ライフサイエンスコーパス: B. subtilis

1 B. subtilis coat proteins (CotY, CotE, CotV and CotW) ex

2 B. subtilis has a thicker layer of peptidoglycan and lac

3 B. subtilis is a soil dwelling organism and mitomycin C

4 B. subtilis joins an ever-expanding group of bacteria, i

5 B. subtilis NusG shifts RNAP to the posttranslocation re

6 B. subtilis PGA and B. licheniformis PGA both elicited m

7 B. subtilis protects itself against sublancin by produci

8 B. subtilis QST713 produces the lipopeptides in a ratio

9 B. subtilis strains lacking SpoVAF or SpoVAEa and SpoVAF

10 B. subtilis, E. coli, and pga-deleted E. coli carrying t

11 study of selectivity toward B. cereus 11778, B. subtilis, Legionella pneumophila, and Salmonella Typh

12 lant Arabidopsis thaliana, B. cereus PK6-15, B. subtilis PK5-26 and B. circulans PK3-109 significantl

13 A B. subtilis strain lacking c-di-AMP is not viable at hig

14 We enriched the phosphatase activity from a B. subtilis cell extract and suppose that dephosphorylat

15 associated GR operon, and transcription of a B. subtilis D gene was controlled by RNA polymerase sigm

16 Here, we report a cryo-EM structure of a B. subtilis transcription activation complex comprising

17 Here we explain how to produce a B. subtilis SSB probe that exhibits 9-fold fluorescence

18 By screening a B. subtilis knock-out library for deficiency in acetylat

19 What is special about B. subtilis is the unusually rich repertoire of alternat

20 Our results across B. subtilis, Arabidopsis, E.coli, Drosophila and the DRE

21 ication of metabolic states between adjacent B. subtilis biofilms, providing a possible generalizable

22 P. aeruginosa, B. subtilis and S. aureus were used as a model organism

23 ium, belonging to the species P. aeruginosa, B. subtilis, and S. aureus.

24 ichia coli cells from the root surface after B. subtilis colonization, suggesting a possible protecti

25 Xene membranes reaches more than 73% against B. subtilis and 67% against E. coli as compared with tha

26 hat unlike U34 at the wobble position of all B. subtilis tRNAs of known sequence, U34 in the mutant t

27 Although B. subtilis was no longer detected in the guts of fish e

28 ctions in the interior of the filament among B. subtilis, P. aeruginosa and Salmonella enterica.

29 xpression level and mutation phenotype among B. subtilis strains, suggesting interstrain variation in

30 n the disinfection efficacies of E. coli and B. subtilis .

31 res of the regulatory domains of E. coli and B. subtilis MutL bound to their respective beta-clamps.

32 slinking, we have stabilized the E. coli and B. subtilis MutL-beta complexes and have characterized t

33 glycans in the septal PG of both E. coli and B. subtilis, organisms separated by 1 billion years of e

34 In model bacteria, such as E. coli and B. subtilis, regulation of cell-cycle progression and ce

35 se reconstituted from mycobacterial GyrA and B. subtilis GyrB, which exceeds the activity of M. tuber

36 olanacearum, E. coli, Staphylococcus sp. and B. subtilis, and exhibited activity against pathogens on

37 ing to EF-P modification in B. subtilis, and B. subtilis encodes the first EF-P ortholog that retains

38 rized gene amj (alternate to MurJ; ydaH) and B. subtilis MurJ (murJBs; formerly ytgP) are a synthetic

39 se secreted by non-virulent bacteria such as B. subtilis, can shift the delicate procoagulant-anticoa

40 While enzymes from both organisms assembled B. subtilis Lipid II into glycan strands, only the B. su

41 entified eight gene products that attenuated B. subtilis growth.

42 dark toxicity to the Gram positive bacterium B. subtilis and good photothermal killing efficiency tow

43 graming in the model Gram-positive bacterium B. subtilis.

44 n in cells has not been investigated because B. subtilis Lipid II was not previously available.

45 sphodiesterase GlpQ can discriminate between B. subtilis WTA and LTA.

46 c liquid cultures demonstrates that, in both B. subtilis and P. aeruginosa, a turbulent flow forms in

47 photothermal killing efficiency toward both B. subtilis and Gram negative E. coli, features that dem

48 gma(A), a promoter DNA, and the ligand-bound B. subtilis BmrR, a prototype of MerR-family TFs.

49 all three PGAs elicited IL-8 from iDCs, but B. subtilis PGA also elicited IL-6, and B. licheniformis

50 e elicited IL-8 and IL-6 from monocytes, but B. subtilis PGA also elicited IL-10 and TNF-alpha, where

51 crucial for Arabidopsis root colonization by B. subtilis and provide insights into how matrix synthes

52 nts, such as those presumably encountered by B. subtilis in the soil.

53 Many of the states exhibited by B. subtilis are similar to states observed in other bact

54 egulated in response to biofilm formation by B. subtilis.

55 as the major inhibitory molecule produced by B. subtilis GS67.

56 abF, markedly decreased biotin production by B. subtilis resting cells whereas a strain having a ceru

57 paradox of pulcherriminic acid production by B. subtilis.

58 ed polyamine norspermidine is synthesized by B. subtilis using the equivalent of the Vibrio cholerae

59 data identify the genes and proteins used by B. subtilis to produce PNAG as a significant carbohydrat

60 d specificity while other bacteria (E. coli, B. subtilis, and S. aureus) samples were applied.

61 lity of metabolically active cells (E. coli, B. subtilis, Enterococcus, P. aeruginosa and Salmonella

62 and Zn(II) as substrates and can complement B. subtilis strains defective in the endogenous export s

63 transcription activation complex comprising B. subtilis six-subunit (2alphabetabeta'omegaepsilon) RN

64 Surprisingly, after disruption of decoated B. subtilis spores with lysozyme and fractionation, appr

65 e report here that in a spermidine-deficient B. subtilis mutant, the structural analogue norspermidin

66 scriptomic analysis of a spermidine-depleted B. subtilis speD mutant uncovered a nitrogen-, methionin

67 important for successful competition during B. subtilis pellicle formation.

68 notion that the enzyme is tetrameric during B. subtilis sporulation.

69 Cloning the entire B. subtilis epsHIJK locus into pga-deleted E. coli, Kleb

70 contrast, there were strong correlations for B. subtilis in media supplemented with polyethylene-glyc

71 The layer orders inferred for B. subtilis and B. megaterium are consistent with measur

72 r observation begins to answer, at least for B. subtilis, a long-standing question on the exonucleoly

73 Despite the importance of oxygen for B. subtilis survival, we know little about how populatio

74 ructural features of spermidine required for B. subtilis biofilm formation are unknown and so are the

75 state', provides an alternative strategy for B. subtilis to endure nutrient depletion and environment

76 ved in, but is not absolutely essential for, B. subtilis pellicle formation.

77 rmidine biosynthetic pathway are absent from B. subtilis, confirming that norspermidine is not physio

78 Chimeric proteins derived from B. subtilis AbrB and the Spx C-terminus showed that a 28

79 itive action was observed for fengycins from B. subtilis, as well as the detergent CHAPS, when combin

80 Bacillus pumilus as well as a paralogue from B. subtilis called YweA.

81 is6-LonBs, ClpPBs, and ClpXBs proteases from B. subtilis was analyzed.

82 the same structure as the LanI protein from B. subtilis, SpaI, despite the lack of significant seque

83 Furthermore, B. subtilis EF-P is post-translationally modified with a

84 Furthermore, B. subtilis has been instrumental in the study of hetero

85 Furthermore, B. subtilis PBP1 catalyzed the exchange of both D-amino

86 porulation during growth in gastrointestinal B. subtilis isolates, presumably as a form of survival a

87 In B. subtilis, deletion of a D gene (B. subtilis gerKD [gerKDbs]) adjacent to the gerK operon

88 When we repaired the dtd gene, B. subtilis became resistant to the biofilm-inhibitory e

89 or Bacillus megaterium, although germinated B. subtilis spores were rapidly killed.

90 Production of sublancin gives B. subtilis a major competitive growth advantage over a

91 cteria: S. epidermidis, M. luteus, E. hirae, B. subtilis, and E. coli.

92 quantitative direct bioautography via HPTLC-B. subtilis was shown as a reliable tool for streamlined

93 In B. subtilis and Streptococcus pneumoniae, condensin comp

94 In B. subtilis and T. brucei, ms2ct6A disappeared and remai

95 In B. subtilis, CodY controls dozens of genes, but the thre

96 In B. subtilis, deletion of a D gene (B. subtilis gerKD [ge

97 In B. subtilis, the RBMs flank the region of the chromosome

98 cond identified protein acetyltransferase in B. subtilis We propose that at least one physiological f

99 to BsRppH, a source of redundant activity in B. subtilis has been proposed.

100 stress known to induce sigma(B) activity in B. subtilis.

101 he idea of an important role for c-di-AMP in B. subtilis and suggest that the levels of the nucleotid

102 The disruption of autolysis in B. subtilis cultures by TiO2 NPs suggests the mechanisms

103 athway is a bona fide precursor of biotin in B. subtilis.

104 hologous proteins can substitute for BslA in B. subtilis and confer a degree of protection, whereas Y

105 inactive omega-epsilon-zeta TA cassettes in B. subtilis mutants that were defective for different pr

106 aptation systems contribute to chemotaxis in B. subtilis and whether they interact with one another.

107 Using conditional alleles of condensin in B. subtilis, we demonstrate that depletion of its activi

108 Examination of the genes neighboring cotH in B. subtilis led us to identify two spore coat proteins,

109 r unknown RNA binding protein might exist in B. subtilis that can promote antitoxin/toxin RNA interac

110 hich is one of four known 3' exonucleases in B. subtilis.

111 to Bacillus subtilis CodY when expressed in B. subtilis cells.

112 When expressed in B. subtilis, FrvA increases resistance to iron both in w

113 ability to induce biofilm gene expression in B. subtilis.

114 Seven mutant flagellar filaments in B. subtilis and two in P. aeruginosa capture two differe

115 We found in B. subtilis that the rapid localization of RecA to repai

116 replicative DNA polymerase PolC functions in B. subtilis, we applied photobleaching-assisted microsco

117 e ~50 putative GNAT domain-encoding genes in B. subtilis for their effects on DNA compaction, and ide

118 essed the transcription of selected genes in B. subtilis.

119 motifs to identify novel partners of GpsB in B. subtilis and extend the members of the GpsB interacto

120 Iron and manganese homeostasis in B. subtilis are closely intertwined: a pfeT mutant is ir

121 messenger controls potassium homeostasis in B. subtilis at a global level by binding to riboswitches

122 udied the requirement for topoisomerase I in B. subtilis.

123 des the primary route of magnesium import in B. subtilis and that the other putative transport protei

124 characterized the major enzymes involved in B. subtilis alanine biosynthesis and identified an alani

125 induction imposes severe iron limitation in B. subtilis resulting in derepression of both Fur- and P

126 ugh the emergence of redundant mechanisms in B. subtilis and related organisms.

127 the pathway leading to EF-P modification in B. subtilis, and B. subtilis encodes the first EF-P orth

128 results we suggest that m6A modifications in B. subtilis function to promote gene expression.

129 regulation of cell chaining and motility in B. subtilis.

130 tion of a dominant negative gerD mutation in B. subtilis.

131 ore, we postulate that adaptive mutations in B. subtilis can be generated through a novel mechanism m

132 act dynamically to individualize origins in B. subtilis and, when loaded along eukaryotic chromosome

133 The retention of the indirect pathway in B. subtilis and B. halodurans likely reflects the ancien

134 ee functional RNase H enzymes are present in B. subtilis NCIB 3610 and that the plasmid-encoded RNase

135 nsor that can be used as a helicase probe in B. subtilis and closely related gram positive bacteria.

136 1.1 and delta, an RNAP-associated protein in B. subtilis, bearing implications for the so-far unknown

137 KimA and other potential target proteins in B. subtilis with c-di-AMP.

138 is an additional RNA pyrophosphohydrolase in B. subtilis.

139 We found that loss of RecD2 in B. subtilis sensitized cells to several DNA-damaging age

140 fect, indicating a new function for RecD2 in B. subtilis.

141 s one of the prominent c-di-AMP receptors in B. subtilis.

142 biosynthesis is differentially regulated in B. subtilis from classically studied Gram-negative flage

143 very aspect of transcriptional regulation in B. subtilis.

144 ity control function of IleRS is required in B. subtilis for efficient sporulation and suggests that

145 We show that loss of RER in B. subtilis causes strand- and sequence-context-dependen

146 In particular, uncoupled RNAPs in B. subtilis explain the diminished role of Rho-dependent

147 least for the establishment of cell shape in B. subtilis.

148 ts that govern the entry into sporulation in B. subtilis and discuss how the use of regulated cell de

149 ructure that is a hallmark of sporulation in B. subtilis and other spore-forming Firmicutes.

150 function of the three adaptation systems in B. subtilis.

151 We observe that in B. subtilis, a relA mutant grows exclusively as unchaine

152 Interestingly, we find that in B. subtilis, unlike E. coli where multiple enzymes have

153 Taken together, these data show that, in B. subtilis, a previously uncharacterized posttranslatio

154 Unlike in B. subtilis, SpoIIQ of Clostridium difficile has intact

155 sis against excess environmental xanthine in B. subtilis, suggesting that regulation of XPRT is key f

156 es, among them MutT, NudF, YmaB, and YvcI in B. subtilis We found that in vitro, YvcI converts RNA 5'

157 We purified the AR9 nvRNAP from infected B. subtilis cells and characterized its transcription ac

158 s bound to its natural substrate, the intact B. subtilis peptidoglycan.

159 rol transcription of a gene of interest into B. subtilis.

160 ty by the antitoxin EcMazE diverges from its B. subtilis homolog.

161 uorescent D-amino carboxamide probe to label B. subtilis PG in vivo and found that this probe labels

162 Surfactin not only lysed B. subtilis vesicles, but also vesicles from Bacillus an

163 flagella and the absence of a periplasm make B. subtilis a premier organism for the study of the earl

164 we develop to port this pathway should make B. subtilis easier to engineer in the future.

165 detect regions of high negative charge near B. subtilis, not detected in the topographical SICM resp

166 B. anthracis PGA and PGAs from nonpathogenic B. subtilis subsp. chungkookjang and B. licheniformis Mo

167 -aminopentanol moiety attached to Lys(32) of B. subtilis EF-P that is required for swarming motility.

168 of the SSB C terminus impairs the ability of B. subtilis to form repair centers in response to damage

169 etion of prkC or prpC altered the ability of B. subtilis to grow under gluconeogenic conditions.

170 atly stimulates the endonuclease activity of B. subtilis MutL and supports this activity even in the

171 We suggest that administration of B. subtilis EPS can be used to broadly inhibit T cell ac

172 Experimental and SNP analyses of B. subtilis genomes show mutational footprints consisten

173 ckout mutants and transcriptomic analysis of B. subtilis NCIB 3610 cells revealed that genes from the

174 cally significant reduction in attraction of B. subtilis, with no impact on attraction of A. tumefaci

175 ults show a distinct chemotactic behavior of B. subtilis toward a particular root segment, which we i

176 Biochemical characterization of B. subtilis RecD2 showed that it is a 5'-3' helicase and

177 cture is location dependent; the cylinder of B. subtilis has dense circumferential orientation, while

178 The dispersal of B. subtilis was very limited, particularly under protect

179 Although the genome of B. subtilis encodes three c-di-AMP-producing diadenlyate

180 is required for the efficient germination of B. subtilis spores.

181 ge amounts of c-di-AMP impairs the growth of B. subtilis and results in the formation of aberrant cur

182 nations for the puzzling fact that growth of B. subtilis does not result in the significant accumulat

183 owever, excess serine inhibits the growth of B. subtilis.

184 solutions markedly enhanced inactivation of B. subtilis spores in 10 mM phosphate buffer; increasing

185 s, respectively, whereas the inactivation of B. subtilis spores was slightly enhanced.

186 we report that a gastrointestinal isolate of B. subtilis sporulates with high efficiency during growt

187 or and even to a greater extent than loss of B. subtilis itself.

188 Specifically, a fengycin-defective mutant of B. subtilis GS67 lost inhibitory activity against pathog

189 exopolysaccharide (EPS)-deficient mutant of B. subtilis was used, suggesting that EPS are the protec

190 Using mutants of B. subtilis that prevent flagellum rotation, they measur

191 ancy in the bacitracin resistance network of B. subtilis is a general principle to be found in many b

192 ose, designed from a detailed observation of B. subtilis levansucrase (SacB) acceptor structural requ

193 Thus, the rplJL operon of B. subtilis is regulated by transcription attenuation an

194 S rRNA gene amplicons showed the presence of B. subtilis in the gut during the seven days of probioti

195 g to determine the transcription profiles of B. subtilis strains expressing mutant CodY proteins with

196 ines and genome-wide mutational profiling of B. subtilis lacking RNase HII, the enzyme that incises a

197 portant for the anti-infective properties of B. subtilis and its relatives.

198 perone protein ClpC of the ClpCP protease of B. subtilis Our results further reveal that Gp53 is a ph

199 Finally, the secretome of B. subtilis might be used for the green synthesis of Ag-

200 rong, fungicidal activity and selectivity of B. subtilis QST713 lipopeptides.

201 wing cells, dormant and germinated spores of B. subtilis, and dormant spores of several other Bacillu

202 e no killing or rupture of dormant spores of B. subtilis, Bacillus cereus or Bacillus megaterium, alt

203 he spo0B gene in a delta-knock-out strain of B. subtilis compared with the wild-type.

204 inally, we show that domesticated strains of B. subtilis carry a mutation in sigH, which influences t

205 We found that in wild strains of B. subtilis, surfactin disrupted vesicles while in labor

206 We present the crystal structure of B. subtilis PhoD.

207 3 are sufficient to disrupt the structure of B. subtilis spores resulting in decreased viability.

208 We conclude that the susceptibility of B. subtilis to the biofilm-inhibitory effects of D-amino

209 Here we report the synthesis of B. subtilis Lipid II and its use by purified B. subtilis

210 ease YbeC as the major serine transporter of B. subtilis.

211 tant tRNA binds strongly to the AUA codon on B. subtilis ribosomes but only weakly to AUG.

212 analysis was used to map TiO2 deposition on B. subtilis cell walls and released enzymes, supporting

213 allate (TF3) exhibited inhibitory effects on B. subtilis c-di-AMP synthase, DisA.

214 les and proteins, a field heavily relying on B. subtilis secretion capabilities.

215 Studies on B. subtilis emphasized the genetics and biochemistry of

216 subtilis' or HSBS) was compared to that onto B. subtilis biomass with a low concentration of sulfhydr

217 on of natural products in the model organism B. subtilis and paves the way to the development of futu

218 en oxygen was depleted from stationary phase B. subtilis cultures, ~90% of cells died while the remai

219 the class 2 mutations in both Gram-positive B. subtilis and Gram-negative Escherichia coli.

220 The Gram-positive B. subtilis show a much higher conductivity around the c

221 Like the Gram-positive B. subtilis SpoIIIJ, the conserved arginine was required

222 agreement with structural features present: B. subtilis gyrase is a minimal enzyme, and its subunits

223 ) exogenous norspermidine at 25 muM prevents B. subtilis biofilm formation, (3) endogenous norspermid

224 pendent adhesive properties of the probiotic B. subtilis natto (Bsn).

225 increased the population of matrix-producing B. subtilis cells and that this activity could be abolis

226 show that the extracellular matrix protects B. subtilis colonies from infiltration by P. chlororaphi

227 Purified B. subtilis aconitase bound to the citZ 5' leader RNA in

228 B. subtilis Lipid II and its use by purified B. subtilis PBP1 and E. coli PBP1A.

229 rast, the phylogenetic range of recognizable B. subtilis RppH orthologs appears to be restricted to t

230 reconstituted in vitro by mixing recombinant B. subtilis PxpA, PxpB, and PxpC proteins.

231 ined following immunization with recombinant B. subtilis spores were able to reduce the adhesion of C

232 In addition to regulating B. subtilis biofilm formation, we found that RapP regula

233 attraction of the beneficial rhizobacterium B. subtilis.

234 Second, B. subtilis synthesizes its own siderophore bacillibacti

235 ore, we monitor the motility state of single B. subtilis cells across multiple generations by the exp

236 In the soil-dwelling firmicute species B. subtilis, the RNA pyrophosphohydrolase BsRppH, a memb

237 Thus, the mesophilic species B. subtilis and E. coli share the same sigma1.1 fold, wh

238 ntact with B. subtilis cells, and stimulates B. subtilis sporulation.

239 richia coli (E. coli) and Bacillus subtilis (B. subtilis) by bacterial growth on the membrane surface

240 rium (S. typhimurium) and Bacillus subtilis (B. subtilis) were examined and observed.

241 These results suggest B. subtilis senses restriction of flagellum rotation as

242 We conclude that B. subtilis EPS is an immunomodulatory agent that induce

243 These findings led us to discover that B. subtilis cells that overproduce KinA can bypass the s

244 Furthermore, we find that B. subtilis sporulation observed prior to direct contact

245 ve bacterium Bacillus subtilis We found that B. subtilis sigma1.1 is highly compact because of additi

246 Altogether our data indicate that B. subtilis blocks entry into sporulation in high-salini

247 Here we report that B. subtilis NusG makes sequence-specific contacts with a

248 We show that B. subtilis and E. coli gyrases are proficient DNA-stimu

249 We show that B. subtilis GS67 persists in the C. elegans intestine an

250 obable substrates for Mini-III and show that B. subtilis Mini-III is also involved in intron regulati

251 Using genetic analysis we show that B. subtilis strains lacking the ability to synthesize pu

252 Moreover, modeling studies suggested that B. subtilis sigma1.1 requires minimal conformational cha

253 The B. subtilis replisome is eukaryotic-like in that it reli

254 conventional cobalamin riboswitches and the B. subtilis cobalamin riboswitch reveal that the likely

255 ted to the electrical signal released by the B. subtilis biofilm.

256 his, met, and leu revertants produced by the B. subtilis YB955 parental strain.

257 us and for the first time, characterized the B. subtilis SSB's DNA binding mode switching and stoichi

258 Here, we describe the B. subtilis PfeT protein (formerly YkvW/ZosA) as a P1B4

259 well characterized type I TA system from the B. subtilis chromosome, bsrG/SR4, reveals similarities b

260 TP activation in KtrA, a RCK domain from the B. subtilis KtrAB cation channel, we have found that act

261 of differentiation of null mutants from the B. subtilis ordered knockout collection.

262 S-adenosyl-methionine-I riboswitch from the B. subtilis yitJ gene encoding methionine synthase, can

263 To further explore how the B. subtilis cell wall structure can influence the SICM c

264 Our findings offer novel insight into the B. subtilis phosphate starvation response and implicate

265 reased fluidity and loss of structure of the B. subtilis colony.

266 ase-dependent intracellular signaling of the B. subtilis DDR is achieved via production of L-malic ac

267 Cloning of the B. subtilis epsH-K genes into Escherichia coli with in-f

268 Examination of the B. subtilis genome sequence showed that these EF-P-depen

269 losis gyrase and reaches the activity of the B. subtilis gyrase, indicating that the activities of en

270 The protein components of the B. subtilis matrix include the secreted proteins BslA, w

271 Here, we describe the use of the B. subtilis model system to study the adaptation of thes

272 The bioelectrical environment of the B. subtilis was found to be considerably more negatively

273 ant protein or to TcdA26-39 expressed on the B. subtilis spore surface, cross-react with a number of

274 tilis Lipid II into glycan strands, only the B. subtilis enzyme cross-linked the strands.

275 The materials to produce the B. subtilis SSB probe are commercially available, so the

276 s supported FtsZ assembly, but replacing the B. subtilis FtsZ linker with a 249-residue linker from A

277 ences in mutation rates of genes require the B. subtilis Y-family polymerase, PolY1 (yqjH).

278 having a second role beyond structuring the B. subtilis colony biofilm.

279 on scattering spectra, we confirmed that the B. subtilis cell membrane is lamellar and determined tha

280 cleave their pre-rRNA substrates within the B. subtilis 50S particle.

281 Thus, B. subtilis and probably most bacteria use two distinct

282 Thus, B. subtilis switches from a unicellular to a multicellul

283 luster in several species closely related to B. subtilis hints at the importance of this protein phos

284 than B. anthracis PGA, but only responses to B. subtilis PGA were affected by a TLR6 neutralizing Ab.

285 tly shorter than that of the transcriptional B. subtilis metI RNA.

286 nd that norspermidine is absent in wild-type B. subtilis biofilms at all stages, and higher concentra

287 nt in both pellicle and planktonic wild-type B. subtilis cells and in strains with deletions in the e

288 Here, colonies of wild-type B. subtilis formed a spreading population that induced p

289 s of chromosomally SNAP-tagged and wild-type B. subtilis strains with protein standards of known conc

290 Viable B. subtilis cells were identified and DNAs of two bacter

291 A fluorescent in vivo B. subtilis reporter system identified peptide motifs wh

292 ly under phosphate-limiting conditions, when B. subtilis specifically degrades WTA and replaces it wi

293 e TLR4 signal than B. anthracis PGA, whereas B. subtilis PGA elicited none.

294 To investigate whether B. subtilis encodes yet additional classes of transport

295 The results explain why B. subtilis with its Asn synthetase genes knocked out is

296 system (T6SS) is activated upon contact with B. subtilis cells, and stimulates B. subtilis sporulatio

297 escribes an investigation of this issue with B. subtilis PBP4a.

298 implies that it shares more orthologues with B. subtilis subsp. subtilis NCIB 3610(T) (ANIm values, 8

299 Dynamic tests with B. subtilis and E. coli showed high antibacterial effici

300 ersatile energy generation strategies within B. subtilis biofilms.