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1 en had greater abundances of Veillonella and Actinomyces.
2 ain, and they may represent novel species of Actinomyces.
3 re confirmed as members of genera other than Actinomyces.
4 ng these organisms from other genera such as Actinomyces.
6 r of strains tested) used in this study were Actinomyces (32), Anaerobiospirillum (8), Bacteroides (3
9 ic line that is related to but distinct from Actinomyces, Actinobaculum, Arcanobacterium, and Mobilun
11 zed by presence of human-associated bacteria Actinomyces, Anaerococcus, Finegoldia, Micrococcus, Prev
12 lanomicrobium and human-associated bacteria (Actinomyces, Anaerococcus, Finegoldia, Micrococcus, Prev
14 vant heritability of the microbiome included Actinomyces and Capnocytophaga in monozygotic twins and
15 and Ezakiella in the vaginal microbiome and Actinomyces and Ezakiella in the vulvar microbiome were
16 biomarkers for host immune response, whereas Actinomyces and Microspora were potential biomarkers of
17 dependent intercellular interactions between Actinomyces and oral streptococci as well as host cells
20 up EP-HD100 presented greater proportions of Actinomyces and Streptococcus-like species and lower pro
21 EP-HN019, there were greater proportions of Actinomyces and Streptococcus-like species and lower pro
22 l interactions between oral streptococci and actinomyces and their adherence to tooth surface and the
23 elucidate the fimbrial assembly pathways in Actinomyces and their function in the pathogenesis of di
24 phylococcus burden was increased in CHP, and Actinomyces and Veillonella burdens were increased in IP
26 treptococcus anginosus group, Fusobacterium, Actinomyces, and Aggregatibacter spp., and 117 of 287 (4
27 ivity were also characterized by Prevotella, Actinomyces, and Capnocytophaga species and those free o
28 ous taxa increased, including Fusobacterium, Actinomyces, and Corynebacterium, while others showed si
29 onella, Streptococcus, Prevotella 7, Rothia, Actinomyces, and Gemella) were present in nearly all US
37 , for example, between oral streptococci and actinomyces, are central to dental plaque development.
38 revented recolonization of S. mutans but not Actinomyces, as compared with a control peptide or salin
39 teria phylum, we found several Actinomadura, Actinomyces, Bifidobacterium, Streptacidiphilus and Stre
40 ate coaggregation with oral streptococci and Actinomyces biofilm development requires the class C sor
42 between 1988 and 2014, and cases describing Actinomyces breast infections published in the medical l
43 onsistent with their assignment to the genus Actinomyces, but they did not appear to correspond to an
44 of Prevotella, Fusobacterium, Streptococcus, Actinomyces, Capnocytophaga, Selenomonas, and Veillonell
47 to frequent requests of bronchoscopists for Actinomyces cultures combined with a change in microbiol
49 ong these, we identify a conserved family of Actinomyces-derived defensin-like peptides, termed actif
55 we also demonstrated a complexity within the Actinomyces genus that compromises the biochemical ident
56 th and, in order of decreasing cell numbers, Actinomyces gerencseriae, Bifidobacterium, S. mutans, Ve
57 founders, were observed for A. naeslundii I, Actinomyces gerencseriae, C. gingivalis, E. corrodens, C
59 nd a higher percentage of species similar to Actinomyces gereneseriae, Actinomyces israelli, and Stre
61 rst case of disseminated infection with both Actinomyces graevenitzii and Mycobacterium tuberculosis
63 er, Prevotella, Capnocytophaga, Selenomonas, Actinomyces, Granulicatella, and Atopobium were increase
64 Non-users had a higher relative abundance of Actinomyces, Granulicatella, Haemophilus, Neisseria, Ori
67 types of adhesive fimbriae are expressed by Actinomyces; however, the architecture and the mechanism
71 n intergeneric coaggregation with human oral actinomyces, indicating the specificity of the mutation
72 ive lacrimal gland ductulitis, commonly from Actinomyces infection, should be considered in patients
75 only coisolated with a particular bacterium: Actinomyces israelii was the only Actinomyces spp. coiso
76 a case of spinal cord compression caused by Actinomyces israelii with the coisolation of Fusobacteri
77 species included Actinomyces naeslundii II, Actinomyces israelii, Actinomyces odontolyticus, Veillon
78 ococcus micros, Streptococcus milleri group, Actinomyces israelii, and Arcanobacterium haemolyticum.
79 kia epibionticum and its corresponding host, Actinomyces israelii, as a model system for unlocking th
80 d coinfection of Fusobacterium nucleatum and Actinomyces israelii, resolving these diagnostic discrep
81 treptococcus micros, Actinomyces naeslundii, Actinomyces israelii, Streptococcus sanguis, Streptococc
82 species similar to Actinomyces gereneseriae, Actinomyces israelli, and Streptococcus gordonii when co
84 f a previously undescribed catalase-negative Actinomyces-like bacterium were recovered from human cli
87 between oral viridans group streptococci and actinomyces may play an important role in microbial colo
88 acillus (Haemophilus) actinomycetemcomitans; Actinomyces meyeri was coisolated with Peptostreptococcu
89 gravenitzii, Actinomyces odontolyticus, and Actinomyces meyeri were isolated from respiratory specim
90 bacterium alocis, Actinomyces odontolyticus, Actinomyces meyeri, and Bifidobacterium dentium were all
94 sequence of the chromosomal DNA flanking the Actinomyces naeslundii (formerly A. viscosus) T14V type
96 he Orange-Blue cluster score (which included Actinomyces naeslundii and Eubacterium nodatum) was inve
99 Biofilms of S. mutans, alone or mixed with Actinomyces naeslundii and Streptococcus oralis, were in
100 terium spp., and Tannerella forsythia, while Actinomyces naeslundii and Streptococcus sanguinis were
101 hogen Streptococcus mutans UA159, as well as Actinomyces naeslundii ATCC 12104 and Streptococcus oral
104 tobacillus acidophilus, Lactobacillus casei, Actinomyces naeslundii genospecies (gsp) 1 and 2, total
105 immune response in saliva to colonization by Actinomyces naeslundii genospecies 1 and 2 was studied i
109 in the presence of Streptococcus oralis and Actinomyces naeslundii steadily formed exopolysaccharide
111 ive with antibody against type 2 fimbriae of Actinomyces naeslundii T14V (anti-type-2) were much less
112 saliva of two human oral commensal bacteria, Actinomyces naeslundii T14V and Streptococcus oralis 34,
117 organisms such as Streptococcus gordonii and Actinomyces naeslundii to the saliva-coated tooth surfac
119 ide to support fimbriae-mediated adhesion of Actinomyces naeslundii was explained by the position of
121 f antibodies against Eubacterium nodatum and Actinomyces naeslundii) was inversely associated with al
122 monas gingivalis, Peptostreptococcus micros, Actinomyces naeslundii, Actinomyces israelii, Streptococ
125 terococcus faecalis, Streptococcus gordonii, Actinomyces naeslundii, and Lactobacillus acidophilus),
126 eptococcus sanguis, Haemophilus aphrophilus, Actinomyces naeslundii, Fusobacterium nucleatum, and A.
127 d of species found in healthy oral biofilms (Actinomyces naeslundii, Lactobacillus casei, Streptococc
128 which was composed of Streptococcus sanguis, Actinomyces naeslundii, Porphyromonas gingivalis, and Fu
130 he oral key pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptococcus mutans, and Aggreg
131 romonas gingivalis, Fusobacterium nucleatum, Actinomyces naeslundii, Tannerella forsythia, and Strept
132 Streptococcus sanguis and type 2 fimbriated Actinomyces naeslundii, which bound terminal sialic acid
133 and characterized the urease gene cluster of Actinomyces naeslundii, which is one of the pioneer orga
141 ally significant urinary tract infections by Actinomyces neuii (21%) and Corynebacterium aurimucosum
144 d with decreased weight or growth (including Actinomyces odontolyticus and Prevotella melaninogenica)
147 apnocytophaga species, Fusobacterium alocis, Actinomyces odontolyticus, Actinomyces meyeri, and Bifid
148 ctinomyces lingnae, Actinomyces gravenitzii, Actinomyces odontolyticus, and Actinomyces meyeri were i
149 nomyces naeslundii II, Actinomyces israelii, Actinomyces odontolyticus, Veillonella parvula, Capnocyt
151 many gram-positive Actinobacteria, including Actinomyces oris and Corynebacterium matruchotii, the co
154 As a pioneer colonizer of the oral cavity, Actinomyces oris expresses proteinaceous pili (also call
155 glycosylation pathway in the oral bacterium Actinomyces oris in which sortase and LCP enzymes operat
156 n a lectin-like activity associated with the Actinomyces oris type 2 fimbria, a surface structure ass
157 iphtheriae and FimA of the type 2 pilus from Actinomyces oris unfold and extend at forces that are th
159 ral microbes, including the coaggregation of Actinomyces oris with Streptococcus oralis that helps to
161 implemented a novel cell-based screen using Actinomyces oris, a key colonizer in the development of
162 itial colonizers, Streptococcus gordonii and Actinomyces oris, as well as with Veillonella sp. (early
164 um diphtheriae and the heterodimeric pili in Actinomyces oris, highlighting some newly emerged basic
169 the housekeeping sortase SrtA was lethal for Actinomyces oris; yet, all of its predicted cell wall-an
171 ytophaga species and those free of caries by Actinomyces, Prevotella, Selenomonas, Streptococcus, and
173 he 2.4-kb plasmid pAP1 from Arcanobacterium (Actinomyces) pyogenes had sequence similarity within the
175 lytic exotoxin expressed by Arcanobacterium (Actinomyces) pyogenes, is a member of the thiol-activate
180 a hitherto unknown species within the genus Actinomyces related to, albeit distinct from, a group of
181 hes among the subject groups (Streptococcus, Actinomyces, Rothia, and Atopobium), but there were no i
182 M7, and Treponema spp, and increased that of Actinomyces, Rothia, Haemophilus, Corynebacterium, and S
183 tive adolescents were typically colonized by Actinomyces, Selenomonas, Prevotella, and Capnocytophaga
184 r, Streptococcus, and Neisseria and fewer in Actinomyces, Selenomonas, Veillonella, Campylobacter, an
186 ray included: Gemella sanguinis (p = 0.002), Actinomyces sp. HOT 448 (p = 0.003), Prevotella cluster
187 overabundant in the caries-active group were Actinomyces sp. strain B19SC, Streptococcus mutans, and
190 ntified bacterium showed some resemblance to Actinomyces species and related taxa, but biochemical te
192 hydrophobicity, deficient in binding to oral Actinomyces species and to human fibronectin, and unable
194 ity or membrane localization also eliminated Actinomyces species biofilm development and bacterial co
195 tinomycosis is a chronic infection caused by Actinomyces species characterized by abscess formation,
196 , our data suggest that among the mechanisms Actinomyces species employ to persist in the oral cavity
197 med clinically significant included multiple Actinomyces species in wounds, Propionibacterium species
199 ctions in NHS Lothian between 2005 and 2013, Actinomyces species isolated from breast infections refe
200 data suggest that A. gerencseriae and other Actinomyces species may play an important role in caries
204 a hitherto unknown subline within a group of Actinomyces species which includes Actinomyces bovis, th
205 forsythus, three Capnocytophaga species, six Actinomyces species, four Propionibacterium species, and
206 31 (70%) were clearly assigned to recognized Actinomyces species, including 94 isolates assigned to s
207 th type 2 DM presented higher proportions of Actinomyces species, purple and green complexes, and low
208 had higher prevalences of Streptococcus and Actinomyces species, while A. actinomycetemcomitans-posi
210 een high frequency of SUP and high levels of Actinomyces spp, Streptococcus spp., members of the oran
211 es of incidence and clinical associations of Actinomyces spp. add to the currently sparse knowledge o
217 bacterium: Actinomyces israelii was the only Actinomyces spp. coisolated with Actinobacillus (Haemoph
219 We determined the frequency distribution of Actinomyces spp. recovered in a routine clinical laborat
220 and 299 stored clinical isolates of putative Actinomyces spp. referred to the Anaerobe Reference Unit
221 nd disease associations for 21 genogroups of Actinomyces spp. that provide greater insights into appr
225 erella, and Veillonella and 36 of 37 (97.3%) Actinomyces strains, 42 of 46 (91.3%) B. fragilis group
227 ty and abundance of Neisseria, Leptotrichia, Actinomyces, Streptococcus, Rothia, and Veillonella (com
228 enotypic characterization, we found that the Actinomyces/Streptococcus coaggregation is only abolishe
229 ompromises the biochemical identification of Actinomyces that can be performed in most clinical labor
235 ve moieties of FimA, the shaft fimbrillin of Actinomyces type 2 fimbriae, which uniquely mediates the
236 neric coaggregation between streptococci and actinomyces, type 2 fimbriae of actinomyces recognize RP
237 red with controls, whereas 2 studies each of Actinomyces, Veillonella, and Escherichia coli revealed
238 Animals were desalivated, infected with Actinomyces viscosus and Streptococcus mutans (sobrinus)
239 The oral bacteria Actinomyces naeslundii and Actinomyces viscosus are known to contribute to the init
240 e protein containing the catalytic domain of Actinomyces viscosus sialidase, removes cell surface sia
242 ferences in hydrophobicity or coadherence to Actinomyces viscosus were detected between the mutants a
243 e of a genospecies 2 A. naeslundii, formerly Actinomyces viscosus, a portion of a homologue of this g
244 ticola, and Tannerella forsythia, as well as Actinomyces viscosus, Campylobacter rectus/showae, Prevo
245 s from hamsters which had been infected with Actinomyces viscosus, fed a caries-promoting diet, and r
247 Streptococcus, Prevotella, Pseudomonas, and Actinomyces) were almost absent from the lower GI tract,
248 ly, 3 genera (Treponema, Fretibacterium, and Actinomyces) were identified that could be used for calc
249 ldanamycin (GA) is an antibiotic produced by Actinomyces, which specifically inhibits the function of
251 uently, the interactions of streptococci and actinomyces with the mucinlike domains of these mammalia