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1 cases investigated, 63% were viremic for the B19 virus.
2 rovides details on host recognition of human B19 virus.
3 aging the DNA inside the capsid structure of B19 virus.
4 llas, respectively), HBoV (73% and 36%), and B19 virus (8% and 27%) were recorded for apes, while OWM
5 patocellular carcinoma cell line Hep G2 with B19 virus and assayed for apoptosis by using annexin V s
6 mmunoassay that detects the human parvovirus B19 virus (B19V) immunoglobulin M (IgM) or IgG in the se
7 ncubation with anticapsid antibodies against B19 virus, but not anticapsid antibodies against AAV, in
8                              Parvovirus B19 (B19 virus) can persist in multiple tissues and has been
9  leukemia cell line which can be infected by B19 virus following erythroid differentiation with eryth
10                                              B19 virus-generated pre-mRNAs are transcribed from a sin
11    Thus, we conclude that replication of the B19 virus genome is the primary limiting step governing
12                           Replication of the B19 virus genome, however, introduced either by viral in
13               Further development of the AAV-B19 virus hybrid vector system should prove beneficial i
14                               Infection with B19 virus induced means of 28% of Hep G2 cells and 10% o
15 3 and 9, but not caspase 8, are required for B19 virus-induced apoptosis.
16                       The mechanism by which B19 virus induces liver failure remains unknown.
17 cute fulminant liver failure associated with B19 virus infection was characterized by hepatocellular
18 valuable in the study of the pathogenesis of B19 virus infection.
19  Analysis of caspase involvement showed that B19 virus-inoculated cultures had a significant increase
20      The pre-mRNA processing strategy of the B19 virus is unique among parvoviruses.
21 electron microscopy (cryo-EM) structure of a B19 virus-like particle (VLP) complexed with the antigen
22 report here the high-resolution structure of B19 virus-like particles (VLPs) complexed with the Fab o
23                                    Moreover, B19 virus-like particles were successfully generated in
24  OWMs were uniformly negative (for PARV4 and B19 virus) or infrequently reactive (3% for HBoV).
25                                  Recombinant B19 virus particles were assembled, as evidenced by elec
26 e and circulation of parvoviruses related to B19 virus, PARV4, and HBoV in nonhuman primates, plasma
27  were screened for antibodies to recombinant B19 virus, PARV4, and HBoV VP2 antigens by enzyme-linked
28 previously reported to be a limiting step in B19 virus permissiveness.
29            Hepatocytes are nonpermissive for B19 virus replication.
30 ome replication, internal polyadenylation of B19 virus RNAs at (pA)p is favored in cells which are bo
31 dthrough of (pA)p and the polyadenylation of B19 virus transcripts at the distal site [(pA)d].
32    Blockage of the production of full-length B19 virus transcripts at the internal polyadenylation si
33 enome is the primary limiting step governing B19 virus tropism.
34 ary human erythroid cells by the recombinant B19 virus vector was significantly higher than that by t
35 sis and immunofluorescence demonstrated that B19 virus was able to infect the cells and produce its n