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1 tic peptide that prevents internalization of AMPA-type glutamate receptor.
2 tire human P2X receptor family and the human AMPA-type glutamate receptor.
3 sporter 2 (KCC2) and the excitatory NMDA and AMPA type glutamate receptors.
4 hydroxy-5-methyl-4-isoxazole-propionic acid (AMPA)-type glutamate receptor.
5 NMDA receptors (NMDARs) and Ca2+-impermeable AMPA-type glutamate receptors.
6 al and activate the AIB interneurons through AMPA-type glutamate receptors.
7 control synaptic targeting and insertion of AMPA-type glutamate receptors.
8 y involves activity-dependent trafficking of AMPA-type glutamate receptors.
9 l pentraxin domains mediate association with AMPA-type glutamate receptors.
10 of glutamate receptor type 1 subunits of the AMPA-type glutamate receptors.
11 ell characterized as a negative modulator of AMPA-type glutamate receptors.
12 nase regulates the physiological activity of AMPA-type glutamate receptors.
13 gh removal and dephosphorylation of synaptic AMPA-type glutamate receptors.
14 fficient in promoting synaptic clustering of AMPA-type glutamate receptors.
15 excitatory synaptic transmission mediated by AMPA-type glutamate receptors.
16 ration of a positive allosteric modulator of AMPA-type glutamate receptors.
17 stitutive, internalization of both NMDA- and AMPA-type glutamate receptors.
18 al activity- and PDZ-dependent regulation of AMPA-type glutamate receptors.
19 ptic activity to postsynaptic endocytosis of AMPA-type glutamate receptors.
20 ths of age, which involves calcium-permeable AMPA-type glutamate receptors.
21 c input from bipolar cells through NMDA- and AMPA-type glutamate receptors.
22 via its regulatory effect on trafficking of AMPA-type glutamate receptors.
23 ayed release, a large quantal size, and fast AMPA-type glutamate receptors.
24 ction in the level of synaptically localized AMPA-type glutamate receptors.
25 ation that enhanced the expression levels of AMPA-type glutamate receptors.
26 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.
27 o-3-hydroxyl-5-methyl-4-isoxazolepropionate (AMPA)-type glutamate receptors.
28 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.
29 -hydroxy-5-methylisoxazole-4-propionic acid (AMPA)-type glutamate receptors.
30 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors.
31 se EPSCs were abolished by the antagonist of AMPA-type glutamate receptors, 6-cyano-7-nitro-quinoxali
32 d of synaptic levels of the GluA1 subunit of AMPA-type glutamate receptors after 48 h silencing with
33 the cell surface expression of NMDA-type and AMPA-type glutamate receptors, along with prominent func
34 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPA-Rs), which mediate
40 NMDA-type glutamate receptor (NMDAR) but not AMPA-type glutamate receptor (AMPAR) mediated currents.
42 synaptic strength in brain are dependent on AMPA-type glutamate receptor (AMPAR) recycling, which is
43 ances have been made in our understanding of AMPA-type glutamate receptor (AMPAR) regulation by trans
45 e that the specific intracellular domains of AMPA-type glutamate receptor (AMPAR) subunits are critic
49 ansmission is mediated primarily through the AMPA-type glutamate receptor (AMPAR); the regulation of
50 excitatory synapses where it associates with AMPA-type glutamate receptors (AMPAR) and enhances synap
51 molecules to synapses and in endocytosis of AMPA-type glutamate receptors (AMPAR) in the dendrites o
53 in, mediates homeostatic synaptic scaling of AMPA type glutamate receptors (AMPARs) via its ability t
54 3-hydroxy-5-methyl-4-isoxazolepropionic acid(AMPA)-type glutamate receptors (AMPARs) are the predomin
55 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) at Schaffer coll
56 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (AMPARs) mediate excitato
57 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) mediate the majo
58 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (AMPARs) to synapses is a
62 zation, number, and function of postsynaptic AMPA-type glutamate receptors (AMPARs) are crucial for s
63 ity is the regulated addition and removal of AMPA-type glutamate receptors (AMPARs) at excitatory syn
66 Although the properties and trafficking of AMPA-type glutamate receptors (AMPARs) depend critically
70 ates endocytosis of GluR2 subunit-containing AMPA-type glutamate receptors (AMPARs) in an ATPase-depe
71 ), which activates postsynaptic synthesis of AMPA-type glutamate receptors (AMPARs) in dendrites and
72 /Arg3.1 selectively modulates trafficking of AMPA-type glutamate receptors (AMPARs) in neurons by acc
73 We studied the dynamics of newly synthesized AMPA-type glutamate receptors (AMPARs) induced with lear
76 The synaptic insertion of GluR1-containing AMPA-type glutamate receptors (AMPARs) is critical for s
93 naptic density protein-95 (PSD-95) localizes AMPA-type glutamate receptors (AMPARs) to postsynaptic s
95 xocytic fusion events mediating insertion of AMPA-type glutamate receptors (AMPARs) to the somatodend
97 ynthesis alters endocytosis and recycling of AMPA-type glutamate receptors (AMPARs), implicating PI(3
98 tic strength through changes in postsynaptic AMPA-type glutamate receptors (AMPARs), suggesting the e
101 affinity tags for labeling and manipulating AMPA-type glutamate receptors (AMPARs), which mediate ne
102 ransmission in the CNS is mediated mainly by AMPA-type glutamate receptors (AMPARs), whose biophysica
103 ron synapses were dominated by GluA2-lacking AMPA-type glutamate receptors (AMPARs), with little cont
112 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors and the function of synap
113 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors and the stabilization of
114 wo glycine receptors, one GABA receptor, two AMPA-type glutamate receptors and one purinergic recepto
115 phrenia, RNA editing sites in genes encoding AMPA-type glutamate receptors and postsynaptic density p
116 tentiation was expressed postsynaptically by AMPA-type glutamate receptors and required calmodulin-de
117 oning induce similar changes in postsynaptic AMPA-type glutamate receptors and that occluding these c
118 These results suggest an interaction between AMPA-type glutamate receptors and the gap junction prote
119 ic data implicates Arc in the endocytosis of AMPA-type glutamate receptors and the weakening of synap
120 EAAT2 buffers basal glutamate activation of AMPA-type glutamate receptors and therefore decreases ba
121 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors, and is implicated in mul
122 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors, and thereby enhance fast
123 eds, wave initiation depends increasingly on AMPA-type glutamate receptors, and an ever increasing fr
125 ve IDRA 21 and other positive modulators of (AMPA)-type glutamate receptors are considered potential
129 Several studies have implicated a change in AMPA-type glutamate receptors as being responsible for t
131 orylation cascades that alter the density of AMPA-type glutamate receptors at excitatory synapses; ho
132 ely increases the level of GluA1 subunits of AMPA-type glutamate receptors at the synapses of the nuc
133 tamate input is necessary for clustering the AMPA-type glutamate receptor but not for clustering the
134 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors (but not by blockade of N
135 then the present data suggest that forebrain AMPA-type glutamate receptors can be classified into a l
136 s in the subunit composition of postsynaptic AMPA-type glutamate receptors can be induced at CNS syna
137 o-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA)-type glutamate receptors cause the enhanced respon
138 turation by recruitment of calcium-permeable AMPA-type glutamate receptors (CP-AMPARs) after drug wit
139 quires opening of calcium (Ca(2+))-permeable AMPA-type glutamate receptors (CP-AMPARs) and signaling
145 hydroxy-5-methyl-4-isoxazoleproprionic acid (AMPA)-type glutamate receptors during long-term potentia
149 p) has been implicated in the aggregation of AMPA-type glutamate receptors (GluR) at excitatory synap
150 ydroxy-5-methyl-4-isoaxazole propionic acid (AMPA)-type glutamate receptors (GluR1 and GluR2/3) durin
154 s onto FSIs, which are mediated primarily by AMPA-type glutamate receptors, glutamate release by chol
155 alpha-amino-3-hydroxy-5-methyl-4-isoxazole (AMPA)-type glutamate receptor has recently been demonstr
156 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors have distinct roles in co
157 were used to test if positive modulators of AMPA-type glutamate receptors have regionally differenti
158 no 3-hydroxy-5-methyl-4-isoxazolepropionate (AMPA)-type glutamate receptors in rat brain and to test
159 selectively reduces postsynaptic function of AMPA-type glutamate receptors in a dose-dependent manner
161 ion molecule linked to autism, in organizing AMPA-type glutamate receptors in the calyx of Held synap
163 igate the relationship between the number of AMPA-type glutamate receptors in the PSD and synaptic st
164 azole chemistry enables covalent labeling of AMPA-type glutamate receptors in the same brain regions.
165 investigated whether positive modulators of AMPA-type glutamate receptors influence neurotrophin exp
167 idal cells, TNFalpha drives the insertion of AMPA-type glutamate receptors into synapses, and contrib
168 Here we report that fear conditioning drives AMPA-type glutamate receptors into the synapse of a larg
170 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) type glutamate receptors mediate most fast synapti
171 hydroxy-5-methyl-4-isoxazole propionic acid (AMPA)-type glutamate receptors mediate the majority of e
176 display a dramatic reduction in frequency of AMPA-type glutamate receptor-mediated miniature excitato
177 ls exhibit a large and selective decrease in AMPA-type glutamate receptor-mediated synaptic transmiss
178 city that converge on regulation of NMDA and AMPA-type glutamate receptors (NMDAR, AMPAR), including
179 d hippocampal neurons to aggregate NMDA- and AMPA-type glutamate receptors on each other as a way of
180 eurons results in clusters of both NMDA- and AMPA-type glutamate receptors on hippocampal interneuron
181 cally and probably involves up-regulation of AMPA-type glutamate receptors on hypocretin neurons.
182 Spinal axons, which normally cluster only AMPA-type glutamate receptors on other spinal neurons, c
183 ), an agent used to block desensitization of AMPA-type glutamate receptors, on heterologously express
184 eversibly modifies the kinetic properties of AMPA-type glutamate receptors, on synaptic responses is
185 piny neurons as a primary site of persistent AMPA-type glutamate receptor plasticity by two widely us
186 et neurons of ALa in dorsal pallidum possess AMPA-type glutamate receptor profiles resembling those o
187 units for a very different ion channel - the AMPA-type glutamate receptor - prominently regulating ea
189 e, we show that membrane proteins, including AMPA-type glutamate receptors, rapidly diffuse within th
192 ls with an ampakine, a positive modulator of AMPA-type glutamate receptors, rescues plasticity and re
194 ill training induces an increase of synaptic AMPA-type glutamate receptor subunit 1 (GluA1), there is
195 -4-isoxazolepropionic acid receptor (AMPAR) [AMPA-type glutamate receptor subunit 1 (GluR1 subunit)],
197 PICK1 protein interacts in neurons with the AMPA-type glutamate receptor subunit 2 (GluR2) and with
198 AMPA receptor complexes that contain the AMPA-type glutamate receptor subunit 2 (GluR2) are respo
199 h correlates with a significant reduction of AMPA-type glutamate receptor subunit 2 (GluR2) at the sy
200 similar to the decrease in the number of the AMPA-type glutamate receptor subunit 2/3-immunoreactive
201 KAP5 is important for phosphorylation of the AMPA-type glutamate receptor subunit GluA1 on Ser-845 by
202 sociated with enhanced surface levels of the AMPA-type glutamate receptor subunit GluA2, an effect th
203 ctive effect of EphB2 may be mediated by the AMPA-type glutamate receptor subunit GluA2, which can be
204 ever, silenced neurons could not recruit the AMPA-type glutamate receptor subunit GluR1 as efficientl
205 of the presynaptic marker synaptophysin, the AMPA-type glutamate receptor subunit GluR1, and the puta
206 , and PKA form a signalling complex with the AMPA-type glutamate receptor subunit GluR1, which is lin
209 bridization, we show that mRNAs encoding the AMPA-type glutamate receptor subunits (GluRs) 1 and 2 ar
212 changes is the remodeling of the ionotropic AMPA-type glutamate receptors that underlie fast excitat
213 n is of particular importance with regard to AMPA-type glutamate receptors, the multimeric complexes
214 s, we studied the distributions of NMDA- and AMPA-type glutamate receptors; the NMDA receptor-interac
215 pses in the mammalian cortex lack sufficient AMPA-type glutamate receptors to mediate neurotransmissi
216 d Proteins (TARPs), which mediate binding of AMPA-type glutamate receptors to PSD-95, was increased i
217 he postsynaptic density, tethering NMDA- and AMPA-type glutamate receptors to signaling proteins and
218 rc protein has been demonstrated to regulate AMPA-type glutamate receptor trafficking by recruiting e
220 easing the ubiquitination and degradation of AMPA-type glutamate receptors via a mechanism depending
222 the subunit that limits Ca2+ permeability of AMPA-type glutamate receptors) was markedly and specific
223 -hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors were studied using equili
225 -hydroxy-5-methylisoxazole-4-propionic acid (AMPA)-type glutamate receptors, which become phosphoryla
226 striatum is mediated, in part, by ionotropic AMPA-type glutamate receptors, which are heteromers comp
227 ertension alters dendritic spines containing AMPA-type glutamate receptors within NTS, suggesting tha
229 sent study tested if a positive modulator of AMPA-type glutamate receptors would counteract the behav