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1 ecombination event between flanking intronic Alu sequences.
2 stered within transcribed sense or antisense Alu sequences.
3 ding sequence is 99% made up of a cluster of Alu sequences.
4  of a long inverted repeat composed of human Alu sequences.
5  proteins with human DNA elements containing Alu sequences.
6 CpG dinucleotide hot spots) in the consensus Alu sequences.
7 rodent genome and are analogous to the human Alu sequences.
8 mined that the repeat is flanked by inverted Alu sequences.
9 s and CpG dinucleotides make up about 20% of Alu sequences.
10 t 80-150bp amplicons generated from adjacent Alu-sequences.
11  in having fewer and more evenly distributed Alu sequences (a single pair in each repeat unit) and a
12          The number of published examples of Alu sequences affecting gene control has grown threefold
13                    An IR involving the human Alu sequence (Alu-IR) has been studied as a model of suc
14          Intron 3 of these genes contains an Alu sequence and intron 4 contains a partial mer13 seque
15 her gene (EIF4E2) was intronless, flanked by Alu sequences and 14-base pair (bp) direct repeats, and
16  on the real-time PCR amplification of human alu sequences and exhibits a high sensitivity (25 cells/
17  counts and distribution of genes, Psig, and Alu sequences and G+C variation are evaluated with respe
18              Subfamily classification of the Alu sequences and identification of flanking direct repe
19                                  Full length Alu sequences are present at the centromeric ends of the
20                           Once inserted, the Alu sequences are rarely subject to loss or rearrangemen
21               Several adjacent insertions of Alu sequences are seen at the hprt region.
22                                              Alu sequences are the most abundant short interspersed r
23 2) linearized vectors that contained a human Alu sequence at one end and a telomere sequence at the o
24                               Recombinogenic Alu sequences bracketed the breakpoints in both fusions.
25                                  Transcribed Alu sequences can alter splicing patterns by generating
26  interacting eRNAs and uaRNAs, predominantly Alu sequences, confer the specificity for eRNA-uaRNA pai
27                                              Alu sequences contain an internal RNA polymerase III pro
28                     We have isolated a novel Alu sequence-containing cDNA, designated AD7c-NTP, that
29 endent MethyLight control reactions based on Alu sequences depleted of CpG dinucleotides by evolution
30 f our algorithmic techniques for identifying Alu sequences, gene clusters and other distant regions o
31 re described, there is an incident where one Alu sequence has inserted into the middle poly(A) tract
32                      Distinct subfamilies of Alu sequences have amplified within the human genome in
33 two genes derives from insertion of multiple Alu sequences immediately upstream and downstream and wi
34 rtion of an Alu element in intron 6 with non-Alu sequence in intron 1.
35 s were confirmed by the analysis of a second Alu sequence in intron 10 of the p53 gene.
36 d from alternative splicing that involved an Alu sequence in intron 3 of SULT1C1.
37 omparison of this primordial sequence to the Alu sequence in intron 6 of present-day primates was use
38 They are distinguished by the presence of an Alu sequence in the 3' nontranslated end of the longer s
39                    PCR assays detected human Alu sequences in DNA in pancreas and kidney on day 17 or
40  of a recombination event between homologous Alu sequences in introns 6 and 1.
41 ed between Alu_Ins and pre-existing intronic Alu sequences, including the restoration of SPINK1 mRNA
42 ntaining in their 3'-UTR inverted repeats of Alu sequences (IRAlu).
43                                  A truncated Alu sequence is present at the telomeric end of the prox
44          A fusion mRNA between COL5A1 and an Alu sequence is produced, but no aberrant protein is det
45 nd that editing of transcripts with embedded Alu sequences is a global phenomenon in the human transc
46 hat a recombination event between homologous Alu sequences is responsible for the partial tandem dupl
47                                              Alu sequences may serve as substrates for elevated HR du
48 r distribution and circumstances under which Alu sequences might affect genome stability.
49 bstitution in the last position of the 95 bp Alu sequence of the rhesus monkey, an old world monkey.
50 ne the effects of large deletions within the Alu sequence on retrotransposition.
51                    We have developed a human Alu sequence PCR-based assay to quantitate intravasated
52 n, using TRIzol-based DNA/RNA extraction and Alu sequence qPCR amplification.
53          Exon 8a is derived from an exonized Alu sequence, sharing no homology with exon 8 of the lon
54            We examined RNAs corresponding to Alu sequences that were differentially active during pri
55                               Like the human Alu sequence, the SINEs found in salmonids could provide
56                                              Alu sequences, the most abundant class of large disperse
57 gions of Alu repeats, rather than within the Alu sequences themselves.
58                                          The Alu sequence was less similar to the consensus sequence
59 ated when rodent DNAs or a vector lacking an Alu sequence was used, the YACs were most likely the con
60  unit containing homology to highly repeated Alu sequences was identified in the upstream flanking se
61                         Sixty-two repetitive Alu sequences were identified in introns 1-49.
62 here are now six published examples in which Alu sequences were inserted long ago into human gene reg
63 kpoints in the partner DNAs, suggesting that Alu sequences were relevant to these rearrangements.
64                         However, most of the Alu sequences were unable to bind to PAX6 because of a s
65 encoding region of man is considered rich in Alu sequences: with 49 sequences present within 57 kilob
66 used to target the YZ sequence to repetitive Alu sequences within the human YAC.
67 ription of exon 7 of the CASR containing the Alu sequence yielded the full-length mutant product and