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1 E-ERalpha(vlVMH) neurons through opening the ATP-sensitive potassium channel.
2 l, betaIV-spectrin targets ankyrin-B and the ATP-sensitive potassium channel.
3 ll bodies of mNTS neurones via effects on an ATP-sensitive potassium channel.
4 eceptors (SURx) are required subunits of the ATP-sensitive potassium channel.
5 ough the activation of adenosine pathways or ATP-sensitive potassium channels.
6  parallel with the acquisition of functional ATP-sensitive potassium channels.
7  inhibition of glycolysis, and activation of ATP-sensitive potassium channels.
8 lly identified as an endogenous regulator of ATP-sensitive potassium channels.
9 ific cation channels, chloride channels, and ATP-sensitive potassium channels.
10  family, which associate with Kir6.x to form ATP-sensitive potassium channels.
11 cium-dependent potassium channels and not by ATP-sensitive potassium channels.
12 mily that is associated with Kir 6.x to form ATP-sensitive potassium channels.
13 bition of insulin secretion by activation of ATP-sensitive potassium channels.
14 ads to activation of adenosine triphosphate (ATP)-sensitive potassium channels.
15 as act by inhibiting adenosine triphosphate (ATP)-sensitive potassium channels.
16 by the putative blocker of the mitochondrial ATP sensitive potassium channel, 5-hydroxydecanoate, bef
17 ation and preconditioning-like mitochondrial ATP-sensitive potassium channel activation.
18 ned with an inward rectifier Kir6.2 subunit, ATP-sensitive potassium channel activity is generated.
19 lucose cotransporter SGLT1, or by closure of ATP-sensitive potassium channels after glucose metabolis
20 conditioning with diazoxide, a mitochondrial ATP-sensitive potassium channel agonist, prevented dendr
21 wnstream glucokinase effectors revealed that ATP-sensitive potassium channel and P/Q calcium channel
22 eir supramolecular assemblies, including the ATP-sensitive potassium channel and the peptide-loading
23 gers membrane depolarization both by closing ATP-sensitive potassium channels and because of its upta
24 version to lactate, leading to activation of ATP-sensitive potassium channels and to decreased hepati
25 itric oxide, cyclic guanosine monophosphate, ATP-sensitive potassium channels, and endothelin-1 exert
26  and unclear and may involve Akt activation, ATP-sensitive potassium channels, and nitric oxide, amon
27 vation of adenosine receptors and opening of ATP-sensitive potassium channels appear to play a role i
28  by covalently modifying (sulfhydrating) the ATP-sensitive potassium channel, as mutating the site of
29 nteractions involved in pancreatic beta-cell ATP-sensitive potassium channel assembly.
30 tions between Kir6.2 and SUR1 participate in ATP-sensitive potassium channel assembly.
31 al-ventricle administration of inhibitors of ATP-sensitive potassium channels, but not of antagonists
32                   Constitutive activation of ATP-sensitive potassium channels by diazoxide does not a
33 s that activation of adenosine triphosphate (ATP)-sensitive potassium channels contributes to vascula
34 s the majority of LHA MC4R-GFP neurons in an ATP- sensitive potassium channel-dependent manner.
35 Activation of either protein kinase C or the ATP-sensitive potassium channel has been shown to induce
36 ide, a selective opener of the mitochondrial ATP-sensitive potassium channel, has been shown to elici
37 tegral component of the pancreatic beta-cell ATP-sensitive potassium channel, IKATP, was investigated
38 h DZX, supporting a role for a mitochondrial ATP-sensitive potassium channel in the mechanism of card
39                                   Closure of ATP-sensitive potassium channels in pancreatic islet bet
40                Pharmacological activation of ATP-sensitive potassium channels in the brain normalized
41 ntial clinical consequences of inhibition of ATP-sensitive potassium channels in the heart of diabeti
42    These results suggest that the opening of ATP-sensitive potassium channels in vascular smooth musc
43 le sodium salt of glipizide, an inhibitor of ATP-sensitive potassium channels, in anesthetized and aw
44               In the pancreas, inhibition of ATP-sensitive potassium channels induces release of insu
45 hannel inhibitor), glibenclamide (5 mumol/L, ATP-sensitive potassium channel inhibitor), and iberioto
46 isolation buffer, cardioplegia (CPG)+/-DZX+/-ATP-sensitive potassium channel inhibitor, 5-hydroxydeca
47 nhibitor), 5-hydroxydecanoate (mitochondrial ATP-sensitive potassium channels inhibitor), or glibencl
48 ein kinase G-inhibitor) or glibenclamide (an ATP-sensitive potassium channel-inhibitor) all led to an
49                                          The ATP-sensitive potassium channel is a key molecular compl
50 ich in that dose is a selective inhibitor of ATP sensitive potassium channels (K(ATP)).
51                                          The ATP-sensitive potassium channel (K(ATP) ) links glucose
52                                          The ATP-sensitive potassium channel (K(ATP) channel) couples
53         Furthermore, we found that beta cell ATP-sensitive potassium channel (K(ATP)) channels are re
54 potassium channel subunit (Kir6) to form the ATP-sensitive potassium channel (K(ATP)) complex.
55 KCNJ11 encodes Kir6.2 a major subunit of the ATP-sensitive potassium channel (K(ATP)) expressed in bo
56 a protein that combines with SUR2 to form an ATP-sensitive potassium channel (K(ATP)) expressed in co
57                                          The ATP-sensitive potassium channel (K(ATP)) in mouse coloni
58             We have previously shown that an ATP-sensitive potassium channel (K(ATP)) is expressed in
59                                       Atrial ATP-sensitive potassium channel (K(ATP)) modulation duri
60                                              ATP-sensitive potassium channel (K(ATP)) openers target
61                                          The ATP-sensitive potassium channel (K(ATP)) regulates insul
62 odes Kir6.2, the pore-forming subunit of the ATP-sensitive potassium channel (K(ATP)), are the common
63 erminal transmitter release by actions on an ATP-sensitive potassium channel (K(ATP)).
64                                The beta-cell ATP-sensitive potassium channel (K-ATP channel), which r
65       Loss- or gain-of-function mutations in ATP-sensitive potassium channel (K-ATP)-encoding genes,
66 n beta-cells at a density similar to that of ATP-sensitive potassium channels (K(ATP) channels) and e
67 ites on SUR1 that antagonize the inhibition, ATP-sensitive potassium channels (K(ATP) channels) are d
68                                              ATP-sensitive potassium channels (K(ATP) channels) are h
69                                              ATP-sensitive potassium channels (K(ATP) channels) are h
70                                              ATP-sensitive potassium channels (K(ATP) channels) are i
71                           Opening of cardiac ATP-sensitive potassium channels (K(ATP) channels) is a
72                                              ATP-sensitive potassium channels (K(ATP) channels) of ar
73                                              ATP-sensitive potassium channels (K(ATP) channels) regul
74                                  Sarcolemmal ATP-sensitive potassium channels (K(ATP)) act as metabol
75                                              ATP-sensitive potassium channels (K(ATP)) are formed fro
76                                              ATP-sensitive potassium channels (K(ATP)) are involved i
77 eta-cells demonstrated that leptin activated ATP-sensitive potassium channels (K(ATP)) by increasing
78                IPC may involve activation of ATP-sensitive potassium channels (K(ATP)).
79 ate with the inward rectifier Kir6.x to form ATP-sensitive potassium channels (K(ATP)).
80                                              ATP-sensitive potassium channels (K(ATP); Kir6.x) are a
81                                          The ATP-sensitive potassium channels (K+ATP channels) are he
82                                              ATP-sensitive potassium channels (K-ATP channels) couple
83                                          The ATP-sensitive potassium channel, K(ATP) channel, a funct
84 methylpropanamides 1, were found to activate ATP sensitive potassium channels (KATP) and represent a
85 tion of macrophage polarization by targeting ATP sensitive potassium channels (KATP).
86 e perfused with Ringer solution (control), a ATP-sensitive potassium channel (KATP ) inhibitor, an in
87                        ABSTRACT: Sarcolemmal ATP-sensitive potassium channel (KATP channel) activatio
88 lming majority of evidence suggests that the ATP-sensitive potassium channel (KATP channel) is an imp
89 ells were compared to those of the reference ATP-sensitive potassium channel (KATP channel) openers d
90 1 revealed that the F1388 mutation abolished ATP-sensitive potassium channel (KATP) activity in intac
91                                          The ATP-sensitive potassium channel (KATP) controls insulin
92 g of the lead cardiac selective antiischemic ATP-sensitive potassium channel (KATP) opener (4) are de
93 and KCNJ11, the genes encoding the beta-cell ATP-sensitive potassium channel (KATP), are unresponsive
94                                  Sarcolemmal ATP-sensitive potassium channels (KATP channels) in card
95 axin (Syn)-1A interacts with SUR1 to inhibit ATP-sensitive potassium channels (KATP channels).
96  from the patient of origin, lack functional ATP-sensitive potassium channels (KATP) and also carry a
97                                              ATP-sensitive potassium channels (KATP) are implicated i
98                          In the vasculature, ATP-sensitive potassium channels (KATP) channels regulat
99 ; encoded by ABCC8) and its associated islet ATP-sensitive potassium channel (Kir6.2; encoded by KCNJ
100  is the prototypical opener of mitochondrial ATP-sensitive potassium channels (mitoK(ATP)) and protec
101 we demonstrated that targeting mitochondrial ATP-sensitive potassium channels (mitoK(ATP)) protects n
102                            The mitochondrial ATP sensitive potassium channel (mK(ATP)) is implicated
103 preconditioning induced by the mitochondrial ATP-sensitive potassium channel opener BMS-191095.
104                         ERP shortening by an ATP-sensitive potassium channel opener increases ventric
105 ve cardioplegia based on the hyperpolarizing ATP-sensitive potassium channel opener pinacidil, the pr
106  exposed to simulated ischemia or pinacidil (ATP-sensitive potassium channel opener).
107               Cromakalim (10 micromol/L), an ATP-sensitive potassium channel opener, caused a signifi
108  response to stress that is prevented by the ATP-sensitive potassium channel opener, diazoxide (DZX)
109 C by pinacidil (PIN, 10 micromol/L; n=6), an ATP-sensitive potassium channel opener.
110             In beta cells from the pancreas, ATP-sensitive potassium channels, or KATP channels, are
111 tassium channel (Ir), proteins that comprise ATP-sensitive potassium channels regulating hormone secr
112 th the goal of obtaining an activator of the ATP sensitive potassium channel suitable for use in the
113                                              ATP-sensitive potassium channels, termed KATP channels,
114                                Expression of ATP-sensitive potassium channels was absent in these gli
115               Adenosine-mediated pathways or ATP-sensitive potassium channels were activated by augme
116 ancy; the cells therefore lacked operational ATP-sensitive potassium channels, which results in the f
117 malian SUR genes are associated with K(ATP) (ATP-sensitive potassium) channels, which are involved in

 
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