ライフサイエンスコーパス: AMV

1 AMV and ilarvirus coat protein sequence alignment center

2 AMV and TSV coat proteins, which share little primary am

3 AMV RNA 4 and Arabidopsis HSP21 showed only a slight dep

4 AMV RT binds much tighter to template- primer and has a

5 AMV was discovered in the 1930s as a virus that caused a

6 AMV-RT degraded the RNA to segments 11-12 nt long, and r

7 To detect coat-protein-activated AMV RNA replication, we designed and characterized a sub

8 AUGC sequences that are conserved among AMV and ilarvirus RNAs were among the invariant nucleoti

9 tein interactions are highly conserved among AMV and the ilarvirus RNAs.

10 5' UTRs allowed the systemic transport of an AMV RNA3 carrying a CP mutant defective in virus particl

11 a compensatory evolution experiment using an AMV RNA3 derivative defective in long-distance transport

12 ty with eIF4F, whereas Arabidopsis HSP21 and AMV RNA 4 used both eIF4F and eIF(iso)4F equally well.

13 , although both Rous sarcoma virus (RSV) and AMV could replicate in cultures of either embryonic fibr

14 ned in this myb oncogene were shared between AMV and the avian E26 leukemia virus, but were not conta

15 ws that the record is strongly influenced by AMV via atmospheric circulation anomalies.

16 We found that (th)A is recognized by AMV reverse transcriptase as A, and is deaminated rapidl

17 over twice the length of that synthesized by AMV RT and can synthesize cDNA over 4 times longer than

18 y avian erythroblastosis virus (AEV) than by AMV.

19 We further compare AMV and AMD-derived PNCs for well-characterized polystyr

20 First, AMV was used to demonstrate that all oncogenic viruses d

21 ion of an RNA binding consensus sequence for AMV and ilarvirus coat proteins, provides a framework fo

22 rometry (spICP-MS) and the PNCs derived from AMV using size distributions independently measured by h

23 replication and may explain why heterologous AMV and ilarvirus coat protein-RNA mixtures are infectio

24 We measured the ability of HIV-, AMV- and MuLV-RT to coordinate DNA-dependent DNA synthes

25 Consistent with these results, HIV-, AMV- and MuLV-RT showed relatively higher affinity for R

26 QRT-SDA) of an HIV gag sequence by including AMV reverse transcriptase, a quantitative control sequen

27 rse transcriptases with higher fidelity like AMV-RT, the methylation could either retain the normal n

28 eport the addition of ammonium metavanadate (AMV), a phosphatase inhibitor, to PIA (PIA-AMV) induced

29 ound that when comparing wild-type or mutant AMV RT with the respective M-MLV RT, the avian enzymes r

30 The ability of AMV IN to interact with internal att sequences to mediat

31 ecord of AMV is short, long-term behavior of AMV is unknown, but climatic teleconnections to regions

32 In the case of AMV, it was shown that almost the entire retroviral env

33 Furthermore, the BIR domains of AMV-IAP protein were demonstrated to bind the mammalian

34 dual baculoviral IAP repeat (BIR) domains of AMV-IAP was investigated by using a random-peptide, phag

35 understanding the functional equivalence of AMV and TSV coat proteins in binding RNA and activating

36 ity of atALKBH9B affected the infectivity of AMV but not of CMV, correlating with the ability of atAL

37 organization model as an alternate model of AMV replication that offers an improved fit to the avail

38 binding of the 3'-terminal 39 nucleotides of AMV RNA 3/4 (AMV843-881) to an amino-terminal coat prote

39 oth of which contribute to the warm phase of AMV.

40 Our record provides a reconstruction of AMV for the past ~3 millennia at an unprecedented time r

41 Because the instrumental record of AMV is short, long-term behavior of AMV is unknown, but

42 suggests that the 3' untranslated regions of AMV and ilarvirus RNAs have the potential to fold into p

43 protein's function in the earliest stages of AMV replication.

44 e not required for the systemic transport of AMV but also that BMV MP is competent for the short- and

45 We also propose the use of AMV (instead of nucleus taenia/TnA), AMD, AD, and AI as

46 polymerase activity was observed with MLV or AMV reverse transcriptase, T7 DNA polymerase, or DNA pol

47 r), but not that of RTs derived from MMLV or AMV.

48 Alginate induction in PAO1 on PIA-AMV was correlated with increased proteolytic degradatio

49 (AMV), a phosphatase inhibitor, to PIA (PIA-AMV) induced mucoidy in both these laboratory strains an

50 st a model of alginate induction and the PIA-AMV medium may be suitable for examining early lung colo

51 es being twice as variable during a positive AMV phase than a negative one.

52 a moorei entomopoxvirus-encoded IAP protein (AMV-IAP).

53 For this reason AMV was often used as a prototypic retrovirus in order t

54 tlantic offer the prospect of reconstructing AMV from high-resolution records elsewhere.

55 increased the systemic transport of several AMV RNA3 derivatives carrying different viral MPs associ

56 can synthesize cDNA over 4 times longer than AMV RT in assays with poly(rA) templates.

57 the conformational switch model states that AMV coat protein blocks minus-strand RNA synthesis, whil

58 and the affinity between the 5' UTR and the AMV CP.

59 ncreased cell-to-cell transport for both the AMV RNA3 carrying the BMV MP and that carrying the AMV M

60 A3 carrying the BMV MP and that carrying the AMV MP.

61 Here, the AMV coat protein RNA binding domain is shown to contain

62 eased the relative abundance of m(6)A in the AMV genome, impairing the systemic invasion of the plant

63 ich amino-terminal RNA binding domain of the AMV coat protein lacks previously identified RNA binding

64 also increased the systemic transport of the AMV constructs that have reduced encapsidation capabilit

65 Anomaly dominated by a positive phase of the AMV.

66 e, dominated by a more negative phase of the AMV.

67 are competent to systemically transport the AMV genome without the requirement of the virus particle

68 48 residues and unable to interact with the AMV CP.

69 myeloblastosis virus reverse transcriptase (AMV-RT) or human DNA polymerase beta (pol beta), was sig

70 de of the Atlantic Multidecadal Variability (AMV), with relatively few events during the warm Medieva

71 ch as the Atlantic Multidecadal Variability (AMV), with stratification onset dates being twice as var

72 ale Atlantic multidecadal ocean variability (AMV), including the extreme pre-greenhouse-gas northern

73 al scale (Atlantic multidecadal variability, AMV).

74 ecrosis virus RNA, and alfalfa mosaic virus (AMV) 4, were used in wheat germ in vitro translation ass

75 g-distance movement of alfalfa mosaic virus (AMV) and brome mosaic virus (BMV), its precise function

76 e Bromoviridae family, alfalfa mosaic virus (AMV) and cucumber mosaic virus (CMV).

77 Alfalfa mosaic virus (AMV) and ilarvirus RNAs are infectious only in the prese

78 A defining feature of alfalfa mosaic virus (AMV) and ilarviruses [type virus: tobacco streak virus (

79 The coat proteins of alfalfa mosaic virus (AMV) and the related ilarviruses bind specifically to th

80 Alfalfa mosaic virus (AMV) coat protein and tobacco streak virus (TSV) coat pr

81 scribing regulation of alfalfa mosaic virus (AMV) replication have been tested using biochemical assa

82 The alfalfa mosaic virus (AMV) RNAs are infectious only in the presence of the vir

83 three genomic RNAs of Alfalfa mosaic virus (AMV).

84 type recombinant avian myeloblastosis virus (AMV) and Moloney murine leukemia virus (M-MLV) reverse t

85 ts with purified avian myeloblastosis virus (AMV) IN and retrovirus-like donor substrates containing

86 oncogene of the avian myeloblastosis virus (AMV) is unique among known oncogenes in that it causes o

87 , encoded by the avian myeloblastosis virus (AMV), can induce acute monoblastic leukemia in vivo and

88 d retroviral RT, avian myeloblastosis virus (AMV).

89 btained by using the arithmetic mean volume (AMV).

90 r Hid-induced cell death was suppressed when AMV-IAP was coexpressed.

91 RSV could transform only fibroblasts whereas AMV could transform only hematopoietic cells.

92 a 2:1 preference to insert dA over dC, while AMV-RT incorporated predominantly dC.

93 Second, chickens infected with AMV develop remarkably high white counts and therefore t

94 m and Hid were demonstrated to interact with AMV-IAP in vivo, and Grim- or Hid-induced cell death was