ライフサイエンスコーパス: BOLD signal

1 ement learning model, were regressed against BOLD signal.

2 ps were found between panic symptoms and the BOLD signal.

3 nsights in the neuronal underpinnings of the BOLD signal.

4 equency fluctuations/temporal variability of BOLD signal.

5 ributed to the observed group differences in BOLD signal.

6 100 Hz) LFP bands were informative about the BOLD signal.

7 1.5-2.0 minutes of RS functional MR imaging BOLD signal.

8 explain different components of the observed BOLD signal.

9 flow (CBF) and volume (CBV) that affect the BOLD signal.

10 ity patterns specific to the depth-dependent BOLD signal.

11 subsystems using an MDS in the space of the BOLD signal.

12 ning cell-type-specific contributions to the BOLD signal.

13 emodynamic responses that drive the negative BOLD signal.

14 phy (fbEEG) are related to the resting-state BOLD signals.

15 ty of MVPA to exploit fine scale patterns of BOLD signals.

16 fluctuations (ISFs) in scalp potentials and BOLD signals.

17 local field potential (LFP) activity, and of BOLD signals.

18 as measured by blood oxygen level-dependent (BOLD) signal.

19 es in the blood oxygenation level-dependent (BOLD) signal.

20 ating the blood oxygenation level-dependent (BOLD) signal.

21 ced changes in blood oxygen level-dependent (BOLD) signal.

22 e on the brain blood oxygen level-dependent (BOLD) signal.

23 y measured via blood oxygen level dependent (BOLD) signals.

24 ndencies among blood-oxygen-level-dependent (BOLD) signals.

25 pon brain fMRI blood oxygen level-dependent (BOLD) signals.

26 wer (task-off) blood oxygen level-dependent (BOLD) signals.

27 as measured by blood-oxygen-level-dependent (BOLD) signals.

28 negative blood oxygenation level-dependent (BOLD) signaling.

29 ced response amplitude (percentage change in BOLD signal, 0.65 +/- 0.28 vs 0.89 +/- 0.14; p < 0.01),

30 BOLD signal (7.5 minutes worth) was obtained from 30 sub

31 ency) were computed for each subject at each BOLD signal acquisition duration.

32 bal efficiency stabilized after 2 minutes of BOLD signal acquisition, whereas correlation coefficient

33 ted threshold) stabilized after 5 minutes of BOLD signal acquisition.

34 WM tracts and patterns of condition-related BOLD signal across all GM regions.

35 vestigated the spatial pattern of correlated BOLD signal across eight visual areas on data collected

36 ty, measured by the spatial coherence of the BOLD signal across regions of the brain; and metastabili

37 tinguishable solely from the pattern of fMRI BOLD signals across voxels in the human hippocampus.

38 al patterns of blood oxygen-level-dependent (BOLD) signal across voxels.

39 ing changes in blood-oxygen-level-dependent (BOLD) signal after visual stimulation.

40 Comparing the model activity with BOLD signal allowed us to identify neural correlates of

41 ility (measured as the standard deviation of BOLD signal amplitude) in resting state networks (RSNs)

42 Both BOLD signal and CBV decreased from superficial to deep l

43 functional magnetic resonance imaging (fMRI) BOLD signal and electrocorticographic (ECoG) field poten

44 sly and with high spatiotemporal resolution, BOLD signal and LFP during spontaneous activity in early

45 ates positively with the superficial layers' BOLD signal and that beta-power is negatively correlated

46 s well as blood oxygenation level-dependent (BOLD) signal and cerebral blood volume (CBV) and blood f

47 regional blood oxygenation level-dependent (BOLD) signal and correlated symptoms was compared with i

48 tivity are the blood-oxygen-level dependent (BOLD) signal and surface field potential.

49 conditions on blood-oxygen-level-dependent (BOLD) signal and task-based functional connectivity.

50 alient in blood oxygenation level-dependent (BOLD) signals and correlated within specific brain syste

51 mpirical foundation for the widely-used fMRI BOLD signal, and the features of of MRI define a potent

52 ptom severity, blood-oxygen-level-dependent (BOLD) signal, and dorsolateral prefrontal cortex (DLPFC)

53 different brain regions in the resting-state BOLD signal are thought to reflect intrinsic functional

54 Changes in BOLD signals are sensitive to the regional blood content

55 (LFOs) of the blood oxygen level-dependent (BOLD) signal are gaining interest as potential biomarker

56 ions (LFOs) of blood-oxygen-level-dependent (BOLD) signals are used to map brain functional connectiv

57 then test the purity of the ventral striatal BOLD signal as a model-free report.

58 e glucose and oxygen consumption, and of the BOLD signal as reported in human studies.

59 nduced deactivations in medial temporal lobe BOLD signal (as compared to periods of rest) demonstrate

60 annot be explained by differences in overall BOLD signal, as average BOLD activity was either equival

61 In this way, we indexed BOLD signals associated with an anticipated need to exer

62 orrelated with blood oxygen level-dependent (BOLD) signal associated with each working memory compone

63 erence in blood oxygenation level-dependent (BOLD) signal associated with the selected and rejected s

64 We calculated the time delay between the BOLD signal at each voxel and the whole-brain signal usi

65 0.2 Hz) inversely correlated fluctuations in BOLD signal at rest.

66 ignal such that TMS to the left LO decreases BOLD signal at the stimulation site (LO) while viewing o

67 the neocortex or thalamus, elicits positive BOLD signals at the stimulus location with classical kin

68 The blood oxygen level-dependent (BOLD) signal at primary olfactory cortex (POC) was weake

69 ations of blood oxygenation level-dependent (BOLD) signals between remote brain areas [so-called BOLD

70 minar-resolved blood-oxygen level-dependent (BOLD) signal by combining data from simultaneously recor

71 y and the blood oxygenation level dependent (BOLD) signal can be described as following linear system

72 (2.35 versus 2.37 mL/min per gram; P=0.91), BOLD signal change (17.3% versus 17.09%; P=0.91), and co

73 a priori defined striatal region of interest BOLD signal change during reward anticipation compared w

74 is was mirrored by a monotonically decreased BOLD signal change in dorsolateral prefrontal cortex on

75 A BOLD signal change in right superior parietal cortex was

76 and S2, as measured by the magnitude of the BOLD signal change to tactile stimuli, was reduced marke

77 ship between MRS-visible neurochemicals, the BOLD signal change, and stimulus intensity, we measured

78 gatively (r(2) = 0.09, p < 10(-16)) with the BOLD signal change.

79 ne on striatal blood oxygen level-dependent (BOLD) signal change during anticipation of monetary rewa

80 Mean percent blood oxygen level-dependent (BOLD) signal change was extracted from a priori regions

81 rther assessed blood-oxygen-level-dependent (BOLD) signal change while subjects performed a face-reco

82 motrigine pretreatment prevented many of the BOLD signal changes and the symptoms.

83 ontrast, men exhibited significantly greater BOLD signal changes compared to LF/MC women on bilateral

84 In healthy women, robust task-evoked BOLD signal changes observed under low E(2) conditions w

85 e task-related blood-oxygen level-dependent (BOLD) signal changes during sematic processing and resti

86 rences in blood oxygenation level-dependent (BOLD) signal changes in men compared to EF women, except

87 ng (fMRI) blood oxygenation level-dependent (BOLD) signal changes in response to a mild cold challeng

88 osely parallel blood oxygen level dependent (BOLD) signal changes observed in human functional magnet

89 sity, we measured combined neurochemical and BOLD signals (combined fMRI-MRS) to different image cont

90 We also assessed correlations between BOLD signal contrasts and clinical measures in SZs.

91 tional circuits at rest, the extent to which BOLD signals correlate spatially with underlying neurona

92 w frequent the blood-oxygen-level-dependent (BOLD) signal correlation between two nodes is negative.

93 by inter-areal blood-oxygen-level-dependent (BOLD) signal correlation, as a proxy for communication i

94 ients, areas exhibiting significant delay in BOLD signal corresponded to areas of hypoperfusion ident

95 Temporal profiles of stimulus-evoked BOLD signals covaried with LFP and multiunit spiking in

96 ical domains, testing them on functional MRI BOLD signal data obtained from individuals undergoing va

97 halamus after placebo compared with HCs, and BOLD signal decreases in the left hypothalamus after dru

98 uations in the blood-oxygen-level-dependent (BOLD) signals demonstrate consistent temporal correlatio

99 dataset of individuals, using resting state BOLD signals, demonstrated that a functional entropy ass

100 The blood-oxygen-level-dependent (BOLD) signal, detected in fMRI, reflects changes in deox

101 meaningful and meaningless actions elicited BOLD signal differences at bilateral sites in the supram

102 Blood Oxygenation Level Dependent (BOLD) signal differences between tasks were identified b

103 Blood oxygen level dependent (BOLD) signal differences in response to SS and neutral s

104 (opto-fMRI) in mice to test the linearity of BOLD signals driven by locally induced excitatory activi

105 differences in blood oxygen level dependent (BOLD) signal due to risk factors in 74 middle-aged cogni

106 Successful older participants had higher BOLD signal during encoding than average participants, n

107 easing colonic propionate production reduced BOLD signal during food picture evaluation in the caudat

108 ontaneous low-frequency fluctuations in fMRI BOLD signal during rest from two separate regions key to

109 Whereas these areas also produced changes in BOLD signal during the dynamic warming stimulus on the c

110 functional magnetic resonance imaging (fMRI) BOLD signal during the performance of the Simon task.

111 RI and a blocked design were used to acquire BOLD signals during implicit (task-unrelated) presentati

112 F) in the blood oxygenation level-dependent (BOLD) signal during resting-state fMRI reflects the magn

113 ng of the fMRI blood oxygen level-dependent (BOLD) signal during working memory in healthy subjects c

114 asures of blood oxygenation-level-dependent (BOLD) signals during the performance of dopamine-depende

115 found that the global phase synchrony of the BOLD signals evolves on a characteristic ultra-slow (<0.

116 Our findings provide evidence that the BOLD signal fluctuates with spatial heterogeneity in mal

117 ic brain activity as measured by spontaneous BOLD signal fluctuations and help to understand propofol

118 the neuronal correlates of these reciprocal BOLD signal fluctuations are unknown.

119 rous studies have measured how low-frequency BOLD signal fluctuations from the brain are correlated b

120 interregional correlations of low-frequency BOLD signal fluctuations in 10 high-functioning particip

121 Analyses of intrinsic fMRI BOLD signal fluctuations reliably reveal correlated and

122 0.015 Hz, commensurate with the frequency of BOLD signal fluctuations seen by fMRI, suggesting that q

123 relevance for the neuronal basis of coherent BOLD signal fluctuations, our procedure may translate in

124 low-frequency blood-oxygen-level-dependent (BOLD) signal fluctuations between major divisions of the

125 scular compartments and other factors to the BOLD signal for different magnet strengths and pulse seq

126 a typical rodent fMRI voxel and predict the BOLD signal from first principles using those measuremen

127 group difference in the skewness of the fMRI BOLD signal from the vPMC, suggesting that the relative

128 phase interactions among resting-state fMRI BOLD signals from human subjects.

129 , we investigated the content-specificity of BOLD signals from various brain regions during a VSTM ta

130 dies using the Blood Oxygen-Level Dependent (BOLD) signal from functional Magnetic Resonance Imaging

131 n between blood-oxygenation-level-dependent (BOLD) signals from different brain areas.

132 We examined blood oxygen dependent (BOLD) signal functional connectivity using conventional

133 Although the blood oxygen level-dependent (BOLD) signal has been found to correlate preferentially

134 ns, where blood oxygenation level-dependent (BOLD) signals have been suggested as correlating with qu

135 frequently accompanied by reductions in the BOLD signal in adjacent regions of cortex.

136 A main effect of reduced BOLD signal in bilateral occipital areas was noted acros

137 nd directionality (positive/negative) of the BOLD signal in both contralateral and ipsilateral sensor

138 ent work with fMRI has demonstrated that the BOLD signal in dopaminergic target areas meets both nece

139 Additional analysis revealed that BOLD signal in heteromodal association cortex typically

140 e the accuracy of word report and reduce the BOLD signal in lateral temporal lobe regions.

141 threat probability and magnitude related to BOLD signal in left aHC/entorhinal cortex.

142 In Experiment 2, spatial patterns of BOLD signal in medial temporal and posterior midline reg

143 emoglobin oxygen saturation (sO2) levels and BOLD signal in response to carbogen.

144 Significantly greater percent increases in BOLD signal in response to TMT were observed in the ante

145 Oxytocin administration reduces the BOLD signal in reward-related food motivation brain regi

146 negative variation (CNV) signal and the fMRI BOLD signal in SMA and VS, (2) the underlying causal con

147 nd a subject-by-subject correlation with the BOLD signal in the amygdala.

148 BOLD) signal in BAs 9 and 10, and diminished BOLD signal in the anterior cingulate, thalamus, midbrai

149 nts who changed their minds more showed less BOLD signal in the insula and the amygdala when evaluati

150 he risky Stag choice, AVP down-regulates the BOLD signal in the left dorsolateral prefrontal cortex (

151 n performance was positively correlated with BOLD signal in the left posterior hippocampus, parahippo

152 ite (LO) while viewing objects and increases BOLD signal in the left PPA when viewing scenes.

153 l use measures, were associated with greater BOLD signal in the region that differentiated the FHP an

154 iol, a TD by ACE interaction was observed on BOLD signal in the right DLPFC such that TD increased ac

155 During retrieval of the event, the BOLD signal in the same region was modulated by the pers

156 Among GG-homozygotes, BOLD signal in the subgenual cingulate was greater in MD

157 first provide unbiased evidence that the raw BOLD signal in these regions corresponds closely to a re

158 However, BOLD signal in this region was better explained by avoid

159 by the decision process, our data show that BOLD signal in this same region reflects the choices we

160 D) signals, it remains controversial whether BOLD signals in a particular region can be caused by act

161 d BOLD activations and local correlations of BOLD signals in a resting state, and whether these spati

162 ng state functional connectivity measures of BOLD signals in different brain regions.

163 he living and intact mammalian brain reveals BOLD signals in downstream targets distant from the stim

164 aspartate (NMDA) receptor antagonist-induced BOLD signals in healthy humans and animals to differing

165 By measuring BOLD signals in human primary visual cortex while varyin

166 e found that the spontaneous fluctuations of BOLD signals in key nodes of RSNs are associated with ch

167 We recorded BOLD signals in occipitotemporal visual cortex of human

168 Load-dependent BOLD signals in primary visual cortex (V1) and superior

169 ectively correlated with subsets of cortical BOLD signals in specific task-positive and task-negative

170 Both choices and value-related BOLD signals in striatum, although most often associated

171 neural model that best explains the observed BOLD signals in terms of effective connectivity.

172 based value computation were correlated with BOLD signals in the medial temporal lobe and frontal cor

173 BOLD signals in the rostral medial prefrontal cortex (rm

174 model-free reinforcement learning, with fMRI BOLD signals in ventral striatum notably covarying with

175 matter (WM) despite increasing evidence that BOLD signals in WM change after a stimulus.

176 elevated blood oxygenation-level dependent (BOLD) signal in BAs 9 and 10, and diminished BOLD signal

177 with decreased blood oxygen level-dependent (BOLD) signal in Broca's area.

178 e) or negative blood oxygen level-dependent (BOLD) signal in functional magnetic resonance imaging (f

179 variability of blood oxygen level-dependent (BOLD) signal in individuals from three independent datas

180 cortical blood oxygenation level-dependent (BOLD) signal in response to emotionally salient and neut

181 n-related blood oxygenation level-dependent (BOLD) signal in task-positive GM regions.

182 Blood-oxygen-level-dependent (BOLD) signal in the amygdala was examined in children wi

183 and changes in blood oxygen level-dependent (BOLD) signal in the amygdala with a 3T MRI scanner in 16

184 with the blood oxygenation level-dependent (BOLD) signal in the nucleus accumbens and a subject-by-s

185 Moreover, blood-oxygenation-level-dependent (BOLD) signal in the temporo-parietal junction is modulat

186 found that the blood oxygen level dependent (BOLD) signal in ventromedial prefrontal cortex (vmPFC) w

187 creases in blood-activation-level-dependent (BOLD) signal in WM task-positive brain regions and incre

188 RI) to measure blood oxygen level-dependent (BOLD) signals in 66 infants, 47 of whom were at high ris

189 ations between blood oxygen level-dependent (BOLD) signals in different brain areas.

190 slow intrinsic blood oxygen level dependent (BOLD) signals in normal adults during wake and SWS.

191 Blood-oxygen-level-dependent (BOLD) signals in the rostral anterior cingulate cortex (

192 a context, and blood oxygen level dependent (BOLD) signals in the ventromedial prefrontal cortex (PFC

193 negative blood oxygenation level-dependent (BOLD) signals in these regions.

194 eater deactivation (task-induced decrease in BOLD signal) in medial parietal regions during successfu

195 omponents was also observed in value-related BOLD signaling, in the form of lateralized biases in str

196 in neural synchrony goes hand-in-hand with a BOLD signal increase in the left dorsolateral prefrontal

197 During cataplexy, suprapontine BOLD signal increase was present in the amygdala, fronta

198 Brain areas showing changes in BOLD signal increases (activations) and decreases (deact

199 h aversive and erotic images produced robust BOLD signal increases in bilateral primary and secondary

200 s with more intact WM further showed greater BOLD signal increases in typical "task-negative" regions

201 refrontal cortex, and the nucleus accumbens; BOLD signal increases were also observed at locus cerule

202 illustrated by quantifying variations in the BOLD signal induced by the morphological folding of the

203 tions in regards to fMRI technology: how the BOLD signal inferences the underlying microscopic neuron

204 een photoacoustically derived sO2 levels and BOLD signal intensity (r = 0.937, P = .005) and partial

205 The BOLD signal intensity and activation volume within the P

206 isplayed a correlation between participants' BOLD signal intensity and reaction time that was selecti

207 Changes in PAI and BOLD signal intensity before and after VDA treatment wer

208 irty-six patients were analyzed for relative BOLD signal intensity increase according to the 16-segme

209 Relative BOLD signal intensity increase was significantly lower i

210 kg/min) and assessed quantitatively (using a BOLD signal intensity index [stress/resting signal inten

211 A BOLD signal intensity index threshold to identify ischem

212 Because the BOLD signal is an indirect measure of neuronal activity

213 Our results suggest that aHC BOLD signal is better explained by avoidance behavior th

214 s similarly to classic language regions: (i) BOLD signal is higher during sentence comprehension than

215 rol conditions that are more difficult; (ii) BOLD signal is modulated by phonological information, le

216 croscopic vascular behavior into macroscopic BOLD signals is at the foundation of physiologically inf

217 r activity and blood oxygen level-dependent (BOLD) signal is of critical importance to the interpreta

218 R) imaging T2* blood oxygen level-dependent (BOLD) signal is sensitive to blood oxygen concentration;

219 RI) using blood oxygenation level-dependent (BOLD) signals, it remains controversial whether BOLD sig

220 delay in blood oxygenation level-dependent (BOLD) signals may be sensitive to perfusion deficits in

221 nt cautionary data concerning the quality of BOLD signals measured from the LC using standard functio

222 Whereas some studies have reported that BOLD signals measured in visual cortex are tightly linke

223 However, blood oxygen level-dependent (BOLD) signals measured via fMRI are very slow [9], so it

224 y, a proxy for blood oxygen level-dependent (BOLD) signal measurement in human functional magnetic re

225 itated by the fact that time resolution of a BOLD signal much lower than that of cognitive processes

226 We show that reading increased the top-down BOLD signal observed in the deep layers of the LOTS and

227 t anxiety was positively correlated with the BOLD signal of the right parahippocampal gyrus during th

228 first create nodal signals by averaging the BOLD signals of all the voxels in each region, and to th

229 iterature, the blood oxygen level-dependent (BOLD) signal of functional magnetic resonance imaging st

230 -specific information represented within the BOLD signal on every trial.

231 The BOLD signal originated primarily from venules, and the C

232 increase in tumor sO2 levels (P = .003) and BOLD signal (P = .001).

233 ongly influence the cortical depth-dependent BOLD signal, particularly for gradient-echo BOLD.

234 tudy revealed for the first time contrasting BOLD signal patterns of biased agonists in comparison to

235 hat xanomeline induces robust and widespread BOLD signal phMRI amplitude increases and decreased high

236 pus, where the blood-oxygen-level-dependent (BOLD) signal predicts behavioral measures of memory inte

237 a specific cognitive paradigm, modelling the BOLD signal provided new insight into the dynamic causal

238 ever, the blood oxygenation level-dependent (BOLD) signal provides an indirect measure of neuronal fi

239 ion, which correspond to specific changes in BOLD signal reactivity.

240 addition, we assess MD responsiveness using BOLD signal recruitment and multi-task activation indice

241 ed significant blood oxygen level-dependent (BOLD) signal reductions in the amygdala, hippocampus, in

242 dACC BOLD signals reflected two decision biases-to defer comm

243 However, the BOLD signal reflects changes in blood volume and oxygena

244 1) higher blood oxygenation level-dependent (BOLD) signal related to the eye closure over the visual

245 of the LFP, it is still unclear whether the BOLD signal relates to the activity expressed by each LF

246 Both increases and decreases in BOLD signal reliably followed increases and decreases in

247 esia, as well as rTMS-induced attenuation of BOLD signal response to painful stimuli throughout pain

248 zed but not control mothers showed a greater BOLD signal response to predator odor than a control put

249 lthough applying global signal regression to BOLD signals results in some BOLD anticorrelations that

250 Voxelwise analysis of task-related BOLD signal revealed a significant group-by-abstinence i

251 sirable for interspecies comparison, and the BOLD signal's faster response function promises to be be

252 ns in the blood oxygenation level-dependent (BOLD) signal serve as the basis of functional magnetic r

253 The blood oxygenation level-dependent (BOLD) signal serves as the basis for human functional MR

254 gether, these findings suggest that the vPMC BOLD signal skewness and the temporal relationship of vP

255 s effect is also reflected by changes in the BOLD signal such that TMS to the left LO decreases BOLD

256 Knowledge of the properties of the BOLD signal, such as how linear its response is to senso

257 allodynia produced significant decreases in BOLD signal, suggesting pain-induced activation of endog

258 ous fingers of the two hands elicited higher BOLD signal than stimulation of homologous fingers.

259 The results identified a component of the BOLD signal that can be attributed to significant change

260 us, showed a monotonic variation of the fMRI BOLD signal that scaled with perceived memory strength (

261 een areas, between subjects, or even between BOLD signals that have been preprocessed in different wa

262 100 Hz) neural activity contributes to local BOLD signals, the neural basis of interareal BOLD correl

263 rectly test this assumption by comparing the BOLD signal time courses in each network across differen

264 e-tracking data to a low-temporal resolution BOLD signal to extract responses to single words during

265 eagues use resting-state fluctuations of the BOLD signal to highlight the relevance of networks to hu

266 tionship between cerebral blood flow and the BOLD signal to improve dynamic estimates of blood flow f

267 that psychosis patients showed an increased BOLD signal to LRZ challenge, rather than the decreased

268 at such cross-frequency coupling links local BOLD signals to BOLD correlations across distributed net

269 educe the blood oxygenation level-dependent (BOLD) signal to high-calorie food vs non-food visual sti

270 e behavior and blood oxygen level-dependent (BOLD) signals to decision variables extracted from simul

271 fMRI blood oxygen level-dependent (BOLD) signals to the sight and/or taste of the stimuli w

272 The authors investigated how well BOLD signal tracked prediction error in the striatum and

273 can extract word-level information from the BOLD signal using high-temporal resolution eye tracking.

274 Recording blood oxygenation level-dependent (BOLD) signal using functional magnetic resonance imaging

275 ole-brain blood oxygenation level-dependent (BOLD) signals using functional MRI during an affective S

276 ine model, we show that calcium predicts the BOLD signal, using a model that optimizes a gamma-varian

277 Because previous work investigating BOLD signal variability has been conducted within task-b

278 Fast BOLD scans showed that the elevated BOLD signal variability in AD arises mainly from cardiov

279 We detected a replicable increase in brain BOLD signal variability in the AD populations, which con

280 wn regarding regional changes in spontaneous BOLD signal variability in the human brain across the li

281 onal-MRI-based blood oxygen level-dependent (BOLD) signal variability (SD(BOLD)) in younger and older

282 asy-to-implement analysis approach to assess BOLD-signal variability in event-related fMRI task parad

283 However, higher levels of BOLD-signal variability in the left inferior frontal jun

284 r, our data suggest that neuronally mediated BOLD signal variance generally increases in light sleep.

285 cy fluctuations (fALFF; 0.01-0.08 Hz) of the BOLD signal was compared among the three groups.

286 In the caudate, the reduction in BOLD signal was driven specifically by a lowering of the

287 on, rCBF was measured with [15O]H2O PET, and BOLD signal was measured with fMRI, both during an n-bac

288 BOLD signal was recorded during rest periods before and

289 Conversely, among A-carriers, BOLD signal was smaller in MDD (n = 7) compared with con

290 Resting-state BOLD signal was transformed into frequency space (Welch'

291 occur during early conditioning and amygdala BOLD signaling was unaffected in these patients.

292 The blood oxygen level-dependent (BOLD) signal was measured in a priori brain regions invo

293 The blood-oxygen-level dependent (BOLD) signal was measured to identify regions where the

294 y, only decreases in cerebral blood flow and BOLD signal were seen, and these were maximal in hub reg

295 nal MRI (fMRI) blood-oxygen-level-dependent (BOLD) signal were analyzed using a computational model o

296 rences in blood oxygenation level-dependent (BOLD) signal were found in healthy controls (A allele ca

297 nique opportunity to reconstruct compromised BOLD signal while capturing features of an individual's

298 power was reflected in the amplitude of the BOLD signal, while the relationship between beta and gam

299 At the lowest odorant concentration, the BOLD signals within POC, hippocampus, and insula were si

300 inhibitory population evoked robust positive BOLD signals within striatum, while downstream regions e