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1 e (K(i) = 0.05 +/- 0.02 microM for 4-N-allyl-AlCAR).
2 d after feeding old rats acetyl-L-carnitine (ALCAR).
3 tochondrial metabolites, acetyl-l-carnitine (ALCAR) [0.5% or 0.2% (wt/vol) in drinking water], and/or
4  the dietary addition of acetyl-l-carnitine [ALCAR, 1.5% (wt/vol) in the drinking water] and/or (R)-a
5 nificantly restored CAT-binding affinity for ALCAR and CoA, and CAT activity.
6 in CAT-binding affinity for both substrates, ALCAR and CoA.
7           These results suggest that feeding ALCAR and LA to old rats improves performance on memory
8                 Dietary supplementation with ALCAR and/or LA improved memory, the combination being t
9 copic studies in the hippocampus showed that ALCAR and/or LA reversed age-associated mitochondrial st
10                    Dietary administration of ALCAR and/or LA significantly reduced the extent of oxid
11 s with the CAT substrate acetyl-l-carnitine (ALCAR) and/or the mitochondrial antioxidant precursor R-
12  acid, or an acetyl donor, acetyl-carnitine (ALCAR), elevated histone acetylation levels in the human
13 n, while restoration of SIRT3 with AICAR and ALCAR improved cisplatin-induced mitochondrial dysfuncti
14                                      Feeding ALCAR in combination with LA increased metabolism and lo
15  rats were given a 1.5% (wt/vol) solution of ALCAR in their drinking water for 1 mo, were sacrificed,
16                                              ALCAR increases cellular oxygen consumption, which decli
17               Feeding old rats LA or LA plus ALCAR inhibited lipid peroxidation but did not decrease
18                         To determine whether ALCAR+LA also affected indices of oxidative stress, asco
19                                              ALCAR+LA also increased ambulatory activity in both youn
20                                              ALCAR+LA partially reversed the age-related decline in a
21 n old versus young rats, also declined after ALCAR+LA supplementation and was not significantly diffe
22 estored to the level seen in young rats when ALCAR+LA was given.
23                                      Feeding ALCAR or ALCAR plus LA to old rats significantly restore
24                    Preincubation of CAT with ALCAR or CoA prevented malondialdehyde-induced dysfuncti
25 also greater when compared with old rats fed ALCAR or LA alone.
26                             Feeding ALCAR or ALCAR plus LA to old rats significantly restored CAT-bin
27 or the antioxidant agent acetyl-l-carnitine (ALCAR) restored SIRT3 expression and activity, improved
28                                              ALCAR supplementation increases ambulatory activity sign
29  untreated old rats, further indicating that ALCAR supplementation might increase oxidative stress.
30                                              ALCAR supplementation significantly reverses the age-ass
31                                        Thus, ALCAR supplementation to old rats markedly reverses the
32 % and 50% lower, respectively, in cells from ALCAR-supplemented old rats than in untreated old rats,
33 n WT animals and died, and neither AICAR nor ALCAR treatment prevented death in Sirt3-/- AKI mice.
34 s improved sociability in Kat6b+/- mice, and ALCAR treatment restored learning and memory.