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1                                              BALT HEVs also express the L-selectin ligand PNAd.
2                                              BALT most often accompanied A0 or A1 rejection.
3                                              BALT structures are prevalent around lung airways during
4                                              BALT was found from 9 days to 2431 days after transplant
5                                              BALT-like lesion formation in the lungs of SE2(-/-) mice
6 literans developed in 29% of patients with a BALT-positive biopsy, a percentage not different from th
7 nd B cells can independently be recruited as BALT.
8 lished infection with oxytetracycline caused BALT, but not the lymphatics, to regress.
9 ts had transbronchial biopsies demonstrating BALT.
10 ce treated with P. aeruginosa (P.a.) develop BALT but B cell follicles lack FDCs while still harborin
11 nd adhesion molecules that are essential for BALT induction, organization, and maintenance.
12  Together, our findings provide evidence for BALT as an early life adaptation for mobilizing localize
13 endent induction and maturation pathways for BALT formation.
14 Toll-like receptor pathways are required for BALT induction by P.a., but not MVA, and provide evidenc
15 show that high endothelial venules (HEVs) in BALT express substantial levels of VCAM-1, in marked con
16            LPS-bead(+) cells were present in BALT, suggesting local induction of immune responses.
17 e migration pathways and immune responses in BALT and other bronchopulmonary tissues.
18   Although chronic smoke exposure can induce BALT formation, the impact of donor cigarette use on all
19 rmore, in IL-17-deficient mice, P.a.-induced BALT largely lacks B cells as well as CXCL12-expressing
20 ich seem to be initiated at sites of induced BALT (iBALT).
21           Here, we show that smoking-induced BALT in mouse donor lungs contains Foxp3(+) T cells and
22                                    Inducible BALT (iBALT) can amplify pulmonary or systemic inflammat
23                                    Inducible BALT (iBALT) is associated with immune responses to resp
24          This IL-4/IL-13-dependent inducible BALT model will be useful for investigating the pathophy
25 ome; mucus plugging; and extensive inducible BALT.
26 n mice leads to the development of inducible BALT (iBALT), which is located in peribronchial, perivas
27 des that recruit lymphocytes from blood into BALT.
28 block the homing of B and T lymphocytes into BALT, whereas anti-alpha4 integrin and anti-VCAM-1 mAbs
29 ungs of naive mice and humans typically lack BALT, pulmonary infection in mice leads to the developme
30                           The association of BALT with high-grade acute cellular rejection and with t
31 inal centers consistent with descriptions of BALT.
32 ed at random to evaluate the distribution of BALT lymphocyte subsets immunohistochemically.
33           Immunohistochemical examination of BALT showed helper T cells predominated over cytotoxic T
34 R-2/VEGFR-3 did not prevent the formation of BALT.
35 ansplant procedure and the identification of BALT.
36  molecular requirements for the induction of BALT in pulmonary allografts during tolerance induction
37 required for the formation and maturation of BALT.
38  raises the possibility that the presence of BALT on transbronchial biopsy may be part of the evoluti
39 ymphatic network is restricted to regions of BALT, but lymphatics do not regress when BALT regresses
40 VA) is sufficient to induce highly organized BALT with densely packed B cell follicles containing a n
41 e human disease also develop lymphocyte-rich BALT.
42 alysts: i.e., binaphthyl-allyl-tetrasulfone (BALT) and imidodiphosphorimidate (IDPi).
43                Thus, our work indicates that BALT in donor lungs is plastic in nature and may have im
44  of the bronchus-associated lymphoid tissue (BALT) and infiltration of the pulmonary interstitium wit
45 ence of bronchus-associated lymphoid tissue (BALT) in donor lungs has been suggested to accelerate gr
46 such as bronchus-associated lymphoid tissue (BALT) in the lung, develops spontaneously at sites of ch
47         Bronchus-associated lymphoid tissue (BALT) is occasionally found in the lungs of mice and hum
48 nctive Bronchial Associated Lymphoid Tissue (BALT) not induced by any wild type Bordetella species, a
49 induced bronchus-associated lymphoid tissue (BALT) of tolerant mouse lungs.
50         Bronchus-associated lymphoid tissue (BALT) participates in airway immune responses.
51 grafts, bronchus-associated lymphoid tissue (BALT) plays a major role in the induction and persistenc
52         Bronchus-associated lymphoid tissue (BALT) was originally described as a mucosal lymphoid org
53 wn that bronchus-associated lymphoid tissue (BALT), characterized by the development of peripheral no
54 ow that bronchus-associated lymphoid tissue (BALT), containing B cell follicles, CD4(+) T cells and f
55 sion of bronchus-associated lymphoid tissue (BALT), goblet cell hyperplasia, epithelial hypertrophy,
56 ions of bronchus-associated lymphoid tissue (BALT), where VEGF-C-producing cells were scattered in T-
57 sembled bronchus-associated lymphoid tissue (BALT).
58 ominate bronchus-associated lymphoid tissue (BALT).
59 ry phenotype) but not naive T and B cells to BALT.
60 FA-1, targets specific lymphocyte subsets to BALT.
61                        Mechanisms underlying BALT induction in tolerant pulmonary allografts, however
62  of BALT, but lymphatics do not regress when BALT regresses after antibiotic treatment.
63 he clinical and histologic associations with BALT identified on transbronchial biopsy in human lung a
64 at regulatory Foxp3+ T cells residing within BALT of tolerant pulmonary allografts function to suppre