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1 athogens including human anaplasmosis agent, Anaplasma phagocytophilum.
2 a spp., Rickettsia spp., Ehrlichia spp., and Anaplasma phagocytophilum.
3 burgdorferi sensu lato, Babesia microti, and Anaplasma phagocytophilum.
4 orrelia burgdorferi or the rickettsial agent Anaplasma phagocytophilum.
5 aris transmits the human anaplasmosis agent, Anaplasma phagocytophilum.
6 host stimulation with the rickettsial agent Anaplasma phagocytophilum.
7 used by the obligate intracellular bacterium Anaplasma phagocytophilum.
8 a critical role in the early eradication of Anaplasma phagocytophilum.
9 sease, or a variant strain, Ap-Variant 1, of Anaplasma phagocytophilum.
10 ession sites in both Anaplasma marginale and Anaplasma phagocytophilum.
11 used by the obligate intracellular bacterium Anaplasma phagocytophilum.
12 a focus on the model intracellular bacterium Anaplasma phagocytophilum.
22 nd von Willebrand factor immunofluorescence, Anaplasma phagocytophilum and Anaplasma marginale were s
24 Pennsylvania and tested for the presence of Anaplasma phagocytophilum and Borrelia burgdorferi by PC
26 amine TH 1 and TH 17 immunity infection with Anaplasma phagocytophilum and Citrobacter rodentium resp
27 , O. tsutsugamushi, and infections caused by Anaplasma phagocytophilum and Ehrlichia chaffeensis with
30 gen, seventy-four (40%) were seroreactive to Anaplasma phagocytophilum, and five (2.7%) were seroposi
31 ate substrates, an ankyrin repeat protein of Anaplasma phagocytophilum, AnkA, is delivered into the h
33 Anaplasmosis, a tick-borne illness caused by Anaplasma phagocytophilum (AP), presents with nonspecifi
37 nt outer membrane proteins (P44 proteins) of Anaplasma phagocytophilum are encoded by the p44 polymor
39 intracellular bacteria such as Ehrlichia and Anaplasma phagocytophilum, as well as obligate intracell
40 ted real-time PCR assays for Ehrlichia spp., Anaplasma phagocytophilum, Babesia spp., or Lyme Borreli
41 ngton State suspected of being infected with Anaplasma phagocytophilum because of the finding of moru
42 d 100% sensitivity for the identification of Anaplasma phagocytophilum, Borrelia miyamotoi, Borrelia
43 Ixodes scapularis ticks infected with either Anaplasma phagocytophilum (causative agent of anaplasmos
48 Colonization of neutrophils by the bacterium Anaplasma phagocytophilum causes the disease human granu
49 rrelia burgdorferi and the rickettsial agent Anaplasma phagocytophilum Collectively, we highlight the
52 egative for all tick-borne pathogens tested (Anaplasma phagocytophilum, Ehrlichia canis, and Ricketts
53 hogens in the order Rickettsiales, including Anaplasma phagocytophilum, Ehrlichia canis, E. chaffeens
54 ighly conserved with orthologous proteins in Anaplasma phagocytophilum, Ehrlichia chaffeensis, and Eh
56 from 16S rRNA gene-based genetic variants of Anaplasma phagocytophilum from dogs in the western Unite
57 to determine the presence and prevalence of Anaplasma phagocytophilum human agent (AP-ha) and a gene
58 ptional regulator, tr1, and a homolog of the Anaplasma phagocytophilum, identified here as A. platys
61 n Anaplasma marginale and the human pathogen Anaplasma phagocytophilum, in order to examine the abili
66 tick vector Ixodes scapularis in response to Anaplasma phagocytophilum infection, the causative agent
88 he agent of human granulocytic ehrlichiosis (Anaplasma phagocytophilum) is intensely enzootic in rabb
89 during infection with the rickettsial agent Anaplasma phagocytophilum Macrophages deficient in annex
90 3 of Anaplasma marginale and Anaplasma ovis, Anaplasma phagocytophilum MSP2 (p44), Ehrlichia chaffeen
92 membrane protein of Anaplasma marginale and Anaplasma phagocytophilum pathogens that cause bovine an
95 Borellia burgdorferi, Babesia microti and Anaplasma phagocytophilum rely almost exclusively on a s
97 burgdorferi sensu lato, Borrelia miyamotoi, Anaplasma phagocytophilum, Rickettsia spp., Candidatus N
98 rrelia burgdorferi s.l., Borrelia miyamotoi, Anaplasma phagocytophilum, Rickettsia spp., Candidatus N
99 et al. demonstrate that ticks infected with Anaplasma phagocytophilum show enhanced fitness against
100 first tissue culture isolates of the unique Anaplasma phagocytophilum strain, Ap-Variant 1, were obt
101 ing of the region directly downstream of the Anaplasma phagocytophilum (strain MRK) 16S rRNA gene ide
102 ated first to Anaplasma platys and second to Anaplasma phagocytophilum, supporting its placement in t
103 ligate intracellular Gram-negative bacterium Anaplasma phagocytophilum The disease often presents wit
104 recent studies of the granulocyte pathogen, Anaplasma phagocytophilum, the agent of granulocytic ehr
106 burgdorferi, the agent of Lyme disease, and Anaplasma phagocytophilum, the agent of human anaplasmos
107 s harbor numerous human pathogens, including Anaplasma phagocytophilum, the agent of human granulocyt
108 y proteomics and transcriptome sequencing to Anaplasma phagocytophilum, the agent of human granulocyt
111 increases tick colonization by the bacterium Anaplasma phagocytophilum, the causative agent of human
116 ertain Ixodes ticks, which can also transmit Anaplasma phagocytophilum, the cause of human granulocyt
117 des scapularis tick, which can also transmit Anaplasma phagocytophilum, the cause of human granulocyt
123 ion of the mildly virulent rickettsial agent Anaplasma phagocytophilum through the SNARE proteins Vam
124 owledge regarding the strategies employed by Anaplasma phagocytophilum to evade or subvert neutrophil
125 ic ehrlichiosis was recently reclassified as Anaplasma phagocytophilum, unifying previously described
127 ce-exposed major membrane proteins, P44s, of Anaplasma phagocytophilum were hypothesized to be garner
128 obligate intracellular rickettsial bacterium Anaplasma phagocytophilum, which causes human granulocyt
129 his report, we show that the AnkA protein of Anaplasma phagocytophilum, which is translocated into th