-interac

242 tested the role of the postsynaptic NMDA and AMPA type glutamatergic receptors in the lactate-induced

243 Here we examine how DNE influences AMPA-type glutamatergic neurotransmission in the pre-Bot

244 nent of the cellular machinery that delivers AMPA-type glutamatergic receptors (AMPARs) into synapses

245 AMPA-type gultamate receptors (AMPARs) mediate excitator

246 p segments and helices within a region of an AMPA-type iGluR NTD, which has been identified previousl

247 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type iGluRs using genetically encoded unnatural am

248 have evolved to optimize rapid responses of AMPA-type iGluRs at synapses.

249 lore the allosteric potential for the NTD in AMPA-type iGluRs using coarse-grained simulations.

250 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type ionotropic glutamate receptors (AMPA receptor

251 -isomer has been identified as a competitive AMPA-type ionotropic glutamate receptor antagonist, whil

252 o proper synaptic content and trafficking of AMPA-type ionotropic glutamate receptor homolog GLR-1 in

253 AMPA-type ionotropic glutamate receptors (AMPARs) are ce

254 AMPA-type ionotropic glutamate receptors (AMPARs) are in

255 Phosphorylation and dephosphorylation of AMPA-type ionotropic glutamate receptors (AMPARs) by kin

256 KEY POINTS: The AMPA-type ionotropic glutamate receptors (AMPARs) mediat

257 n the mammalian brain is largely mediated by AMPA-type ionotropic glutamate receptors (AMPARs), which

258 GRIA3 encodes GluA3, a subunit of AMPA-type ionotropic glutamate receptors (AMPARs).

259 with presynaptic neurexins and postsynaptic AMPA-type ionotropic glutamate receptors and induced the

260 ly inhibited by activation of either NMDA or AMPA-type ionotropic glutamate receptors in a calcium-de

261 AMPA-type ionotropic glutamate receptors mediate the maj

262 nsmission is mediated by glutamate acting on AMPA-type ionotropic glutamate receptors.

263 three ionotropic glutamate subfamilies (i.e. AMPA-type, kainate-type, and NMDA-type) assemble as tetr

264 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) type mediate fast excitatory synaptic transmission

265 ent with the role of synaptic trafficking of AMPA-type of glutamate receptors in HSP, Mecp2 KO neuron

266 hdrawal requires activation of NMDA-type and AMPA-type postsynaptic receptors within the abdominal ga

267 The regulation of AMPA-type receptor (AMPAR) abundance in the postsynaptic

268 -hydroxy-5-methylisoxazole-4-propionic acid (AMPA)-type receptors is required for synaptopathy, and h

269 At many excitatory synapses, AMPA-type receptors (AMPARs) are not statically situated

270 AMPA-type receptors (AMPARs) are rapidly inserted into s

271 blished homeostatic increase in postsynaptic AMPA-type receptors (AMPARs), we performed a series of e

272 Dynamic regulation of AMPA-type receptors at the synapse is proposed to play a

273 5-HT increased the functional expression of AMPA-type receptors in the motor neuron.

274 tion 5-HT causes the insertion of additional AMPA-type receptors into the postsynaptic membrane of se

275 mulation, IHCs release glutamate to activate AMPA-type receptors on these myelinated type-I neurons,

276 glutamate receptors, rather than changes in AMPA-type receptors or membrane excitability.

277 ns, LTP depends instead on calcium-permeable AMPA-type receptors.

278 ng pathway and glutamate ionotropic receptor AMPA type subunit 2(GluR2) transportation in the pathoge

279 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type subunit 2 (GRIA2) in neurons depended on FUS

280 B (GRIN2B) and glutamate ionotropic receptor AMPA-type subunit 2 (GRIA2).

281 While the cellular localization of AMPA-type subunits in the basal ganglia has been well ch

282 ase in dendritic spine density and increased AMPA-type synaptic responses.